This document provides an overview of the macroscopic and microscopic anatomy of the gingiva. It discusses the different types of gingiva including the marginal, attached, and interdental gingiva. Microscopically, it describes the oral, sulcular, and junctional epithelium, as well as the development of the junctional epithelium during tooth eruption. It also reviews the cells and layers present in the gingival epithelium, the epithelium-connective tissue interface, and components of the gingival connective tissue.
seminar on gingiva
contents:
Introduction
Definition
Development of gingiva
Macroscopic anatomy
Microscopic anatomy
Blood supply
Lymphatic drainage
Nerve supply
Correlation of clinical and microscopic features
Repair/healing of gingiva
Age changes
Gingival diseases
Clinical considerations
Conclusion
References
THIS PRESENTATION INCLUDES:
INTRODUCTION
MAIN BLOOD SUPPLY BRANCHES TO PERIODONTIUM
BLOOD SUPPLY TO MAXILLARY TEETH AND PERIODONTIUM
BLOOD SUPPLY TO MANDIBULAR TEETH AND PERIODONTIUM
VENOUS DRAINAGE OF MAXILLARY AND MANDIBULAR TEETH AND PERIODONTIUM
BLOOD SUPPLY TO EACH COMPONENT OF PERIODONTIUM
CLINICAL SIGNIFICANCE OF BLOOD SUPPLYING THE PERIODONTIUM
CLINICAL CORELATIONS WITH GINGIVITIS AND PERIODONTITIS
CONCLUSION
REFERENCES
seminar on gingiva
contents:
Introduction
Definition
Development of gingiva
Macroscopic anatomy
Microscopic anatomy
Blood supply
Lymphatic drainage
Nerve supply
Correlation of clinical and microscopic features
Repair/healing of gingiva
Age changes
Gingival diseases
Clinical considerations
Conclusion
References
THIS PRESENTATION INCLUDES:
INTRODUCTION
MAIN BLOOD SUPPLY BRANCHES TO PERIODONTIUM
BLOOD SUPPLY TO MAXILLARY TEETH AND PERIODONTIUM
BLOOD SUPPLY TO MANDIBULAR TEETH AND PERIODONTIUM
VENOUS DRAINAGE OF MAXILLARY AND MANDIBULAR TEETH AND PERIODONTIUM
BLOOD SUPPLY TO EACH COMPONENT OF PERIODONTIUM
CLINICAL SIGNIFICANCE OF BLOOD SUPPLYING THE PERIODONTIUM
CLINICAL CORELATIONS WITH GINGIVITIS AND PERIODONTITIS
CONCLUSION
REFERENCES
Introduction
A sound knowledge of the anatomy of the periodontium and the surrounding hard and soft structures is essential to determine the scope and possibilities of surgical periodontal procedures and to minimize their risks.
Blood vessels, and nerves located in the vicinity of the periodontal surgical field, are particularly important during various surgical procedures.
Arterial Supply
Common Carotid Artery
Carotid Sinus & Carotid Body
Applied Anatomy of CCA
CAROTID PULSE :
CCA may be compressed against the carotid tubercle of transverse process of C6 vertebra about 4cm above the sternoclavicular joint.
External Carotid Artery
Generally it lies anterior to the Internal Carotid Artery.
It is the chief artery of supply to structures in the front of neck, oral cavity and in the face.
In carotid triangle
Crossed superficially by:
Cervical branch of facial nerve
Hypoglossal nerve
Facial, lingual &superior thyroid vein
Deep to artery lies:
Wall of pharynx
Superior laryngeal nerve
Ascending pharyngeal artery
Above the carotid triangle
ECA lies deep in the substance of parotid gland
Branches
Lingual Artery
Principal artery of tongue.
Arises anteromedially from ECA opposite the tip of greater cornu of hyoid bone.
Divided into three parts by hyoglossus muscle.
Applied anatomy
Sublingual artery injury occurs in premolar & molar region, when sharp instrument or rotating disks slips off a lower molar & injure the floor of mouth.
Sublingual and submental arteries may course anteriorly in close proximity to the lingual plate, and branches of these blood vessels enter accessory foramina along the lingual cortex.
Hofschneider et al (1999)
Inadvertent penetration through the lingual cortical plate into the floor of the mouth while preparing an osteotomy can cause arterial trauma, thereby resulting in development of a sublingual or submandibular hematoma
Flanagan D. et al.2003
Facial Artery
ORIGIN: Arises from the ECA just above the tip of greater cornua of hyoid bone.
COURSE:
Runs upwards in neck as cervical part ;
On face as facial part.
