Normal puerperium - Obstetrical and Gynecological NursingJaice Mary Joy
The word puerperium is originated from the Latin words ‘puer’ – child and ‘pams’ – bringing forth.
Also known as the post-partum, post-natal, or post-delivery period.
The mother during puerperium is termed as puerpera.
In this slide you will get to know about what is second stage of labor and what are cardinal movements in mechanism of labor and its management are discussed in this slide
Prolonged labor is the inability of a woman to proceed with childbirth upon going into labor. Prolonged labor typically lasts over 20 hours for first time mothers, and over 14 hours for women that have already had children.
Normal puerperium - Obstetrical and Gynecological NursingJaice Mary Joy
The word puerperium is originated from the Latin words ‘puer’ – child and ‘pams’ – bringing forth.
Also known as the post-partum, post-natal, or post-delivery period.
The mother during puerperium is termed as puerpera.
In this slide you will get to know about what is second stage of labor and what are cardinal movements in mechanism of labor and its management are discussed in this slide
Prolonged labor is the inability of a woman to proceed with childbirth upon going into labor. Prolonged labor typically lasts over 20 hours for first time mothers, and over 14 hours for women that have already had children.
Labour induction
Induction of labour
Guidelines on induction of labour
Guidelines on labour induction
induction of labour is not risk free
prostaglandins for induction of labour
Bishop score
Cervical ripening techniques
mechanical and pharmacological induction of labour
Post dates induction
options for cervical ripening
oral vs. vaginal misoprostol
advantages diadvantages and techniques for induction of labour
gynecology & obstetrics
Labour induction methods
review of guidelines for labour induction
This topic contains definition, incidence, varieties, causes, risk factors, dangers, diagnosis, prognosis, prevention and management of inversion of uterus.
INTRODUCTION
DEFINITION
TYPES
CAUSES
MANAGEMENT-Management of 3rd stage bleeding
Actual management
MANAGEMENT OF 3RD STAGE BLEEDING
Steps of management
1. Placental site bleeding-
To palpate the fundus and massage the uterus to make it hard. The massage is to be done by placing four fingers behind the uterus and thumb in front.
To start crystalloid solution (NS or RL) with oxytocin (1L with 20 units) at 60 drops per minute and to arrange for blood transfusion if necessary.
Oxytocin 10 unit IM or methergine 0.2 mg is given intravenously.
To catheterize the bladder.
To give antibiotics (Ampicillin 2gm and Metronidazole 500mg IV)
2. Management of traumatic bleed
The uterovaginal canal is to be explored under general anesthesia after the placenta is expelled and haemostatic sutures are placed on the offending sites.
STEPS OF MANUAL REMOVAL OF PLACENTA
The patient is placed in lithotomy position. With all aseptic measures, the bladder is catheterized.
One hand is introduced into the uterus in cone shaped manner following the cord. While introducing the hand, the labia are separated by the fingers at the other hand.
Counter pressure on the uterine fundus is applied by the hand placed over the abdomens. The abdominal hand should steady the fundus and guide the movement of the fingers inside the uterine cavity till the placenta is completely separated.
Feta Distress is a condition that describes inadequate oxygen delivery to the fetus during pregnancy or labor with resultant fetal hypoxia, abnormal fetal heart patterns and acidosis. It is one of the most common life threatening fetal conditions in the field of obstetrics with associated high fetal morbidity and mortality. Understanding the basics of this condition, including the pathogenesis and management by the maternal and child health care providers is therefore crucial towards reducing the associated short and long term sequelae of fetal distress. This power point is a key stimulant to Medical students and Doctors involved in providing maternal and child health care to further reading and understanding about fetal distress.
Labour induction
Induction of labour
Guidelines on induction of labour
Guidelines on labour induction
induction of labour is not risk free
prostaglandins for induction of labour
Bishop score
Cervical ripening techniques
mechanical and pharmacological induction of labour
Post dates induction
options for cervical ripening
oral vs. vaginal misoprostol
advantages diadvantages and techniques for induction of labour
gynecology & obstetrics
Labour induction methods
review of guidelines for labour induction
This topic contains definition, incidence, varieties, causes, risk factors, dangers, diagnosis, prognosis, prevention and management of inversion of uterus.
