The document discusses various topics related to indirect pulp capping, direct pulp capping, and pulpotomy procedures. Indirect pulp capping involves sealing a tooth with a deep carious lesion using a protective material to stimulate healing and repair, avoiding direct pulp exposure. It is aimed at maintaining pulp vitality. Direct pulp capping places a material directly on an exposed pulp to encourage reparative dentin formation. Pulpotomy involves removing part of the coronal pulp as an emergency procedure to preserve the remaining radicular pulp. Various materials used and techniques for each procedure are described.
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
A brief description of all topics to recent advances,SDD, host modulation and diabetes, host modulation in smokers, chemically modified tetracyclines, bisphosphonates
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
The presentation features the pulp reparative and regenerative procedures which can be carried out in immature teeth. It involves development of mature tooth from an immature one by root formation and root fixation as a preparatory phase for root canal treatment.
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
The presentation features the pulp reparative and regenerative procedures which can be carried out in immature teeth. It involves development of mature tooth from an immature one by root formation and root fixation as a preparatory phase for root canal treatment.
Dense Evaginatus: Management Using Novel Materials A Case ReportQUESTJOURNAL
ABSTRACT: Dens evaginatus is an uncommon developmental anomaly of human dentition characterized by a projection of enamel and dentin that usually encloses pulp tissue. Most commonly found as the tubercle on the occlusal surface of mandibular premolars and lingual surface of anterior teeth.Due to occlusal trauma this tubercle tends to fracture thus exposing the pathway to the pulp chamber of teeth. This case reports about the presentation of dens evaginatus in mandibular premolar 35 which was associated with open apex and chronic apical periodontitis. Root canal treatment was performed with tooth 35. DFDBA apical barrier and Biodentine as an apical plug was placed showing successful management of the same.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Phone Us ❤85270-49040❤ #ℂall #gIRLS In Surat By Surat @ℂall @Girls Hotel With...
Pulp Therapy by nishtha
1.
2. INDEX SNO. TOPIC PAGE NO. 1) INDIRECT PULP CAPPING A) DEFINITION B)OBJECTIVES C)INDICATIONS D) CONTRAINDICATIONS E) INDIRECT PULP THERAPY F) INDIRECT PULP CAPPING TECHNIQUE 14 16 19 20 21 26 2) DIRECT PULP CAPPING A)DEFINITION B) OBJECTIVES C) INDICATIONS D) CONTRAINDICATIONS E) DIRECT PULP CAPPING TECHNIQUE 43 45 46 48 54
3. SNO. TOPIC PAGE NO. F) HISTOLOGIC CHANGES AFTER PULP CAPPING G)MATERIALS USED IN DIRECT PULP CAPPING H)LIMITATIONS OF DIRECT PULP CAPPING IN PRIMARY TEETH 59 65 79 3) PULPOTOMY A)DEFINITIONS OF PULPOTOMY B) CLASSIFICATION OF PULPOTOMY C) OBJECTIVE,INDICATIONS CONTRAINDICATIONS D) PULPOTOMY IN PRIMARY TEETH 84 86 88 90
4. SNO. TOPIC PAGE NO. E) FORMOCRESOL PULPOTOMY,HISTORY, COMPOSITION OF FORMOCRESOL,PREPARATION, MECHANISM OF ACTION,HISTOLOGIC FEATURES DEVITALIZATION DISADVANTAGES OF FORMOCRESOL F) ELECTROSURGICAL PULPOTOMY,PROCEDURE 91 100
5. SNO. TOPIC PAGE NO. G) LASER PULPOTOMY,TWO VISIT DEVITALIZATON, INDICATIONS,CONTRAINDICATIONS, MATERIAL USED, PROCEDURE H) MODIFIED FORMOCRESOL PULPOTOMY, PRESERVATION, GLUTARALDEHYDE PULPOTOMY I) ADVANTAGES OF GLUTARALDEHYDE OVER FORMOCRESOL,ATTRIBUTES OF GLUTARALDEHYDE 104 106 107
10. Ref 16, page 22 DEFINITION OF THE PRINCIPAL TERMS USED IN PULPAL PROTECTION AND VTAL PULP THERAY TERM DEFINITION Pulp cap Treatment of an exposed vital pulp in which the pulpal wound is sealed with a dental material, such as calcium hydroxide or MTA, to facilitate the formation of reparative dentine and maintenance of vital pulp. Direct pulp cap A dental material placed directly on a mechanical or traumatic vital pulp exposure. Step wise caries excavation A material is placed on a thin partition of remaining carious dentin that if removed might accidentally expose the pulp (for immature permanent tooth) Pulpectomy (pulp extirpation) The complete surgical removal of the vital pulp Pulpotomy( pulp amputation) The surgical removal of the coronal portion of the vital pulp as a means of preserving vitality of the remaining radicular portion is usually is performed as an emergency procedure for temporary relief of symptoms or therapeutic measure.
11. Partial pulpotomy(shallow pulpotomy; cvek pulpotomy) The surgical removal of the small diseased portion of vital pulp as the means of preserving the remaining corona and radicular pulp tissue. apexification Inducing a calcified or artificial barrier in a root with an open apex or the continued apical development of an incompletely formed root in teeth with a necrotic pulp. apexogenesis A vital pulp therapy procedure performed to enable continued physiologic development and formation of the root end; term frequently used to describe vital pulp therapy that encourages the continuation of this process. Ref 16, page 22
34. Gross caries was removed and calcium hydroxide was placed over the remaining caries. Tooth was restored with amalgam and not reentered for complete caries removal for 3 months Sclerotic dentine can be seen below the remaining caries and the covering of calcium hydroxide . The tooth was reentered and the remaining caries was removed a sound dentine barrier is observed at the base of the cavity. A new amalgam restoration was placed after complete caries removal. RADIOGRAPH OF THE FIRST PERMANENT MOLAR (Ref 4, pg 395)
40. ▪ Highly demineralized ▪ Unremineralizable ▪ Superficial layer ▪ Lacking sensation ▪ Stained by 0.5% fuschin or i.e. 1.0% acid red solution Ultrastructure : intertubular dentin greately demineralized, with irregular scattered crystals. Presence of deteriorated collagen fibers that have only distinct cross bands and no interbands. ▪ Should be excavated ▪ Intermediately demineralized ▪ Remineralizable collagen ▪ Deeper layer ▪ Sensitive ▪ Does not stain Ultrastructure : intertubular dentin Partially demineralized, but apatitie crystals bound like fringes to the Sound fibers with distinct Cross bands and interbands. ▪ Should be left remineralize. Ref 1, pg 401 Infected dentin Affected dentin
53. Exposure to bleeding of molar Hard tissue Formation of the exposure Histological section showing hard tissue Formation following 90 days with a calcium Hydroxide cement DIRECT PULP CAPPING Ref 20
94. Composition of formocresol :Buckley’s formula Cresol – 35% Glycerol – 19% Formaldehyde – 19% Water – 31% Preparation: currently we use 1/5th conc.of Buckley’s formula,which is prepared by the following method: 3 parts glycerine (90ml)+1 part distilled water (30ml)=Diluent (120ml) 4 parts Diluent (120ml)+1 part Buckley’s formocresol of 1/5th strength [Ref.5,pg.185] To prepare a 1:5 concentration of this formula,first thoroughly mix 3 parts of glycerine with1 part of distilled water,then add 4 parts of this preparation to 1 part Buckley’s formocresol,and thoroughly mix again [Ref.1,pg.405]