This document provides an outline and overview of clubfoot (congenital talipes equinovarus) including its causes, types, assessment using the Pirani scoring system, and management using the Ponseti method. It begins with definitions of clubfoot and describes the components of the deformity. It then discusses the causes, epidemiology, classification based on etiology and treatment stage, and clinical assessment. Major sections cover the Ponseti treatment method including manipulation, serial casting, tenotomy, and bracing. It concludes with details on recurrence prevention and management.
Dr. Anisuddin Bhatti Paediatric Orthopaedic Surgeon DR. Ziauddin University Karachi presented talk on Congenital Vertical Talus at AKU karachi on August 2023 in Orthopaedic Review course. Acknowledged for some text material & photo taken from Published literature.
Dr. Anisuddin Bhatti Paediatric Orthopaedic Surgeon DR. Ziauddin University Karachi presented talk on Congenital Vertical Talus at AKU karachi on August 2023 in Orthopaedic Review course. Acknowledged for some text material & photo taken from Published literature.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
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3. What is clubfoot?
ØIt is also known as its Latin name,
Congenital Talipes Equinovarus. It is an
easily recognisable deformity due to the
abnormal shape and position of the feet.
ØRigid congenital deformity of the foot
ØCongenital Talipes Equinovarus (CTEV)
.
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4. Types
A. Based on Eithology
1. Idiopathics
Ø Most common type
Ø No associated with other congenital conditions
2.Postural
Ø the deformity is very flexible and is thought to be due to
intrauterine crowding
Ø The deformity can be corrected easily by the examiner
Ø Not considered as a real club foot
Ø It usually improves by teaching parents some stretching and by
itself with time
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5. Con…
3. Syndromic
Ø Some congenital conditions are associated with club
foot deformity e.g. arthrogrogryposis ,diastrophic
dwarfism
4.Neuromascular conditions
§ Myelomengiocle
§ Cerebral palsy
ü Equinovarus deformity due to muscle spasticity
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6. Con…
B. Based on treatment stage
1.Untreated
2.Treated
3.Resistant
4.Recurrent
5.Neglected
6.Complex club foot
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7. Con…
• Untreated :affected child is under 2 years of age
and had no or very little treatment
• Treated: affected Childs feet have corrected with
ponseti method and they have completed the
casting phase
• Resistant :child has previously untreated clubfoot
and that does not correct with ponseti method this
usually syndromic and surgery may be necessary
• Recurrent :children who show signs of deformity
in previously treated club foot
oUsually treated through casting and surgery
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8. Con…
• Neglected :child older than two years who had little or no
treatment usually severe soft tissue contracture and bony
deformities
o Ponseti treatment has some success but may requires
surgery
• Complex :club foot treated by other than ponseti
technique
complicated by additional pathology or scaring
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9. Clubfoot cause(Etiology)
Ø Most often idiopathic
Ø Muscular , neurological disorders
and connective tissue theories have
been proposed
Ø Hereditary influence
Photo credit: CURE International,
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10. • Average 1-2 cases/1000
• Globally
– 200,000 new cases/year
– 80% in low and middle
income countries
• More common in boys
• 50% of the cases bilateral
• 3 % occurrence in siblings
Incidence
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12. The midfoot - CAVE
• Cavus – 1st ray is plantarflexed in relation to
the hind foot
• High arch foot
• Adductus – the forefoot is adducted in
relation to the hind foot
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13. The hind foot deformity - CAVE
• Varus – The heel is inverted in relation to the tibia
• Equinus – The entire foot points downward in
relation to the tibia
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14. Why do we assess?
vTo diagnose
vShows the severity of the Clubfoot
vHelps to monitor treatment progress
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15. Con….
vHow do we assess club foot?
• Observation for signs
• History taking
• Examination (pirani score)
Ø By making the environment comfortable
Eg. Position the child at mother’s lap
Ø Undress the child
Ø Playing
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16. Overview of the Ponseti treatment - Basic
Ø Manipulation
Ø Casts after Manipulations
Ø Tenotomy
Ø The Bracing
16
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17. Overview of the Ponseti treatment - Basic
Manipulation + Cast (5-7 days)- 4 times or more
–(6-7 times max )
Correction of the Cavus, Mid foot Inversion,
and Heel Varus 17
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18. Tenotomy of the Achilles tendon + Cast (3 weeks)
Correction of the Rigid Equinus
18
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19. Bracing
a)full-time = 23 hours (3
months)
b) Sleeping time = 14-16 hours
(2-4 years)
19
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20. Manipulation
Ø The technique of the manipulation is based on the
understanding of the described coupling of movements
Ø To correct the hindfoot we need to manipulate the
mid foot.
