Shigella species and other pathogens can cause food poisoning and gastrointestinal infections through contaminated food or water. Shigella is a gram-negative rod that causes dysentery through invasion and infection of the colonic epithelial cells. Symptoms include abdominal pain, bloody stool, and diarrhea. Treatment involves antibiotics like ampicillin or fluid/electrolyte replacement. Prevention relies on proper hygiene, sanitation, and avoiding contaminated foods/water.
Non-typhoidal Salmonellosis, is one of the most common and widely distributed foodborne diseases, with tens of millions of human cases occurring worldwide every year.
In the United States, the incidence of NTS infection has doubled in the past 2 decades.
In 2009 there were 14 million cases of NTS.
Escherichia coli species are components of the
Normal animal and human colonic flora;
Flora of a variety of environmental habitats, including long-term care facilities (LTCFs) and hospitals.
E.coli are the cause of most nosocomial infections.
Shigellosis = inflammation of intestines (especially the colon) with accompanying severe abdominal cramps, tenesmus and frequent, low-volume stools containing blood, mucus and fecal leukocytes.
Non-typhoidal Salmonellosis, is one of the most common and widely distributed foodborne diseases, with tens of millions of human cases occurring worldwide every year.
In the United States, the incidence of NTS infection has doubled in the past 2 decades.
In 2009 there were 14 million cases of NTS.
Escherichia coli species are components of the
Normal animal and human colonic flora;
Flora of a variety of environmental habitats, including long-term care facilities (LTCFs) and hospitals.
E.coli are the cause of most nosocomial infections.
Shigellosis = inflammation of intestines (especially the colon) with accompanying severe abdominal cramps, tenesmus and frequent, low-volume stools containing blood, mucus and fecal leukocytes.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
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Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
2. GENERAL CHARACTERISTICS
Gram negative rods, non-lactose
fermenters ( pale colonies on Mac
Conkeys medium)
Capsule "K" and Somatic ``O``
antigen
Exotoxins:
1. Shiga toxin.
2. Entero toxin (similar to
E. coli
LT)
3.
4.
5.
6. 1. S. dysenteriae A
Type 1 (Asia, Japan, Central America)
May go systemic.
Type 2 (Europe) Less severe form
2. S. flexneri B Most common.
Worldwide.
3. S. boydi C Worldwide.
4. S. sonnei D Most common in USA (2-
year olds)
8. EPIDEMIOLOGY:
Reservoir: Strict human pathogen.
Human colon, food, feces, but doesn't
live long outside of host.
`` Food, fingers, flies and
feces``
Infectious dose: less than 100
organisms .
Incubation period: 1-4 days .
Oral >>> Stomach >>> Small
Intestine >>>large intestine
>>>Feces>>>Oral
9. PATHOGENESIS:
Invasion of colonic epithelial cells -
Reproduction (108 to 109/ml)
within 12 hours in distal small
bowel. In 1 to 4 days invades
large intestine.
Ulceration (purulent with PMN's)
<<Not systemic and usually self-
limiting, lasts a few days to a
month>>
10. DIAGNOSIS:
Selective media needed for isolation.
Stool culture for isolation.
Plate rapidly, organisms die.
Selective media:
Mac Conkey agar
(XLD) Xylose-Lysine-Deoxycholate
agar
EMB
Salmonella Shigella (SS) agar
13. Gastrointestinal Tract
Infections and Food
Poisoning.
Food borne and Water borne
diseases.
Varieties of Microbes cause G.I.T.
infections.
MUSHROOMS FUNGI
PARASITES BACTERIA & VIRUSES
14. Worldwide
Diarrheal diseases
second only to
Respiratory
diseases as cause
of Adult death.
Leading cause of
child death in
Asia, Africa and
South America.
Each year 5
million children
(13,600/day) die
of Diarrheal
diseases.
15. HOW TO INVESTIGATION OF FOOD
POISONING ?
SOURCE
SYMPTOMS
INCUBATION PERIOD
MECHANISM
``TREATMENT PREVENTION AND
CONTROL``
16. 1. Staph. aureus gram positive coccus
Dairy & bakery products like,
mayonnaise, sauces pastries ice
creams, processed meat etc. Salads
also common source. Heat stable
Rapid onset with short duration1-8
hours to 24 hours. Vomiting, little or
no diarrhea, no fever.
