Practical pediatric quiz - Kaun Banega WinnerGaurav Gupta
Interactive quiz based on mentimeter platform for IAP Chandigarh Annual meeting in Dec 2017.
Great success for practising paediatricians in general,
Also a great teaching experience
Practical pediatric quiz - Kaun Banega WinnerGaurav Gupta
Interactive quiz based on mentimeter platform for IAP Chandigarh Annual meeting in Dec 2017.
Great success for practising paediatricians in general,
Also a great teaching experience
Frank Lockie, paediatric intensivist, discusses how kids are just little adults at Bedside Critical Care Conference 4 (Cairns, 2013)
The podcasts accompanying these slides will be uploaded onto www.intensivecarenetwork.com and libsyn.
Screening for any disorder in individuals is a strategy used for identifying a disease before the onset of signs or symptoms, thus enabling earlier detection and management with the aim to reduce morbidity and mortality.
Typical & atypical clinical presentations of COVID-19 in childrenMoosaAllawati1
A brief presentation about some typical symptoms in children diagnosed with COVID-19 in Oman along with atypical or unusual presentations of the disease in the same age group in the USA and Bahrain.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
4. Station-2
• X-ray of a newborn who
died immediately after
birth
• Parents are reluctant to
give consent for post
mortem investigation
• What is the likely
diagnosis?
• What investigations can
help in diagnosis?
8. Station - 4
• What is the diagnosis?
• What is the name given
to this typical chest X-
ray presentation?
• What is the clinical
presentation of the
infant with this
diagnosis?
9. Station 4
• Supra cardiac type of TAPVD
• “Snow man sign”
• Cyanosis, CCF, Recurrent chest infections
• Pulmonary HTN
12. Station 6
• Write 4 uses in
paediatric practice
• Dose in Nephrotic
syndrome
• Name 4 adverse effects
• Contraindications
13. Station - 6
• Steoid dependent and resistant Nephrotic
syndrome
• ITP
• SLE
• NHL
• Dose – 375 mg/M^2
• Back pain,oedema,chills,allergic rash
• Arrhythmia, ongoing infection, severe
immunocompromised state
14. Station - 7
• Comment about this
compound muscle action
potential recorded from
abductor pollicis brevis
following median nerve
stimulation(figure B)
• Diagnosis?
• Mention two other
diagnostic methods
• Mention at least three
treatment methods
16. Station - 8
• Jacob is 10 month old male
infant. He was born
weighing 4.2 kg and had an
uneventful perinatal period.
• What is the probable
diagnosis?
• What are the components
of this syndrome?
• Which chromosome carries
the gene responsible for
this disorder?
• What complications do you
expect in such a child?
17. Station – 8
• Beckwith Widemann syndrome
• Omphalocele,macroglossia,microcephaly,visce
romegaly,hemihypertrophy
• 11(11p15.5)
• Tumours –
wilms,hepatoblastoma,gonadoblastoma,adren
al carcinoma,rhabdomyosarcoma
18. Station - 9
• What is the schedule for polio vaccination for an
unimmunized 9 month old child with IPV?
• What is the recommendation for IPV in an
adolescent visiting a country with a polio
outbreak?
• What is your advise regarding typhoid conjugate
vaccine in a child previously taken typhoid
polysaccharide vaccine?
• What is New Rabies post exposure vaccination
schedule WHO-2018?
19. Station - 9
• 2 doses of IPV at 4 weeks interval
• Unimmunised – 0,1 and 6 months IPV
• Previuosly immunised – single booster dose
IPV
• TCV 25 mcg 4 weeks later
• 0,3,7 and 14 – NO 28 Day schedule
20. Station - 10
• Diagnosis?
• Complications?
• Mention the drug of
choice,route,dose and
duration
• One yr old male child
with high fever-15 days
off &on ,occasional loose
stools.
