An inguinal hernia occurs when abdominal contents bulge through the inguinal canal. There are two main types - indirect which are congenital and direct which are acquired. Diagnosis is made through history and physical exam assessing for reducibility and impulse on coughing. Treatment options include watchful waiting for small reducible hernias or surgical repair through open or laparoscopic techniques. The Lichtenstein tension-free repair using mesh placement is commonly performed.
Inguinal Hernia is the commonest problem in General surgery. All medical students should know everything about this common problem. In this ppt presentation I have covered all the details regarding Inguinal hernia thoroughly.
OPEN INGUINAL HERNIA REPAIR- OPERATIVE SURGERY
#surgicaleducator #operativesurgery #openinguinalherniarepair #usmle #babysurgeon #surgicaltutor
Dear viewers,
• Greetings from “Surgical Educator”
• Because of the popular demand by viewers of the YouTube channel “Surgical Educator”, I have decided to create and upload videos on common surgeries.
• I have already uploaded videos on open and Laparoscopic Appendicectomy, Thyroidectomy, Modified Radical Mastectomy and open and
Laparoscopic Cholecystectomy
• In this video today, I have discussed Open Inguinal Hernia Repair.
• However, these videos are not real surgeries but the theoretical aspect of operative surgery like going through an atlas of operative surgery.
• Along with these videos, I recommend you to watch real operative surgery videos as well and I will give a link for each surgery in the end of the video as end-cards, which I think will be very useful.
• This will give a very good opportunity for the surgical trainees to mentally rehearse various surgical steps in a sequential manner prior to actual surgery. You can watch the video in the following links:
• surgicaleducator.blogspot.com
• youtube.com/c/surgicaleducator
• Thank you for watching the videos.
Femoral hernia is the third common hernia after inguinal and incisional hernias. The swelling in femoral hernia is below and lateral to pubic tubercle. It is more common in females. Strangulation is very common in this hernia.
Inguinal Hernia is the commonest problem in General surgery. All medical students should know everything about this common problem. In this ppt presentation I have covered all the details regarding Inguinal hernia thoroughly.
OPEN INGUINAL HERNIA REPAIR- OPERATIVE SURGERY
#surgicaleducator #operativesurgery #openinguinalherniarepair #usmle #babysurgeon #surgicaltutor
Dear viewers,
• Greetings from “Surgical Educator”
• Because of the popular demand by viewers of the YouTube channel “Surgical Educator”, I have decided to create and upload videos on common surgeries.
• I have already uploaded videos on open and Laparoscopic Appendicectomy, Thyroidectomy, Modified Radical Mastectomy and open and
Laparoscopic Cholecystectomy
• In this video today, I have discussed Open Inguinal Hernia Repair.
• However, these videos are not real surgeries but the theoretical aspect of operative surgery like going through an atlas of operative surgery.
• Along with these videos, I recommend you to watch real operative surgery videos as well and I will give a link for each surgery in the end of the video as end-cards, which I think will be very useful.
• This will give a very good opportunity for the surgical trainees to mentally rehearse various surgical steps in a sequential manner prior to actual surgery. You can watch the video in the following links:
• surgicaleducator.blogspot.com
• youtube.com/c/surgicaleducator
• Thank you for watching the videos.
Femoral hernia is the third common hernia after inguinal and incisional hernias. The swelling in femoral hernia is below and lateral to pubic tubercle. It is more common in females. Strangulation is very common in this hernia.
Inguinal hernia presentation
by Shariatyfar MD
based on schwartz principles of surgery 11th edition
Qom university of medical sciences
winter 2017
email me at Mohammadali.shariatyfar@hotmail.com for Download
Good luck
Simple notes on definition of abdominal hernias in general, as well as clinical features and management of inguinal hernias.
Brief explanation of hernia repair methods (laparoscopic, open surgery)
Out of a variety of Digestive System diseases, Hernia is common and associated with obesity. the presentation gives a brief overview regarding the management of hernias in clinical surgical departments of Hospitals.
Inguinal hernia presentation
by Shariatyfar MD
based on schwartz principles of surgery 11th edition
Qom university of medical sciences
winter 2017
email me at Mohammadali.shariatyfar@hotmail.com for Download
Good luck
Simple notes on definition of abdominal hernias in general, as well as clinical features and management of inguinal hernias.
