Detailed history and its evaluation , examination of spine in general and local with special tests in cervical , thoracic outlet syndrome , lumbar spine and SI joint with diagrams, neurological examination both sensory and motor.
Spinal stenosis is a degenrative spine disorder in which the AP and transverse diameter are decreased causing neural compression and symptoms of chronic & acute nerve compression
Back pain
Etiology
Anatomical & pathophysiological concepts
Diagnostic approach
Clinical approach
Red flags & yellow flags
Investigations
Back pain in children & elderly
Detailed history and its evaluation , examination of spine in general and local with special tests in cervical , thoracic outlet syndrome , lumbar spine and SI joint with diagrams, neurological examination both sensory and motor.
Spinal stenosis is a degenrative spine disorder in which the AP and transverse diameter are decreased causing neural compression and symptoms of chronic & acute nerve compression
Back pain
Etiology
Anatomical & pathophysiological concepts
Diagnostic approach
Clinical approach
Red flags & yellow flags
Investigations
Back pain in children & elderly
Entrapment Neuropathies in Upper Limb.pptxNeurologyKota
This presentation is about the entrapment syndrome of upper limb giving an insight regarding diagnosis clinically as well as electrophysiologically and
its management.
This is a short presentation on one of the most common entrapment neuropathy carpal tunnel syndrome. This presentation also provides information on its causes, epidemiology,diagnosis and management of carpal tunnel syndrome.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
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Carpal Tunnel Syndrome
1. CARPAL TUNNEL SYNDROME
DR G AVINASH RAO
FELLOW HAND AND MICROSURGERY
DEPARTMENT OF PLASTIC SURGERY, SKIMS ,
SRINAGAR
2. INTRODUCTION
Carpal tunnel syndrome– most common
compressive neuropathy of upper extremity.
Symptoms of median nerve compression–
were first described (distal radius fractures) by
Sir James Paget in 1854.
The term carpal tunnel--- coined—Mersch
8 decades later.
3. Anatomy
Carpal tunnel is bordered dorsally by concave
arch of carpus & volarly by transverse carpal
ligament (TCL).
10 structures pass--- 9 flexor tendons &
median nerve.
Median nerve is most superficial structure,
entering just radial to midline.
4.
5.
6. Recurrent motor branch.
It usually originates in extraligamentous
position distal to TCL.
Extraligamentous (46%)
Subligamentous(31%)
Transligamentous (23%)
7. The PALMAR CUTANEOUS BRANCH OF THE MEDIAN
NERVE lies between the FCR and PL tendons in the distal
forearm, but its branches may be found upto 6mm ulnar
to the thenar crease in the palm.
8. ETIOLOGY
Idiopathic
Women > men (2-3:1)
Age – 30-60 (MC)
Use of vibrating hand tools, smokers,
High body mass index (BMI) – obesity.
Wrist ratio – If the anterior to posterior distance is ≥70% of the
medial to lateral distance, there is a significant association with
idiopathic CTS.
Pregnant women – Symptoms relieve after Delivery,
Children – Rare (macrodactyly, lysosomal storage disorders, strong
family history – predisposes).
9. Trauma caused by repetitive hand motions
especially in works requiring repeatetive
forceful finger and wrist flexion and extension.
Habitual sleeping posture in which the wrist is
kept acutely flexed.
10. FACTORS INVOLVED IN PATHOGENESIS
OF CTS. Kerwin G, Williams CS, Seiler JG: The pathophysiology of
carpal tunnel syndrome, Hand Clin 12:243, 1996.
11. Pathogenesis
Median nerve is susceptible to compression
within carpal canal because of unyielding fibro
osseous borders.
Normal pressure--- 2.5mm Hg.
Elevation of carpal tunnel pressures impedes
epineurial blood flow, and nerve function is
impaired
A decrease in epineural blood flow &
edematous changes occur--- pressure reaches
20-30 mm Hg.
At > 30 mm Hg, nerve conduction diminishes,
12. Diagnosis of CTS
History & physical examination are the key.
CTS is primarily a clinical Diagnosis.
Pain (nocturnal > day) , deep aching/throbbing
Numbness , tingling and Paresthesia in typical
median nerve distribution.
Daytime paresthesias– elicited with activities
involving prolonged wrist flexion/ extension.
Shaking & Exercises--- sometimes REDUCE
symptoms.
B/L CTS - Check opposite hand for early
diagnosis
Atypical presentation– paresthesias in radial digits
13. CHRONIC CTS
Chronic median nerve compression--- gritty or
numb sensation in fingers.
grip & pinch weakness, &
diminished finger dexterity with H/o dropping
objects (Prominent Thenar wasting).