Symptomatic.
Hygienic measures.
17. 2. Bacillus cereus
gram positive rod.
Food poisoning: Bacteria or Enterotoxin in
food.
``Reheated fried rice or Chinese
rice``
Heat-stable Enterotoxin …mainly vomiting.
Heat-labile Enterotoxin
mainly diarrhea by stimulation of
adenylate cyclase. (increases cAMP).
Incubation period 2-16 hours. Vomiting or
Diarrhea; no fever.
Symptomatic: Rice should not be reheated.
Sauces creams prepared and stored
properly.
18. 3. Clostridium perfringens
gram positive rods
Spores in soil and food
Enterotoxin.
Heat-labile protein.
Inhibits glucose transport & damages int.
epithelium causes protein loss into the
intestinal lumen.
Diarrhea; little or no vomiting; no fever.8-16
hours incubation
Symptomatic No antibiotics , Foods
adequately cooked.
19. 4. Clostridium botulinum gram positive rod.
Canned foods.
In newborns ingestion of honey as``
GHUTTI``
Protein prevents release of acetylcholine at
neuro-muscular junction causing flaccid
paralysis;
Botulism; flaccid Paralysis
of muscles; ptosis of eyelids, facial muscle
paralysis.
Heat labile toxin:destroyed by proper
cooking
Trivalent Antitoxin AB&E along with
respiratory support. Proper sterilization and
vacuum packed cooked food for canning.
Discard swollen cans.
20. 5. Vibrio cholera gram negative
curved rod .
Ingestion with contaminated water
or food.
Contaminated water supplies,
Lack of septic tanks springs, wells
become infected during floods.
Cholera; Severe Diarrhea; Rice
water stools;
Massive IV fluids.
Strict Public health measures,
Boiled or chlorinated water in
epidemics.
Cholera Pots.
Tetracyclines shorten exertion of
bacteria.
Rehydration.
21.
22. 6. Enterotoxigenic (ETEC) E. coli
contaminated water or cells.
Traveler's Diarrhea; watery, self limited
diarrhea, vomiting,
cramps, nausea,
low-grade fever, 1-3 days duration.
Also known as Turista Antibiotics not
advocated.
Septran can shorten illness. Rehydration.
7. Enteropathogenic. (EPEC) E.coli
(EPEC) Ingestion of contaminated water or
food. No toxins24-48 hours Infant
diarrhea with fever, nausea, vomiting
nonbloody diarrhea.
23. 9. Enteroaggregative (EAggEC)
E. coli Ingestion contaminated water.
Persistent watery diarrhea with
vomiting. and dehydration in Infants.
Can lead to bloody stools.
10. Enteroinvasive (EIEC) same
11. Enterohemmorhagic (EHEC) E. coli
(hemolytic uremic syndrome)
[HUS]).
26. 13, Clostridium difficile gram positive rod.
Treatment of a patient with antibiotics
loss of normal flora allowing
Cl. difficile to
overgrow. Especially after Abdominal
surgery, abrupt onset of bloody
diarrhea fever and abdominal pain.
Antibiotic associated or
Pseudomembranous colitis.
Causative agent should be withdrawn
esp. Clindamycin.
Treatment with Vancomycin. Fluid
replacement
27. 14. Vibrio parahemolyticus
slightly curved gram negative rod.
Ingestion of contaminated undercooked
or raw seafood.
Organisms grow in gut and invade
epithelium.
Most common cause of diarrhea in
Japan. Pseudo membranous colitis.
Self limiting Avoid undercooked or
contaminated sea food.
28. 14. Rotavirus fecal-oral contamination
No toxins Winter diarrhea-fever,
abdominal pain and Vomiting (mostly
in Infants.)
Fluid replacement, Prevent infection from
spreading in nurseries.
15. Norwalk Agent fecal-oral
contamination. No toxins. Summer
diarrhea- fever, abdominal pain,
diarrhea, and vomiting.
Rehydration
29. 16 . Giardia lamblia Intestinal parasite
Ingestion of Contaminated water or food.
Tooth brushes .
