This document provides information about congenital talipes equinovarus, or clubfoot. It begins with definitions and descriptions of the deformities associated with clubfoot. It then discusses the epidemiology, causes, bony abnormalities, pathological anatomy, clinical features, classifications systems including Pirani and Dimeglio, treatment including serial casting and the Ponseti method as well as surgical options. Radiographic images are included to illustrate the deformities. The goal of treatment is to produce a plantigrade, supple foot that functions well, and the Ponseti method is now the standard non-operative treatment approach.
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http://www.davidsfeldmanmd.com/specialties/avascular-necrosis-hip
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Achilles tendon is the strongest tendon of body. There are many causes of its rupture. It can be acute or chronic rupture. Management of chronic rupture by semitendinosus tendon is mentioned here.
Avascular necrosis (AVN) or Aseptic Necrosis of the hip is caused by a disruption to the hip’s blood supply which results in the deterioration and often collapse of the ball of the thigh bone (femoral head). Early identification and treatment of the condition increases the likelihood that a patient’s hip will recover. Surgery may be required in severe cases to repair or revascularize (restore circulation) the hip or to replace the hip in neglected/end stage cases.
http://www.davidsfeldmanmd.com/specialties/avascular-necrosis-hip
Avascular necrosis (AVN) of the femoral head is a pathologic process that results from interruption of blood supply to the bone. AVN of the hip is poorly understood, but this process is the final common pathway of traumatic or nontraumatic factors that compromise the already precarious circulation of the femoral head. Femoral head ischemia results in the death of marrow and osteocytes and usually results in the collapse of the necrotic segment
Dr. Anisuddin Bhatti Paediatric Orthopaedic Surgeon DR. Ziauddin University Karachi presented talk on Congenital Vertical Talus at AKU karachi on August 2023 in Orthopaedic Review course. Acknowledged for some text material & photo taken from Published literature.
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1. Speaker: Dr. S. M. Sufi Shafi-Ul-Bashar
Assistant Registrar Surgery
MMCH
2. Another name:
Congenital Talipes Equinovarus
The term ‘talipes’ is derived from talus (Latin
= ankle bone) and pes (Latin = foot).
Equinovarus is one of several different
talipes deformities.
3.
4. What is CTEV??
• Idiopathic clubfoot
• Club foot Causing
CAVE
-Midfoot Cavus/ increase in height
-Forefoot Adductus
-Hindfoot Varus
-Hindfoot Equinus/ plantarflex
7. Causes-unknown:
• Germ defect.
• Arrested development.
• Neuromuscular disorder in neurological disorders
and neural tube defect.
• Postural deformity caused by tight packing
in an overcrowded uterus.
8. Bony abnormalities
• Talus:
Head & neck deviated medially &
plantarward
Body rotated externally in the
ankle mortise
Body extruded anteriorly
Smaller than normal
9. Bony abnormalities contd.
• Navicular:
Medially displaced
Close to medial malleolus
Articulates with medial
surface of head of talus
• Calcaneus
Anterior portion lies beneath
the head of talus causin
gvarus and equinus of heel
In equinus
Rotated medially
10. Bony abnormalities contd.
• Cuboid
Displaced medially
on the dysmorphic
distal end of the
calcaneus
• Talonavicular joint
In inversion
12. Pathological Anatomy
Neck of Tallus
-pointing downward
and deviates
medially
Body of Tallus
- Rotated outward
Posterior part of calcaneum
-held close to fibula by CF ligt
-tilted into equinus and varus
-rotated medially beneath
ankle
Navicular and forefoot
-shifted medially
-rotated into
supination
(composite varus
deformity)
13. Pathological Anatomy
• Skin and soft tissue of calf and medial side of
foot are short and underdeveloped.
• If not corrected early, secondary growth
changes occur in the bones-PERMANENT.
14. Clinical Features
• Heel is small and high.
• Deep creases appear posteriorly and medially.
• Abnormal thin calf.
15. • Varying degree of resistance / fixed deformity
when try to dorsiflex and evert the foot.
Normal baby foot
17. CLASSIFICATION
Two of the more commonly used classifications
by
1.Pirani et al.
and 2.Diméglio et al.
are based solely on physical examination requiring
no radiographic measurements or other special
studies.
