Canine Rickets
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Rickets is a disease affecting young growing animals characterized by defective calcification of bones leading to deformity of the skeleton. It is caused by factors disturbing the metabolism of calcium, phosphorus, and vitamin D. Clinical signs include bowed legs, enlarged joints, stiffness, and susceptibility to bone fractures. Diagnosis can be confirmed with radiological examination showing a lack of bone density and abnormal bone structure. Treatment involves dietary supplementation with calcium, vitamin D, and phosphorus as well as exposure to sunlight.
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5. Conditions affecting bone mineralization, mineral density, and viability such as osteoporosis, rickets, and fractures.
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This document discusses a case of a 33-year-old Asian lady presenting with 3 months of generalized bony pain. Laboratory investigations found elevated alkaline phosphatase and low calcium and phosphate levels. The document then reviews osteomalacia, discussing the pathology, signs and symptoms, diagnosis, comparisons to other conditions, and x-ray findings like Looser zones and codfish vertebrae. Osteomalacia results from defective bone mineralization due to vitamin D deficiency or phosphate deficiency.
CALCIUM AND ITS CLINICAL IIMPORTANCE
The document discusses calcium's biochemical functions including roles in bone and teeth formation, muscle contraction, blood coagulation, nerve transmission, and enzyme activation. It describes calcium requirements, sources, absorption factors, homeostatic regulation by parathyroid hormone, calcitonin, and vitamin D, and excretion. It also examines diseases like hypercalcemia, hypocalcemia, rickets, osteoporosis, and osteopetrosis.
Calcium metabolism
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RICKETS.pptx
Rickets is a disease of growing bones in children caused by vitamin D deficiency or lack of calcium and phosphate. It results in soft, weak bones that can deform. Symptoms include bowed legs, fractures, and delayed growth. Diagnosis involves lab tests showing low calcium, vitamin D, and phosphate levels as well as characteristic bone changes on x-rays like widened growth plates. Treatment is vitamin D supplements and ensuring adequate calcium and phosphate intake. Prevention involves vitamin D supplementation, sunlight exposure, and a nutritious diet.
Minerals and trace elements.ppt
The document discusses several important minerals and trace elements required by the human body. It covers macro minerals like calcium, phosphorus, sodium, and potassium which are needed in larger amounts. It also discusses trace minerals or micro minerals like iron, zinc, iodine and copper which are needed in smaller quantities. For each mineral, it provides information on sources, recommended dietary allowances, functions in the body, deficiency and toxicity risks. It emphasizes the role of minerals in bone health, energy production, cell function, blood pressure regulation and other vital processes.
Nutri report
This document discusses calcium and phosphorus metabolism and disorders related to their imbalance. It notes that hypercalcemia and hypercalciuria can lead to kidney stones. Phosphorus is the second most abundant mineral in the body after calcium, and its levels are regulated by hormones like vitamin D, calcitonin, and parathyroid hormone. An imbalance can result in disorders like osteomalacia and osteoporosis. Osteomalacia is caused by vitamin D or phosphorus deficiency and results in softening of bones. Osteoporosis is caused by calcium or estrogen deficiencies and results in reduced bone density and mass. Risk factors, signs, treatments, and prevention strategies are described.
Bones (applied physiology)
This document discusses three bone diseases: osteoporosis, rickets, and osteomalacia. It provides details on the causes, risk factors, symptoms, diagnosis, and treatment of osteoporosis. It explains that osteoporosis is a disease where loss of bone density leads to fragile bones that fracture easily. Rickets causes weak, soft bones in children due to vitamin D deficiency and inadequate mineralization. Osteomalacia is the same disease as rickets but occurs in adults.
RICKETS.pptx
This document provides an overview of rickets, including:
- It is a disorder of defective bone mineralization caused by vitamin D deficiency that disturbs calcium and phosphorus homeostasis.
- Those at risk include children with dark skin and those exclusively breastfed without vitamin D supplementation.
- Vitamin D is synthesized in the body from sun exposure and obtained through diet. A deficiency can be caused by inadequate intake or absorption.
- Symptoms include bone deformities, muscle weakness, and fractures. Diagnosis involves blood tests showing low vitamin D and elevated alkaline phosphatase. Treatment is vitamin D supplementation.
CM & R.pptx
This document discusses calcium metabolism and rickets. It begins by introducing calcium's role in the body and dietary requirements. It then covers absorption, regulation by parathyroid hormone and calcitonin, and roles in bone mineralization. Rickets is defined as a childhood disease caused by calcium and vitamin D deficiencies, leading to softened, deformed bones. Causes, clinical features, investigations, management, and surgical options are described.
Metabolic bone diseases for students
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Presented by Usama Ragab Youssif
Internal Medicine Lectures for 5th-year medical students
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Bone metabolism by Dr. binod Chaudhary.pptx
Normal bone metabolism, calcium metabolism, phosphate metabolism, calcium balance, phosphate balance, bone markers
Vitamin D 1.ppt
Vitamin D is a fat-soluble vitamin obtained through exposure to sunlight, food sources like fatty fish and supplements. It plays an important role in calcium absorption and bone health. Deficiency can lead to rickets in children and osteomalacia in adults characterized by soft, weak bones and skeletal deformities. Recommended daily intake amounts vary from 200-600IU depending on age. Both deficiency and excess can impact multiple body systems.
Calcium metabolism disorders
Calcium is essential for bone health, muscle function, and other bodily processes. Its levels are tightly regulated by parathyroid hormone (PTH), vitamin D, and calcitonin. Disorders can result from too little or too much calcium and include rickets, osteomalacia, osteoporosis, hypercalcemia, and hypocalcemia. Hyperparathyroidism is a common cause of hypercalcemia due to excessive PTH levels and can result from primary or secondary hyperfunction of the parathyroid glands. A case example is presented of a woman with chronic fatigue and mood changes found to have hypercalcemia due to long term thiazide use increasing calcium resorption.
metabolicbonedisease-150401111940-conversion-gate01-converted.pptx
This document summarizes several metabolic bone diseases. It begins by describing bone composition and cells. It then discusses calcium, phosphate, parathyroid hormone, vitamin D, calcitonin, and alkaline phosphatase metabolism. Several metabolic bone diseases are covered in detail, including rickets/osteomalacia, osteoporosis, Paget's disease, and renal osteodystrophy. For each disease, causes, clinical presentations, investigations, and management are summarized.
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- 2. MUDASIR BASHIR Div. Of vety surgery & Radiology I.V.R.I izatnagar…………………….
- 4. Structure of bone
- 12. CALCIUM HOMEOSTASIS DIETARY CALCIUM INTESTINAL ABSORPTION ORGAN PHYSIOLOGY ENDOCRINE PHYSIOLOGY dietary habits, supplements BLOOD CALCIUM BONE KIDNEYS URINE THE ONLY “IN” THE PRINCIPLE “OUT” ORGAN PHYS. ENDOCRINE PHYS. ORGAN, ENDOCRINE
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