Tortuous course—
In neck allows free movements of pharynx during deglutition,
On face allows free movements of mandible , lips, & cheek during mastication & facial expressions, escapes traction & pressure during movements.
Cervical part :
Cervical part runs upwards on superior constrictor of pharynx deep to the posterior belly of digastric.
It grooves the posterior border of submandibular gland, makes S-bend [2 loops]
1st winding down over submandibular gland &
then up over the base of mandible.
Facial part:
The vessel enters the face by winding around the base of the mandible, and by piercing the deep cervical fascia,at the anteroinferior angle of the masseter muscle, here it can be palpated & is called as anaesthetist’s artery. Using contracted masseter as a landmark, pulse of facia
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
alveolar bone in health with microscopic features and details about bone formation, resorption also includes bone remodelling and changes after extraction
As the periodontium consists of cementum and alveolar bone and periodontitis poss an immense insult to the periodontium, it is of utmost importance for a periodontist and implantologist to understand the basic molecular biology of cementum and alveolar bone to manage the cases of periodontitis more effectively.
Macroscopic features of Gingiva.
This presentation will help and let u know about the Development and Macroscopic features of gingiva in detail. Thank you.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
• Introduction
• Definitions
• Macroscopic Features
• Microscopic Features
• Blood supply
• Nerve supply
• Lymphatic drainage
• Role of epithelium in defence mechanism
• Oxygen consumption of gingiva
• Correlation of Macroscopic with microscopic features
• Conclusion
Introduction
A sound knowledge of the anatomy of the periodontium and the surrounding hard and soft structures is essential to determine the scope and possibilities of surgical periodontal procedures and to minimize their risks.
Blood vessels, and nerves located in the vicinity of the periodontal surgical field, are particularly important during various surgical procedures.
Arterial Supply
Common Carotid Artery
Carotid Sinus & Carotid Body
Applied Anatomy of CCA
CAROTID PULSE :
CCA may be compressed against the carotid tubercle of transverse process of C6 vertebra about 4cm above the sternoclavicular joint.
External Carotid Artery
Generally it lies anterior to the Internal Carotid Artery.
It is the chief artery of supply to structures in the front of neck, oral cavity and in the face.
In carotid triangle
Crossed superficially by:
Cervical branch of facial nerve
Hypoglossal nerve
Facial, lingual &superior thyroid vein
Deep to artery lies:
Wall of pharynx
Superior laryngeal nerve
Ascending pharyngeal artery
Above the carotid triangle
ECA lies deep in the substance of parotid gland
Branches
Lingual Artery
Principal artery of tongue.
Arises anteromedially from ECA opposite the tip of greater cornu of hyoid bone.
Divided into three parts by hyoglossus muscle.
Applied anatomy
Sublingual artery injury occurs in premolar & molar region, when sharp instrument or rotating disks slips off a lower molar & injure the floor of mouth.
Sublingual and submental arteries may course anteriorly in close proximity to the lingual plate, and branches of these blood vessels enter accessory foramina along the lingual cortex.
Hofschneider et al (1999)
Inadvertent penetration through the lingual cortical plate into the floor of the mouth while preparing an osteotomy can cause arterial trauma, thereby resulting in development of a sublingual or submandibular hematoma
Flanagan D. et al.2003
Facial Artery
ORIGIN: Arises from the ECA just above the tip of greater cornua of hyoid bone.
COURSE:
Runs upwards in neck as cervical part ;
On face as facial part.
Tortuous course—
In neck allows free movements of pharynx during deglutition,
On face allows free movements of mandible , lips, & cheek during mastication & facial expressions, escapes traction & pressure during movements.
Cervical part :
Cervical part runs upwards on superior constrictor of pharynx deep to the posterior belly of digastric.
It grooves the posterior border of submandibular gland, makes S-bend [2 loops]
1st winding down over submandibular gland &
then up over the base of mandible.
Facial part:
The vessel enters the face by winding around the base of the mandible, and by piercing the deep cervical fascia,at the anteroinferior angle of the masseter muscle, here it can be palpated & is called as anaesthetist’s artery. Using contracted masseter as a landmark, pulse of facia
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
alveolar bone in health with microscopic features and details about bone formation, resorption also includes bone remodelling and changes after extraction
As the periodontium consists of cementum and alveolar bone and periodontitis poss an immense insult to the periodontium, it is of utmost importance for a periodontist and implantologist to understand the basic molecular biology of cementum and alveolar bone to manage the cases of periodontitis more effectively.
Macroscopic features of Gingiva.
