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Spinal Cord Injury
General Principles of Spine Injuries
Epidemiology
 the fourth leading cause of death in the US
 C-spine injuries
MVAs - 50%
falls - 25%
sports activities - 10%
 60% of all spine injuries in children;
upper cervical spine.
 C5-C6 is the most commonly injured level in
adults.
Ranges of Motion (in degrees)
Level Flexion/
Extension
Lateral
Flexion
Rotation
O -C1 25 5 5
C1-C2 20 5 40
C2-C3 10 10 3
C3-C4 15 11 7
C4-C5 20 11 7
C5-C6 20 8 7
C6-C7 17 7 6
C7-T1 9 4 2
Stability
 Definition
1. The ability of the spine under physiologic
loads to prevent displacements which
would injure or irritate neural tissue.
2. Instability is the loss of the ability of the
spine to tolerate physiological loading
without incurring neurological deficit, pain,
or progre ssive structural deficit.
Initial evaluation and treatment
 ABC's
 Stablization for transport
don't move patient before stabilization of cervical
spine.
 In hospital
 Usual trauma protocol
 Traction
rule of thumb is 5 pounds per spinal level above the
fracture/dislocation.
60-80 pounds are usually the upper limit in any case.
Pharmacologic treatment of SCI
1.Hypertension
adequate volume and normotension
2. Methylprednislone
within 8 hours of the SCI protocol
30mg/kg initial IV bolus over 15 minutes
followed by a 45 minute pause,
and then a 5.4 mg/kg/hr continuous infusion.
3. Naloxone
4. Triliazad
General Imaging Features and
Determination of Instability
Plain Films
 AP, lateral, obliques
 open mouth
 Swimmer's view
Radiographic Criteria
Upper limits of
prevertebral soft tissue
level of C1: 10 mm
level of C4: 7 mm
level of C6-7: 20 mm
Inter-spinous process widening
(on AP radiographs)
More than 1.5 times the interspinous
distance at the levels above and below is
abnormal.
 C1-C2 distance between posterior cortex of
C1 arch and odontoid is maximally 3 mm in
adults and 4.5 mm in children.
 Normal spinal canal diameter is 17 ± 5 mm
Stenosis is present if <13 mm.
 anterior subluxation of 3 mm of one body on
another (or >20% of the AP distance)
indicates instability.
 angulation greater than 11° is suggestive of
instability.
Other Investigations
 flexion and extension views, tomograms
CT, CT/myleogram, and MRI
 MRI essential for suspected spinal cord injury
all soft tissues (disk and ligamentous
structures) are shown in much better detail.
Injuries of the Upper Cervical Spine
Odontoid Anatomy
 Steele's rule of thirds
The dens, subarachnoid space, and
spinal cord each occupy 1/3 of the area
of the canal at the level of the atlas.
Ligaments
External
1. posterior occipito-atlantal ligament
(ligamentum flavum ends at C1).
2. anterior occipito-atlantal ligament.
3. ligamentum nuchae.
4. anterior longitudinal ligament.
Internal
1. cruciform ligament( transverse ligament )
2. accessory ligaments
3. apical ligament
4. alar ligaments
5. posterior longitudinal ligament
( tectorial membane )
Types of injuries
I. Atlanto-occipital dislocation.
II. Condylar fractures.
III. Atlanto-axial dislocation.
IV. Atlas fractures.
V. Odontoid fractures.
VI. Hangman's fractures.
VII. C3 fractures.
Atlas fractures
 cervicomedullary
junction remains
unchanged
because of capacity
of spinal canal.
 Jefferson’s fracture
Clinical Features
 isolated C1 fractures rarely have associated
cord injury.
 symptoms
neck tenderness
need neck support
pharyngeal protuberance.
dysphagia.
Imaging
 Spence's Rule
If, on plain films, the distance of excursion
of lateral masses is 7 mm or more,
there is a transverse ligament rupture.
Treatment
 Halo immobilization in virtually all cases
except when Spence's rule exceeded
Odontoid Fractures
 Odontoid fractures
are the M/C
fractures of C2.
Classification
Type I An oblique fracture line through the upper part of the
odontoid process representing an avulsion
fracture where the alar ligaments attach. Stable,
high rate of fusion.
Type II A fracture at the junction between the odontoid
process and the body of the axis. Unstable, high
rate malunion.
Type IIA Similar to type II but with fragments of bone present
at the fracture site.
Type III A fracture that extends down into the cacellous bone
of the body of the axis and in reality is a fracture
of the body of C2. Stable, with high rate of fusion.