Ø The initiator for the correction of the whole foot is
the abduction of the mid foot.
Ø The manipulation can be done on the mother’s lap
and should not cause pain for the child
20
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21. Manipulation technique
1. Stabilize the Talus by placing the thumb over the
head of the Talus .This provides a pivot point around
which the foot is abducted.
2. Manipulate the foot by abducting the foot in
supination with the other hand without any discomfort
to the child.
3. Hold the correction with gentle pressure for a few
seconds, then release, and repeat one or two more
times.
21
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24. CASTS AFTER MANIPULATIONS
Ø The cast is applied after the manipulation and
immobilizes the foot in order to stretch the tight
ligaments.
Ø Always use long leg casts in 90° knee flexion!
Ø Avoid external rotation of the knee!
Ø Always mould well at the heel, Malleoli, and sole of
the foot.
Ø Never cast the foot in a pronated position!
Ø Never hold the heel.
Ø Never attempt to correct the equinus before
achieving the full abduction (aim for 70°).
24
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25. First cast:
Correction of the Cavus:
1. Stabilize the Talus by placing the thumb over the
lateral part of the head of the Talus.
2. Elevate the first metatarsal and achieve a
homogeny supination of the forefoot in line with the
mid foot.
3. Put padding and plaster on by holding this
position and molding well.
– In small babies, correction usually occurs with the
first cast.
– A severe cavus in a stiff foot will need 2 or 3 cast
changes for correction. 25
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26. First cast: Correction of the Cavus
Stabilize the Talus
Elevate the first ray
Holding this position
26
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27. Following casts
Correction of Mid foot Inversion and Heel Varus
v Each cast aim for more abduction of the supinated foot -
70° at the end.
1. Stabilize the Talus by placing the thumb over the lateral
part of the head of the Talus.
2. Hold the supinated foot in abduction while applying the
cast. The correction of the Calcaneus will be achieved
because of coupling.
3. Apply padding and plaster by holding the corrected
position and molding well.
27
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29. A
B
Note the correction of the adductus [A] and heel varus [B] occurs
when the foot is everted and abducted around the head of the talus
29
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31. Removal of casting
• Should be done in the clinic just before the next step
of the treatment
1. Soak the cast in water
2. Remove the cast: above the knee then below
31
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32. TENOTOMY
Ø Percutaneous tenotomy of the achilles
tendon
Ø It corrects the rigid ankle foot equinus./
Ø Equinus when cavus, adductus, and varus
are fully corrected but ankle dorsiflexion
remains less than 10 degrees above neutral.
Ø Make certain that abduction is adequate for
performing the tenotomy.
32
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33. • Cont.
• The Pirani Severity Scoring Method indicates when
sufficient correction has been obtained:
– Mid foot Contracture Score under 1
• Hind foot Contracture Score is about1
• LHT = 0 (70° abduction, older children maybe
less)
• Never perform a tenotomy, if the heel is in varus,
because not enough correction has been achieved.
33
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35. Casting after tenotomy
• A cast will be applied right after the tenotomy had
been done and has to stay on for 3 weeks: 70°
abduction, 10- 15° dorsiflexion.
35
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36. THE BRACING
Ø The bracing is a very important and a
critical part of the treatment
Ø Failure to use the brace in the correct
way and for the required time is the
most common cause of recurrence!
Ø The brace needs to be put on
immediately after the last casts are
removed.
36
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37. Bracing protocol
• Bracing time for small children (who haven't walked before
the treatment is started):
– For first 3 months: full-time (except bathing = 23 hrs a
day)
– For 4 years : sleeping time (night and during day sleeps)
• Bracing time for older children of walking age (about 2-4
years):
– 18 hours a day for 3 months (6 hours out of the brace
during the day time for playing, moving, walking,
bathing, etc.)
– Sleeping time (= 14-16 hours a day) until the age of 5
37
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38. Common Bracing Type
The Abduction Brace (Steenbeek
Type)
• The brace needs to have both
shoes attached to a bar in order to
prevent a recurrence
• Children should not stand or walk
in this brace. The brace is not
designed for this purpose.