This presentation will help and let u know about the Development and Macroscopic features of gingiva in detail. Thank you.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
• Introduction
• Definitions
• Macroscopic Features
• Microscopic Features
• Blood supply
• Nerve supply
• Lymphatic drainage
• Role of epithelium in defence mechanism
• Oxygen consumption of gingiva
• Correlation of Macroscopic with microscopic features
• Conclusion
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
macroscopic/ clinical features of gingiva
With video clips
Short video descriptions
Lecture for 3rd BDS students
Periodontology
Periodontics aspect
Clinical features of the gingiva
Described here are the clinical features of gingiva, the various stages of gingivitis and the clinical features associated with them. The microscopic features have been described on a different slide presentation.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
3. Development of junctional epithelium
Cells present in gingival epithelium
Keratinisation,types
Nonkeratinocytes
Epithelium connective tissue interface
Gingival connective tissue
Gingival fibres-Function,Types
Clinical features of gingiva and their correlation with microscopic features
Color
Contour
Shape
Consistency
Surface texture
Position
Blood supply of gingiva
Nerves
Lymphatics
References
4. The periodontium is a structural and
functional group of tissues that together
facilitate the anchorage of teeth in jaws
There are four tissues that constitute the
periodontium
1. Periodontal ligament
2. Alveolar bone
3. Gingiva
4. Cementum
5.
6. CARANZA-Is the part of oral mucosa that covers the alveolar
processes of jaw and surrounds the neck of the teeth.
SCHROEDER-It is a combination of epithelium and
connective tissue and is defined as that portion of oral mucous
membrane, which in complete post eruptive dentition of a
healthy young individual, surrounds and is attached to the
teeth and the alveolar processes.
GRANT-Is the part of mucous membrane attached to the
teeth and the alveolar processes.
LINDHE-Is that part of masticatory mucosa covering the
alveolar processes and the cervical portions of teeth.
12. Also known as free gingiva
It is coral pink, dull surface, firm in
consistency
It is the terminal edge or border of the gingiva
surrounding the teeth like a collar.
It is not fixed to the basal periosteum of
alveolar bone .
It is usually 1mm wide and forms the soft
tissue wall of gingival sulcus
13. It is the distance between the mucogingival junction and the projection on
the external surface of the bottom of the gingival sulcus or the periodontal
pocket
It is continous with the margnal gingiva
It is firm reselient and tightly bound to the underlying tissues of the
alveolar bone
Mucogingival junction
Important landmark between the coronally positioned attached gingiva and
the apically placed alveolar mucosa
Absent on the palatal gingiva of the maxilla due to an absence of a freely
movable alveolar mucosa on the palate.
14. DIMENSIONS OF ATTACHED GINGIVA (ON
FACIALASPECTS)
REGION DIMENSION(mm)
Maxillary incisor region 3.5-4.5
Mandibular incisor region 3.3-3.9
Maxillary 1st premolar 1.9
Mandibular 1st premolar Approx 1.8
15.
16. The width of attached gingiva increases with age and supraerupted teeth
Reduced/Absent attached gingiva may be due to:
base of the pocket is close to the mucogingival line
18. It gives support to the marginal gingiva
It provides attachment or a solid base for the
movable alveolar mucosa for the action of
lips,cheeks,tongue
It can withstand frictional and functional
stresses of mastication and tooth brushing
It acts as barrier for passage of inflammation
It provides resistance to tensional stresses
19. Functionally by passive movement of lips and
cheeks(Ochsenbein et al 1974,Cohen 1964)
Measure the amount of width of attached
gingiva using probe(substraction method)
Staining method
Roll method
20. Also known as interdental papilla
Portion of gingiva located in the space
between two adjacent teeth
If a diastema is present the gingiva is firmly
bound over the interdental bone and forms a
smooth, rounded surface without interdental
papilla
21. pyramidal col
Where there is one papilla with its tip
immediately beneath the contact
point
•Which represents as a depression
that connects facial and a lingual
papilla and conforms to the shape of
interproximal contact
•Non keratinized epithelium
•Susceptible to inflammation and
disease progression
22. Marginal gingiva is separated from an
attached gingiva by a shallow line called as
free gingival groove
Located corresponding to the level of
cementoenamel junction(CEJ)
Present in only about 30%-40%
Free Gingival Groove
23. Shallow space/crevice around the tooth
bounded by the surface of the tooth on one
side and epithelium lining the free margin of
the gingiva on the other side
V shaped and rarely permits the entrance of a
periodontal probe.
The probing depth 2-3mm
24.
