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VOLKMANN ISCHAEMIC CONTRACTURE
Presenter. Dr. Sikandar kumar Mahto
PG 1ST Year Resident
NMCTH, Birgunj
Moderator: Dr. Prashant Thakur
Definition
Volkmann`s Ischemic contracture is a permanent shortening of the
forearm muscles resulting from injury that gives rise to a claw-like
deformity of the hand, wrist and fingers.
Historical Background
In 1881, Richard Von Volkmann described irreversible contracture of
the hand and forearm muscles.
In 1906, Hildebrand first used the term Volkmann`s ischaemic
contracture.
In 1909, Thomas reviewed 112 published cases of volkmann
contracture and found closed fractures to be the most common cause.
In 1914, Murphy was the first to suggest fasciotomy might prevent
volkmann contracture.
Epidemiology
• The incidence of Volkmann’s contracture is low.
• Its prevalence is 0.5%, which means it is a rare disease.
• Occurs most commonly in children and follows injuries particularly to
the elbow.
Etiology
• The intracompartmental pressure occurs when there is a bulging
caused by a trauma.
• Thus, there is not enough space for muscles, nerves and blood vessels
that lie within this fascia.
• This results in vascular defects and defects on nerves.
Possible causes can be
• fractures of the forearm
• Animal bite,
• bleeding disorders,
• burns,
• excessive exercise and
• injections of medications at the forearm
Relevant anatomy
The flexor group of
muscles in the anterior
forearm may be divided
into:
• superficial group
• deep group
Superficial group consists
• Pronator teres
• Flexor carpi radialis
• Flexor digitorum superficialis
• Palmaris longus
• Flexor carpi ulnaris
Deep group consists
• Flexor pollicis longus
• Pronator quadratus
• Flexor digitorum profundus
Pathophysiology
• Volkmann's Contracture is usually seen in children with displaced
supracondylar fractures of the humerus or forearm.
• It results from severe injury to the deep tissues and muscles of the
volar compartment secondary to increased compartmental pressures
• Volkmann's contracture is the end result of prolonged ischaemia of
muscles and nerves in the forearm.
• The muscles undergoes necrosis, fibrosis and contracture
Clssification
There are three main classification systems widely in use:
1. Holden classification
2. Tsuge classification
3. Zancolli classificationa
Holden classification (1979)
This system is based on the site of the vascular compression.
Type I
• The cause of compression is proximal to the site of ischemia
• Example supracondylar fracture resulting in distal ischemic damage
Type II
• There is direct localized compression in the distal segment, resulting
in a localized increase in compartmental pressure
• Example-tight bandage applied after forearm fracture
Tsuge classification (1975) [modification of
Seddon classification (1964)]
This system is based on the degree of the involvement of muscle
groups in the forearm.
Mild type:
• Localized
• Involves the deep flexor compartment, more commonly the FDP of
the middle and ring fingers and the FPL.
• Nerve involvement is absent or insignificant
Tsuge classification…
Moderate type'classic or typical type‘
• Involves all FDP and FPL tendons, with partial involvement of the FDS
• Nerve involvement is always present
• Sensory impairment is more common in the median nerve
distribution than the ulnar nerve
• Intrinsic minus deformity is common
Tsuge classification…
Severe type
• Involves all of the digital and wrist flexors and a varying amount of
extensor muscles.
• Nerve involvement is severe, with total loss of sensation and total
intrinsic palsy
Zancolli classification (1979)
This system is based on the degree of involvement of the intrinsic
muscles of the hand.
• Type I: Contracture involving forearm muscles with normal intrinsic
muscles
• Type II: Contracture involving forearm muscles with paralysis of
intrinsic muscles.
• Type III: Contracture involving forearm muscles with contracture of
intrinsic muscles
• Type IV: Combined type
Clinical Manifestations
The clinical presentation includes the five "Ps" which are:
• Pain
• Pulselessness
• Pallor
• Paresthesia
• Paralysis
Clinical Manifestations
Special findings include:
• Pain with passive stretching of the flexors
• Elbow flexion
• Forearm pronation
• Finger flexion
• MCPJ in extension
• The wrist is in palmar flexion
Clinical Manifestations
• Palpation of the affected region creates pain .
• Also, upon palpation, firmness of the tissues can be felt .
• Swelling
• Decreased sensation
Volkmann’s sign
• In classical claw hand deformity in
established VIC .