• Full-time bracing (for small
children) or 18 hours bracing (for
older children) is only for 3
months.
• After that time, the children must
wear the brace at sleep times.
38
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39. Bracing Adjustment
• Bilateral clubfoot:
– Both feet in 70° abduction
• Unilateral clubfoot:
– Clubfoot in 70° abduction
– Normal foot in about 45°
abduction.
• Overcorrected clubfoot (e.g.
hypermobile, low muscle tone
child or atypical clubfoot) and
corrected clubfoot that develop
excessive heel valgus and
external tibial torsion:
– Both feet in about
45°abduction 39
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40. Recurrence
Reasons for recurrences
• Failure to wear the brace
– Failure to wear the brace: more than 80% of
recurrences.
– Proper wearing the brace: only about 6% of
recurrences.
• Muscle imbalance and ligament stiffness
– The Gastrosoleus tendon unit doesn’t grow as fast
as the Tibia which causes (>) :?????
• Failure in the treatment
– Not enough abduction or dorsiflexion have been
achieved.
40
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41. Prevention of Recurrences
Ø Strict bracing according to the bracing protocol.
Ø Stretching of the Gastrocnemius muscle (daily
exercise done by the parents).
Ø Squatting with heels on the floor: stretches Tibialis
posterior muscle.
41
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42. The Pirani score
• 6 components
– Severity score of 0,
0.5 or 1
• Measure and record
deformity
• Valid and reliable
• Good intra/inter-
observer reliability
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46. Pirani scoring – Hindfoot contracture score
(HFCS)
Posterior Crease
(PC)
0, 0.5,
1
Empty Heel (EH) 0, 0.5,
1
Rigid Equinus (RE) 0, 0.5,
1
HFCS 0-3
§ Observe, look,
f e e l a n d
measure
§ Score with the
f o o t g e n t l y
corrected
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66. Definition
Ø Torticollis literally means “twisted neck or “wryneck”
and is derived from the Latin words “torquere”
(twisted) and “collum” (neck) (Stellwagen 2004, cited
by Burch C. etal).
Ø The generic term “torticollis” is not a specific
diagnosis but a word used to describe the twisted
neck posture. It may be a clinical sign of one of a
variety of underlying pathologies, some benign and
some quite serious
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67. Classifications of torticollis
torticollis
congenital CMT
CMT
acquired
Skeletal
disorders
Neurological and
physiological
disorders
Spasmodic
occular
CNS lesions and
others
Soft tissue
contracture
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68. Acquired Cont.
Ø Spasmodic torticolis is an extremely painful chronic
neurological movement disorder causing the neck to
involuntarily turn to the left ,right ,upwards and
downwards
Ø This conditions is also referred to as cervical dystonia
Ø It’s a disease of middle aged and later
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69. Cont.
Ø Occular- the result of over action of the inferior
oblique muscle which elevates and rotates laterally.
The head is turned to the opposite side in order to
abduct the eye on the same side.
* Only occurs after 18 months of age
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70. CMT
Ø Congenital muscular Torticollis (CMT)- a postural
deformity detected at birth or shortly after birth ,
primarily resulting from unilateral shortening and
f i b r o s i s o f t h e S t e r n o c l e i d o - m a s t o i d
muscle(SCM).(Karmel-Ross 2006
Ø Unilateral contracture of the Sternocleidomastoid
(SCM)
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71. Infant with left congenital muscular
Torticollis( CMT)
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72. Cont.
Ø Over 80% of all infants presenting with a Torticollis
posture will be found to have congenital muscular
Torticollis (CMT). In the remaining 20%, the
Torticollis posture may represent a sign of a more
serious underlying condition.
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73. Cont.
Ø CMT is the third most frequently occurring
musculoskeletal condition in infants with a reported
incidence of 0.4% to 1.9%.
Ø CMT is often seen in combination with metatarsus
adductus and developmental hip dysplasia
Ø The pathophysiology and etiology of sternocleid-
omastoid impairment in CMT is still unknown
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74. Etiology
The cause of CMT is unknown, there are several
theories , the most advanced ones are;
v Intrauterine mal positioning of the neck with
resultant local ischemia of died sternocledomastoid.
