Upper respiratory tract
infections
Medical Surgical Nursing 1
2024 COHORT
February 2025
By
D. Kamalizeni
Learning objectives
1. Outline the common URTIs
2. Describe the etiology and pathophysiology of common
upper respiratory tract infections
3. Describe clinical manifestations of common upper
respiratory tract infections
4. Describe the complications of common upper respiratory
tract infections
5. Describe the nursing management of patients with
common upper respiratory tract infections
Overview of the upper respiratory tract
Structures of the upper respiratory tract include:
• the nose,
• the nasal cavity,
•pharynx,
•epiglottis,
•Larynx
• trachea.
Key features liable to pathogen infestation include:
Nasal mucosa: a sticky mucous membrane that lines
the nasal cavity and traps foreign particles
Para nasal sinuses:
mucosal-lined, air filled cavities in cranial bones
(frontal, ethmoidal, sphenoidal & maxillary bones)
that surround the nasal cavity; secrete mucus that
passes to the back of the nose to moisten the inhaled
air.
Pharynx or throat
Overview of upper respiratory tract infections
(URI)
• Represents the most common acute illness evaluated in the
outpatient settings.
• Range from the common cold—typically a mild, self-limited
ailment to life-threatening illnesses such as epiglottitis.
• Viruses account for most URIs
• Appropriate management of URI may consist of
reassurance, education, and instructions for symptomatic
home treatment
Definition of common URIs
• Rhinitis: Inflammation of the nasal mucosa
• Sinusitis: Inflammation of the paranasal sinuses, including
frontal, ethmoid, maxillary, and sphenoid
• Influenza (Flu) Generalized, acute, febrile, viral disease
associated with upper and lower respiratory infections
• Nasopharyngitis (rhinopharyngitis or the common cold):
Inflammation of the nares, pharynx, tonsils
Definition of common URIs CTs
•Pharyngitis: Inflammation of the pharynx,
•Epiglottitis (supraglottitis): Inflammation of the
superior portion of the larynx and supraglottic area
•Laryngitis: Inflammation of the larynx
•Laryngotracheitis: Inflammation of the larynx &
trachea.
•Tracheitis: Inflammation of the trachea
The common occurring upper respiratory
conditions
• Rhinitis
• Influenza
• Sinusitis
The common occurring upper respiratory
conditions CTs…,
Rhinitis
• Inflammation of the nasal mucosa
• Can be allergic or viral in origin
 Allergic Rhinitis
• Most often, a reaction of the nasal mucosa to a specific antigen
• Caused by allergy to pollens from trees, flowers, crops
• Has therefore a seasonal trend
Rhinitis CTs
 Acute Viral rhinitis
• Also referred to as common cold
• Caused by virus that invade the upper respiratory tract
• Highly infectious condition spread by air born droplets
• Most prevalent in winter months
• Can be aggravated by chills, fatigue, emotional stress
• When uncomplicated, it is a self-resolving condition hence ABs are
not necessary.
Rhinitis CTs
Pathophysiology
• Invasion of the nasal mucosal by the causative agent induces
inflammatory rxn
• There is also leukocytes infiltration and tissue edema because of
capillary permeability and vasodilation.
• This inflammatory response may be confined to the nasal
membranes or spread to regions where the nasal mucosa extends:
(nasolacrimal/tear ducts, paranasal sinuses, oropharynx)
• Can also progress to the chest as the nasal mucosal is continuous
with the respiratory tract
Clinical manifestations of rhinitis
•Nasal congestion causing obligatory mouth
breathing
•Thin watery nasal discharge
•Irritating/itchy nasal cavity
•Sneezing
•Altered sense of smell
Clinical manifestations of rhinitis cts…,
•Ocular manifestations may include profuse
tearing, bilateral conjuctival edema
•When inflammatory responses extend to the
Para nasal sinuses, the air in the sinuses gets
absorbed resulting into feeling of partial vacuum
and sinus headache localized over the inflamed
regions
Clinical manifestations of rhinitis CTs
Extension of the inflammation to the oropharynx may
present with:
• Cough
• Hoarseness of voice
• Snoring
• Recurrent need to clear the throat
Decreased hearing, a sensation of fullness or popping in the
ears may be evident of eustachian tube involvement
Complications of rhinitis
Mostly linked with acute viral rhinitis and these
include:
•Sinusitis
•Otitis media
•Pharyngitis
•Tonsillitis
•Lung infections
Diagnostic approaches
Mostly through:
• Hx taking: ( onset, aggravating factors: environmental related)
• Physical examination; routine head to toe exam with focus on the
general appearance & presentation of the clinical manifestations; the
goal being to asses extent and severity of organ involvement
Ocular involvement
Extent of nasal membrane inflammation
Oropharyngeal involvement
• Check of vital signs
• Review of lab results (FBC;)
Treatment plan for rhinitis
•Aimed at blocking symptoms
•Maintaining optimal functioning.