INTRODUCTION
DEFINITION
TYPES
CAUSES
MANAGEMENT-Management of 3rd stage bleeding
Actual management
MANAGEMENT OF 3RD STAGE BLEEDING
Steps of management
1. Placental site bleeding-
To palpate the fundus and massage the uterus to make it hard. The massage is to be done by placing four fingers behind the uterus and thumb in front.
To start crystalloid solution (NS or RL) with oxytocin (1L with 20 units) at 60 drops per minute and to arrange for blood transfusion if necessary.
Oxytocin 10 unit IM or methergine 0.2 mg is given intravenously.
To catheterize the bladder.
To give antibiotics (Ampicillin 2gm and Metronidazole 500mg IV)
2. Management of traumatic bleed
The uterovaginal canal is to be explored under general anesthesia after the placenta is expelled and haemostatic sutures are placed on the offending sites.
STEPS OF MANUAL REMOVAL OF PLACENTA
The patient is placed in lithotomy position. With all aseptic measures, the bladder is catheterized.
One hand is introduced into the uterus in cone shaped manner following the cord. While introducing the hand, the labia are separated by the fingers at the other hand.
Counter pressure on the uterine fundus is applied by the hand placed over the abdomens. The abdominal hand should steady the fundus and guide the movement of the fingers inside the uterine cavity till the placenta is completely separated.
Feta Distress is a condition that describes inadequate oxygen delivery to the fetus during pregnancy or labor with resultant fetal hypoxia, abnormal fetal heart patterns and acidosis. It is one of the most common life threatening fetal conditions in the field of obstetrics with associated high fetal morbidity and mortality. Understanding the basics of this condition, including the pathogenesis and management by the maternal and child health care providers is therefore crucial towards reducing the associated short and long term sequelae of fetal distress. This power point is a key stimulant to Medical students and Doctors involved in providing maternal and child health care to further reading and understanding about fetal distress.
Physiological Process that occur in a woman who has given birth up to 6wks postpartum, abnormal processes and their risk factors, clinical assessment and management
Lastly a brief review of anatomy of the breast
The presentation contain:
Normal puerperium ; Physiology, Duration
Postnatal assessment and management
Promoting physical and emotional well-being
Lactation management
Immunization
Family dynamics after child-birth.
Family welfare services; methods, counseling
Follow-up
Records and reports
Similar to Puerperium lecture by Associate Prof.Dr. Aisha Elbareg (20)
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
Follow us on: Pinterest
Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Factory Supply Best Quality Pmk Oil CAS 28578–16–7 PMK Powder in Stockrebeccabio
Factory Supply Best Quality Pmk Oil CAS 28578–16–7 PMK Powder in Stock
Telegram: bmksupplier
signal: +85264872720
threema: TUD4A6YC
You can contact me on Telegram or Threema
Communicate promptly and reply
Free of customs clearance, Double Clearance 100% pass delivery to USA, Canada, Spain, Germany, Netherland, Poland, Italy, Sweden, UK, Czech Republic, Australia, Mexico, Russia, Ukraine, Kazakhstan.Door to door service
Hot Selling Organic intermediates
Factory Supply Best Quality Pmk Oil CAS 28578–16–7 PMK Powder in Stock
Puerperium lecture by Associate Prof.Dr. Aisha Elbareg
1. Associate Clinical Prof. Dr. Aisha M. El-Bareg, MD, PhD .
Senior Consultant in (Obs & Gyn)/ Reproductive Medicine
Faculty of Medicine, Misurata .LIBYA
Puerperium for 4th year med.students
2. Normal Puerperium
Puerperium : Is a period during which the
anatomical and physiological changes of
pregnancy returns to pre-pregnancy state.
Time: approximately 6 weeks (± 42 days) after
delivery
3. The post partumof PuerperiumStages
period has been divided into:
o The immediate Puerperium, the first 24
hours after parturition; when acute post
anesthetic or post delivery complications
may occur.
o The early Puerperium, which extends until
the first week post partum.
o The remote Puerperium, which includes the
period of time required for involution of the
genital organs through the sixth weeks
postpartum.
6. Immediately after labor, the woman is in a state
of physical fatigue in many cases, slight shivering,
muscular tremors and chattering of teeth occur
for about 10 – 15 minutes.
1. Temperature
Slight reactionary rise in the first 24 hrs (<38⁰C)
for <24hrs.
