Tmj/certified fixed orthodontic courses by Indian dental academyIndian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Basics about TMJ ( development fuction movement etc ) with classification of tmj disorders and stress on tmj examination.
Also covers muscles of masstication
Tmj/certified fixed orthodontic courses by Indian dental academyIndian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Basics about TMJ ( development fuction movement etc ) with classification of tmj disorders and stress on tmj examination.
Also covers muscles of masstication
Temporomandibular joint anatomy and functionDR POOJA
diarthrodial joint
The masticatory system is the functional unit of the body primarily responsible for chewing, speaking and swallowing. Components also play a major role in tasting and breathing.
The system is made up of bones, joints, ligaments, teeth and muscles.
In addition ,there is an intricate neurologic controlling system that regulates and coordinates all these structural components.
The Temporomandibular joint (TMJ) is formed by the articulation between the articular eminence and the anterior part of the glenoid fossa of the squamous part of temporal bone above and the condylar head of the mandible below.
The TMJ contains a fibrous intraarticular disk that is interposed between the articular surface and functions as a shock absorber.
The TMJ is a compound joint that can be classified by anatomic type as well as by function.
Anatomically the TMJ is a diarthrodial joint, which is a discontinuous articulation of two bones permitting freedom of movement that is dictated by associated muscles and limited by ligaments.
It is also a synovial joint, lined on its inner aspect by a synovial membrane, which secretes synovial fluid. The fluid acts as a joint lubricant and supplies the metabolic and nutritional needs of the non-vascularized internal joint structures.
Functionally the TMJ is a compound joint, composed of four articulating surfaces:
articular facets of the temporal bone
articular facets of the mandibular condyle
superior surface of the articular disk
inferior surface of the articular disk.
The articular disk divides the joint into two compartments. The lower compartment permits hinge motion or rotation and hence is termed ginglymoid.
The superior compartment permits sliding (or translatory) movements and is therefore called arthrodial. Hence the temporomandibular joint as a whole can be termed ginglymoarthrodial.
SYNONYMS
Craniomandibular joint/ articulation
Mandibular joint
Bicondylar joint
Modified ball and socket joint
Compound joint
Diarthroidal joint
Temporomandibular joint Disorder in oral pathology HIMANSHU DHAKAD
In this slide we talk about temporomandibular joint disorders
Its introduction,classification , anatomy , histology in details
Also cover articular disorder
Traumatic disturbances
Non articular disorders
Development disturbance etc.
Temporomandibular joint anatomy and functionDR POOJA
diarthrodial joint
The masticatory system is the functional unit of the body primarily responsible for chewing, speaking and swallowing. Components also play a major role in tasting and breathing.
The system is made up of bones, joints, ligaments, teeth and muscles.
In addition ,there is an intricate neurologic controlling system that regulates and coordinates all these structural components.
The Temporomandibular joint (TMJ) is formed by the articulation between the articular eminence and the anterior part of the glenoid fossa of the squamous part of temporal bone above and the condylar head of the mandible below.
The TMJ contains a fibrous intraarticular disk that is interposed between the articular surface and functions as a shock absorber.
The TMJ is a compound joint that can be classified by anatomic type as well as by function.
Anatomically the TMJ is a diarthrodial joint, which is a discontinuous articulation of two bones permitting freedom of movement that is dictated by associated muscles and limited by ligaments.
It is also a synovial joint, lined on its inner aspect by a synovial membrane, which secretes synovial fluid. The fluid acts as a joint lubricant and supplies the metabolic and nutritional needs of the non-vascularized internal joint structures.
Functionally the TMJ is a compound joint, composed of four articulating surfaces:
articular facets of the temporal bone
articular facets of the mandibular condyle
superior surface of the articular disk
inferior surface of the articular disk.
The articular disk divides the joint into two compartments. The lower compartment permits hinge motion or rotation and hence is termed ginglymoid.
The superior compartment permits sliding (or translatory) movements and is therefore called arthrodial. Hence the temporomandibular joint as a whole can be termed ginglymoarthrodial.
SYNONYMS
Craniomandibular joint/ articulation
Mandibular joint
Bicondylar joint
Modified ball and socket joint
Compound joint
Diarthroidal joint
Temporomandibular joint Disorder in oral pathology HIMANSHU DHAKAD
In this slide we talk about temporomandibular joint disorders
Its introduction,classification , anatomy , histology in details
Also cover articular disorder
Traumatic disturbances
Non articular disorders
Development disturbance etc.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
3. Ginglymo diarthrodial joint.
Synovial joint between head of the mandible and
mandibular fossa on the under surface of the
squamous part of the temporal bone.
Separated into upper and lower cavities by
fibrocartilaginous disc within joint.
7. Histology of articulating surfaces
• Bony surface of head of the condyle is made of dense
compact bone with cancellous bone in the center.
• Articular fossa is lined by a thin layer of compact
bone.
• Articular eminence has a core of cancellous bone
covered by a layer of compact bone.
8.