After breech delivery.
vHemi atlas, rare congenital anomaly of formation of
the first cervical vertebra may cause progressive
torticollis.
vInjury to the neck during delivery
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75. Cont.
vIntrauterine pressure on the neck due to
positioning in the womb
vTrauma to sternomastoid lead to loss of blood
supply to muscle-------------fibrosis
vHaemorrhage , swelling and degeneration of the
muscle fibres
vAbnormality of blood supply to the fetus----- lead
to scar formation of sternomastoid muscles
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76. Symptoms
ü The head is tilted to the affected side
ü The chin is turned away from the affected side
ü Face develops asymmetrically
ü Neck mass is usually notice soon after birth but may
not found after several weeks(gradually disappears)
ü Limited ROM of the neck
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77. Congenital(CMT)...
q CMT is often classified into three clinical
groups:
1. Sternocleido mastoid tumor (SMT), when a definitive mass
or tumor is palpable within the SCM muscle,
2. muscular Torticollis (MT), when contracture of the SCM
muscle is present but no palpable mass is present, and
3. positional Torticollis (POST), when both contracture of the
SCM muscle and a palpable mass are absent. Cheng
etal.2001, Van Vlimmerenetal.2006
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79. PT assessment and treatment
• History
* birth/delivery
* presence of skull asymmetry at birth
* presence of facial asymmetry at birth
• Current Health
*feeding *positioning
• Torticollis/Plagiocephaly History
*onset *changes in symptoms
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81. • Clinical Observations *Resting posture
*Motor development
• Range of Motion *Cervical
*upper and lower
extremity
• Palpation
*SCM
*Trapezius
*Scalenes
• PROM of the neck is performed in all
directions, AROM is tested by using attracting
toy
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82. Management
Ø When CMT is inadequately managed infants may
develop progressive limitation of cervical movement
along with craniofacial asymmetry, compensatory
scoliosis, delayed achievement of early motor
milestones and functional asymmetry similar to
hemiplegia
Ø physiotherapists are the most frequently using first
line conservative management for infants with CMT
• The majority (>90%) of infants and children
diagnosed with CMT achieve good to excellent
outcome with conservative treatment
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83. PT TX
Goals:
• Prevent development of contracture
• Stretch tight muscles
• Strength the antagonist muscles including
contralateral sternomastoid and neck muscles
• Prevent delay of normal neck activities
• Encourage normal posture
• Facilitate normal righting reactions
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84. PT TX
Ø Ice, ultrasound, massage and stretching are all
effective in reducing spasm.
Ø Instructions in posture and resting positions are
helpful in limiting degree of distress and spasm
Ø Passive stretching
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85. Con….
• Baby position: The baby is placed on a padded table in supine
with affected side away from therapist
• Grasp : one hand fixing the shoulder of the baby allowing the
head to side flexion to perform stretch
• Head gently but firmly stretched into side flexion to the non
affected side ,rotations to affected side for a count of 8(ten
seconds)
Ø Relax slightly for a few seconds
Ø Repeat
Ø Several times ,4 or 5 times daily
Ø Comfortable with breast feeding
• The stretch should be gradually and therapist grasp should be
gentle not harmful
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86. Active exercises:
Ø After stretching, therapist should encourage the
child to active correction and full ROM of head
and neck muscles
Ø As the head control develop, Facilitate head
righting to improve head side bending and
rotation, facilitate righting by using ball with
baby supine or prone position
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87. Splinting
Ø Cap and jacket Splint to keep the baby head in full
stretched position , not preferable since long time use
hinders the active correction of neck muscles
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89. Surgical treatment:
ü Tenotomy of sternomastoid above the
attachment of the clavicle if the contracture
persists treatment
Post operative PT:
ü Immediately after surgery: the child lies without
pillow, sand bag prevent head from returning to
the asymmetrical position, Cap jacket is advised
until the child can maintain head in midline
position;l
Ø Stretching and active correction are started after
36 hours from surgery
Ø Facilitate normal righting reactions
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90. Home advice
ü Explain to parents not only the purpose of the
treatment but also the practical ways of making
treatment at home
ü The mother should be taught how to stretch the
sternomastoid muscle and how to facilitate the
movements
ü The baby should be encouraged to turn head
away from abnormal posture.
ü The baby should be encouraged to sleep on
one side rather than supine
ü While awake ,place baby so that he/she has turn
his head in the desired direction
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