• This test consists of extending the
wrist, which exaggerates the
deformities and on flexion the
deformities appear less prominent
Treatment
Mild type:
• Dynamic splinting
• Physiotherapy- functional training and active use of muscle
• If multiple tendon involved- muscle sliding operation or wrist
resection
• If single i.e.pronater teres- excision
Treatment…
Moderate contracture:
• Muscle sliding operation
• Neuronolysis
• Excision of fibrotic tissue
• Volar transfer of dorsal wrist extensors-when no movement of finger
has been retained e.g. BR,ECRL
Treatment…
Severe contracture:
• Early excision of all necritic tissue with neurolysis
• Tendon transfer-to restore function e.g. BR to FPL,ECRL to FDP
• Free innervated muscle transfer using gracilis muscle- if motors to
restore finger flexion are unavailable
Oishi and Ezaki
• Recommended two stage procedure
Stage 1.
Initial muscle debridement and neurolysis
Stage 2
free functioning gracilis transfe – after return of sensation and intrinsic
function hand.
Established Intrinsic Muscle Contractures of
Hand
Mild contracture:
• MCP joint can be passively extended completely
• Positive intrinsic muscle tightness test- i.e. extended PIP joint while
MCP extended.
Established Intrinsic Muscle Contractures of
Hand…
More severe:
• Interosseus muscle- viable and contracted
• Intrinsic muscle tightness test- positive
• Active spreading of fingers- possible
• Contracted muscles released from MC shafts by muscle sliding
operation
Established Intrinsic Muscle Contractures of
Hand…
More severe:
• Interosseus muscle- viable and contracted
• Intrinsic muscle tightness test- positive
• Active spreading of fingers- possible
• Contracted muscles released from MC shafts by muscle sliding
operation
Established Intrinsic Muscle Contractures of
Hand…
• Most severe:
• Intrinsic muscles – contracted and also necrosed and fibrosed
• Each muscle must be divided to release of contracture with
• Capsulotomies and tendon transfer
References
• Campbell
• Apley & solomons
• Essential orthopaedics by Maheshwari
• Netter atalas
Volksman ischemic Contracture orthopaedics .pptx

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Volksman ischemic Contracture orthopaedics .pptx

  • 1. VOLKMANN ISCHAEMIC CONTRACTURE Presenter. Dr. Sikandar kumar Mahto PG 1ST Year Resident NMCTH, Birgunj Moderator: Dr. Prashant Thakur
  • 2. Definition Volkmann`s Ischemic contracture is a permanent shortening of the forearm muscles resulting from injury that gives rise to a claw-like deformity of the hand, wrist and fingers.
  • 3. Historical Background In 1881, Richard Von Volkmann described irreversible contracture of the hand and forearm muscles. In 1906, Hildebrand first used the term Volkmann`s ischaemic contracture. In 1909, Thomas reviewed 112 published cases of volkmann contracture and found closed fractures to be the most common cause. In 1914, Murphy was the first to suggest fasciotomy might prevent volkmann contracture.
  • 4. Epidemiology • The incidence of Volkmann’s contracture is low. • Its prevalence is 0.5%, which means it is a rare disease. • Occurs most commonly in children and follows injuries particularly to the elbow.
  • 5. Etiology • The intracompartmental pressure occurs when there is a bulging caused by a trauma. • Thus, there is not enough space for muscles, nerves and blood vessels that lie within this fascia. • This results in vascular defects and defects on nerves.