•Prevent complications
Key rx approaches include:
Environmental control: identifying and avoiding
triggers of the allergic rxns (allergic rhinitis)
Drug therapy: ( anti-histamines and
decongestants;eg phenylephrine o.25%, 2 drops in
each nostril); to manage symptoms: reading
assignment on other specific drugs in use
Supportive care: Increase fluid intake to liquefy
secretions & counter loss from obligatory mouth
breathing
Nursing management of patients with rhinitis
•Nursing interventions are directed towards relief
of uncomfortable symptoms
•Increasing fluid intake is key to aid in liquefying
secretions
•Administering the ordered antihistamines and
decongestants helps to reduce severity of
symptoms
Nursing management of patients with Rhinitis
CTs
Nursing diagnoses
• Defined according to specific patient presentation
• Obvious ones could be:
Ineffective breathing pattern……..; further defined in the
context of the presence of the following: (Obligatory mouth
breathing, dyspnea, snoring)
Eg: Ineffective breathing pattern related to nasal mucosal
membrane congestion secondary to the disease
process(inflammation) evidenced by obligatory mouth
breathing
Nursing diagnosis CTs
Goal of care: Patient to breath at ease and through the nostrils 1 hour
after nursing interventions.(specify the easiness of the breathing)
Nursing interventions:
• Explain condition to patient to gain cooperation
• Elevate head of bed to 45 degrees to facilitate mucous drainage
• Assess environment for presence of offending allergen and remove it
if possible
• Administer the prescribed decongestant( specify name, dose etc etc)
• Emphasize importance of nasal breathing
Nursing diagnoses CTs
Other nursing diagnoses to address the following:
•Fluid intake
•Nutrition
•Health maintenance (need to prevent
symptoms)
•Susceptibility to infections like infective otitis
media, infective conjunctivitis
Patient education
Key areas to include:
• Disease process; reinforce the concept of self-care and self
management of the disease
• Environmental control measures and patient`s
responsibilities
• Medications in use, side effects and rationale for use of such
medications
• Importance of monitoring the symptoms, response to
therapies, any difficulties, new symptoms etc etc
Influenza
Disease overview
• Also termed as Flu
• Generalized, acute, febrile, viral disease associated with
upper and lower respiratory infections
• There are 3 known groups of flu viruses ( A, B & C), all with
many mutagenic strains; ie; have a remarkable ability to
change over time; rendering it a widespread disease
• C believed to have little pathogenic potential.
Pathophysiology
• Flu viruses are inhaled in mucus droplets from infected
persons
• These then penetrate the surface of upper resp tract
mucosal cells causing cell lysis and destruction of the ciliated
epithelium
• This compromises viscosity of mucosa which facilitate spread
of virus containing exudate to the lower resp tract
• An interstitial inflammation and necrosis of the bronchiolar
and alveolar result, filling the alveoli with an exudate
containing leukocytes, erythrocytes
Pathophysiology CTs
• Regeneration of epithelium slowly begins after 5th
day of the viral
infestation reaching maximum within 9 – 15 days; at which time
mucous production and cilia begin to appear.