• O/E – Liver-4cm,spleen-
5cm,sick looking, BP
60/40, cervical lymph
nodes 0.5 x 0.5 cm
• Pallor +
• Hb-8,MCV-82,MCH
28,MCHC 29,WBC 2600,
P56 L39 M5,Platelets 1.1L
21. Station -10
• Severe malaria(Complicated Malaria)
• Cerebral oedema,DIC,AKI
• IV Artesunate
• 2.4 mg/kg – 0,12 and 24 hrs then daily till
patient can tolerate oral medications---ACT -
3days
22. Station -11
• CT of a 3 month old
breast fed infant who was
brought to hospital with
history of focal seizure
with no h/o trauma
• Most probable diagnosis
• How would you confirm
etiological diagnosis?
name 3 tests
• Mention any preventive
measure for this
condition
23. Station -11
• ICH with midline shift
• Late onset Vitamin K def
• Abnormal PT and APTT,raised PIVKA
• Vit K prophylaxis at birth
24. Station - 12
• A neonate admitted in NICU with persistent
pulmonary hypertension. He is on ventilator with
FiO2 100%,PIP 35,PEEP 6, MAP 14, and SpO2 85%
and he has following lab values(ABG)
• Ph – 7.22, PCO2 50, PO2 50, HC03 14, Na 136, k
4, Cl 103
• What is interpretation of ABG
• Calculate OI and OSI
• What are the indications of ECMO in terms of
(i)AaDO2 , (ii) OI
25. Station 12
• Mixed acidosis with hypoxemia
• 28 and 14
• (i) oxygen gradient >600 mm hg for 12 hrs
• (ii)OI>40
26. Station - 13
• Total population of children in an area -1300
• Protected against measles by vaccination – 800
• Total cases of measles in one year – 10
• Primary cases – 2
• No of susceptible children coming in contact of primary
cases -15
• Secondary cases that received infection from primary
cases with in the infective period – 8
• Calculate attack rate of measles
• Calculate secondary attack rate of measles
27. Station - 13
• No of cases/spell in a year//no at risk of
measles
• 10/(1300-800)=10/500 = 2%
• Secondary attack rate = secondary cases / no
of susceptible children coming in contact
• 8/15 = 53%
28. Station -14
• 14 yr old boy with giddiness and palpitation
• write characteristic ECG findings
• Diagnosis?
• Treatment
29. Station -14
• Short PR interval
• Slurring of upstroke of QRS complex
• WPW syndrome
• Beta –blockers
• Radiofrequency ablation
30. Station 15
• A 14 yr old boy is brought to Paediatric OPD with
concerns of delayed puberty. On examination he
has preadolescent male genitalia with scanty
axillary hairs, and few fine pubic hairs.
• What is tanner staging of this child?
• What is the first visible sign of puberty in males?
• At what tanner stage does a male child achieve
peak growth velocity?
32. Station - 16
• This 11 month old male child presented with pallor, dark
pigmentation of nail folds, loss of motor mile stones, lethargy and
coarse tremors
• What is your diagnosis?
• How will you manage this child?
• How will you treat tremors?
36. Station - 18
42 day old male infant
Fever with seizures
Blindness
Infantile Spasms
Macrocephaly
CSF protein-rasied;20 lymphocytes
CSF ADA – raised
Diagnosis?
Treatment?
38. Station - 19
• A Mother is being counselled for prevention of
allergy. She wants to know which of the following
could be (A) food intolerance (B) food
hypersensitivity.
• i. Lactose intolerance
• Ii.Urticaria
• Iii.Heiner syndrome
• Iv.Celiac disease
• V.Irritable Bowel syndrome
40. Station 20
• 4 yr old female child presented with gradually increasing
difficulty in breathing with cough and fever since past 10
hours. She had one episode of haemoptysis and 1-2
episodes of malena 24 hours back. on examination, she is
pale, HR- 162, RR 50. Examination of respiratory system
reveals bilateral wheezing. Examination of blood shows
microcytic hypochromic anaemia with reticulocytosis.
Serum iron was found to be low.
• What is the diagnosis?
• What is diagnostic investigation findings?
• Which cardiac condition resemble such symptomatology?