  • 6. Possible causes can be • fractures of the forearm • Animal bite, • bleeding disorders, • burns, • excessive exercise and • injections of medications at the forearm
  • 7. Relevant anatomy The flexor group of muscles in the anterior forearm may be divided into: • superficial group • deep group
  • 8. Superficial group consists • Pronator teres • Flexor carpi radialis • Flexor digitorum superficialis • Palmaris longus • Flexor carpi ulnaris
  • 9. Deep group consists • Flexor pollicis longus • Pronator quadratus • Flexor digitorum profundus
  • 10. Pathophysiology • Volkmann's Contracture is usually seen in children with displaced supracondylar fractures of the humerus or forearm. • It results from severe injury to the deep tissues and muscles of the volar compartment secondary to increased compartmental pressures • Volkmann's contracture is the end result of prolonged ischaemia of muscles and nerves in the forearm. • The muscles undergoes necrosis, fibrosis and contracture
  • 11. Clssification There are three main classification systems widely in use: 1. Holden classification 2. Tsuge classification 3. Zancolli classificationa
  • 12. Holden classification (1979) This system is based on the site of the vascular compression. Type I • The cause of compression is proximal to the site of ischemia • Example supracondylar fracture resulting in distal ischemic damage Type II • There is direct localized compression in the distal segment, resulting in a localized increase in compartmental pressure • Example-tight bandage applied after forearm fracture
  • 13. Tsuge classification (1975) [modification of Seddon classification (1964)] This system is based on the degree of the involvement of muscle groups in the forearm. Mild type: • Localized • Involves the deep flexor compartment, more commonly the FDP of the middle and ring fingers and the FPL. • Nerve involvement is absent or insignificant
  • 14. Tsuge classification… Moderate type'classic or typical type‘ • Involves all FDP and FPL tendons, with partial involvement of the FDS • Nerve involvement is always present • Sensory impairment is more common in the median nerve distribution than the ulnar nerve • Intrinsic minus deformity is common
  • 15. Tsuge classification… Severe type • Involves all of the digital and wrist flexors and a varying amount of extensor muscles. • Nerve involvement is severe, with total loss of sensation and total intrinsic palsy
  • 16. Zancolli classification (1979) This system is based on the degree of involvement of the intrinsic muscles of the hand. • Type I: Contracture involving forearm muscles with normal intrinsic muscles • Type II: Contracture involving forearm muscles with paralysis of intrinsic muscles. • Type III: Contracture involving forearm muscles with contracture of intrinsic muscles • Type IV: Combined type
  • 17. Clinical Manifestations The clinical presentation includes the five "Ps" which are: • Pain • Pulselessness • Pallor • Paresthesia • Paralysis
  • 18. Clinical Manifestations Special findings include: • Pain with passive stretching of the flexors • Elbow flexion • Forearm pronation • Finger flexion • MCPJ in extension • The wrist is in palmar flexion
  • 19. Clinical Manifestations • Palpation of the affected region creates pain . • Also, upon palpation, firmness of the tissues can be felt . • Swelling • Decreased sensation
  • 20. Volkmann’s sign • In classical claw hand deformity in established VIC . • This test consists of extending the wrist, which exaggerates the deformities and on flexion the deformities appear less prominent
  • 21. Treatment Mild type: • Dynamic splinting • Physiotherapy- functional training and active use of muscle • If multiple tendon involved- muscle sliding operation or wrist resection • If single i.e.pronater teres- excision
  • 22. Treatment… Moderate contracture: • Muscle sliding operation • Neuronolysis • Excision of fibrotic tissue • Volar transfer of dorsal wrist extensors-when no movement of finger has been retained e.g. BR,ECRL
  • 23. Treatment… Severe contracture: • Early excision of all necritic tissue with neurolysis • Tendon transfer-to restore function e.g. BR to FPL,ECRL to FDP • Free innervated muscle transfer using gracilis muscle- if motors to restore finger flexion are unavailable
  • 24. Oishi and Ezaki • Recommended two stage procedure Stage 1. Initial muscle debridement and neurolysis Stage 2 free functioning gracilis transfe – after return of sensation and intrinsic function hand.
  • 25. Established Intrinsic Muscle Contractures of Hand Mild contracture: • MCP joint can be passively extended completely • Positive intrinsic muscle tightness test- i.e. extended PIP joint while MCP extended.
  • 26. Established Intrinsic Muscle Contractures of Hand… More severe: • Interosseus muscle- viable and contracted • Intrinsic muscle tightness test- positive • Active spreading of fingers- possible • Contracted muscles released from MC shafts by muscle sliding operation
  • 27. Established Intrinsic Muscle Contractures of Hand… More severe: • Interosseus muscle- viable and contracted • Intrinsic muscle tightness test- positive • Active spreading of fingers- possible • Contracted muscles released from MC shafts by muscle sliding operation
  • 28. Established Intrinsic Muscle Contractures of Hand… • Most severe: • Intrinsic muscles – contracted and also necrosed and fibrosed • Each muscle must be divided to release of contracture with • Capsulotomies and tendon transfer
  • 29. References • Campbell • Apley & solomons • Essential orthopaedics by Maheshwari • Netter atalas