Epidemiology
 about 10-15% of all cervical spine fractures.
 In children, these consitute about 75% of all
C-spine injuries.
Clinical Features
 need high index of suspicion in all trauma
patients
 many signs and symptoms are non-specific
 vertebral artery compression may cause brain
stem ischemic symptoms.
 most patients unwilling to go from supine to
sitting position without supporting their heads
with their hands.
Imaging
 open mouth views
 tomography or CT
 saggital and coronal
reconstructions
have superseded
tomography
Treatment and results
 Halo traction, maximum of 5-10 pounds to
achieve reduction
 Surgery
Type I : no fusion required
Type III : no fusion required
(>90% fuse with Halo immobilization)
Type II : several factors important in
decision making
1. Patient age.
sixty years of age is the usually quoted cutoff
for conservative management.
2. Displacement
If >6 mm and >60 years, 85% nonunion rate.
3. Age of fracture
greater than 2 weeks seems to be the cutoff
for high union rate
Techniques for C1/C2 Fusion
 C1/C2 fusions can
be considered even
if there is a
unilateral fracture of
the atlas ring, or
unilateral fracture of
the lateral mass of
the atlas.
Hangman's Fracture
 When submental knots with a measured
corporal drop are used to hang someone, a
classic hangman's C2 fracture is the result.
 Pathophysiology and Classification
Hangman's fracture involves a bilateral arch
fracture of C2 (pars interarticularis i.e, the
pedicle) with variable C2 on C3
displacement.
Type Description Mechanism of Injury
Type I Stable. Fractures through
the pars with less than
2 mm displacement of
C2 on C3.
Axial loading and then
extension.
Type II Unstable. Significant
displacement of C2 on
C3 (>2 mm or >118
angulation). Disruption
of PLL.
Axial loading, extension, and
then rebound flexion.
Type
IIA
Less displaced but more
angulated than II.
Same as II.
Type III Pure flexion injuries. C2-
C3 facet capsules are
disrupted with
angulation and
anterolisthesis.
Compression and flexion.
Clinical Features
 not highly specific symptoms,
diffuse neck pain with stiffness
 relative sparing of the spinal cord because of
the capacious bony canal.
Treatment
 type I may be treated in a rigid collar for
12 weeks.
 remainder are all treated initially with Halo
immobilization.
 up to 5% will eventually require surgery.
Surgical Indications
 inability to reduce fracture
 failure to maintain reduction in Halo vest.
 C2-3 disk herniation with spinal cord
compromise.
 established non-union (late)
Surgical Procedures
1. C1-C3 arthrodesis
2. C2 pedicle screws.
3. C2-C3 anterior discectomy with fusion
and plate.
Injuries of the Lower Cervical Spine
Classification
I. flexion-dislocation
II. flexion-compression
III. compression burst
IV. extension
Criteria for Instability
 White and Panjabi also concluded that
horizontal motion between vertebrae should
not exceed 2.7 mm
(3.5 mm with standard X-ray magnification),
and angular motion should not exceed 11 mm.
Three column model
 failure of 2 of the 3
columns implies that
there will be instability.
 1. anterior column
2. middle column
3. posterior column
Flexion Dislocation Injuries
A) unilateral facet
subluxation
B) bilateral facet
dislocation
Indications for surgery
1. failure to obtain reduction
2. failure to maintain reduction
3. pseudoarthrosis
4. purely ligamentous injury
5. re-subluxation
6. any patient with jumped facets
Flexion Compression Injuries
A) wedge compression
B) teardrop flexion fracture
Compression Burst Fractures
 characterized by
axial loading injuries
to a spine which is
held in a neutral or
very slightly flexed
position.
 immobilized by
traction with a Halo
vest.
Extension Injuries
 may occur either in distraction or compression.
Surgical Considerations
 absolute Indications
1. inability to obtain reduction
2. inability to maintain reduction
 indications for emergent surgery
1. progression of neurological deficit
2. complete myelographic or MRI CSF block
3. fragments in spinal canal in incomplete SCI
4. necessity for decompressing a cervical nerve root
5. open or penetrating trauma
6. non-reducible fractures
Contraindications for emergent
surgery
1. complete SCI
2. central cord syndrome.
3. medically unstable patient.