• Before complete regeneration, the compromised epithelium is prone
to bacterial invasion and this can result into pneumonia
• If virus specific antibodies are adequate, the initial viral invasion can
be aborted at the port of entry
• The disease is usually self limiting; acute symptoms last 2 – 7 days and
are followed by convalescent period of about 1 wk
Clinical manifestation
Onset usually abrupt and characterized by:
• fever,
• cough,
• headache,
• sore throat
If uncomplicated, symptoms may subside within 7 days
Complications
Mostly pneumonia
Diagnostic approaches
• Hx taking:
Hx of having travelled to flu prone regions
Sudden onset fever which rises and falls
Hx of general body pains, running nose, cough, sore throat
• Routine physical examination:
Check vital signs: high temp, increased resp rate
Check for conjunctivitis, erythema of soft palate
• Review lab investigation results(FBC, sputum culture)
Treatment Plan
•Antipyretic for fever: ASA 600 mgs tds,
•Decongestants for nasal congestion eg
phenylephrine o.25%, 2 drops in each nostril
may be prescribed
•Increase in fluid intake
•Adequate rest
Nursing management
•Primary goals of nursing care are directed
at:
Relief of symptoms
Prevention of secondary infection:
• Disease is highly contagious hence
adherence to standard IP remains key
Nursing management CTs
• Nursing diagnoses are patient specific and commonly with regard to:
Ineffective airway clearance
Potential for fluid deficit
Activity intolerance
Susceptibility to infection; pneumonia
Altered comfort
Transmission trend of the disease: highly infectious condition
Patient education
Key Info to include:
•Bed rest during acute phase
•Need for isolation
•Force fluid intake
•Alertness to symptoms of secondary
infection
Sinusitis
Overview of sinuses
•Sinuses are air filled spaces in the skull and facial
bones.
•Make up the upper part of the respiratory tract from
the nose into the throat.
•Are located in the forehead (frontal sinuses), inside the
cheekbones (maxillary sinuses), and behind the nose
(ethmoid and sphenoid sinuses).
Overview of sinuses CTs
Overview of sinuses
Overview of sinuses
Sinusitis
Disease overview
• An inflammatory process that produce changes in the mucosa of the
sinus (Inflammation of the tissues lining the sinuses)
• Can be caused by bacterial, viral or allergic conditions
• Frequently follows a common cold as infection spread from the nasal
cavity to the sinus.
• Forceful nasal blowing can also force infected materials into the
sinuses
• Swimming and diving can also cause acute onset of sinusitis
Pathophysiology
• The nasal mucosa extend to the paranasal sinuses
• Therefore nasal cavity infections spread to these sinuses causing
sinusitis (inflamed sinuses)
• When infected materials block the passageways connecting the sinuses
to the nasal cavity, the air in the sinuses gets absorbed; resulting into a
partial vacuum and sinus headache localized over the inflamed area.
• With repeated attacks or infections remain unresolved/treated, the
mucosal lining of the sinus may become permanently damaged leading
to chronic suppurative sinusitis characterized by continued purulent
nasal discharge
Clinical manifestation of sinusitis
•Pain/pressure on the affected site/sinus
•Purulent nasal discharge
•Nasal congestion & obstruction
•Fever, general malaise
Treatment plan for sinusitis
Use of nasal Decongestants
• Nasal sprays and nasal decongestants can be used for relief of the symptoms of acute
sinusitis.
• These medications help shrink the inflamed tissues and allow secretions and air to pass
through more easily.
• Over-the-counter nasal spray decongestants should only be used for a maximum of
three days.
• Prolonged use can cause tissues to become more inflamed and lead to a disorder called
rhinitis medicamentosa.
• Consult the doctor before using any drugs to treat sinusitis.
• Combinations of oral medications and nasal anti-inflammatories may be better options.
Treatment plan CTs
• Use of antibiotics Usually Unnecessary
• Most cases of sinusitis are triggered by viruses such as the common cold
virus hence can not respond to antibiotics.
• Antibiotics should only be used in cases of sinusitis where a bacteria
pathogen is suspected and documented by a culture of the mucus from
your sinuses.
• Home remedies can help relieve some symptoms of sinusitis. (Breathing
in warm humidified air can help decrease symptoms of sinusitis.)
• If symptoms are due to allergies, over-the-counter antihistamines may
help.
Nursing management
•Assessment approach; similar to above conditions
•Nursing diagnoses may include issues of:
Alteration in comfort
Sensory perceptual alteration: olfactory
Sleep pattern disturbances
Compromised breathing pattern
Nursing interventions
May include:
• Bed rest
• Elevating head of bed to promote drainage of secretions
• Applying warm compresses prn for pain relief
• Administering the ordered analgesics,, antihistamines, Abs as may be
ordered
• IEC on self care and management of the condition
IEC
Sinusitis Prevention
• Sinusitis may not be completely avoided; but there are ways to
prevent it in some cases:
Avoid smoking.
