Basic
Electrocardiography
By
Dr. Ahmed Easa
What is ECG
►It is recording of electrical activity of the
heart.
Basic concepts
► The normal resting excitable
cardiac cells have differential
charge distribution with their
outside being positive in
relation to their inside at
rest (Polarized state).
► With activation the charge distribution reverses
and this reversal propagates through the cardiac
cells producing a propagated action potential(
Depolarized state).
Basic concepts
►The junction between the depolarized and
polarized portion on the surface of cardiac
myocyte form an electric dipole.
Basic concepts
►The voltage produced by cardiac dipole can
be recorded by applying electrode over the
surface of the heart.
Basic concepts
Currents and Voltages
► At rest, Vm is
constant
► No current flowing
► Inside of cell is at
constant potential
► Outside of cell is at
constant potential
++++++++++++++++++
------------------------------
A piece of cardiac muscle
outside
inside
0 mV
+-
Currents and Voltages
► During AP upstroke,
Vm is NOT constant
► Current IS flowing
► Inside of cell is NOT
at constant potential
► Outside of cell is
NOT at constant
potential
++++------------------------
------++++++++++++++
A piece of cardiac muscle
outside
inside
Some positive
potential
+-
current
AP
An action potential propagating
toward the positive ECG lead
produces a positive signal
More Currents and Voltages
A piece of cardiac muscle
outside
current
+-
No voltage
+-
A negative voltage reading
------++++++++++++++
inside
++++------------------------
More Currents and Voltages
current
-------------------------------
A piece of totally depolarized
cardiac muscle
outside
inside
+++++++++++++++++++
Vm not changing
No current
No ECG signal
+++++++-------------------
A piece of cardiac muscle
outside
inside
------------+++++++++++
During Repolarization
+-
Some negative potential
Repolarization spreading toward
the positive ECG lead produces
a negative response
►If the wave of depolarization spreads towards
the electrode --- positive deflection is recorded.
►If the wave of depolarization spreads away
from the electrode --- negative deflection is
recorded.
►If the wave of depolarization spreads
orthogonal to the electrode --- biphasic
deflection is recorded.
►If the wave of repolarization spreads towards
the electrode --- negative deflection is
recorded.
►If the wave of repolarization spreads away from
the electrode --- positive deflection is recorded.
►If the wave of repolarization spreads
orthogonal to the electrode --- biphasic
deflection is recorded.
Dipole voltage
2
cos( )
dipole
M
V
r


------++++++++++++++
inside
++++------------------------
Observation
point
►The cardiac dipoles activating the
individual cardiac myocyte add vectorially
and are represented by a single vector.
►The direction of this vector changes
continuously with time giving rise to
instantaneous vector .
Surface Electrocardiographic leads
*12 standard leads
► More leads can be added for special purposes
► The are subdivided into
 Limb leads
 Chest leads
Surface Electrocardiographic leads
► They are of 2 types
 Bipolar record voltage or potential difference between
2 sites in the heart as standard limb leads I,II,III)
 Unipolar leads directly record the voltage at the site
of application ( Augmented limb leads and chest
leads)
► Standardization:-
►Paper speed 25 mm/sec always look for ( rapid speed 50
mm/sec may lead to false diagnosis of bradycardia).
►10 mm = 1 milivolt
Limb Leads
► Limb leads
 Record electrical activity in
the frontal plane
 Two types
►Nonaugmented Lead I,II,III.
►Augmented aVR, aVL, aVF
Chest Leads
► Position of chest leads:-
 V1 right fourth intercostal space at sternal edge
 V2 left fourth intercostal space at sternal edge
 V3 midway between V2 & V4
 V4 left fifth intercostal space in midclavicular line
 V5 in anterior axillary line on the same horizontal level
of V4
 V6 in mid axillary line on the same horizontal level of V4
► They record the electrical activity in the horizontal
plane
V1 V2
V4 V5 V6
V3
Anatomical relations of the leads
►Anterior wall of LV ---V1-V4
►Lateral wall of LV ----- 1,aVL,V5 & V6
►Inferior wall of LV----- 2,3,aVF
►RA & LV cavity ----- aVR & V1
►RV ---- V3R & V4R
How to comment on ECG
►1-Rate
►2-Rhythm
►3-Cardiac axis.
►4- Analysis of individual waves, segments
and intervals.
►5- ECG findings specific to disease states.
1- RATE
► Normally 60-100 Bpm
► Less than 60 ---- Bradycardia
► More than 100 --- tachycardia
► How to calculate heart rate from ECG
► 1-Regular:-
► Measure the number of small squares between 2 successive R waves
and divide 1500 upon it.
► Measure the number of large squares between 2 successive R waves
and divide 300 upon it.
► One small square = 40 msec
► One large square = 200 msec
► 2-Irregular
► Count the number of R waves in 10 seconds and multiply by 6.
2-Rhythm
►The normal cardiac rhythm is regular
►Irregular rhythm may be present in
 Atrial fibrillation
 MAT
 Atrial flutter with varying conduction
 PVCs, PACs
 Mobitz type 1 second degree AV block
3-Cardiac electrical axis
►The electric axis is a fundamental and general
term
►It can be applied to any wave or segment e.,g(
P, QRS,T,U,ST segments)
►Due to 3 dimensional cardiac geometry, the
axis can be calculated in the 3 orthogonal
planes (Frontal, Sagittal & Horizontal)
►The simplest & commonest form of axis
calculation is the mean frontal plane QRS.
 Normal mean frontal QRS axis (-30 ---- +110)
 Left axis deviation< -30
 Right axis deviation > 110
3-Cardiac electrical axis
How to calculate the QRS axis
►1-The easiest way
► Hold the SINGLE lead ECG strip with your hands
► Put your right hand over lead III
► Put your left hand over lead I
► Look for QRS in lead 1& lead 3
 Positive in lead 1 negative in lead 3 ---LAD
 Positive in lead 3 negative in lead 1 ---RAD
 If both negative ( extreme axis deviation) NO MAN
LAND
 If both positive the axis is within normal
►2- More difficult way
►Choose a lead with a biphasic QRS.
►The axis lie perpendicular to this lead.
►Look for the perpendicular leads or the leads
close to the perpendicular leads.
►Normally there is only 2 perpendicular leads for
any lead you choose.
►The lead that shows positive QRS, the axis is in
that direction
3-Exact calculation of mean frontal
QRS axis
► 1- Pick up 2 orthogonal leads (
Lead 1 & aVF)
► 2- For lead 1 calculate the net
QRS voltage ( - for negative
waves Q&S, + sign for positive
waves R)
► 3- For lead aVF do the same
and multiply the resultant by
2/3
► 4-Divide net QRS voltage in aVF
over net QRS voltage in lead 1.
► 5- The angle the mean frontal
QRS vector make with the
horizontal axis is given by
2
arctan
3
aVF
I
QRS
QRS