``Upper part of intestine `` Duodenum
Attach to microvillus of small intestine
Giardiasis- fatty, foul smelling diarrhea,
Flatulence(gas) ,
Metronidazole 10 gms in 10 days
Secondal short treatment 4 tabs,
Symptomatic.
30. 17. Shigella S.sonnei S. flexneri S.
boydii, S. dysenteriae . Transmitted by
the 4-F's;``
food, fingers, feces, and flies.``
Fever and cramping Abdominal pain is
Prominent. Diarrhea usually appears after
48hours, with dysentery supervening about
2 days later.
Amoxil and Septran in severe cases.
Electrolyte and fluid replacement
31. 18 . Salmonella typhi, Salmonella
paratyphi A, B, and C, Salmonella
cholerasuis : Ingestion of fecally
contaminated food or water. Able to
survive in neutrophils. Typhoid fever,
paratyphoid or enteric fever.
abdominal pain and
discomfort,
constipation bloody feces nausea,
vomiting, Diarrhea
Antibiotics like Amoxil, Chloramphenicol
Quinolones etc. Soft diet. Fluids
32. 19. Salmonella enteriditis 1500 serotypes
gram negative rod Fecal-oral
transmission contaminated water,
Food (poultry, eggs, or dairy
products). Nausea vomiting Diahorrea
Antibiotics not indicated
except in infants
Fluid and electrolyte
replacement.
33. 20 . Campylobacter jejuni coma or seagull-
shaped gram negative rod. Animal to
human transmission, contaminated
water,
raw milk and food especially poultry.
Verotoxin - similar to the Shigella toxin.
2-10 days
bloody diarrhea. Self limiting,
usually recovers in 5-10 days.
Erythromycin in severe cases
34. 21.Yersinia enterocolitica and Yersinia
pseudotuberculosis.
Spread via contaminated meat or milk.
Ingestion of uncooked or undercooked
meat invasins allows entry in cells in the
Payer’s patches, diarrhea, fever,
abdominal pain ,
If severe treat with Gentacin.
Fluid and electrolyte replacement
35. 22. Francisella tularensis
Ingestion of uncooked or under cooked
mammals. Tularemia a zoonosis disease,
of bird fanciers.
Present in bird droppings. No toxins.
Continuous fever (without chills or sweating
(muscle pains), Severe headaches,
Hepatosplenomegaly
Usually no vomiting or Diarrhea.
Myalgias Streptomycin.
Symptomatic treatment
36. 23 . Helicobacter pylori
curved rod,
fecal oral spread
Peptic ulcer, Gastritis results in dyspepsia,
Cramps, nausea, vomiting and Flatulence.
Triple drug therapy, Antacids Bismuth
sulphate.
37. 24. Entamoeba histolytica parasite
protozoa
Transmitted by the
4-F's; food, fingers, feces, and flies
dysentery= bloody Mucoid diarrhea,
flask shaped ulcers in the Colon.
Can cause Liver Abscess.
Metronidazole Surgery may be required.
38. 25. Listeria
monocytogenes
present all around in the
environment.
It has also been found in
low numbers in many
foods.
In certain foods, such as
cheeses and pâtés, it
may be present in
higher numbers.
Eating foods
containing high levels
of Listeria
monocytogenes is
generally the cause
of illness
39. Prevention:
``Hand washing is the most
effective means of
preventing diarrheal
infections that are passed
from person to person. ``
Dirty hands carry infectious germs into
the body when a child bites his nails or
puts any part of his hands into his
mouth.
Children should wash their hands
frequently, especially after using the
toilet and before eating.
Clean bathroom surfaces .
40. Food and water also can carry
infectious germs: 08 tips
1. Be sure to wash fruits and
vegetables thoroughly before eating.
2. Wash kitchen counters and cooking
utensils after they have been in
contact with raw meat, especially
poultry.
3. Refrigerate meats as soon as
possible after bringing them home
from the supermarket, and cook them
until they are no longer pink.
4. After meals, refrigerate cooked
leftovers as soon as possible.
41. 5. When traveling, never drink from
streams, springs.
6. In our country , it may be safer to drink
only bottled water and drinks rather than
water from a tap.
7. Exercise caution when buying prepared
foods from street vendors, especially if no
local health agency oversees their
operations.
8. Pets, especially reptiles, can spread
germs if their feeding areas are not
separate from family eating areas.