18. Pirani Classification of Clubfoot Deformity
SCORE
PHYSICAL EXAMINATION
FINDINGS
0 0.5 1.0
Curvature of lateral border of
foot
Straight Mild distal
curve
Curve at
calcaneocuboid
joint
Severity of medial crease
(foot
held in maximal correction)
Multiple fine creases One or two
deep
creases
Deep creases
change
contour of arch
Severity of posterior crease
(foot
held in maximal correction)
Multiple fine creases One or two
deep
creases
Deep creases
change
contour of arch
Medial malleolar-navicular
interval(foot held in maximal
correction)
Definite depression felt Interval
reduced
Interval not
palpable
Palpation of lateral part of
headof talus (forefoot fully
abducted)
Navicular completely
“reduces”; lateral talar
head cannot be felt
Navicular
partially
“reduces”;
lateral head
less palpable
Navicular does
not
“reduce”;
lateral talar
head easily felt
19. Pirani Classification of Clubfoot Deformity
SCORE
PHYSICAL EXAMINATION
FINDINGS
0 0.5 1.0
Emptiness of heel (foot and
ankle
in maximal correction)
Tuberosity of
calcaneus
easily palpable
Tuberosity of
calcaneus
more difficult to
palpate
Tuberosity of
calcaneus not
palpable
Fibula-Achilles interval (hip
flexed,knee extended, foot and
ankle maximally corrected)
Definite
depression felt
Interval reduced Interval not
palpable
Rigidity of equines (knee
extended, ankle maximally
corrected)
Normal ankle
dorsiflexion
Ankle dorsiflexes
beyond neutral, but
not fully
Cannot dorsiflex
ankle
to neutral
Rigidity of adductus (forefoot is
fully abducted)
Forefoot can be
overcorrected into
abduction
Forefoot can be
corrected beyond
neutral, but not fully
Forefoot cannot
be corrected to
neutral
Long flexor contracture (foot
andankle held in maximal
correction)
MTP joints can be
dorsiflexed to 90
degrees
MTP joints can be
dorsiflexed beyond
neutral but not fully
MTP joints
cannot be
dorsiflexed to
neutral
21. a. b.&c.
Talipes equinovarus – x-rays The left foot is abnormal. In the
anteroposterior view (a) the talocalcaneal angle is 5degrees, compared
to 42 degrees on the right. In the lateral views, the left talocalcaneal
angle is 10 degrees in plantarflexion (b) and 15 degrees in dorsiflexion
(c).
22. d. e.
Talipes equinovarus – x-rays
In the normal foot the angle is unchanged at 44
degrees,whatever the position of the foot (d,e).
23. Treatment:
Aim is to produce and maintain a plantigrade,
supple foot that will function well.
After sucessful treatment foot should
Look good
Feel good
Move good
Measure good
24. Treatment contd.
Non Operative Operative
• Serial Manipulative and Casting
(Ponseti’s method)
• -Posteromedial tissue release and
tendon lengthening
• -medial opening or lateral column-
shortening osteotomy, or cuboidal
decancellation
• -triple arthrodesis
• -tallectomy
25. Goal-Rotate leg laterally around the fixed tallus
Serial Manipulative and Casting
(Ponseti method)
Increase the supination deformity of
forefoot
26. Serial Manipulative and Casting
(Ponseti method)
• Each cast - four basic steps
1 2 3 4
Manipulate and
hold
Apply padding
and hold
Apply cast and
mould
Complete above
knee
27. Ponseti method
Based on kinematic of the subtalar joint.
1st concept : the whole foot moves under the talus “calcaneo-
pedis block”
2nd concept : fore foot and hind foot are corrected
simultaneously by abduction
Equinus correction :
– mostly close tenotomy
28. Order of correction (CAVE)
-Midfoot cavus
-Forefoot adductus
-Hindfoot varus
-Hindfoot equinus
Ponseti method
33. Ponseti method
A B C D
Technique of Ponseti casting for clubfoot correction
A, First cast ; note positioning of forefoot to align with heel, with outer edge
of foot tilted even farther downward because of
Achilles tendon tightness.
B, Second cast; is applied with outeredge of foot still tilted downward and
forefoot moved slightly outward.
C, Third cast; Achilles tendon is stretched bringing outer edgeof foot into
more normal position as forefoot is turned farther outward.
D, Final cast; Achilles tendon is stretched more with footpointed upward.
35. Follow up protocol
• 2 weeks: to troubleshoot compliance issues
• 3 months: to graduate to the nights and naps protocol
• Every 4 months: until age 3 years to monitor compliance and
check for relapses
• Every 6 months: until age 4 years.
• Every 1 to 2 years: until skeletal maturity
36. Surgery in clubfoot
• Resistant clubfoot( non-responsive to serial casting and
manipulation)
• Persistently deformed clubfoot(non-operative correction
inadequately done with/without compliant bracing)
• Relapsed clubfoot( initially satisfactorily corrected that recurs
in part or whole)
• Neglected clubfoot( no treatment given till age of 2 yrs)
37. General Principles
• Goal: address all pathoantomic structures
• Decision regarding timing & extent
• Posteromedial-plantar-lateral release: all deformities present.
• Posterior release: straight lateral border, flexible forefoot and
hindfoot, and palpable gap between medial malleolus and
navicular tuberosity.
38. Follow up
Wound inspection done under sedation at 1 week.
Foot held in neutral, plantigrade position and cast
applied – above knee.
Cast kept for 4 – 6 weeks.
Cast removed along with any K wires, if applied
during surgery for stabilisation.
AFO(Ankle foot orthroses brace) given for 6
months.
39. Conclusion
• Proper understanding of the patho-anatomy is
a must.
• Ponseti method is now the standard
treatment method.
• Indications of surgery limited but well defined.