1. The document discusses various types of upper limb trauma including fractures of the clavicle, humerus, forearm, and distal radius as well as dislocations of the shoulder and elbow.
2. Key fracture classifications discussed include the Allman classification for clavicle fractures, Neer classification for proximal humerus fractures, and Bado's classification for Monteggia fractures.
3. Common mechanisms of injury involve falls onto an outstretched arm. Imaging workup involves radiographs to identify fracture patterns and displacements.
Dislocation of joint is very tricky. In this presentation radiological evaluation of Dislocation of various joints will be discussed.
This is one of the best pictoral review of important joint dislocations
Dislocation of joint is very tricky. In this presentation radiological evaluation of Dislocation of various joints will be discussed.
This is one of the best pictoral review of important joint dislocations
Monteggia fractures and neglected cases
A simple presentation to understand the fracture and its classifications and answer some coomonly asked questions regarding the neglected cases managment
JOINT DISLOCATION of hip knee and shoulder PART-2.pptxrammmramm000
JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip kn
Elbow is the most common joint to dislocate in children. Posterior dislocation is most common.
Simple dislocations are those without fracture.
Complex dislocations are those that occur with an associated fracture
this ppt is based on clinical anatomy related with upper limb which will help all medical students to understand the upper limb related clinical situations for the diagnostic purposes.
Monteggia fractures and neglected cases
A simple presentation to understand the fracture and its classifications and answer some coomonly asked questions regarding the neglected cases managment
JOINT DISLOCATION of hip knee and shoulder PART-2.pptxrammmramm000
JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip kn
Elbow is the most common joint to dislocate in children. Posterior dislocation is most common.
Simple dislocations are those without fracture.
Complex dislocations are those that occur with an associated fracture
this ppt is based on clinical anatomy related with upper limb which will help all medical students to understand the upper limb related clinical situations for the diagnostic purposes.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
4. Clavicle Fractures
• Mechanism
• Fall onto shoulder (87%)
• Direct blow (7%)
• Fall onto outstretched hand (6%)
• The clavicle is the last ossification center to
complete (sternal end) at about 22-25yo.
7. Clavicle Fractures
• Allman Classification of Clavicle Fractures
• Type I Middle Third (80%)
• Type II Distal Third (15%)
• Differentiate whether ligaments attached to
lateral or medial fragment
• Type III Medial Third (5%)
11. Proximal Humerus Fractures
• Epidemiology
• MC fracture of the humerus
• Higher incidence in the elderly, related to
osteoporosis
• Females 2:1 males
• Mechanism of Injury
• MC fall onto an outstretched arm from standing
height
• Younger patient typically present after high
energy trauma such as Motor Vehicle Accident
14. Humeral Shaft Fractures
• Mechanism of Injury
• Direct trauma MC - MVA
• Indirect trauma -fall on an outstretched hand
• Fracture pattern depends on stress applied
• Compressive- proximal or distal humerus
• Bending- transverse fracture of the shaft
• Torsional- spiral fracture of the shaft
• Torsion and bending- oblique fracture usually
associated with a butterfly fragment
16. Humeral Shaft Fractures
• Holstein-Lewis Fractures
• Distal 1/3rd fractures
• May entrap or lacerate radial nerve as the
fracture passes through the intermuscular
septum
19. Forearm Fractures
• Epidemiology
• Highest ratio of open to closed than any other
fracture except the tibia
• males > females,MC secondary to MVA,
contact sports, altercations, and falls
• Mechanism of Injury
• Commonly associated with mva, direct trauma
missile projectiles, and falls
20. Forearm Fractures
• Radiographic Evaluation
• AP and lateral radiographs of the forearm
• Don’t forget to examine and take x-ray of the
elbow and wrist
23. BADO’s
Classification
• Type I : Anterior dislocation of the radial
head
• is dislocated anteriorly and the ulna
has a fracture in the diaphyseal or
proximal metaphyseal area.
Most common type
24. BADO’s
Classification
Type II : Posterior dislocation: The radial
head is posterior/posterolaterally
dislocated, the ulna is usually fractured in
the metaphysis.
Associated with nerve palsy (PIN) and
poor prognosis
25. BADO’s
Classification
• Type III: Lateral dislocation : There is
lateral dislocation of the radial head with a
metaphyseal fracture of the ulna.
26. BADO’s
Classification
Type IV : Anterior dislocation with radius
shaft fracture
the pattern of injury is the same as with
a type I injury, with the inclusion of a
radius shaft fracture below the level of
the ulnar fracture.
27. MECHANISM OF INJURY
Type I:forced pronation of forearm
Type II:axial loading of forearm with flexed
elbow
Type III – forced abduction of elbow
Type IV - Type I mechanism in which radial
shaft additionally fails
29. GALEAZZI FRACTURE OR PIEDMONT
FRACTURE
• The combination of fracture of the distal or
middle third of the shaft of the radius and
dislocation of the distal radioulnar joint.
