The stages of infectious disease include incubation, prodrome, illness, and convalescence. During incubation, pathogens grow and multiply without symptoms appearing. Prodrome involves nonspecific symptoms before specific symptoms of illness. Illness is when signs and symptoms specific to the infection are present. Convalescence occurs as acute symptoms disappear. An infection develops when an infectious agent enters a host through a portal of entry, multiplies within the host, and exits through a portal of exit, facilitated by the mode of transmission between reservoirs. Nurses follow infection control practices to break this chain and prevent infection.
Stages of Infection, Comparison of Viral and Bacterial Infection /w QuizJustinJiYeon
(Mapeh report unit 3 lesson 3)Stages of Infection, Comparison of Viral and Bacterial Infection. What is the diffirence between Viral and Bacterial infection? With Quiz at the last. Exact need for your report. Detailed.
Chain of infection is a process in which a favorable condition is required for micro-organism to spread or transfer from reservoir to a susceptible host.
Stages of Infection, Comparison of Viral and Bacterial Infection /w QuizJustinJiYeon
(Mapeh report unit 3 lesson 3)Stages of Infection, Comparison of Viral and Bacterial Infection. What is the diffirence between Viral and Bacterial infection? With Quiz at the last. Exact need for your report. Detailed.
Chain of infection is a process in which a favorable condition is required for micro-organism to spread or transfer from reservoir to a susceptible host.
Infectious Agents
Infectious agents are tricky little invaders. They sneak in, use our body, and often make us sick in the process.
These causes infectious disease.
They are collectively known as Pathogens
Infectious Agent No. 1
Bacteria
Bacteria
Bacteria
Bacteria
What are Infectious Agents?
An Infectious Agent is something that infiltrates another living thing, like you. When an infectious agent hitches a ride, you have officially become an infected host.
There are four main classes of Infectious Agents. These fab four can infect all sorts of living things.
This ppt contains all the information about Modes of disease transmission. It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved), and everyone who is interested in knowing about it
the chain of infection, if we think of it as an actual chain , is made up of six different links: pathogen, reservoir, portal of exit, means of transmission, portal of entry and new host.
A nosocomial infection is contracted because of an infection or toxin that exists in a certain location, such as a hospital. People now use nosocomial infections interchangeably with the terms health-care associated infections (HAIs) and hospital-acquired infections. For a HAI, the infection must not be present before someone has been under medical care.
The most common types of HAIs are:
-urinary tract infections (UTIs)
surgical site infections
-gastroenteritis
-meningitis
-pneumonia
This is an Embedded version of the presentation. This Presentation is Updated with some new graphics and interiors,it will help everyone to understand the lesson "Why do we fall ill".
Infectious Agents
Infectious agents are tricky little invaders. They sneak in, use our body, and often make us sick in the process.
These causes infectious disease.
They are collectively known as Pathogens
Infectious Agent No. 1
Bacteria
Bacteria
Bacteria
Bacteria
What are Infectious Agents?
An Infectious Agent is something that infiltrates another living thing, like you. When an infectious agent hitches a ride, you have officially become an infected host.
There are four main classes of Infectious Agents. These fab four can infect all sorts of living things.
This ppt contains all the information about Modes of disease transmission. It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved), and everyone who is interested in knowing about it
the chain of infection, if we think of it as an actual chain , is made up of six different links: pathogen, reservoir, portal of exit, means of transmission, portal of entry and new host.
A nosocomial infection is contracted because of an infection or toxin that exists in a certain location, such as a hospital. People now use nosocomial infections interchangeably with the terms health-care associated infections (HAIs) and hospital-acquired infections. For a HAI, the infection must not be present before someone has been under medical care.
The most common types of HAIs are:
-urinary tract infections (UTIs)
surgical site infections
-gastroenteritis
-meningitis
-pneumonia
This is an Embedded version of the presentation. This Presentation is Updated with some new graphics and interiors,it will help everyone to understand the lesson "Why do we fall ill".
There is an important distinction among the 3 terms, prevention, control, and eradication. Prevention is of most immediate concern to the individual veterinarian in private practice. Prevention can be defined as inhibiting the introduction of disease into an area, herd, or individual.
