Overview on midline diastema and its unesthetic effects
Presented by : Anamika Thorat
Guided by : Dr. Rehan Khan
Dept. of Pediatric Dentistry
SDDCH PBN
self correcting anomalies in the development of occlusion. this ppt includes the anomalies of a child's developing occlusion which get corrected by itself in some time as the development continues. This includes Retrognathic mandible,infantile swallow,anterior open and deep bite,etc. these topics are important in BDS final examination
self correcting anomalies in the development of occlusion. this ppt includes the anomalies of a child's developing occlusion which get corrected by itself in some time as the development continues. This includes Retrognathic mandible,infantile swallow,anterior open and deep bite,etc. these topics are important in BDS final examination
Diagnostic aids in endodontics /certified fixed orthodontic courses by India...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
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State of the art comprehensive training-Faculty of world wide repute &Very affordable.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
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Diagnostic aids in endodontics /certified fixed orthodontic courses by India...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
It include proximal stripping, Diagnostic aids, advantages, disadvantages, periodontal consideration, procedure for proximal stripping. Expansion, extraction, Distalization in detail as method of gaining space, Extra-oral, Intra-oral method for gaining space. uprighting, derotation of posterior teeth. proclination of anterior teeth.
Similar to Midline diastema in children and adults (20)
IMPORTANCE OF GUMS
मसूड़ों का महत्व
दांतों की आसपास की संरचनाSurrounding structures of teeth
मसूड़ा (Gingiva)
गिंगिवा आपके दांतों के नीचे नरम, गुलाबी ऊतक है।
मसूड़े की सूजन (GINGIVITIS)
मसूड़े की सूजन तब होती है जब आपके मसूड़े लाल, सूजन और छूने या ब्रश करने पर वे आसानी से खून बह सकता है। यह घटनाओं की एक श्रृंखला में पहला चरण है जो पट्टिका के निर्माण के साथ शुरू होता है।
यदि ठीक से इलाज नहीं किया जाता है, तो यह पीरियडोनिटिस (Periodontitis) और दांत के नुकसान के साथ समाप्त हो सकता है क्योंकि ऊतक जो चारों ओर से घेरे हुए है और दांतों का समर्थन करता है।
मसूड़ों की हानि ( Gingival Recession)
मसूड़ों की सिकुड़न के कारण दांतों की जड़ों में मसूड़ों की मंदी होती है। यह घर्षण, क्षरण, पेरियोडोंटल बीमारी या सर्जरी के कारण होता है।
मुंह से दुर्गंध (Halitosis)
हैलिटोसिस मौखिक या जठरांत्र मूल की खराब सांस है।
कारण :
तंबाकू
खाना
शुष्क मुँह
दांत की सफाई
क्रैश डाइट्स
दवा
Why Mouthwash ? माउथवॉश क्यों ?
सांसों की बदबू (Halitosis)
मसूड़े की सूजन (Gingivitis)
पट्टिका (Plaque removal)
शुष्क मुँह (Dry Mouth)
पीले या फीके दांत (Discoloured teeth)
मसूड़ों में कमी (Gingival Recession)
Important contents of Mouthwash
Fluoride –
फ्लोराइड यह घटक दाँत क्षय से लड़ता है और
तामचीनी को मजबूत करता है।
Cetylpyridinium Chloride –
साइटिलपिरिडिनियम क्लोराइड।
यह खराब सांस को खत्म करता है और बैक्टीरिया को मारता है।
Chlorhexidine –
यह पट्टिका को कम करता है और
मसूड़े की सूजन को नियंत्रित करता है।
Essential Oils –
आवश्यक तेल। कुछ माउथवॉश में
आवश्यक तेलों में पाए जाने वाले यौगिक होते हैं, जैसे मेन्थॉल (पेपरमिंट), नीलगिरी, और थाइमोल (थाइम), जिसमें ऐंटिफंगल और जीवाणुरोधी गुण होते हैं
Carbamide Peroxide –
कार्बामाइड पेरोक्साइड या हाइड्रोजन पेरोक्साइड। यह घटक दांतों को सफेद करता है।
Home Remedies
अनानास का रस ( Pineapple Juice )
पानी (Water)
दही (Curd)
ग्रीन टी (Green Tea)
बेकिंग सोडा के साथ घर का बना माउथवॉश (Home made mouthwash with baking soda)
सिरका के साथ घर का बना माउथवॉश
( Home made mouthwash with vinegar
गम कसैला (Gum Astringent / GumPaint)
एस्ट्रिंजेंट प्रोटीन को जमाकर, मसूड़ों के रक्तस्राव को रोकने में मदद करते हैं।
You can contact us directly in case of any help needed from my side
Dr. Harsh S. Shah
(Dental Surgeon)
Contact – 7776096239
Email- dr.shahsdentalclinic@gmail.com
Sterilization and disinfection in Dentistry Dr. Harsh Shah
An overview of significance of sterilization in safety of patients and view on all the methods being followed for sterilization and disinfection in todays' practice.