Avoid dry environments
Use a humidifier when needed
Drink plenty of fluids
Seek treatment for chronic allergies that can trigger sinus
inflammation
Other URIs (reading assignment)
1. Nasopharyngitis
2. Pharyngitis
3. Laryngitis
4. Epiglottitis
5. Lryngotrcheitis
6. Tracheitis
Key issues on URTIs
•URIs involve direct invasion of the mucosa lining
the upper airway.
•Bacterial/viral inoculation occurs when a person
directly inhales respiratory droplets from an
infected person who is coughing or sneezing.
Key issues cts
After inoculation, viruses and bacteria encounter several barriers ( physical,
mechanical, humoral, and cellular immune defences):
• Hair lining the nose filters and traps some pathogens
• Mucus coats much of the upper respiratory tract, trapping potential
invaders
• The angle resulting from the junction of the posterior nose to the pharynx
causes large particles to impinge on the back of the throat
• Ciliated cells lower in the respiratory tract trap and transport pathogens
up to the pharynx; from there they are swallowed into the stomach
Key issues CTs
•Adenoids and tonsils contain immune cells that
respond to pathogens.
•Antigen/antibody rxns act to reduce infections
throughout the entire respiratory tract.
• Resident and recruited macrophages, monocytes,
neutrophils, and eosinophils coordinate to engulf and
destroy invaders.
Key issues CTs
• A host of inflammatory cytokines mediates the immune response to
invading pathogens.
• Normal nasopharyngeal flora, including various staphylococcal and
streptococcal species, help to defend against potential pathogens.
Note:
Patients with suboptimal humoral and phagocytic immune function are
at increased risk for contracting URI, and they are at increased risk for
a severe or prolonged course of disease.
Inflammation (chronic or acute) from allergy predisposes individuals to
URI.
References
• Med/surg texts

Upper_resp_tract_edited_for_2024_cohort[1].pptx

  • 1.
    Upper respiratory tract infections MedicalSurgical Nursing 1 2024 COHORT February 2025 By D. Kamalizeni
  • 2.
    Learning objectives 1. Outlinethe common URTIs 2. Describe the etiology and pathophysiology of common upper respiratory tract infections 3. Describe clinical manifestations of common upper respiratory tract infections 4. Describe the complications of common upper respiratory tract infections 5. Describe the nursing management of patients with common upper respiratory tract infections
  • 3.
    Overview of theupper respiratory tract Structures of the upper respiratory tract include: • the nose, • the nasal cavity, •pharynx, •epiglottis, •Larynx • trachea.
  • 4.
    Key features liableto pathogen infestation include: Nasal mucosa: a sticky mucous membrane that lines the nasal cavity and traps foreign particles Para nasal sinuses: mucosal-lined, air filled cavities in cranial bones (frontal, ethmoidal, sphenoidal & maxillary bones) that surround the nasal cavity; secrete mucus that passes to the back of the nose to moisten the inhaled air. Pharynx or throat
  • 5.
    Overview of upperrespiratory tract infections (URI) • Represents the most common acute illness evaluated in the outpatient settings. • Range from the common cold—typically a mild, self-limited ailment to life-threatening illnesses such as epiglottitis. • Viruses account for most URIs • Appropriate management of URI may consist of reassurance, education, and instructions for symptomatic home treatment
  • 6.
    Definition of commonURIs • Rhinitis: Inflammation of the nasal mucosa • Sinusitis: Inflammation of the paranasal sinuses, including frontal, ethmoid, maxillary, and sphenoid • Influenza (Flu) Generalized, acute, febrile, viral disease associated with upper and lower respiratory infections • Nasopharyngitis (rhinopharyngitis or the common cold): Inflammation of the nares, pharynx, tonsils
  • 7.
    Definition of commonURIs CTs •Pharyngitis: Inflammation of the pharynx, •Epiglottitis (supraglottitis): Inflammation of the superior portion of the larynx and supraglottic area •Laryngitis: Inflammation of the larynx •Laryngotracheitis: Inflammation of the larynx & trachea. •Tracheitis: Inflammation of the trachea
  • 8.