25. After the enamel formation is complete , the enamel is covered with
REE(reduced enamel epithelium),which is attached to the tooth by a basal
lamina & hemidesmosomes
When tooth penetrates oral mucosa,the REE unites with oral mucosa and
transformed into JE
JE proceeds in an apical direction ,forming a shallow groove,the gingival sulcus
between circumference of tooth & gingiva that encircles the newly erupted tip of
the crown
Gingival sulcus deepens as a result of seperaton of the REE from actively
erupting tooth & JE attains its position at CEJ of fully erupted tooth
26.
27. The gingival sulcus contains a fluid that seeps into it from the gingival
connective tissue through the thin sulcular epithelium
Inflammatory exudate derived from the periodontal tissues
Found in sulcus/periodontal pocket between tooth and marginal gingiva
Composed of serum and locally generated materials such as tissue
breakdown poducts,inflammatory mediators and antibodies directed
against dental plaque bacteria
Plays special part in maintaining the structure of junctional epithelium and
the antimicrobial defense of periodontium
In healthy sulcus,the amount of GCF is very less
During inflammation ,the GCF flow increases . The increasd GCF flow
contributes to host defense by flushing bacterial colonies and their
metabolites away from the sulcus
The man route of gcf diffusion is through the basement menbrane and then
through the junctional epithelium into the sulcus
28. Significance of gingival crevicular fluid
1. To assess the severity of gingival diseases, the effectiveness of periodontal
therapy and oral hygiene, the healing following gingival surgery, and the
effectiveness of oral hygiene
2. To evaluate the rate of local destruction, to assess the permeability of
junctional and sulcular epithelium, and to assess the relationship between
periodontal and systemic diseases
Factors stimulating gingival crevicular fluid flow
1. Gingival inflammation, mastication of coarse food, pocket depth,
intracrevicular scraping, scaling, and histamine topical application
2. Enzymes and sex hormones: female sex hormones increases the gingival fluid
flow
3. Circadian periodicity: gradual increase in gingival fluid amount from 6 AM to
10PM
4. Post- periodontal surgery, restoratve procedure, strip placement, mobility,
increased body temperature and salivary contamination
5. Ovulation ,hormonal contraceptives and smoking
29. Method of collection
1. Absorbing paper strips
2. Preweighed twisted threads
3. Micropipettes
4. Crevicular washings
Composition of GCF
1. Celluar elements
1. Epithelial cells
2. Leukocytes
3. Bacteria
2. Electrolytes
1. Na:K
2. Flouride,calcium,iodine and phosphorous
3. Organic compounds
1. Carbhohydrates
2. Proteins
3. Immunoglobulis
4. Complement
5. Cytokines
6. Metabolic and bacterial products
30.
31. The thickness of the gingiva in the faciopalatal /faciolingual dimension.
Reduced gingival thickness is one of the factors that can cause periodontal
attachment loss and marginal tissue recession
The term periodontal biotype introduced by Seibert and Lindhe categrized the
gingiva into “thick flat”and “thin scalloped”biotypes
Thick biotype
broad zone of keratinized tissue
flat gingival contour which indicates thick underlying bony architecture
More resilient to any inflammation or trauma
Thin biotype
Thin band of the keratinized tissue
Scalloped gingival contour , which indicates thin bony architecture
More sensitive to inflammation or trauma
32.
33.
34. Histological examination of gingiva exhibited following
structures:
Gingival epithelium
Epithelium connective tissue interface
Gingival connective tissue or lamina propria
36. Called as outer epithelium
Keratinized stratified squamous type epithelium
It covers the crest and outer surface of marginal gingiva and surface of
the attached gingiva
The oral epithelium consists of following types of cellular layers
Oral Epithelum
37. 1. Stratum basale:
Cells either cylindrical or cuboidal
Found immediately next to connective tissue and separated by a basement membrane
Germinative layer, hence it can divide
When two daughter cells are formed by cell division, an adjacent older basal cell is
pushed into the spinous cell layer and starts as a keratinocyte to transverse the
epithelium
2. Stratum spinosum:
prickle cell layer
Polyhedral cells with short cytoplasmic processes are present
Uppermost cells from this layer contain granules called as keratinosomes or odland
bodies
3. Stratum granulosm:
Cells are flattened in plane parallel to the gingival surface
Keratohylain granules associated with keratin formation round in shape and appear
within in cytoplasam of the cell
4. Stratum corneum:
Closely packed flattened cells, lost nuclei.
Cells are densely packed with tonofilaments
Rounded bodies probably representing lipid droplets appear within the cytopasam of the
cell
38.