9. Articular fibrous covering
• Composed of 4 layers
1. Articular zone
2. Proliferative zone
3. Fibrocartilagenous zone
4. Calcified zone
10. Articlular zone
• Most superficial zone
• Composed of fibrous tissue
consisting of fibroblasts
scattered in a dense type 1
collagen fibers arranged in
bundles
11. Proliferative zone
• Second zone
• Highly cellular and composed
of undifferentiated
mesenchymal tissue
• This layer is responsible
for proliferation of
articular capsule in response
to functional demand
12. Fibrocartilagenous zone
• Bundles of collagen fibers arranged in a crossing
pattern and some in radiating pattern
• This layer provides resistance against compressive
or lateral surfaces
13. Calcified zone
• Made up of chondrocytes and chondroblasts
distributed throughout the articular cartilage
• This zone provides an active site for remodeling
activity as endosteal bone growth proceeds
15. Articular capsule
• It is a dense collagenous sheet or sac that encloses
the joint space
• It is circumferentially attached to the rim of
glenoid fossa and articular eminence above and to
the neck of the condyle below
16. Histology of articular capsule
• Composed of two layers:
1. Outer fibrous layer of dense connective tissue
2. Inner layer termed synovial membrane
17.
18. Synovial membrane
• Lines the inner aspect of fibrous capsule and
therefore forms the lining of joint cavity
• Consists of two layers:
1. Inner cellular intimal layer
2. Highly Vascular subintimal layer
20. Intimal layer
• Consists of few layers (1-4) of synovial cells,
distributed in an amorphous intercellular matrix.
• These cells are not attached to each other and do
not form a continuous layer.
• Three types of cells are mainly seen
21. • Type A cells: macrophage like cells:
- have an irregular outline with plasma membrane
- cytoplasm is rich in mitochondria, golgi apparatus,
lysosomes
- type A cells have phagocytic properties
22. Type B cells (fibroblast like cells or secretory
cells)
• These cells are rich in RER and involved in
synthesis of hyaluronic acid which is added to
synovial fluid
Third type: cells have cellular morphology between
the other two types
23.
24. Synovial fluid
• Joint cavity contains appxomiately 1ml of SF
• Formed by diffusion of plasma from the rich
capillaries of subintimal layer
• Proteins and hyaluronic acid secreted by
fibroblasts is also added
• Also contain few synovial cells and defense cells
25. Functions of synovial membrane
1. Lubrication: by providing a fluid environment for
the joint
2. Nutritive: provides nutrition to avascular fibrous
covering and center part of articular disc
3. Regulatory: controls movement of nutrients,
electrolytes
26. 4. Secretory: intimal cells secrete proteins and
hyaluronic acid to the synovial fluid
5. Phagocytic: Type A cells are phagocytic and
therefore help in debriding
27. ARTICULAR DISC
Biconcave oval
structure located
between the condyle
and articular surface of
temporal bone.
The disc is thinnest at
the central zone and
thicker in posterior and
anterior border zone
28. Thick posterior and anterior band act as wedge
giving the disc a self seating capacity with the
condyle functioning against the intermediate zone
Contour of the disc with in the thick bands helps to
prevent displacement of the disc from the condyle
during translation
29. • Four distinct regions namely:
1. Anterior band
2. Intermediate zone
3. Posterior zone
4. Bilaminar region
30. • The shape of articular disc conforms to the articular
surfaces which it is opposed
• The upper contour of the disc is concave in the
anterior region to fit under articular eminence.
• Convex posteriorly and loosely rest against articular
fossa
• Lower surface of disc is concave thus adapting the
upper surface of mandibular condyle
31.
32. • Articular disc divides the joint space into
1. Upper compartment called Temporo-discal
which is between disc and temporal fossa
2. Lower compartment called condylo-discal
situated between disc and condyle
33. • Upper joint space allows translatory movement
anteriorly along the slopes of the articular
eminence to produce an anterior and inferior
movement of the jaw.
• Lower joint space allows the rotational movement
of head of the condyle which is also called as hinge
movement
34. Histology of articular disc
• Composed of dense fibrous tissue with tightly
packed interlacing collagen fibers.
• The fibroblasts are elongated with long processes.
• Few elastic fibers may be present
• The center portion of disc is devoid of blood
vessels and nerves while periphery is highly
vascular.
35. Functions of articular disc
• Divide the joint cavity into two compartments and
therefore allowing different types of mandibular
movements
• Since it is soft tissue component between two hard
tissue components of the joint, it prevents rubbing
or friction between these components
36. • Acts as a cushion against heavy load and therefore
helps in shock abosrption
• Stabilizes the condyle by filling up the space
between the articulating surfaces
• Proprioceptive nerve fibers present in the anterior
and posterior portion of disc help in regulating
movements of condyle
38. Temporomandibular ligament or
lateral ligament
• Fan shaped ligament functioning to reinforce the
lateral wall of the articular capsule
• Limits the lateral and posterior movements of the
joint
39. • It is attached superiorly to the articular tubercle and
runs posterio inferiorly and laterally to attach to
neck of mandible
40. Accessory ligaments
• Two accessory ligaments are:
1. Sphenomandibular ligament:
• Extends form the spine of sphenoid to the lingula and
lower margin of mandibular foramen
• It represents residual perichondrium of meckel’s
cartilage
• Overmovements of mandible is limited by this
ligament
41. 2. Stylomanibular ligament:
• Extends from lateral border of the styloid process
to the posterior border of ramus of the manible
above its angle
• It represents the thickened part of the investing
layer of deep fascia of the neck
• Helps in limiting protrusive movement