RSD/ CRPS.
14. Clinical Evaluation
Thorough physical examination--- including
cervical spine & entire upper extremities.
(Double crush phenomenon)
Soft tissues are assessed for skin & muscle
atrophy.
Cold intolerance, dryness & unusual textures
in radial digits signify disruption of sympathetic
fibres carried by median nerve.
16. Tinel’s sign
The examiner taps directly over the carpal
tunnel with his or her long and index
fingers.
A positive test consists of paresthesia or pain
in a median nerve distribution.
17. Phalen’s test
The patient’s wrist is held in a flexed position
for upto a minute or until onset of symptoms.
A positive test consists of the onset of
numbness or paraesthesia in the median
nerve distribution.
18.
19. Carpal tunnel compression test
/ Durcan’s Test
The examiner applies direct pressure to the carpal
tunnel with his or her thumb for upto 1minute or until
onset of symptoms. A positive test consists of the onset
of numbness or paresthesia in the median nerve
distribution.
More specific (90%) and more sensitive (87%) than either
the Tinel or Phalen test.
21. Semmes-Weinstein
monofilaments
Monofilament evaluator size was started from
2.83 to 6.65.
2.83 – Green – Normal
3.61 – Blue – Diminished light touch
4.32 – Purple – Diminished protective
sensation
4.56 – Red – Loss of protective
sensation
23. Two-point discrimination
STATIC
• Determine minimal separation of two distinct points
when applied to palmar fingertip
• Innervation density of slow-adapting fibers
• Failure to determine separation of at least 5 mm
DYNAMIC
• As above, with movement of the points
• Innervation density of fast-adapting fibers
• Failure to determine separation at least 4 mm
BOTH INDICATE ADVANCED NERVE
24. Katz & Stirrat hand diagram
Ryan P. Calfee, MD, Ann Marie Dale, PhD, Daniel Ryan, MS, Alexis Descatha, MD,
Alfred Franzblau, MD, Bradley Evanoff, MD
25.
26. The MOST SENSITIVE TESTS - Durkan nerve
compression, the hand diagram score, night
pain, and Semmes-Weinstein testing after a
Phalen test.
The MOST SPECIFIC TESTS were the hand
diagram and Tinel sign.
30. Szabo et al determined a probability of 0.86 in
correctly diagnosing CTS in presence of
positive median nerve compression test,
positive hand diagram, night pain, & abnormal
Semmes-Weinstein monofilament testing.
31. NCS & EMG
It tells weather pt has the disease
It is useful to judge severity of disease
Prognostic value after treatment
If NCS shows axonal loss then surgery has to be
advised.
Diagnose incomplete release or Iatrogenic Nerve
injury
Double crush syndrome (cervical myelopathy /
pronator syndrome)
Medicolegal issues
If pt has obvious thenar muscle wasting.
32. NCS &EMG
According to the American Association of Electrodiagnostic
Medicine recommendations:
Median nerve distal sensory latency, upper limit of
normal – 3.6 ms
Difference between the median and ulnar nerve distal
sensory latencies, upper limit of normal – 0.4 ms
Distal motor latency over the thenar, upper limit of
normal – 4.3 ms
Median motor nerve conduction velocity – lower limit of
normal – 49 m/s
Median sensory nerve conduction velocity – lower limit
– 49 m/s
33. SONOGRAPHY
Non invasive
Presence of median nerve edema
Measurements of cross-sectional area of median nerve at carpal tunnel
inlet proximally and outlet distally were taken
The shape, size, echogenecity and relationship of median nerve to
overlying retinaculum
Amount of synovial fluid and any presence of masses (cysts /lumps)
Anatomy of median nerve and continuity
Considered in reccurent cases (not routinely done)
35. The cut-off value for pathological cross-
sectional area of median nerve is 9.4 mm
square.
Mild – 9.4 to 11.3
Moderate –11.3 to 13.5
Severe – 13.5 and above
36. OTHER INVETIGATIONS
Imaging studies
Three-view radiographs of the wrist
(posteroanterior, lateral, oblique) plus carpal tunnel
view: obtained when there is antecedent wrist trauma.
MRI (diffusion tensor imaging) - not routinely used for
diagnosis. A major advantage of MRI is its high soft-
tissue contrast, which gives detailed images of bones
and soft tissues.