39. Nonkeratinized stratified squamous epithelium
Lines the gingival sulcus
Apically bounded by the junctional epithelium,coronally it meets the outer gingival epithelium at the
height of gingival margin
Lacks granulosum , corneum strata,K1,K2 and K10 to K12 cytokeratins
It contains K4 and K13 so called esophageal type cytokeratins
Also express K19
Does not contain merkel cells
Lack of keratinization makes this area particularly susceptible to influences from microorganism
It has the potential to keratinize if
It is reflected and exposed to the oral cavity
Bacterial flora of the sulcus is totally eliminated
Act as semipermeable membrane, through which injurious bacterial products pass into the gingival and
tissue fluid from gingiva seeps into the sulcus
No rete pegs
40. Collar like band of stratified squamous non keratinizing epithelium
3 to 4 layers thick in early life ,the number of layers increase with age to 10 or even 20 layers
The cells can be grouped in two strata:
Basal layer: Facing the connective tissue
Suprabasal layer: Extending to the tooth surface
The length of juntional epithelium 0.25 to 1.35 mm
The junctional epithelium is formed by the confluence of the oral epithelium and the reduced
enamel epithelium during tooth eruption
K19 and K5,K14-Stratification-specific cytokeratins
It exhibits lower glycolytic enzyme, lacks acid phosphatase activity
41. The junctional epithelium is attached to the tooth
surface- internal basal lamina
It is attached to the gingival connective- external
basal lamina
THE DENTOGINGIVAL UNIT:
The attachment of the junctional epithelium to the
tooth is reinforced by the gingival fibres, which
brace the marginal gingiva against the tooth
surface
For this reason the juctional epithelium and the
gingival fibers are considered together as a
functional unit
42. Hypothesis given to explain mode of attachment of epithelium
to tooth surface
1.GOTTLIEB 1921:gingiva is organically united to surface of
enamel. He termed it as epithelial attachment
2.ORBAN1953:stated that the separation of the epithelial
attachment cells from the tooth surface involved preparatory
degenerative changes in the epithelium
3.WAERHAUG 1960:presented a concept of epithelial cuff, he
concluded that gingival tissues are closely adapted but not
organically united
3.SCHROEDER AND LISGARTEN 1971: Based on
transmission electron microscopic studies,they proved the
existence of a hemidesmosomal basement lamina attachment
between the tooth and the cells of the so called cells of
epithelial attachment
43. Firmly attached to the tooth surface forming an epithelial barrier
against plaque bacteria
Allows access of gingival fluid, inflammatory cells, and components
of the immunologic host defense to the gingival margin
Exhibits rapid turnover contributing t the host-parasite
equilibrium and the rapid repair of damaged tissue
Have an endocytic capacity equal to that of macrophages and
nuetrophils and that this activity may be protective in nature
44. As an erupting tooth approaches the oral
epithelium, the enamel epithelium rapidly
proliferates forming the thick reduced enamel
epithelium.
As the crown erupts further, the reduced enamel
epithelium overlying the enamel fuses with the
oral epithelium, undergoes transformation and
establishes the dentogingival junction forming
the junctional epithelium cells
45. The junctional epithelium maintains a direct
attachment to the tooth surface.
During eruption, contact is established
between the reduced enamel epithelium and
oral gingival epithelium.
Epithelial cells of junctional epithelium
contact with the tooth surface by internal basal
lamina and separated from the connective
tissue by the external basal lamina
46.
47. DIFFRENCE BETWEEN ORAL,SULCULAR AND JUNCTIONAL EPITHELIUM
ORAL SULCULAR JUNCTIONAL
KERATINIZATION Keratinised Non keratinised Non keratinised
RETE PEGS Present Absent Absent
STRATUM
GRANULOMAAND
CORNEUM
Present Lacking Lacking
MERKEL CELLS Present Absent Absent
LANGERHANS
CELLS
Present Few Absent
TYPE IV COLLAGEN
IN BASAL LAMINA
Present Absent Absent
TIGHT JUNCTIONS More Few Few
ACID
PHOSPHATASE
ACTIVITY
Present Lacking Lacking
GLYCOLYTIC
ENZYME ACTIVITY
High Lower Lower
INTERCELLULAR
SPACE
Narrower Narrower Wider
48. Cells present namely are keratinocytes and non
keratinocytes
Keratinocytes:
90% of the total gingival cell population
Orginate from the ectodermal germlayer
It have nucleus,cytosol,ribosome and golgi
apparatus
It have melanosomes,which are the pigment
bearing granules present with in these cells only
and not in the other cells of the periodontium
49. The main function of gingival epithelium is to protect the deep
structures,this is achieved by proliferation and diffrentiation of the
keratinocytes
Proliferation :it takes place by mitosis in the basal layer
Diffrentiation:involves process of keratinization ,which consists of
progressions of biochemical and morphologic events that occur in basal
layer
The main morphologic changes are:
Progressive flattening of cells
Increase in tonofilaments
Intercellular junctions coupled to the production of keratohyalin
granules
Disappearence of nucleus
50. Expression and synthesis of keratin proteins in
the basal layer of cell,their chemical
compositon in the upper layer and their
interaction with keratohylin granules and
formatin of filamentous matrix structure in the
interior of corneocyte and strengtening of the
envelope.