• counterpart of the Monteggia fracture-
dislocation
• also known as a reverse Monteggia fracture.
31. Mechanism of injury
• as indirect trauma : due to a fall on an outstretched
hand (FOOSH) with a superimposed rotation force
• Rotation determines direction of angulation
– Pronation flexion injury ( dorsal angulation )
– Supination extension injury (volar angulation)
• direct trauma to the wrist, typically on the
dorsolateral aspect
32. Types
• Type I
• apex volar
• Caused by axial loading of forearm in
supination
• dorsal displacement of radius and volar
dislocation of distal ulna
33. • Type II
• apex dorsal
• fractures are caused by axial loading of
forearm in pronation
• anterior displacement of radius and dorsal
dislocation of distal ulna
35. Greenstick fracture
• incomplete fractures of long bones
• young children, MC less than 10 years of age.
• MC mid-diaphyseal, affecting the forearm and
lower leg.
• distinct from torus fractures.
36. Mechanism
Greenstick fractures - force applied to a bone results in bending of
the bone such that the structural integrity of the convex surface is
overcome.
disintegration of the cortex results in fracture of the convex surface.
the bending force applied does not break the bone completely and
the concave surface of the bent bone remains intact.
This can occur following an angulated longitudinal force applied
down the bone (e.g. an indirect trauma following a fall on an
outstretched arm), or after a force applied perpendicular to the
bone (e.g. a direct blow).
different, and much less common, than the torus fracture that
results in buckling of the cortex on the concave side of the bend
and an intact convex surface.
37.
38. Greenstick fracture
• Radiographic features
• Plain radiograph
• usually mid-diaphyseal
• occur in tandem with angulation
• incomplete fracture, with cortical breach of
only one side of the bone.
40. Distal Radius Fractures
• Epidemiology
• MC fractures of the upper extremity
• Common in younger and older patients as a result of
direct trauma such as fall on an outstretched hand
• Increasing incidence due to aging population
• Mechanism of Injury
• MC a fall on an outstretched extremity with the wrist in
dorsiflexion
• High energy injuries- significantly displaced, highly
unstable fractures
42. Definition:
• It was first described by Abraham colles in 1814.
• Colles fracture is the fracture at the distal end of
radius, at its cortico cancellous junction(about 2cm
from the distal articular surface).
• It is not just the fracture of distal radius but
the fracture dislocation of the inferior radio-ulnar
joint.
• Most common age group is above forty years,
occuring most commonly in women.
44. Patho-Anatomy:
• Displacement: The fracture line runs transversely
at the cortico-cancellous junction. In many cases
one or more displacements may occur as follows.:
• Impaction of fragments
• Dorsal displacement
• Dorsal tilt
• Lateral displacement
• Lateral tilt
• Supination
45. Clinical features:
• Pain
• Swelling
• Deformity- There is classic ‘dinner-fork
deformity’ seen in colles’ fracture.
• Radial styloid process lies in the same level or
little higher than the ulnar styloid process.
47. Diagnosis:
•It is important to differentiate Colles’ fracture
from other fractures occurring at the same site,
such as Smith’s fracture, Barton’s fracture by
looking at the displacements.
48. X-RAY:
• Lateral view
• Dorsal tilt- It can be detected by looking at
the direction of distal articular surface
• AP view
• Lateral tilt- similarly it can be detected by
looking at the articular surface if it faces
medially it is normal,if it becomes horizontal or
faces laterally ,a lateral tilt is present.
57. Shoulder Dislocations
• Posterior Dislocation
• Adduction/Flexion/IR at time of injury
• Electrocution and Seizures cause overpull of
subscapularis and latissimus dorsi
• Look for “lightbulb sign” and “vacant glenoid”
sign
• Reduce with traction and gentle anterior
translation
58.
59. Shoulder Dislocations
• Inferior Dislocations
• Hyperabduction injury
• Arm presents in a flexed “asking a question”
posture
• High rate of nerve and vascular injury
• Reduce with in-line traction and gentle
adduction
68. Normal Alignment
• Anterior humeral line- line drawn along
anterior surface of humeral cortex should pass
through the middle third of the capitellum
• Radiocapitellar line- Line drawn through the
proximal radial shaft and neck should pass
through to the articulating capitellum
69.
70.
71.
72. Elbow Dislocations
• Epidemiology
• 11-28% of injuries to the elbow
• Posterior dislocations most common
• Highest incidence in the young 10-20 years and usually
sports injuries
• Mechanism of injury
• Most commonly due to fall on outstretched hand or elbow
resulting in force to unlock the olecranon from the trochlea
• Posterior dislocation following hyperextension, valgus
stress, arm abduction, and forearm supination
• Anterior dislocation ensuing from direct force to the
posterior forearm with elbow flexed
73. Elbow Dislocations
• Radiographic Evaluation
• AP and lateral elbow films should be obtained
both pre and post reduction
• Careful examination for associated fractures