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VENOUS ULCER
By Muweesi Ismail
Dip OPM UIAHMS
Outline
◦ Definition and introduction
◦ Risk factors
◦ Pathophysiology
◦ Signs and symptoms
◦ Diagnosis/investigations
◦ Treatment
◦ Complications
Defintion and introduction
◦ Venous ulcer is an ulcer due to ambulatory chronic venous hypertension
◦ Its due to varicose veins ie the long saphenous veins and short saphenous/perforators veins or a post
phlebitic limb
◦ Post phlebitic limb consists of veins that have been partially recanalised following a DVT which causes
increased venous pressure around the ankle through perforators
◦ Its common around the ankles(gaiter’s zone)
◦ Its most common in women with varicose veins
Risk factors
◦ Varicose veins
◦ History of DVT in the legs
◦ Old age, being tall, being female
◦ Family history of venous insufficiency
◦ Obesity
◦ Smokin
pathophysiology
o Venous ulcer arises from dysfunction of venous valves allowing back flow causing increases venous pressure.
The imbalance between arterial and venous pressures leads to pooling.
o Venous stretching allows extravasation of proteins, inflammatory exudates, occlusion with ischaemia and free
radical release hence contributing to ulceration and poor wound healing
Signs and symptoms
◦ Ulcer initially painful but later becomes painless
◦ Ulcer is often vertically oval with sloping edges
◦ Commonly located on medial side of ankle but not above the middle athird of leg
◦ Floor is covered with pale or often without any granulation tissue
◦ Tenderness is often seen at the base of the ulcer.
◦ It doesnt pentrate deep fascia
◦ Edema
◦ Presence of varicosities
◦ Venous dermatitis
◦ Inguinal lymph nodes are often enlarged.
◦ Ulcer heals but often reforms again. Scarring is common due to repeated healing and recurrent ulcer formation.
This unstable scaring of long duration may lead to squamous ceell carcinoma.(Marjolin ulcer)
Investigatins
Colour duplex Ultrasonography ( gold standard in venous disease)
Xray ( R/O osteomyelitis)
Biopsy and histology (suspected malignant)
General and systemic examination
Venogram
Geneneral principles of managing ulcers
1. Ensure adequate and balanced perfusion
2. Manage wound (debridement, dressing)
3. Treat associated infection
4. Ensure adequate nutrition
5. Find and treat cause
6. Find and treat associated and risk factors
7. Educate the patient
8. Follow up and protect from recurrence
Treatment of venous ulcers
Bisgaard regimen (4Es)
Education
Elevation
Elastic compression
Evaluation
◦ Artificial skin (may be combined c comp)
◦ Surgical correction of superficial venous reflux
Complications
◦ Chronic pain
◦ Impaired mobility(often due to pain)
◦ Infection eg osteomyelitis and septicemia
◦ Allergic contact dermatitis
◦ Malignancy leading to amputation and death
◦ cellulitis
Thank you for your attention………..

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venous ulcer.pptx

  • 1. VENOUS ULCER By Muweesi Ismail Dip OPM UIAHMS
  • 2. Outline ◦ Definition and introduction ◦ Risk factors ◦ Pathophysiology ◦ Signs and symptoms ◦ Diagnosis/investigations ◦ Treatment ◦ Complications
  • 3. Defintion and introduction ◦ Venous ulcer is an ulcer due to ambulatory chronic venous hypertension ◦ Its due to varicose veins ie the long saphenous veins and short saphenous/perforators veins or a post phlebitic limb ◦ Post phlebitic limb consists of veins that have been partially recanalised following a DVT which causes increased venous pressure around the ankle through perforators ◦ Its common around the ankles(gaiter’s zone) ◦ Its most common in women with varicose veins
  • 4. Risk factors ◦ Varicose veins ◦ History of DVT in the legs ◦ Old age, being tall, being female ◦ Family history of venous insufficiency ◦ Obesity ◦ Smokin
  • 5. pathophysiology o Venous ulcer arises from dysfunction of venous valves allowing back flow causing increases venous pressure. The imbalance between arterial and venous pressures leads to pooling. o Venous stretching allows extravasation of proteins, inflammatory exudates, occlusion with ischaemia and free radical release hence contributing to ulceration and poor wound healing
  • 6. Signs and symptoms ◦ Ulcer initially painful but later becomes painless ◦ Ulcer is often vertically oval with sloping edges ◦ Commonly located on medial side of ankle but not above the middle athird of leg ◦ Floor is covered with pale or often without any granulation tissue ◦ Tenderness is often seen at the base of the ulcer. ◦ It doesnt pentrate deep fascia ◦ Edema ◦ Presence of varicosities ◦ Venous dermatitis ◦ Inguinal lymph nodes are often enlarged. ◦ Ulcer heals but often reforms again. Scarring is common due to repeated healing and recurrent ulcer formation. This unstable scaring of long duration may lead to squamous ceell carcinoma.(Marjolin ulcer)
  • 7.
  • 8. Investigatins Colour duplex Ultrasonography ( gold standard in venous disease) Xray ( R/O osteomyelitis) Biopsy and histology (suspected malignant) General and systemic examination Venogram
  • 9. Geneneral principles of managing ulcers 1. Ensure adequate and balanced perfusion 2. Manage wound (debridement, dressing) 3. Treat associated infection 4. Ensure adequate nutrition 5. Find and treat cause 6. Find and treat associated and risk factors 7. Educate the patient 8. Follow up and protect from recurrence
  • 10. Treatment of venous ulcers Bisgaard regimen (4Es) Education Elevation Elastic compression Evaluation ◦ Artificial skin (may be combined c comp) ◦ Surgical correction of superficial venous reflux
  • 11. Complications ◦ Chronic pain ◦ Impaired mobility(often due to pain) ◦ Infection eg osteomyelitis and septicemia ◦ Allergic contact dermatitis ◦ Malignancy leading to amputation and death ◦ cellulitis
  • 12. Thank you for your attention………..