Also known as CORNIFICATION
-The journal of western society of periodontology,1979
51. Three types of surface keratinization can occur
in the gingival epithelium
1.Orthokeratinization
2.Parakeratinization
3.Nonkeratinization
52. Complete keratinization of superficial horny
layer
No nuclei in stratum corneal layer
Well defined stratum granulosum
Few areas of outer gingival epithelium
53. Intermediate stage of keratinization
Most prevalent surface area of the gingival
epithelium
Stratum cornea retains pyknotic nuclei
Keratohyalin granules are disperesed rather
than giving rise to a stratum granulosum
54. • Viable nuclei in superficial layers
• Has neither granulosum nor cornea strata
• Layers of non keratinized epithelium
Stratum superficiale
Stratum intermedia
Stratum basale
55. The various nonkeratinocytes or clear cells are langerhans
cells,merkel cells and melanocytes
Melanocytes
Melanocytes are dendritic cells located in the basal and
spinous layers of the gingival epithelium . They synthesize
melanin in organelles called premelanosomes or
melanosomes . These contain tyrosinase, which
hydroxylates tyrosine to dihydroxy phenylalanine (dopa),
which in turn is progressively converted to melanin.
Melanin granules are phagocytosed and contained within
other cells of the epithelium and connective tissue, called
melanophages or melanophores.
56. Langerhans Cells
Langerhans cells are dendritic cells located among keratinocytes
at all suprabasal levels . They belong to the mononuclear phagocyte
system (reticuloendothelial system) as modified monocytes derived
from the bone marrow. They contain elongated granules and are
considered macrophages with possible antigenic properties.
Langerhans cells have an important role in the immune reaction as
antigen-presenting cells for lymphocytes. They contain g-specific
granules (Birbecks' granules) and have marked adenosine
triphosphatase activity. They are found in oral epithelium of normal
gingival and in smaller amounts in the sulcular epithelium; they are
probably absent from the junctional epithelium of normal gingiva. It
stains with: Gold chloride, ATPase, Immuno florescent markers .
57. Merkel Cell
Merkel cells are located in the deeper layers of
the epithelium, harbor nerve endings, and are
connected to adjacent cells by desmosomes. They
have been identified as tactile preceptors. Stained by
PAS
58. Boundary where epithelial tissue meets with connective tissue.
Deep extensions of an epithelium that reach down into connective
tissue are called epithelial ridges or retepegs
Finger like extensions of connective tissue that extend up into the
epithelium are called as connective tissue papillae
Epithelial connective tissue interface is composed of lamina lucida
and lamina densa
Lamina lucida is an electrolucent zone of 25-45nm width and
composed of glycoprotein laminin
Laminin densa is an electrodense zone of 40-60nm where type IV
collagen is present
59.
60. TIGHT JUNCTION/ZONAE OCCLUDENS: Formed by the fusion of external
leaflets of adjacent cell membranes
ADHESIVE JUNCTIONS:
1. Cell to cell:
zonula adherens
desmosomes:most common type of junction
2. Cell to matrix
focal adhesions
hemidesmosomes
COMMUNICATING (GAP) JUNCTIONS:
Intercellular pipes/channels bridge both adjacent membranes and intercellur space
Intercelluar space in gap junction is approx 3nm
Majority pathway for direct intercellular communication
61.
62. Also called as lamina propria
Composed of
1. Ground substance
2. Cells
3. Gingival fibres
Layers of connective tissue
1. Papillary layer
2. Reticular layer
63. Fills space between fibres and cells
Amorphous
High water content
Composed of:
Proteoglycans:
Decorin
Biglycan
Versican
Heparin sulfate proteoglycans and syndecans
CD44
Glycoprotein:
Fibronectin
Laminin
64. The different types of cells present in the
connective tissue are
o Fibroblasts
oPolymorphonuclear cells
oLymphocytes and Plasma cells
oMonocytes and macrophages
oMast cells
oOsteoblast and Osteoclasts
oCementoblasts and cementoclasts
65. Most abundant cells
They make 65% of the total cell volume of the gingival
connective tissue
Derived from undifferentiated progenitor mesenchmal
cells, that are present in the follicle
Elongated or spindle shaped cells
Have prominent golgi apparatus, rough endoplasmic
reticulum, mitochondria,vacoules and vesciles
It synthesize and secrete various cytokines, growth factors
and metabolic products
All these play a vital role in the development,
maintanence and repair of gingival connective tissue.