Surgical techniques
 posterior approaches
 anterior approaches
 cervical orthoses
Thoracolumbar Spine Fractures
Epidemiology
 represent 40% of all spine fractures
 majority due to motor vehicle accidents.
 grouped into
thoracic (T1-T10)
thoracolumbar (T11-L1)
lumbar fractures (L2-L5)
 60% occur between T12 and L2
Biomechanics
 In general, compression causes burst
fractures, flexion causes wedge fractures,
rotation causes fracture dislocations, and
shear causes seatbelt type fractures.
 thoracic region is inherently more stable
because of the rib cage and ligaments
linking the ribs and spine.
 the three column model states that failure of
two or more columns results in instability.
 the middle column is crucial, its mode of
failure distinguishes the four types of spine
fractures.
Imaging
 most important initial study is a complete
plain film series of the spine.
 5-20% of fractures are multiple.
 CT is used to define the bony anatomy in the
injured area.
 MRI essential to define spinal cord anatomy
and also for assessing ligamentous injury.
Flexion Compression Fractures
 most common type of fracture.
 failure of the anterior column due to flexion and
compression.
 a minimum of 30% loss of ventral height is
necessary to appreciate a kyphotic deformity
on x-rays.
 generally considered
stable.
 usually managed
with
bedrest
analgesics
early mobilization
Burst Fractures
 17% of major spinal fractures
 between T1-T10, most burst fractures are
associated with complete neurological deficit.
 bony fragments may be retropulsed into the
canal (25% of cases) and associated
fractures of the posterior elements are
common.
 failure of anterior and middle column defines
these as unstable.
Management
 those fractures with canal compromise in the
presence of neurological deficit should be
treated with surgical decompression and
fusion.
 the treatment of burst fractures with canal
compromise and no neurological deficit is
controversial.
 generally, management is based upon
determination of stability.
 those fractures with >50% loss of vertebral
body height, or if the posterior column injury
includes a facet fracture or dislocation are
considered unstable.
 commonly sited indications for surgery are
either,
i) angular deformity >15-40 degrees
ii) canal compromise >40-70%
Seat Belt Injuries
 6% of major spinal
injuries.
 caused by
hyperflexion and
distraction of the
posterior elements.
 the middle and posterior columns fail in
distraction with an intact anterior hinge.
 unstable in flexion but will not present with an
anterior subluxation, which would indicate
that the ALL is disrupted
(i.e. a fracture-dislocation).
 PLL is also intact and sometimes fracture
fragments are moved back from the canal
simply by distraction.
Management
 treatment of mainly osseous injuries is
bracing while mainly ligamentous injuries are
treated with posterior fusion.
Fracture Dislocations
 19% of major spinal
fractures.
 anterior and cranial
displacement of the
superior vertebral body
with failure of all three
columns.
Extension Distraction Fractures
 rare injuries.
 hyperextension force tears the ALL and leads
to separation of the disc.
 all are unstable and require fixation.
Treatment
 By Degree
according to Denis, first degree is mechanical
instability, second degree is neurolgical, and
third is both.
 first degree: manage with external orthosis
i) >30 degree wedge compression fracture
ii) Seat belt injuries.
 second degree: mixed category
 third degree: all require surgery
i) fracture dislocations
ii) burst fractures who fail non-operative
management
iii) burst fractures who develop new
neurological deficit
Non-operative Therapy
 external orthosis for 8-12 weeks
serial radiographs every 2-3 weeks for the
first 3 months, then 4-6 week intervals until
6 months and at 3 month intervals for 1 year.
Indications for operative therapy
1. progressive neurological deficit
2. spinal cord compression
3. dural laceration
4. unstable spine
Spinal Shock
Definition
 loss of all cord function distal to the site
of injury (motor, sensory, sympathetic).
Etiology
1. trauma
2. vascular
3. infection
Pathophysiology
 sudden loss of descending, tonic input from
the rubrospinal, vestibulospinal and the
corticospinal tracts.
Natural history
 can last days, weeks, or occasionally months.
 there is loss of facilitatory inputs which
gradually return via sprouting, membrane
hyperexcitability, and residual connections.
 the stretch reflexes begin to return (flexors in
the upper extremities and extensors in the
lower extremities).
4. return of bulbocavernosus reflex heralds time
course out of spinal shock.
5. mass reflexes return with cutaneous stimuli,
or bladder distension and include spasms,
colon and bladder evacuation, hypertension,
profuse sweating.
6. return of sacral reflex for bladder and bowel.
Treatment
1. maintain adequate intravascular volume
2. maintain blood pressure (spinal cord arterial
pressure).
3. keep bladder well decompressed; attend to
bowel routine.
4. surgical stabilization if indicated or surgical
decompression.