    The common occurringupper respiratory conditions • Rhinitis • Influenza • Sinusitis
  • 9.
    The common occurringupper respiratory conditions CTs…, Rhinitis • Inflammation of the nasal mucosa • Can be allergic or viral in origin  Allergic Rhinitis • Most often, a reaction of the nasal mucosa to a specific antigen • Caused by allergy to pollens from trees, flowers, crops • Has therefore a seasonal trend
  • 10.
    Rhinitis CTs  AcuteViral rhinitis • Also referred to as common cold • Caused by virus that invade the upper respiratory tract • Highly infectious condition spread by air born droplets • Most prevalent in winter months • Can be aggravated by chills, fatigue, emotional stress • When uncomplicated, it is a self-resolving condition hence ABs are not necessary.
  • 11.
    Rhinitis CTs Pathophysiology • Invasionof the nasal mucosal by the causative agent induces inflammatory rxn • There is also leukocytes infiltration and tissue edema because of capillary permeability and vasodilation. • This inflammatory response may be confined to the nasal membranes or spread to regions where the nasal mucosa extends: (nasolacrimal/tear ducts, paranasal sinuses, oropharynx) • Can also progress to the chest as the nasal mucosal is continuous with the respiratory tract
  • 12.
    Clinical manifestations ofrhinitis •Nasal congestion causing obligatory mouth breathing •Thin watery nasal discharge •Irritating/itchy nasal cavity •Sneezing •Altered sense of smell
  • 13.
    Clinical manifestations ofrhinitis cts…, •Ocular manifestations may include profuse tearing, bilateral conjuctival edema •When inflammatory responses extend to the Para nasal sinuses, the air in the sinuses gets absorbed resulting into feeling of partial vacuum and sinus headache localized over the inflamed regions
  • 14.
    Clinical manifestations ofrhinitis CTs Extension of the inflammation to the oropharynx may present with: • Cough • Hoarseness of voice • Snoring • Recurrent need to clear the throat Decreased hearing, a sensation of fullness or popping in the ears may be evident of eustachian tube involvement
  • 15.
    Complications of rhinitis Mostlylinked with acute viral rhinitis and these include: •Sinusitis •Otitis media •Pharyngitis •Tonsillitis •Lung infections
  • 16.
    Diagnostic approaches Mostly through: •Hx taking: ( onset, aggravating factors: environmental related) • Physical examination; routine head to toe exam with focus on the general appearance & presentation of the clinical manifestations; the goal being to asses extent and severity of organ involvement Ocular involvement Extent of nasal membrane inflammation Oropharyngeal involvement • Check of vital signs • Review of lab results (FBC;)
  • 17.
    Treatment plan forrhinitis •Aimed at blocking symptoms •Maintaining optimal functioning. •Prevent complications
  • 18.
    Key rx approachesinclude: Environmental control: identifying and avoiding triggers of the allergic rxns (allergic rhinitis) Drug therapy: ( anti-histamines and decongestants;eg phenylephrine o.25%, 2 drops in each nostril); to manage symptoms: reading assignment on other specific drugs in use Supportive care: Increase fluid intake to liquefy secretions & counter loss from obligatory mouth breathing
  • 19.
    Nursing management ofpatients with rhinitis •Nursing interventions are directed towards relief of uncomfortable symptoms •Increasing fluid intake is key to aid in liquefying secretions •Administering the ordered antihistamines and decongestants helps to reduce severity of symptoms
  • 20.
    Nursing management ofpatients with Rhinitis CTs Nursing diagnoses • Defined according to specific patient presentation • Obvious ones could be: Ineffective breathing pattern……..; further defined in the context of the presence of the following: (Obligatory mouth breathing, dyspnea, snoring) Eg: Ineffective breathing pattern related to nasal mucosal membrane congestion secondary to the disease process(inflammation) evidenced by obligatory mouth breathing
  • 21.
    Nursing diagnosis CTs Goalof care: Patient to breath at ease and through the nostrils 1 hour after nursing interventions.(specify the easiness of the breathing) Nursing interventions: • Explain condition to patient to gain cooperation • Elevate head of bed to 45 degrees to facilitate mucous drainage • Assess environment for presence of offending allergen and remove it if possible • Administer the prescribed decongestant( specify name, dose etc etc) • Emphasize importance of nasal breathing
  • 22.