Responsible for the resorption of collagen fibres ,thus
playing important role in collagen homeostasis.
66. The connective tissue fibres are produced by the fibroblasts and can be divided into
Collagen fibres
Reticulin fibres
Oxytalan fibres
Elastic fibres
Collagen fibre types: Type I,III,IV,V,VI
Type I: It predominates,gives tensile strength
Type III:
1.fetal collagen
2.important in the early phases of wound healing.
3.partly responsible for the maintanence of space in healing matrix
Type IV: present in lamina densa layer of the basement membrane of the epithelium.
Type VI
1.distributed with the elastin fibres along the blood vessels.
2.impart rigidity required to maintain the elastic blood vessel wall from undergoing
permanent deformation.
3.acts as anchoring fibrils and helps to reinforce epithelial attachment to the
underlying connective tissue
67. To brace the marginal gingiva firmly against
tooth
To provide rigidity to necessary to withstand
the forces of mastication without being
deflected away from the tooth surface
To unite the free marginal gingiva with the
cementum of the root and the adjacent
attached gingiva
68. There is a specific of orientation and
organization of the collagen fibre bundles in
the gingival connective tissue, also referred to
as fiber apparatus of gingiva.
These are named according to their location,
origin and insertion
69. Name of fiber
group
Origin and orientation Supposed function
Principal groups
Dentogingival From cementum, splay laterally into lamina
propria
Provide gingival support
Alveologingival From periosteum of the alveolar crest, splay
coronally into lamina propria
Attach gingiva to bone
Dentoperiosteal From cementum near the cementoenamel
junction, into periosteum of the alveolar
crest
Anchor tooth to bone; protect
periodontal
ligament
Circular Within free marginal and attached gingiva
coronal
to alveolar crest, encircle each tooth (“purse
string)
Maintain contour and
position of free
marginal gingiva
Transeptal From interproximal cementum coronal to
alveolar
crest, course mesially and distally in
interdental
area into cementum of adjacent tooth
Maintain relationships of
adjacent
teeth; protect interproximal
bone
70. Secondary Groups
Periostogingival From periosteum of the lateral aspect of
alveolar
process, splay into attached gingiva
Attach gingiva to bone
Interpapillary Within interdental gingiva (gingival papilla),
orofacial course
Provide support for
interdental gingiva
Transgingival Within attached gingiva, intertwining along the
dental arch between and around the teeth
Secure alignment of teeth
in arch
Intercircular From cementum on distal surface of a tooth,
splaying buccally and lingually around adjacent
tooth and inserting on mesial cementum of next
tooth
Stabilize teeth in arch
Intergingival Within attached gingiva, immediately subjacent
to epithelial basement membrane, course
mesiodistally
Provide support and
contour of attached
gingiva
Semicircular From cementum on mesial surface of tooth,
course distally, insert on cementum of distal
surface of same
None intuitively obvious
71.
72.
73. The color of attached and marginal gingiva is generally described as “coral
pink”
Produced by the vascular supply ,the thickness,degree of keratinization of
epithelium, the presence of pigment cells
The attached gingiva is demarcated from the adjacent alveolar mucosa on
the buccal aspect by a clearly defined mucogingival line
DIFFRENCES BEWEEN ALVEOLAR MUCOSA AND ATTACHED GINGIVA
ALVEOLAR MUCOSA ATTACHED GINGIVA
COLOR Red Pink
SURFACE
TEXTURE
Smooth and shiny Shiny
EPTHELIUM Thinner non keratinised .Rete
pegs absent
Thicker parakeratinised .Rete pegs
present
CONNECTIVE
ISSUE
More loosely arranged.More
blood vessels
Not so loosely arranged.Moderate blood
vessels
74. The gingiva is considered the most frequently pigmented tissue in the oral cavity
Gingival pigmentation may be classified as Physiologic or Pathologic
Physiologic gingival pigmentation
All patients except albinos have some degree of physiologic melanin distribution throughout epidermis
Eumelanin is present in large amounts in individuals with dark skin and hair .