STERILIZATION AND DISINFECTION , INFECTION CONTROL IN DENTISTRY ,
Solitary oral ulcers and systemic diseasesDr. Harsh Shah
A brief overview of different ulcerative lesions seen in the oral cavity linked to the dangerous systemic diseases and preventive measures for the disease before it turns lerhal
SDDCH, Parbhani
Non –pharmacological behavior management in childrenDr. Harsh Shah
Overview on nonpharmacological managent of behaviour in children
Presented by : Mayuri Karad
SDDCH Parbhani
Guided by : Dr. Rehan Khan
Dept, of Pediatric and preventive dentistry
Apexogenesis & apexification in pediatric dentistryDr. Harsh Shah
SDDCH Parbhani
Presented by : Vipul GIratkar
Dept. of Pediatric dentitstry
Guided by . Dr. Rehan Khan
DIscussion regarding apexification and apexogenesis
Tongue thrust and mouth breathing habits in childrenDr. Harsh Shah
Overview on mouth breathing and tongue thrusting in children leading to ill effects
Presented by : Pratiksha Ahire
Guided by : Dr. Rehan Khan
Dept. of Pediatric Dentistry
SDDCH PArbhani
Dental Fluorosis : double sided sword
Overview of this deadly disease in this presentation
Presented by: Shubham Shegokar
Guided by : Dr. Rehan Khan
Pediatric Dentitstry
Growth and development of mandible in childrenDr. Harsh Shah
a brief idea about the development of mandible for indian students looking for a quick review from dentistry department
all the best to students
Presented by : Harsh SHah
Dept. of Orthodontics
SDDCH PBN
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
3. DEFINITION
Definition of Diastema:
Diastema is defined as a space between adjacent
teeth.
Definition of Midline Diastema:
Midline diastema refers to anterior midline spacing
which is greater than 0.5 mm, between the two
central incisors.
4. CAUSES OF MIDLINE DIASTEMA
1) Transient Malocclusion
A midline spacing can occur during the mixed
dentition period associated with the eruption of
permanent canines.This stage is called as ugly
duckling stage,corrects itself as the developing
permanent canines erupt further.(Broadbent
phenomenon)
5. 2) Tooth material arch length discrepancy:
Conditions such as missing teeth,microdontia,peg
shaped laterals,macrognathia,extractions with resultant
driftring of adjacent teeth may affect midline diastema.
3) Unerupted Mesiodens:
Presence of an unerupted mesiodens between the
two central incisors also predispose to midline diastema.
Microdontia Unerupted mesiodens
6. 4) Abnormal frenal attachment:
The presence of a thick and fleshy labial frenum can
give rise to midline diastema.
5) Proclination:
Proclination of a teeth as a result of habits such as
thumb sucking or tongue thrusting can cause midline
diastema along with genralised spacing.
Abnormal frenum attachment Proclination
7. 6) Midline Pathology:
Soft tissue and hard tissue pathologies such as
cysts,tumors,odontomes may cause midline diastema.
7) Iatrogenic cause:
Midline diastema can occur when certain therapeutic
procedures such as rapid maxillary expansion are
undertaken.
8. 8) Racial predisposition:
Presence of midline spacing also has a racial &
familial background.The negroid race shows the greatest
incidence of midline diastema.
9) Ectopic maxillary canines:
Absence of canines from their normal position can
facilitate distal drift & tilt of incisors with space opening &
there is the associated lack of physiological pressures to
upright the lateral & central roots that normally closes the
diastema.
9. DIAGNOSIS
A proper history and clinical examination should be
done.
Measure the mesiodistal width of the teeth which
will help in determining the tooth material-arch
length discrepancy.
Blanch test-done by pulling the upper lip
outwards.Presence of a thick & fleshy frenum is
confirmed by the blanching of the tissue in the
incisive papilla region palatal to central incisor.
10. ` Check for any precious oral habit.
Periapical radiograph-Presence of notching in the
interdental alveolar bone as seen on a radiograph is also
diagnosing midline pathology that cause spacing.
11. MANAGEMENT OF MIDLINE DIASTEMA
Treatment of midline diastema is done in three
phases:
1) Removal of cause
2) Active treatment
3) Retention.
12. 1)REMOVAL OF CAUSE:
First phase involves removal of the etiology.
Habits should be eliminated using fixed or removeable
habit brekers.
ETIOLOGY TREATMENT PLAN
• Normal developing dentition(ugly
duckling stage)
• Self correcting after the eruption
of permanent canines.
• Spontaneous closure seems to
occur with less frequency in-
a) generalised sparing
b) Initial diastema of more than 3
mm.
• Familial incidence • Appliance therapy
• Parafunctional habits
-flaccid lips & poor muscle tone.
a)Correction of the habits has been
known to spontaneously correct the
diastema.
13. ETIOLOGY TREATMENT PLAN
-tongue thrust may cause anterior
open bite & diastema
-thumb/digit sucking over a
prolonged period.
b)In cases of excessive
diastema,correction of the diastema
can be simultaneously carried out
with the habit breaking appliance.