- The End -

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pinal cord injury.ppt

  • 2.
  • 3. General Principles of Spine Injuries
  • 4. Epidemiology  the fourth leading cause of death in the US  C-spine injuries MVAs - 50% falls - 25% sports activities - 10%  60% of all spine injuries in children; upper cervical spine.  C5-C6 is the most commonly injured level in adults.
  • 5. Ranges of Motion (in degrees) Level Flexion/ Extension Lateral Flexion Rotation O -C1 25 5 5 C1-C2 20 5 40 C2-C3 10 10 3 C3-C4 15 11 7 C4-C5 20 11 7 C5-C6 20 8 7 C6-C7 17 7 6 C7-T1 9 4 2
  • 6. Stability  Definition 1. The ability of the spine under physiologic loads to prevent displacements which would injure or irritate neural tissue. 2. Instability is the loss of the ability of the spine to tolerate physiological loading without incurring neurological deficit, pain, or progre ssive structural deficit.
  • 7. Initial evaluation and treatment  ABC's  Stablization for transport don't move patient before stabilization of cervical spine.  In hospital  Usual trauma protocol  Traction rule of thumb is 5 pounds per spinal level above the fracture/dislocation. 60-80 pounds are usually the upper limit in any case.
  • 8. Pharmacologic treatment of SCI 1.Hypertension adequate volume and normotension 2. Methylprednislone within 8 hours of the SCI protocol 30mg/kg initial IV bolus over 15 minutes followed by a 45 minute pause, and then a 5.4 mg/kg/hr continuous infusion. 3. Naloxone 4. Triliazad
  • 9. General Imaging Features and Determination of Instability
  • 10. Plain Films  AP, lateral, obliques  open mouth  Swimmer's view
  • 12. Upper limits of prevertebral soft tissue level of C1: 10 mm level of C4: 7 mm level of C6-7: 20 mm
  • 13. Inter-spinous process widening (on AP radiographs) More than 1.5 times the interspinous distance at the levels above and below is abnormal.
  • 14.  C1-C2 distance between posterior cortex of C1 arch and odontoid is maximally 3 mm in adults and 4.5 mm in children.  Normal spinal canal diameter is 17 ± 5 mm Stenosis is present if <13 mm.
  • 15.  anterior subluxation of 3 mm of one body on another (or >20% of the AP distance) indicates instability.  angulation greater than 11° is suggestive of instability.
  • 16. Other Investigations  flexion and extension views, tomograms CT, CT/myleogram, and MRI  MRI essential for suspected spinal cord injury all soft tissues (disk and ligamentous structures) are shown in much better detail.
  • 17. Injuries of the Upper Cervical Spine
  • 18. Odontoid Anatomy  Steele's rule of thirds The dens, subarachnoid space, and spinal cord each occupy 1/3 of the area of the canal at the level of the atlas.
  • 19. Ligaments External 1. posterior occipito-atlantal ligament (ligamentum flavum ends at C1). 2. anterior occipito-atlantal ligament. 3. ligamentum nuchae. 4. anterior longitudinal ligament.
  • 20. Internal 1. cruciform ligament( transverse ligament ) 2. accessory ligaments 3. apical ligament 4. alar ligaments 5. posterior longitudinal ligament ( tectorial membane )
  • 21.
  • 22. Types of injuries I. Atlanto-occipital dislocation. II. Condylar fractures. III. Atlanto-axial dislocation. IV. Atlas fractures. V. Odontoid fractures. VI. Hangman's fractures. VII. C3 fractures.
  • 23. Atlas fractures  cervicomedullary junction remains unchanged because of capacity of spinal canal.  Jefferson’s fracture
  • 24. Clinical Features  isolated C1 fractures rarely have associated cord injury.  symptoms neck tenderness need neck support pharyngeal protuberance. dysphagia.
  • 25. Imaging  Spence's Rule If, on plain films, the distance of excursion of lateral masses is 7 mm or more, there is a transverse ligament rupture.
  • 26. Treatment  Halo immobilization in virtually all cases except when Spence's rule exceeded
  • 27. Odontoid Fractures  Odontoid fractures are the M/C fractures of C2.
  • 28.
  • 29. Classification Type I An oblique fracture line through the upper part of the odontoid process representing an avulsion fracture where the alar ligaments attach. Stable, high rate of fusion. Type II A fracture at the junction between the odontoid process and the body of the axis. Unstable, high rate malunion. Type IIA Similar to type II but with fragments of bone present at the fracture site. Type III A fracture that extends down into the cacellous bone of the body of the axis and in reality is a fracture of the body of C2. Stable, with high rate of fusion.