Serologic studies
No blood tests specifically support diagnosis of CTS
Diabetes & Hypothyroidism are common diseases---
FBS & Thyroid function test.
37. AAOS GUIDELINES – for Non Sx
Rx
Considered in early CTS.
Trial of conservative Rx for 2-7weeks if it fails,
attempt one more time.
Local Sterid and splint recommended prior to
surgery
Neurotonics – Placebo affect
Massage / acupunture /any other conservative
options not recommended
38. Kaplan, Glickel, and Eaton - five important factors
in determining the success of nonoperative
treatment:
(1) age older than 50 years,
(2) duration longer than 10 months,
(3) constant paresthesia,
(4) stenosing flexor tenosynovitis,
(5) a positive Phalen test result in less than 30
seconds.
0 - Two thirds of patients were cured by medical treatment
1 - 59.6% were cured.
2 - 83.3% when two factors were noted
3 - 93.2% did not experience any improvement
No patient with four or five factors was cured by medical
management.
39. Treatment – Conservative
options
Nerve Gliding Exercises.
Local Triamcinolone (steroid) Injection
Night Splints.
Strict control of medical illness
Treatment of double crush syndrome
contraversial
Oral medications
Diuretics, NSAIDs, oral corticosteroids & vitamin B6.--- thought to decrease
interstitial fluid pressure within carpal canal.
40. Celiker et al compared the effectiveness of
NSAIDs & splinting with corticosteriods
injections in treating CTS.
Found that both methods of treatment led to
statistically significant improvement in
symptoms at 2 months.
41. Aufiero et al cited several studies supporting &
disproving the efficacy of vit B6.
Only 2 studies were randomized & blinded in
design--- no improvement.
42. Corticosteroid injection
Gelberman et al found single injection
improved CTS symptoms in 76% of pts after 6
weeks.
However only 22 % remained symptom free at
1 year.
Effective in mild CTS symptoms,< 1
year,normal sensibilty testing & only minor
electrodiagnostic study abnormalities.
43. Transient elevation in blood glucose can be
anticipated--- thus a less soluble corticosteroid
preparation (triamcinolone).
Diabetic pts should be instructed to monitor
their serum glucose.
No absolute contradiction to injection during
third trimester of uncomplicated pregnancy or
healthy breast feeding woman.
44. Immobilization of wrist at night & intermittently
during the day.
Pressure in carpal tunnel is lowest with wrist in
2* -/+ 9* of extension & 2* +/- 6* of ulnar
deviation.
Splinting
45. Ultrasound Therapy
In randomized study ultrasound improved
symptoms at 2 weeks, 7 weeks, 6 months.
However another study demonstrated no
appreciable benefit at 2 weeks from this form
of treatment.
47. Exercises
Nerve & tendon gliding exercises enhance
venous blood flow & decrease pressure within
carpal tunnel.
Rozmaryn et al evaluated 240 pts with CTS,
half of whom were instructed to perform nerve
& tendon gliding exercises.
Patients who did not do these exercises, 71
% eventually underwent carpal tunnel release
surgery, in other group 43% had surgery.
48. AAOS GUIDELINES- for Sx
Rx
Regardless of technique used – complete
release of Flexor Retinaculum is
recommended
Epineurotomy , Neurolysis, Tenosynovectomy
are not recommended in all cases
Wrist immobilization is not required
49. Surgical treatment - Options
OPEN CTS RELEASE
MINI OPEN CTS RELEASE
ENDOSCOPIC CTS RELEASE
- SINGLE PORTAL (AGEE)
- TWO PORTAL (CHOW)
51. OPEN CTS RELEASE
The palmar incisions should be well ulnar.
A curved incision ulnar and parallel to the thenar
crease but the palmar cutaneous branch of the
median nerve proximally may be more at risk of
injury.
Maintain longitudinal orientation so that the incision is
generally to the radial border of the ring - fourth ray.
Incise and reflect the skin and subcutaneous tissue.
Identify the palmar fascia from the wrist flexion crease
distally.
52. And the distal forearm antebrachial fascia proximally by
subcutaneous blunt dissection.
Split the palmar fascia and expose the underlying transverse
carpal ligament and carefully divide it and avoid damage to the
median nerve and its recurrent branch
Fibers of the transverse carpal ligament can extend distally
farther than expected. The flexor retinaculum includes the distal
deep fascia of the forearm proximally, the transverse carpal
ligament, and the aponeurosis between the thenar and
hypothenar muscles.
A successful carpal tunnel release usually requires division of all
54. Mark surgical incision with a skin pen
The longitudinal incision - just distal to the distal wrist
flexion crease and slightly ulnar to the midline of the
wrist.