• Tooth size discrepancies
a) Excessive anterior vertical
overlap.
b) Excessive vertical maxillary
alveolar growth.
c) Retrognathic mandible or a
prognathic mandible
• First intrusion of the maxillery
incisors followed by retraction of
incisors to close the diastema.
• If however cephalogram indicates
an excessively long lower face or
a class 3 growth trend.functional
therapy may be the treatment of
choice.
• Frenal attachments • Generally advocated that the
diastema should be closed as far
as possible before going in for
frenectomy.the reason cited is that
the surgery be performed
before,the surgical scar tissue
maintains the diastema.
• Mesio-distal angulation of teeth. • the correction of the crown
angulation(tipping)will close the
14. ETIOLOGY TREATMENT PLAN
• Tooth anomalies
(supernumerary tooth,peg
laterals,absence of laterals)
• Supernumerary-removal of the
supernumerary followed by a
closure of the diastema is done.
• Peg shaped laterals-the
diastema can be corrected
orthodontically followed by
esthetic restoration of the peg
shaped laterals.
• Absence of laterals-
a)The space for missing laterals if
detected early,may be initially be
maintained and at later that be
replaced with fixed prosthesis.
b)Another option is to
orthodontically more the canine
into the space of the missing
laterals,followed by a carefull
recountring of the cuspid &the first
bicuspid.
• pathological • Systemic phase (if required)
followed by appilance therapy.
16. Second phase-active treatment
a) It can be done using removeable appliances or
fixed appliances.
b) The principle applied here is of reciprocal
anchorage(in fixed)
c) The types of movement are either bodily or more
commonly by tipping.
17. 2)REMOVEABLE APPLIANCES
An active plate can be utilized for this purpose.the
plate incorporates palatal finger springs passing
distally to the central incisors, such that the loop is
opposite to the direction of movement.
A split labial bow is a modification which can be
utilized to close the diastema,(made up of 0.7mm
hard stainless steel wire.
Disadvantage-space may be created between the
laterals.
in split labial bow,bows are extended upto the
buccal aspect of the opposite central incisors.
18. In cases where there is an increased overjet
accompying the diastema, a hawley’s plate with an
active labial bow can be used to retract the incisors
& thus close the space.
Drawback of using removeable appliance-
only tipping movements can be achieved.
19. 3)FIXED APPLIANCES
Fixed appliance incorporating elastics or springs
bring about the most rapid correction of midline
diastema.
Elastics can be streched between the two central
incisors in order to close the space.
M shaped springs incorporating helices can be
inserted into the central incisors brackets.the spring
is activated by closing the helices.
A stainless steel band with a bracket or more
commonly a bracket may be banded to tooth and
elastics utilised to bring the central incisors
towards each other.
20. For the purpose of bodily movement of the teeth, it
is suggested than an edgewise bracket with a
simple looped partial archhwire made from a
retangular wire be tied under the tension into both
the brackets.
22. Third phase of treatment involves retaining the
treated malocclusion.
4)RETENTION
Midline diastema is easy to treat but difficult to
retain.
In order to prevent a relapse,a long term retention
is required in these cases.
23. Most orthodontic recommended long term retention
using suitable retainers.
Since prolonged retention is indicated,it is advisable
to use lingual bonded retainers.
The other retainers that can be used include
bonded retainers & hawley’s retainer.
24. OTHER TREATMENT MEASURES
Role of cosmetics restorations:
a) Esthetic composite resins are generally used to
close the midline diastema especially in adult
patients.
b) It requires gradual composite build up on the
mesial surface & stripping of the distal surface of
central & lateral incisors in order to achieve a
natural shape & size of teeth.
25. COMPOSITE BUILD UP:
a)Composite build up on
mesial aspect of the
central incisors & stripping
of the distal surface-
b)Composite build up on
the mesial aspect of the
lateral incisors & stripping
of the distal surface-
27. Prosthesis/crowns:
a) Presence of peg shaped laterals or teeth with
other anomalies of shape & size require prosthetic
rehabilitation.
b) Missing teeth should be replaced with fixed or
removeable prosthesis.
c) Crowns on central incisors can close a midline.
28. CONCLUSION
Midline diastema although being a transient
condition become persistent at times which needs
to be treated with special care & concern due to the
high propencity of relapse.
Treating midline diastema efficiently & effectively is
important for overall well being of individual as it
affects the esthetics of the person & has an
important effect on overall personality of an
individual.
29. Effective diastema treatment requires correct
diagnosis of its etiology & intervention relevant to
the specific etiology.
Correct diagnosis includes medical & dental
histories, radiographic & clinical examinations, and
possibly tooth size evaluations.
Timing is often important to achieve satisfactory
results.
30. RECENT ADVANCES IN CLOSURE OF MIDLINE
DIASTEMA:
Direct closure with composite layering technique.
Laminate veneers.
Adhesive Restorations.
Micro magnetic retainers.