Pathologic gingival pigmentation
a) Endocrine diseases:Addison’s disease,Albright syndrome,Acromegaly,Nelson’s syndrome
b) Heavy metals:e.g.lead,bismuth,mercury,silver arsenic and gold
c) Kaposis sarcoma
d) Drug induced:
chloroquine,quinine,minocycline,zidovudine,chlorpromazine,ketoconazole,bleomycin,cyclophosphamide
e) Post inflammatory pigmentation
f) Smoking associated melanosis
g) Hemangioma
h) Amalgam tatoo
i) Graphite tatoo
j) Nevocellular nevus and blue nevus
k) Oral melanocanthoma
l) Mucosal melanoms
m) HIV oral melanosis
n) Haemchromatosis
o) Gingival tatoo
76. It depends on the
shape of the teeth
their alignment in the arch
Location and size of the area of proximal contact
Dimension of facial and lingual gingival embrasures
Marginal gingiva:Scalloped outline
Attached gingiva:festooned appearance with intermittent
prominence corresponding to contour of roots
Teeth placed
labially-the normal arcuate contour is accentuated and the
gingiva located further apically
lingually-gingiva is horizontal and thickened
77. Shape of intergingival papilla governed by
Contour of the proximal tooth surfaces
Location of gingival embrassure
Shape of gingival embrassure
Narrow inter
dental gingiva
Broad interdental
gingiva
Proximal surface Flat Flare
Roots Close together Wide
Interdental bone Thin mesiodistally Broad
78. Firm and resilient
The collagenous nature of the lamina
propria and its continuity with the
mucoperiosteum of the alveolar bone
determine the firmness of the attached
gingiva
The gingival fibres contribute to the
firmness of the gingival margin
79. Textured surface similar to an orange peel referred as
stippled
It is best viewed by drying the gingiva
Attached gingiva is stippled ;marginal gingiva not
The central portion of the interdental papillae is usually
stippled,but the marginal borders are smooth
Stippling is produced by alternate rounded
protuberances and depressions in the gingival surface
Stippling is a form of adaptive specialization or
reinforcement of function
80. Refers to the level at which the gingival margin is attached to the tooth
Tooth erupts into the oral cavity the margin and sulcus are at the tip of crown;
as eruption progresses they are seen closer to the root
Active eruption-movement of teeth in the direction of occlusal plane
Passive eruption-exposure of the tooth by apical migration of gingiva
Gottlieb and Orban: active and passive eruption go hand in hand
Active eruption is coordinated with attrition;the teeth erupt to compensate for
tooth substance worn away by attrition
Attrition reduces the clinical crown and prevents it from becoming
dispropotionately long in relation to the clinical root, thus avoiding excessive
leverage on periodontal tissue
Rate of active eruption keeps pace with tooth wear in order to preserve
vertical dimension
Exposure of teeth via the apical migration of the gingiva is called gingival
recession or atrophy
81.
82. Consists of an intricate web of arteries that originate from
the carotid artery
Buccal mucosa-tiny divisions of the buccal artery and
posterior superior alveolar artery
Palate-greater palatine artery, division of the descending
palatine artery originating from the maxillary artery
Floor of the mouth, mandibular lingual gingiva-
sublingual & submental arteries
Labial gingiva overlying the mandible –small divisions
from the inferior alveolar artery(incisive and mental artery)
Labial gingiva overlying maxilla-anterior superior
alveolar artery
83.
84. The innervation of the oral gingiva is unique
because all the nerves supplying the gingival
mucosa originate from the mandibular and
maxillary branches of the trigeminal nerve
Mandible
Labial part-buccal branch of mandibular nerve,and incisive
branch of mental nerve
Lingual part-lingual nerve
Maxilla
Labial part –posterior,middle,and anterior –superior alveolar
nerve
Palatal part-greater palatine and nasopalatine
85.
86.
87. AREA LYMPH NODE
Mandibular incisor area Submental lymph nodes
Maxillary palatal gingiva Deep cervical lymphnodes
Buccal gingiva of maxilla and
buccal &lingual gingiva in
mandibular premolar area
Submandibular lymphnode
88.
89. Clinical periodontology-10,11,12th edition Caranza
Clinical periodontology and Implant dentistry –Niklaus P.Lang,Jan Lindhe
Text book of periodontics –Shalu Bathla
A textbook of periodontics and implantology –Nitin Saroch
Journal of dentistry –Attached gingiva…Then and Now.Vol:4,Issue:1
Tissues and cells of periodontium Periodontology 2000, Vol. 3, 1993, 9-38
Histochemistry of gingiva :A review article International Journal of
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Development Of Periodontium: A Star In The Making!!! International
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Anatomy,Head and Neck,Oral gingiva-Adam Koller,Amit Sapra
Gingival Biotype Assessment in a Healthy Periodontium:Transgingival
Probing Method .Journal of Clinical and Diagnostic Research
Gingival Crevicular fluid: An overview.Journal of Pharmacy & Bioallied
Sciences
Gingival pigmentation(Cause,treatment and histological preview) Future
dental journal vol 3,2017