Brief explanation of hernia repair methods (laparoscopic, open surgery)
Out of a variety of Digestive System diseases, Hernia is common and associated with obesity. the presentation gives a brief overview regarding the management of hernias in clinical surgical departments of Hospitals.
Seminar presentation by 5th year Medical Student under the supervision of a pediatric surgery specialist from HRPZ II. Reference as mentioned in the slide.
Inguinal and femoral hernia:
A hernia is a protusion of a viscus or a part of viscus through and abnormal opening in the walls of its containing cavity. Details of inguinal hernia and few slides on other types of hernia.
This is a case study done by me as a part of my in-service education progamme in my institution...hope this may help all nurses who wants to do a case study.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
6. BOUNDARIES
Anterior
external oblique aponeurosis
Posterior
fusion of the transversalis fascia
and transversus abdominus
muscle,
Superior
arch formed by the fibers of the
internal oblique muscle.
Inferior
inguinal ligament
7. DEFENCE MECHANISM OF INGUINAL
CANAL
Obliquity of inguinal canal
Arching of conjoint tendon
Shutter mechanism of internal oblique
‘Ball valve mechanism’ due to contraction of cremaster muscle which plugs
to superficial ring
When External oblique muscle contracts intercrural fibers of superficial ring
appose causing ‘Slit valve mechanism’
Hormones
16. INDIRECT INGUINAL HERNIA
Through the internal ring
of inguinal canal
Congenital
Patent processus
vaginalis
~5% Lifetime risk
Higher risk of
strangulation than direct
18. ACCORDING TO EXTENT
INDIRECT INGUINAL HERNIA
INCOMPLETE
BUBONOCELE: Here sac is confined to the inguinal canal
FUNICULAR: Here sac crosses the superficial ring but does not
reach the bottom of the scrotum.
Complete : Here sac descends to the bottom of scrotum
19. INDIRECT
Can occur at any age from childhood
to adults
Occurs in pre-existing sac
Protrusion through deep ring
Pyriform/oval in shape, descends
obliquely
Can become complete
Neck of sac is narrow and lateral to
inferior epi- artery
Sac is anterolateral to cord
Ring occlusion is positive
Commonly unilateral but can be
bilateral
Obstruction/strangulation is common
Sac should be opened during surgery
DIRECT
Common in elderly
Always acquired
Herniation through posterior wall
Globular/round in shape, descends
directly forward
Descent down in the scrotum is rare
Wide neck and medial to inf epigastric
artery
Ring occlusion test is negative
Commonly bilateral
Obstruction/strangulation is rare
Sac is not necessarily opened unless
obstruction is present
20. PENTALOON HERNIA
(DOUBLE,SADDLE,ROMBERG)
Here both direct and indirect inguinal sacs
are present and clinically present as direct
hernia
During surgery , indirect sac may be
missed and so leads to recurrent hernia
through retained or unidentified indirect
sac
Here both medial and lateral sacs
straddle the inferior epigastric artery
21. Sliding hernia
Here posterior wall of the
sac is not only formed by
parietal peritoneum but
also by sigmoid colon with
its mesentery on left side;
cecum on right side and
often with a portion of
bladder.
It occurs exclusively in
males and mainly on left
side.
22. CLINICAL CLASSIFICATION
REDUCIBLE
Can be reduced by the patient,surgeon or by it self when pt. lies
supine. Has expansile impulse on coughing.
IRREDUCIBLE
Cant be returned to the abdomen, usually due to adhesion b/w
sac and its contents or over crowding of the contents.
OBSTRUCTED
Bowel is obstructed but there in no interference to the blood
supply.
23. STRANGULATION
When blood supply of contents of hernia is seriously impaired leading to
formation of gangrene.
Obstruction impaired venous return Congestion Further
dilatation of bowel which becomes purple colored Fluid collects in the
sac Eventually arterial supply is compromised Bowel becomes dark
brownish with friable wall Bacteria migrate to fluid of sac
Perforation occurs at site of constriction ring Peritonitis
24. CAUSES OF STRANGULATION
Narrow neck
Adhesions
Irreducibility
Long time large hernia with adhesions
25. RICHTERS HERNIA
This is the partial enterocele when only the anti-mesenteric margin of the
gut is strangulated in the sac.