Slight rise may occur in the 3rd day-breast
engorgement.
7. 2. Pulse: Normal but tachycardia if infection or
hemorrhage.
3. Cardiovascular:
Blood volume returns to non-pregnant levels by
the tenth days of Puerperium.
Cardiac output ↑(immediately after delivery) →
slowly declines→ reach late pregnancy levels 2
days postpartum→ normal 2-6 weeks.
8. 4. Urinary tract
Polyuria in the first few days- excrete fluid
retained during pregnancy
Retention may occur in the first few days due
to:
Lax abdominal wall
Painful episiotomy or laceration
Atony of the bladder & urethral compression
by oedema or hematoma
9. 4. Urinary tract
Dilatation of UT resolves in 12 weeks.
Renal function return to normal by 6th weeks.
Lactosuria may appear.
SUI may follow difficult labour.
10. 5. GIT
Constipation:
Atony of intestinal muscle
lax abdominal wall
Decrease fluid and food intake
Painful episiotomy- reflex constipation
11. 6. blood changes
All changes become gradually reversed
RBC & Hb become normal
Increased risk of TE in the first few days
7. Skin changes
Increased sweating- eliminate excess fluids
Decreased pigmentation
12. 8. Body wt
Decreased during labour- expulsion of contents
Decreased during puerperium- loss of fluid
9. Joints
Laxity gradually decreased
13. 10. Breasts
Larger, firmer, heavier
Lactogenesis is initially triggered by the
delivery of the placenta (E↓P↓and prolactin).
Colostrum appears for first 3 days
Milk appear later, 3rd or 4 day
Breast become engorged, painful & tender
Suckling relieves this discomfort
15. 11. Abdominal wall
Gradually return of tone- exercise
Striae- become white- albicans
12. After pain
Painful uterine contraction
In first few days-control blood loss
↑during suckling due to release of oxytocin
16. 13. Menstruation
Resumption of ovulation influenced by lactation
Variable in lactating women
Starts within 6-8 weeks in non-lactating women
19. 1. Uterus
Weight:
From 1000 gm just after delivery to 50-100
gm by the end of 6th weeks (involution)
Levels:
Immediately after delivery- at umbilicus
After 1 wk- between umbilicus & SP
After 2 wk- at SP
Then gradually ↓to pre-pregnancy size.
20. Structural changes
Uterine Muscle
Autolysis of excess muscle
↓ in muscle size
Uterine Blood vessels
Obliteration, thrombosis
Absorbed & replaced by elastic fibers
21. Decidua
Shedding as a result of ischemia
Endometrial regeneration from the basal
layer of the decidua by the 10th day.
Placental site takes 6 wks.
Lochia: vaginal discharge after labour
1st wk- lochia rubra
2nd wk- lochia serosa
3rd wk- lochia alba
22. 2. Cervix
Closed in 1 wk- return to its original form and
consistency- become slit shaped EO
Failure to close- retained POC
3. Vulva, vagina
↓ laxity of vaginal wall, pelvic floor by the end
of 6 wks.
Vaginal rugea reappear in 3rd wk.
Persistent weakness predisposes to genital
prolapse
23.
24. Management of perineum
Goals in the Puerperium
Prevention of infection
Careful nursing & observation of the newborn
Initiation of breast feeding
Exercise to prevent prolapse
Giving mother support and sympathy
25. Routine postpartum care
Monitor for:
Vital signs, PV bleeding, contraction of
uterus, appearance of lochia, perineal
inspection, signs of infection, urinary and
bowel function, examination of legs, breast.
Encourage mobilization and eat regular high
fiber diet.
Continue with multivitamins.
26.
27.
28. Pain relief- analgesics, sedatives.
Care of vulva & perineum.
Care of the breasts.
Hemorrhoids- symptomatic relief during
immediate postpartum period, using
creams, local anesthetics.
29. Cesarean delivery
Pain and discomfort from abdominal incision-
ice and pain relief medication.
Heparinization until mobilization
Investigations
HB, hematocrit
baby’s blood type in Rh –ve women and
administration of anti D.
30. Patient education
Care of the baby-feeding, diapering, bathing,
what you expect from the baby in terms of
sleep, urination, bowel, eating.
Support and guidance to breastfeeding-
encourage breast feeding immediately and
every 2-3 hrs.