Extend Distally - 2.0 to 3.0 cm in line with the third
web space.
Expose the transverse carpal ligament - by splitting
the parallel palmar fascia fibers and retract
hypothenar fat ulnarly.
Intrinsic muscles obscure the midline of the TCL and
can be released from their origin and reflected away
55. Carefully open the carpel tunnel by division of
the TCL with a no15 blade. Ensure complete
release.
The TCL divided in such a way that 3 to 4 mm
of it is left attached to the hamate hook to
avoid flexor tendon ulnar subluxation.
Make sure the contents of the carpal tunnel
are not adherent to the undersurface of the
TCL.
Close the incision in routine fashion and apply
a compressive dressing.
56. Endoscopic carpal tunnel
release
Problems related to endoscopic carpal tunnel
release include
(1) a technically demanding procedure;
(2) a limited visual field that prevents inspection
of other structures;
(3) The vulnerability of the median nerve, flexor
tendons, and superficial palmar arterial arch;
(4) The inability to control bleeding easily;
(5) The limitations imposed by mechanical
failure.
57. Agee, McCarroll, and North - 10 guidelines for the
single-incision ECTR
1. Know the anatomy.
2. Never over commit to the procedure.
3. Ascertain that the equipment is working properly.
4. If scope insertion is obstructed, abort the procedure.
5. Ascertain that the blade assembly is in the carpal
tunnel and not in the Guyon canal.
6. If a clear view cannot be obtained, abort the
procedure.
7. Do not explore the carpal canal with the scope
8. If the view is not normal, abort the procedure.
9. Stay in line with the ring finger.
10. When in doubt, get out.
58. CONTRAINDICATIONS FOR ECTR (DESCRIBED
BY CHOW)
(1) The patient requires neurolysis,
tenosynovectomy, Z-plasty of the transverse
carpal ligament, or decompression of the
Guyon canal.
(2) The surgeon suspects a space-occupying
lesion or other severe abnormality of the
muscles, tendons, or vessels in the carpal
tunnel.
(3) The patient has localized infection or severe
hand edema, or the vascular status of the
upper extremities is tenuous.
59. Fischer and Hastings further added
(1) Revision surgery for unresolved or recurrent
carpal tunnel syndrome
(2) Anatomic variation in the median nerve.
(3) Previous tendon surgery or flexor injury that
would cause scarring in the carpal tunnel.
(4) Limitation of wrist extension is (endoscopic
instruments cannot be introduced into the
carpal tunnel).
AS CONTRAINDICATIONS FOR ECTR.
64. Open vs endoscopic
Open vs endoscopic
Endoscopic– shorten recovery time
However no substantial differences in final
outcome.
One study in 25 pts, one hand open & another
endoscopic was done 3 months later, no
significant differences
With ECTR - Less palmar scarring and ulnar
“pillar” pain, rapid and complete return of
strength, and return to work and activities at
least 2 weeks earlier.
65. Post-OP Rehabilitation
Wrist immobilisation after carpal tunnel
surgery--- no benefit in pain relief or surgical
outcome.
Active motion exercises of wrist & fingers are
to be encouraged.
67. ECTR COMPLICATIONS
Intraoperative injury to flexor tendons.
Injury to median, ulnar, and digital nerves.
Injury to superficial palmar arterial arch.
ECTR -Technically demanding procedure
68. Young pt with symptoms of CTS and decreased
grip stength consider – CTS release + Flexor
Tenosynovectomy.
If symptoms persists in previously operated case
(tingling and Paresthesia with Tinels positive) –
Consider sonography - incomplete release /
Neuroma in continuity formation 20 to iatrogenic
injury
Elderly with chronic CTS and thenar wasting and
inability to do finer activities – consider CTS with
Tendon Transfer for thumb opposition
(opponensplasty)
Considerations before Sx
69. UNCOMMON – REQUIRE
ATTENTION
BE AWARE OF POTENTIAL ANOMALIES:
connections between the FPL and the index
FDP tendon;
Anomalous FDS;PL, hypothenar, lumbrical
muscle bellies;
Median and Ulnar nerve branches and
interconnections.
RARE – BIFID MEDIAN NERVE
70. Summary
CTS– common problem.
Several risk factor are associated.
Thorough history & physical examination is the
key.
Non-surgical & surgical techniques are
beneficial.
Both open & endoscopic have same results.
Look for S/O CRPS (RSD) in Chronic cases,
Before surgery.