26. MAYDLS HERNIA
When a W-shaped loop of gut lies in the hernia sac and the intervening
loop is strangulated within the main abdominal cavity
Hernial sac containing a strangulated MECKELS diverticulum. It can
progress to gangrene .
LITTRES HERNIA
28. NYHUS CLASSIFICATIONSYSTEM
Type I INDIRECT HERNIA; internal abdominal ring normal; typically in infants, children, smalladults
Type II
INDIRECT HERNIA; internal ring enlarged without impingement on the floor of theinguinal
canal; does not extend to the scrotum
Type IIIA DIRECT HERNIA; size is not taken intoaccount
Type IIIB
INDIRECT HERNIA that has enlarged enough to encroach upon the posterior inguinalwall;
INDIRECT SLIDINGOR SCROTAL HERNIAS are usually placed in this category because theyare
commonly associated with EXTENSIONTOTHE DIRECT SPACE; also includes PANTALOON
HERNIAS
Type IIIC FEMORAL HERNIA
Type IV
RECURRENT HERNIA; modifiers A–D are sometimes added, which correspondTOINDIRECT,
DIRECT, FEMORAL,AND MIXED,RESPECTIVELY
29. EPIDEMIOLOGY
One of the most common surgical procedures
Incidence:
~5-10% lifetime
75% of abdominal wall hernias
Male > Female
Indirect > Direct
Right > Left
1/3 may develop a contralateral inguinalhernia
30. ETIOLOGY
Multifactorial
Weakness in abdominal wall musculature
PRESUMEDCAUSESOF GROIN HERNIATION
Coughing Valsalva's maneuvers
Chronic obstructive pulmonary disease Ascites
Obesity Upright position
Straining Congenital connective tissue disorders
Constipation Defective collagen synthesis
Prostatism Previous right lower quadrant incision
Pregnancy Arterial aneurysms
Birthweight <1500 g Cigarette smoking
Family history of a hernia Heavy lifting
Physical exertion (?)
32. HISTORY
More common in males(20:1)
Dragging pain and swelling in
the groin which is better seen
while coughing and standing.
History of
Constipation
Cough and Smoking
Urological symptoms
Duration
Progressiveness
Any h/o surgery
(appendectomy)
Reccurent hernia or
contralateral
33. PHYSICAL EXAMINATION
Proper exposure from umbilicus to
mid thigh
Inspection
Standing
lying
Palpation
Can you get above the swelling
Reducibility
Cough impulse
Head or leg raise to look for
abdominal muscle tone
Internal Ring Occlusiontest
34. EXAMINATION
Zieman’s test
index finger on deep ring
middle finger on superficial ring
ring finger on femoral opening
Ring invagination test
little/index finger is invaginated from
Bottom of scrotum gradually pushed to enter
In superficial inguinal ring, impulse on coughing is felt
At tip of invaginated finger.
35. EXAMINATION
Always abdominal, urological and respiratory examination is done to rule
out any precipitating factor like Chronic Bronchitis , Ascites, Stricture
urethra and BPH.
40. MANAGEMENT PRINCIPLES
Not all hernias require surgical repair
Small hernias can be more dangerous than large
Pain, tenderness and skin colour changes imply high risk of strangulation
Femoral hernia should always be repaired
41.
42. CONSERVATIVE MANAGEMENT
Aimed at alleviating symptoms such as
pain, pressure, and protrusion of abdominal
contents
Assuming a recumbent position
Truss, an elastic belt or brief
44. TAXIS
The patient is sedated and placed in aTrendelenburg position.
The hernia sac is grasped with both hands, elongated, and then
milked back through the hernia defect.
Pressure applied to the most distal portion of the sac will cause the
contents to mushroom and prevent reduction.
45. STRANGULATED HERNIA
Femoral > Indirect > Direct
Fever, leukocytosis, and hemodynamic instability.
The hernia bulge usually is very tender, warm, and may exhibit
red discoloration.