If not breastfeeding-prevent breast
engorgement
31. Postpartum pelvic floor and abdominal wall
exercise.
Resuming normal physical and sexual
activity.
Advise for pregnancy spacing- Birth control
Follow up visit at 6 weeks after delivery.
34. Excessive blood loss during or after third
stage of labour. The average is 500 ml at
vaginal delivery and 1000 ml at C.S.
10% reduction in the hematocrit level or the
need for transfusion.
PPH complicate 4-6 % of all deliveries
)hemorrhage (PPHPostpartum.1
35. Control of bleeding after delivery
Contraction and retraction
of middle uterine muscle
Closure of the sinuses at
the placental site
Clotting mechanism
36. Early PPH:
Occurring within the first 24 hrs after delivery
Uterine atony, lower genital tract laceration,
retained POC, uterine rupture, uterine
inversion, placenta accreta, coagulopathy.
1. Postpartum hemorrhage (PPH)
37. Late PPH
Frequently occurs 1-2 weeks after delivery
but may up to 6 weeks postpartum.
Retained POC, sub -involution of placental
site, infection, hormonal contraception,
coagulopathy, choriocarcinoma.
US, examination under GA and evacuation
of ROC
Antibiotics
38. Management of PPH
History and examination: antepartum and
intrapartum identification of risk factors for PPH.
All women should be closely monitored within 2
hrs of delivery.
Shout for help- urgently mobilize all available
personnel.
Rapid assessment, general look, response, vital
sings
39.
40.
41. Management of PPH
Message the uterus to expel blood and blood
clots
Start an IV infusion, send blood for Hb, Hct, x-
matching and keep at least 4 units of blood
infuse IV fluids until the blood is ready.
Urinary catheter
Identify the cause treat accordingly
42. . Puerperal pyrexia2
Rise of temperature of 38 ⁰C or more on 2
occasions at 24 hrs apart (not first 24hrs)
within first 10 days following delivery.
44. Causes of P. pyrexia
Puerperal sepsis (most dangerous)
Breast engorgement (most common)
Breast infection
Urinary tract infection
Surgical wound
Thrombophlebitis or Deep venous thrombosis
Respiratory infection
General diseases- typhoid, malaria
45. Puerperal sepsis
Puerperal pyrexia due to genital infection
Mode of infection
Endogenous - vaginal flora
Exogenous- attendants, instruments
Autogenous- Blood born- tonsils
46. Site of infection
1ry site
Placental site
Laceration
Uterus- remnants of POC
2ry site-spread from 1ry sites
Parametritis
Salpingoopheritis
Peritonitis, pelvic abscess
thrombophlebitis
47. Causative organisms
Usually mixed gram +ve and –ve
Anaerobic strept
Group A hemolytic strep.-severe
Staph- suppuration & pus formation
E coli, Cl welchii, tetani
Gonorrhea, Chlamydia.
51. B. Abdominal and vaginal examination
Infected laceration
The wound is red, hot, tender, swollen
Greenish yellow discharge
Puerperal endometritis
Suprapubic or generalized tenderness
Uterus is subinvoluted
Lochia is excessive and offensive
52. Parametritis
Indurated tender mass lateral to the uterus
Parametric abscess-softening of the mass
Salpingo-ophoritis
Bilateral tenderness
Pain on moving the cervix
53. Thrombophlebitis
Deep seated pelvic tenderness
If extended to the femoral vein - Pain
and tenderness along the course of the
vessels
Limb is swollen, cyanosed, hot.
54. Investigation
o Swabs taken from vagina and cervix
o Urine analysis, C/S
o Blood culture at peak of fever
o USS for retained product of conception
o Doppler USS for DVT
o Chest x-ray, blood film for malaria
o Widal test for typhoid
55. Treatment
Supportive
• IV fluids
• Analgesics, antipyretics
Antibiotics
Evacuation of infected placenta
Promotion of abscess drainage
Anticoagulants for DVT
57. Breast infection
Engorgement:
Occurs on day 2-4 pp
Vascular and lymphatic stasis
Both breasts are heavy, painful, warm, firm
and tender
Conservative: encourage breastfeeding, ice
packs, high-fitting bras, analgesia.