    Nursing diagnoses CTs Othernursing diagnoses to address the following: •Fluid intake •Nutrition •Health maintenance (need to prevent symptoms) •Susceptibility to infections like infective otitis media, infective conjunctivitis
  • 23.
    Patient education Key areasto include: • Disease process; reinforce the concept of self-care and self management of the disease • Environmental control measures and patient`s responsibilities • Medications in use, side effects and rationale for use of such medications • Importance of monitoring the symptoms, response to therapies, any difficulties, new symptoms etc etc
  • 24.
    Influenza Disease overview • Alsotermed as Flu • Generalized, acute, febrile, viral disease associated with upper and lower respiratory infections • There are 3 known groups of flu viruses ( A, B & C), all with many mutagenic strains; ie; have a remarkable ability to change over time; rendering it a widespread disease • C believed to have little pathogenic potential.
  • 25.
    Pathophysiology • Flu virusesare inhaled in mucus droplets from infected persons • These then penetrate the surface of upper resp tract mucosal cells causing cell lysis and destruction of the ciliated epithelium • This compromises viscosity of mucosa which facilitate spread of virus containing exudate to the lower resp tract • An interstitial inflammation and necrosis of the bronchiolar and alveolar result, filling the alveoli with an exudate containing leukocytes, erythrocytes
  • 26.
    Pathophysiology CTs • Regenerationof epithelium slowly begins after 5th day of the viral infestation reaching maximum within 9 – 15 days; at which time mucous production and cilia begin to appear. • Before complete regeneration, the compromised epithelium is prone to bacterial invasion and this can result into pneumonia • If virus specific antibodies are adequate, the initial viral invasion can be aborted at the port of entry • The disease is usually self limiting; acute symptoms last 2 – 7 days and are followed by convalescent period of about 1 wk
  • 27.
    Clinical manifestation Onset usuallyabrupt and characterized by: • fever, • cough, • headache, • sore throat If uncomplicated, symptoms may subside within 7 days Complications Mostly pneumonia
  • 28.
    Diagnostic approaches • Hxtaking: Hx of having travelled to flu prone regions Sudden onset fever which rises and falls Hx of general body pains, running nose, cough, sore throat • Routine physical examination: Check vital signs: high temp, increased resp rate Check for conjunctivitis, erythema of soft palate • Review lab investigation results(FBC, sputum culture)
  • 29.
    Treatment Plan •Antipyretic forfever: ASA 600 mgs tds, •Decongestants for nasal congestion eg phenylephrine o.25%, 2 drops in each nostril may be prescribed •Increase in fluid intake •Adequate rest
  • 30.
    Nursing management •Primary goalsof nursing care are directed at: Relief of symptoms Prevention of secondary infection: • Disease is highly contagious hence adherence to standard IP remains key
  • 31.
    Nursing management CTs •Nursing diagnoses are patient specific and commonly with regard to: Ineffective airway clearance Potential for fluid deficit Activity intolerance Susceptibility to infection; pneumonia Altered comfort Transmission trend of the disease: highly infectious condition
  • 32.
    Patient education Key Infoto include: •Bed rest during acute phase •Need for isolation •Force fluid intake •Alertness to symptoms of secondary infection
  • 33.
    Sinusitis Overview of sinuses •Sinusesare air filled spaces in the skull and facial bones. •Make up the upper part of the respiratory tract from the nose into the throat. •Are located in the forehead (frontal sinuses), inside the cheekbones (maxillary sinuses), and behind the nose (ethmoid and sphenoid sinuses).
  • 34.
    Overview of sinusesCTs Overview of sinuses
  • 35.
  • 36.
    Sinusitis Disease overview • Aninflammatory process that produce changes in the mucosa of the sinus (Inflammation of the tissues lining the sinuses) • Can be caused by bacterial, viral or allergic conditions • Frequently follows a common cold as infection spread from the nasal cavity to the sinus. • Forceful nasal blowing can also force infected materials into the sinuses • Swimming and diving can also cause acute onset of sinusitis
  • 37.