  • 30. Epidemiology  about 10-15% of all cervical spine fractures.  In children, these consitute about 75% of all C-spine injuries.
  • 31. Clinical Features  need high index of suspicion in all trauma patients  many signs and symptoms are non-specific  vertebral artery compression may cause brain stem ischemic symptoms.  most patients unwilling to go from supine to sitting position without supporting their heads with their hands.
  • 32. Imaging  open mouth views  tomography or CT  saggital and coronal reconstructions have superseded tomography
  • 33. Treatment and results  Halo traction, maximum of 5-10 pounds to achieve reduction  Surgery Type I : no fusion required Type III : no fusion required (>90% fuse with Halo immobilization) Type II : several factors important in decision making
  • 34. 1. Patient age. sixty years of age is the usually quoted cutoff for conservative management. 2. Displacement If >6 mm and >60 years, 85% nonunion rate. 3. Age of fracture greater than 2 weeks seems to be the cutoff for high union rate
  • 35. Techniques for C1/C2 Fusion  C1/C2 fusions can be considered even if there is a unilateral fracture of the atlas ring, or unilateral fracture of the lateral mass of the atlas.
  • 36.
  • 37. Hangman's Fracture  When submental knots with a measured corporal drop are used to hang someone, a classic hangman's C2 fracture is the result.  Pathophysiology and Classification Hangman's fracture involves a bilateral arch fracture of C2 (pars interarticularis i.e, the pedicle) with variable C2 on C3 displacement.
  • 38. Type Description Mechanism of Injury Type I Stable. Fractures through the pars with less than 2 mm displacement of C2 on C3. Axial loading and then extension. Type II Unstable. Significant displacement of C2 on C3 (>2 mm or >118 angulation). Disruption of PLL. Axial loading, extension, and then rebound flexion. Type IIA Less displaced but more angulated than II. Same as II. Type III Pure flexion injuries. C2- C3 facet capsules are disrupted with angulation and anterolisthesis. Compression and flexion.
  • 39. Clinical Features  not highly specific symptoms, diffuse neck pain with stiffness  relative sparing of the spinal cord because of the capacious bony canal.
  • 40. Treatment  type I may be treated in a rigid collar for 12 weeks.  remainder are all treated initially with Halo immobilization.  up to 5% will eventually require surgery.
  • 41. Surgical Indications  inability to reduce fracture  failure to maintain reduction in Halo vest.  C2-3 disk herniation with spinal cord compromise.  established non-union (late)
  • 42. Surgical Procedures 1. C1-C3 arthrodesis 2. C2 pedicle screws. 3. C2-C3 anterior discectomy with fusion and plate.
  • 43. Injuries of the Lower Cervical Spine
  • 45. Criteria for Instability  White and Panjabi also concluded that horizontal motion between vertebrae should not exceed 2.7 mm (3.5 mm with standard X-ray magnification), and angular motion should not exceed 11 mm.
  • 46. Three column model  failure of 2 of the 3 columns implies that there will be instability.  1. anterior column 2. middle column 3. posterior column
  • 47. Flexion Dislocation Injuries A) unilateral facet subluxation B) bilateral facet dislocation
  • 48.
  • 49.
  • 50. Indications for surgery 1. failure to obtain reduction 2. failure to maintain reduction 3. pseudoarthrosis 4. purely ligamentous injury 5. re-subluxation 6. any patient with jumped facets
  • 51. Flexion Compression Injuries A) wedge compression B) teardrop flexion fracture
  • 52. Compression Burst Fractures  characterized by axial loading injuries to a spine which is held in a neutral or very slightly flexed position.  immobilized by traction with a Halo vest.
  • 53. Extension Injuries  may occur either in distraction or compression.
  • 54. Surgical Considerations  absolute Indications 1. inability to obtain reduction 2. inability to maintain reduction  indications for emergent surgery 1. progression of neurological deficit 2. complete myelographic or MRI CSF block 3. fragments in spinal canal in incomplete SCI 4. necessity for decompressing a cervical nerve root 5. open or penetrating trauma 6. non-reducible fractures
  • 55. Contraindications for emergent surgery 1. complete SCI 2. central cord syndrome. 3. medically unstable patient.