Taxis should not be applied to strangulated hernias as a
potentially gangrenous portion of bowel may be reduced into the
abdomen without being addressed
47. PRINCIPLES OF SURGICAL REPAIR
Reduction of the hernia content into the abdominal cavity
Excision and closure of peritoneal sac
Reapproximation of the walls of the neck of the hernia
Permanent reinforcement of the abdominal wall defect with sutures or
mesh
48. SURGICAL OPTIONS
Herniotomy : excision of hernia sac
in children
Hernirraphy : strengthening of posterior wall of inguinal canal
Hernioplasty : placement of mesh
52. BASSINI REPAIR
Is frequently used for
indirect inguinal
hernias and small direct
hernias
The conjoined tendon of
the transversus abdominis
and the internal oblique
muscles is sutured to the
inguinal ligament
53. LYTLE’S REPAIR
Only internal ring is narrowed
by placing interrupted sutures
over the medial side of the
ring to the fascia transversalis
54. MCVAY REPAIR
inguinal and femoral
canal defects
The conjoined tendon is
sutured to Cooper’s
ligament from the pubic
cubicle laterally
55. SHOULDICE
REPAIR
Multilayered
Transversalis fascia is incised,
lower flap is sutured to
posterior part of upper flap
Upper flap is sutured to
inguinal ligament.
Then conjoint tendon and
inguinal ligament is further
approximated by two layers in
front of cord .
Then Ext Oblique apo is sutured
in two layers.
56. MALONY DARN REPAIR
Posterior wall is re-inforced
with monofilament nylon
darn. The Darn is made in
crisscross fashion between
the conjoint tendon and
inguinal ligament
Excellent results.
Most commonly performed
in countries where mesh is
too expensive
57. MESH
Gross structure
Net meshes or flat sheets
Synthetic mesh
Polypropylene, polyester and polytetrafloroethylen PTFE
Weight and porosity
Dense or heavy weight and Light weight, Large pore meshes
Biological mesh
Sheets of sterilized, decellularised, non immunogenic connective tiisue
59. LICHTENSTEINTENSION- FREE
REPAIR
Initial part of surgery is same as Bassini repair.
Once hernia has been removed and any medial defect closed, a piece of
mesh measuring 8-15cm is placed over posterior wall, behind the
spermatic cord and is split to wrap around the spermatic cord at deep
inguinal ring.
Loose sutures with Prolone hold mesh to inguinal ligament and conjoint
tendon
ADVANTAGES
Lowered recurrence rates
Accelerated postoperative delivery
60. Open plug/device/complex
mesh repair.
Gillbert mesh repair ;
plug and
patch, internal ring is
plugged by a cone shaped
piece of prolene mesh
PHS (Prolene hernia
system)
onlay and
sublay sandwich technique
62. Transabdominal Preperitoneal
Procedure (TAPP)
Surgeon enters the peritoneal cavity incises the peritoneum above the
hernia defects and reflects it away from the muscles entering Pre-
peritonealy
Hernia is reduced and a 10*15 cm mesh is placed just deep to the
abdominal wall extending across midline into retropubic space and 5cm
lateral to the deep inguinal ring.
63. Totally Extraperitoneal (TEP) Repair
Space is created just deep to the
abdominal msucles without entering
the peritoneal cavity
Hernia is reduced and mesh is
inserted preperitonealy.
64. RECURRENCE
Bassini repair 10%
Around 1% for Shouldice repair
Hernioplasty 1-3%
Most recurrences are of the same type as the original
hernia
Recurrence Factors
Patient
Technical
Tissue
65. RECURRENCE
Patient factors
malnutrition, immunosuppression, diabetes, steroid
use, and smoking.
Technical factors
mesh size, prosthesis fixation, and technical proficiency of
the surgeon.
Tissue factors
wound infection, tissue ischemia, and increased tension
within the surgical repair
66. COMPLICATIONS
The overall risk of complications of inguinal
hernia repair is low.
CommonComplications
EARLY: pain, bleeding, urinary retention, anaesthesia
related
Medium : seroma, wound infection
Late: chronic pain, testicular atrophy, recurrance