Control is a more appropriate term when disease is already present. Control efforts consist of the steps taken to reduce the problem to a tolerable level.
Eradication is the final step in disease control efforts; it consists of complete elimination of the disease-producing agent from a defined geographic region.
- http://www.onemedicine.tuskegee.edu/
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
1. STAGES OF INFECTIOUS DISEASE:
INCUBATION;
Time from entrance of pathogen into the body to appearance of first symptoms;
during this time pathogens grow and multiply
PRODROME:
Time from onset of nonspecific symptoms such as fever, malaise, and fatigue to
move specific symptoms
ILLNESS:
Time during which child demonstrates signs and symptoms specific to an infection
type
CONVALESCENCE:
Time when acute symptoms of illness disappear
The Nature of Infection
The straightforward definition of an infection is the entry and
multiplication of an infectious agent in the tissue of a host. In other
words, an infection is an overgrowth of bad bacteria. The infectious
agent is called a pathogen.
2. The presence of a pathogen does not mean that an infection will
necessarily begin. The development of an infection happens in a cycle
that depends on all elements of the cycle being present. There are six
elements in the chain of infection:
• Infectious agent – A microbial organism with the ability to cause
disease. The greater the organism's virulence (ability to grow
and multiply), invasiveness (ability to enter tissue) and
pathogenicity (ability to cause disease), the greater the
possibility that the organism will cause an infection. Infectious
agents are bacteria, virus, fungi, and parasites.
• Reservoir – A place within which microorganisms can thrive and
reproduce. For example, microorganisms thrive in human beings,
animals, and inanimate objects such as water, table tops, and
doorknobs.
• Portal of exit – A place of exit providing a way for a microorganism
to leave the reservoir. For example, the microorganism may
leave the reservoir through the nose or mouth when someone
sneezes or coughs. Microorganisms, carried away from the body
by feces, may also leave the reservoir of an infected bowel.
• Mode of transmission – Method of transfer by which the organism
moves or is carried from one place to another. The hands of the
health care worker may carry bacteria from one person to
another.
• Portal of entry – An opening allowing the microorganism to enter
the host. Portals include body orifices, mucus membranes, or
breaks in the skin. Portals also result from tubes placed in body
cavities, such as urinary catheters, or from punctures produced
by invasive procedures such as intravenous fluid replacement.
• Host – A person who cannot resist a microorganism invading the
body, multiplying, and resulting in infection. The host is
susceptible to the disease, lacking immunity or physical
resistance to overcome the invasion by the pathogenic
microorganism.
An infection will only develop if this chain stays intact. As nurses we
follow infection prevention and control practices to break the chain so
that infection will not develop.
Contusion Discoloration and edema of eyelids
Corneal abrasion May require ointment to smooth injury or
antibiotic ointment
Eyelid injury Vision is usually unaffected
3. Foreign body Requires careful removal only by a health care
professional
Scleral hemorrhage Erythema that resolves gradually without
intervention
VISION AND HEARING MILESTONES:
FUNCTIONAL HEARING Eye color Binocular vision Visual acuity of 20/20
SIGNS AND SYMPTOMS OF CHILDREN WITH HEARING LOSS AT (A) INFANT, (B)
YOUNG CHILD, (C) OLDER CHILD STAGES:
INFANT:
-Wakes only to touch, not environmental noises
-Does not startle to loud noises
-Does not turn to sound by 4 months of age
-Does not babble at 6 months of age
-Does not progress with speech development
YOUNG CHILD:
-Does not speak by 2 years of age
-Communicates needs through gestures
-Does not speak distinctly, as appropriate for his/her age
-Displays developmental (cognitive) delays
-Prefers solitary play
-Displays immature emotional behavior
-Does not respond to ringing of the telephone or doorbell
-Focuses on facial expressions when communicating
OLDER CHILD:
-Often asks for statements to be repeated
-Is inattentive or daydreams
-Performs poorly at school
-Displays monotone or other abnormal speech
-Gives inappropriate answers to questions excepts when able to view face of
speaker