  • 56. Surgical techniques  posterior approaches  anterior approaches  cervical orthoses
  • 58. Epidemiology  represent 40% of all spine fractures  majority due to motor vehicle accidents.  grouped into thoracic (T1-T10) thoracolumbar (T11-L1) lumbar fractures (L2-L5)  60% occur between T12 and L2
  • 59. Biomechanics  In general, compression causes burst fractures, flexion causes wedge fractures, rotation causes fracture dislocations, and shear causes seatbelt type fractures.
  • 60.  thoracic region is inherently more stable because of the rib cage and ligaments linking the ribs and spine.  the three column model states that failure of two or more columns results in instability.  the middle column is crucial, its mode of failure distinguishes the four types of spine fractures.
  • 61. Imaging  most important initial study is a complete plain film series of the spine.  5-20% of fractures are multiple.  CT is used to define the bony anatomy in the injured area.  MRI essential to define spinal cord anatomy and also for assessing ligamentous injury.
  • 62.
  • 63. Flexion Compression Fractures  most common type of fracture.  failure of the anterior column due to flexion and compression.  a minimum of 30% loss of ventral height is necessary to appreciate a kyphotic deformity on x-rays.
  • 64.  generally considered stable.  usually managed with bedrest analgesics early mobilization
  • 65. Burst Fractures  17% of major spinal fractures  between T1-T10, most burst fractures are associated with complete neurological deficit.  bony fragments may be retropulsed into the canal (25% of cases) and associated fractures of the posterior elements are common.  failure of anterior and middle column defines these as unstable.
  • 66.
  • 67.
  • 68. Management  those fractures with canal compromise in the presence of neurological deficit should be treated with surgical decompression and fusion.
  • 69.
  • 70.  the treatment of burst fractures with canal compromise and no neurological deficit is controversial.  generally, management is based upon determination of stability.  those fractures with >50% loss of vertebral body height, or if the posterior column injury includes a facet fracture or dislocation are considered unstable.
  • 71.
  • 72.  commonly sited indications for surgery are either, i) angular deformity >15-40 degrees ii) canal compromise >40-70%
  • 73.
  • 74.
  • 75.
  • 76. Seat Belt Injuries  6% of major spinal injuries.  caused by hyperflexion and distraction of the posterior elements.
  • 77.  the middle and posterior columns fail in distraction with an intact anterior hinge.  unstable in flexion but will not present with an anterior subluxation, which would indicate that the ALL is disrupted (i.e. a fracture-dislocation).  PLL is also intact and sometimes fracture fragments are moved back from the canal simply by distraction.
  • 78. Management  treatment of mainly osseous injuries is bracing while mainly ligamentous injuries are treated with posterior fusion.
  • 79. Fracture Dislocations  19% of major spinal fractures.  anterior and cranial displacement of the superior vertebral body with failure of all three columns.
  • 80. Extension Distraction Fractures  rare injuries.  hyperextension force tears the ALL and leads to separation of the disc.  all are unstable and require fixation.
  • 81. Treatment  By Degree according to Denis, first degree is mechanical instability, second degree is neurolgical, and third is both.
  • 82.  first degree: manage with external orthosis i) >30 degree wedge compression fracture ii) Seat belt injuries.  second degree: mixed category  third degree: all require surgery i) fracture dislocations ii) burst fractures who fail non-operative management iii) burst fractures who develop new neurological deficit
  • 83. Non-operative Therapy  external orthosis for 8-12 weeks serial radiographs every 2-3 weeks for the first 3 months, then 4-6 week intervals until 6 months and at 3 month intervals for 1 year.
  • 84. Indications for operative therapy 1. progressive neurological deficit 2. spinal cord compression 3. dural laceration 4. unstable spine
  • 86. Definition  loss of all cord function distal to the site of injury (motor, sensory, sympathetic).
  • 88. Pathophysiology  sudden loss of descending, tonic input from the rubrospinal, vestibulospinal and the corticospinal tracts.
  • 89. Natural history  can last days, weeks, or occasionally months.  there is loss of facilitatory inputs which gradually return via sprouting, membrane hyperexcitability, and residual connections.  the stretch reflexes begin to return (flexors in the upper extremities and extensors in the lower extremities).
  • 90. 4. return of bulbocavernosus reflex heralds time course out of spinal shock. 5. mass reflexes return with cutaneous stimuli, or bladder distension and include spasms, colon and bladder evacuation, hypertension, profuse sweating. 6. return of sacral reflex for bladder and bowel.
  • 91. Treatment 1. maintain adequate intravascular volume 2. maintain blood pressure (spinal cord arterial pressure). 3. keep bladder well decompressed; attend to bowel routine. 4. surgical stabilization if indicated or surgical decompression.