?Cabergoline for non-breastfeeding
58. Mastitis
Defined as inflammation of the mammary
gland
Associated with
milk stasis and cracked nipples
Primiparity, incomplete emptying of the
breast, improper nursing technique
Causes: Staphylococcus aureus, staph.
Epidermitis, s saprophyticus,
streptococcus viridans, and E coli.
59. S & S: fever, myalgia, erythema, warmth,
swelling, and breast tenderness
treatment:
Encourage breast feeding with proper
positioning of the infant, support breasts with
a binder or brassiere, apply cold compress
Analgesia, Antibiotic therapy
Drain if abscess
61. Wound infection
infection of the perineal wound-episiotomy or
laceration-associated with infected lochia,
fecal contamination, and poor hygiene,
Caused by vaginal flora.
Infection of the abdominal incision- after CS-
contamination with vaginal flora. Also, s.
aureus, mycoplasma species.
63. Risk factors:
diabetes, PET, obesity, anemia,
Chorioamnionitis, prolonged labour,
prolonged ROM, prolonged operation time,
excessive blood loss.
Treatment:
Perineal infection: symptomatic relief-
NSAID, local anesthetic spray, sits path,
drain of abscess and antibiotics
CS wound- drainage and inspection of
underlying fascia, antibiotics
64. Septic pelvic thrombophlebitis
Venous inflammation with thrombus formation
in association with fever that unresponsive to
antibiotic therapy.
Preceded by endometritis
Risk factors: low socioeconomic status,
cesarean birth, prolonged rupture of
membranes, and excessive blood loss.
65. DD: pyelonephritis, appendicitis, broad ligament
hematoma, adnexal torsion, pelvic abscess,
drug fever, collagen vascular disease, and
pelvic abscess.
Management
Urinalysis, C/S, CBC with differentials
Imaging: CT scan and MRI- ovarian and
iliofemoral involvements
Treatment with antibiotics and/or
anticoagulants
66. Other causes of fever in Puerperium
Pneumonia, malaria, typhoid, hepatitis
67. Endocrine disorders.3
Postpartum thyroiditis:
transient destructive lymphocytic thyroiditis
occurring within the first year after delivery.
Thyrotoxicosis phase: 1-4 months pp, self-
limited
Hypothyroidism phase: 4-8 months pp
Self-limiting, no tt unless sever.
69. Sheehan syndrome
Ischemia, congestion, and infarction of
pituitary gland resulting in panhypopituitrism
caused by severe PPH at the time of delivery.
Abnormal lactation, amenorrhea, signs of
cortisol and thyroid hormone deficiency
Hormonal assays
Hormonal replacement.
71. 4. Psychiatric disorders
Postpartum blue (50-70%)
A transient disorder arise in the first 2 weeks
pp and the lasts hours to weeks.
characterized by bouts of crying and
sadness, restlessness, mood lability,
insomnia.
Resolve by day 10, provide support and
education
72. 4. Psychiatric disorders
Postpartum depression (PPD)- 10-15%
Develop 2-3 months pp and resolve slowly
over the following 6-12 months.
Insomnia, lethargy, loss of libido, negative
feeling toward the infant, suicidal or homicidal
ideas..etc..
73. Risk factors: history of depression or PPD,
Supportive care and reassurance.
Drugs: antipsychotics
74. 4. Psychiatric disorders
Postpartum psychosis - 0.14-0.26 %
Refers to a group of severe and varied
disorders that elicit psychotic symptoms.
Schizophrenia or manic depression.
Risk factors: young age, primiparity, personal
or family history of mental illness.
75. Symptoms start 10-14 days pp.
Treatment
Involve hospitalization and psychiatric
consultation
Drugs and/or electroconvulsant therapy
(ECT)
Recurrence: 25-30%
77. Symphysis pubis diastasis
Separation of SP
Spontaneous or surgical
Symphyseal pain, waddling gait, pubic
tenderness, a palpable infrapubic gap
Best rest, anti-inflammatory agents,
physiotherapy, pelvic corset to provide
support and stability.
79. Avoid prolonged standing and prolonged sitting.
Apply well-fitted below knee support stocking
before ambulating in the morning.
Ask mother to elevate her leg on pillow while
taking supine lying position.
Intermittent compression, Bandaging.
Not to sit with leg crossed or knee flexed.
Management of varicose vain