    Pathophysiology • The nasalmucosa extend to the paranasal sinuses • Therefore nasal cavity infections spread to these sinuses causing sinusitis (inflamed sinuses) • When infected materials block the passageways connecting the sinuses to the nasal cavity, the air in the sinuses gets absorbed; resulting into a partial vacuum and sinus headache localized over the inflamed area. • With repeated attacks or infections remain unresolved/treated, the mucosal lining of the sinus may become permanently damaged leading to chronic suppurative sinusitis characterized by continued purulent nasal discharge
  • 38.
    Clinical manifestation ofsinusitis •Pain/pressure on the affected site/sinus •Purulent nasal discharge •Nasal congestion & obstruction •Fever, general malaise
  • 39.
    Treatment plan forsinusitis Use of nasal Decongestants • Nasal sprays and nasal decongestants can be used for relief of the symptoms of acute sinusitis. • These medications help shrink the inflamed tissues and allow secretions and air to pass through more easily. • Over-the-counter nasal spray decongestants should only be used for a maximum of three days. • Prolonged use can cause tissues to become more inflamed and lead to a disorder called rhinitis medicamentosa. • Consult the doctor before using any drugs to treat sinusitis. • Combinations of oral medications and nasal anti-inflammatories may be better options.
  • 40.
    Treatment plan CTs •Use of antibiotics Usually Unnecessary • Most cases of sinusitis are triggered by viruses such as the common cold virus hence can not respond to antibiotics. • Antibiotics should only be used in cases of sinusitis where a bacteria pathogen is suspected and documented by a culture of the mucus from your sinuses. • Home remedies can help relieve some symptoms of sinusitis. (Breathing in warm humidified air can help decrease symptoms of sinusitis.) • If symptoms are due to allergies, over-the-counter antihistamines may help.
  • 41.
    Nursing management •Assessment approach;similar to above conditions •Nursing diagnoses may include issues of: Alteration in comfort Sensory perceptual alteration: olfactory Sleep pattern disturbances Compromised breathing pattern
  • 42.
    Nursing interventions May include: •Bed rest • Elevating head of bed to promote drainage of secretions • Applying warm compresses prn for pain relief • Administering the ordered analgesics,, antihistamines, Abs as may be ordered • IEC on self care and management of the condition
  • 43.
    IEC Sinusitis Prevention • Sinusitismay not be completely avoided; but there are ways to prevent it in some cases: Avoid smoking. Avoid dry environments Use a humidifier when needed Drink plenty of fluids Seek treatment for chronic allergies that can trigger sinus inflammation
  • 44.
    Other URIs (readingassignment) 1. Nasopharyngitis 2. Pharyngitis 3. Laryngitis 4. Epiglottitis 5. Lryngotrcheitis 6. Tracheitis
  • 45.
    Key issues onURTIs •URIs involve direct invasion of the mucosa lining the upper airway. •Bacterial/viral inoculation occurs when a person directly inhales respiratory droplets from an infected person who is coughing or sneezing.
  • 46.
    Key issues cts Afterinoculation, viruses and bacteria encounter several barriers ( physical, mechanical, humoral, and cellular immune defences): • Hair lining the nose filters and traps some pathogens • Mucus coats much of the upper respiratory tract, trapping potential invaders • The angle resulting from the junction of the posterior nose to the pharynx causes large particles to impinge on the back of the throat • Ciliated cells lower in the respiratory tract trap and transport pathogens up to the pharynx; from there they are swallowed into the stomach
  • 47.
    Key issues CTs •Adenoidsand tonsils contain immune cells that respond to pathogens. •Antigen/antibody rxns act to reduce infections throughout the entire respiratory tract. • Resident and recruited macrophages, monocytes, neutrophils, and eosinophils coordinate to engulf and destroy invaders.
  • 48.
    Key issues CTs •A host of inflammatory cytokines mediates the immune response to invading pathogens. • Normal nasopharyngeal flora, including various staphylococcal and streptococcal species, help to defend against potential pathogens. Note: Patients with suboptimal humoral and phagocytic immune function are at increased risk for contracting URI, and they are at increased risk for a severe or prolonged course of disease. Inflammation (chronic or acute) from allergy predisposes individuals to URI.
  • 49.