 
  
 
I
avF+60III+120
avL-30avR-150
-90
+90
+/-180 0
LAD
RAD
Extreme axis deviation
No man”s land

+
+
-
-
Abnormal QRS axis
► Left Axis Deviation (LAD): > -30o (i.e., lead II is mostly 'negative')
 Left Anterior Fascicular Block (LAFB): rS complex in leads II, III, aVF, small q in
leads I and/or aVL, and axis -45o to -90o
 Some cases of inferior MI with Qr complex in lead II (making lead II 'negative')
 Inferior MI + LAFB in same patient (QS or qrS complex in lead II)
 Some cases of LVH ( hypertrophy mainly involves the posterobasal part of LV)
 Some cases of LBBB
 Ostium primum ASD and other endocardial cushion defects
 Some cases of WPW syndrome (large negative delta wave in lead II)
► Right Axis Deviation (RAD): > +90o (i.e., lead I is mostly 'negative')
 Left Posterior Fascicular Block (LPFB): rS complex in lead I, qR in leads II, III,
aVF (however, must first exclude, on clinical basis, causes of right heart
overload; these will also give same ECG picture of LPFB)
 Many causes of right heart overload and pulmonary hypertension
 High lateral wall MI with Qr or QS complex in leads I and aVL
 Some cases of RBBB
 Some cases of WPW syndrome
 Children, teenagers, and some young adults
 Bizarre QRS axis: +150o to -90o (i.e., lead I and lead II are both negative)
(No man land)
► Consider limb lead error (usually right and left arm reversal)
► Dextrocardia
► Some cases of complex congenital heart disease (e.g., transposition)
► Some cases of ventricular tachycardia
4-Waves, intervals and segments
P wave
► Represents atrial depolarization
► Criteria of normal P wave
 Upright in lead II & avF
 Negative in lead avR
 Variable in lead aVL
 Biphasic in V1 with initial positive deflection and late
negative deflection
 Not more than 0.25 mv in amplitude
 Not more than 120 msec in duration
 2.5 x 2.5 small squares
 If notched look for the distance between 2 notches –
normally less than or equal to one small square
P-R interval
► Represents time taken by the impulse to travel via
the AV node, His bundle, Bundle branches and
Purkinje fibers.
► Measured from beginning of P wave to the
beginning of QRS complex
► Normally 120-220 msec ( 3-5.5 small squares)
► Prolonged in AV block (see later)
► Shortened in preexcitation syndromes (see later)
QRS
►Q wave:- Any initial negative deflection
►R wave any initial positive deflection
►S wave negative wave following the R wave.
►Small letters are used if the amplitude of
the wave is less than 5 mv and capital
letters are used if the amplitude is more
than 5 mm.
QRS
►Sequence of ventricular activation
 1- Septal depolarization
►Initial vector directed across the interventricular
septum from left to right
►Produces small r wave in V1 and small q in V5,V6
 2-Ventricular free wall depolarization
►Simultaneous depolarization of LV & RV.
►Because normally the LV is thicker than the RV its
electrical forces predominate
►This lead to large S wave in V1 & large R wave in
lead V6
QRS
►Transitional Zone
 Zone at which there is change from
predominantly negative QRS to predominantly
positive QRS
 Usually between V3-V4
 Delayed transitional zone– shifted to V5&V6---
clockwise rotation in RV enlargement
 Early transitional zone– shifted to V1&V2---
counterclockwise rotation in LV enlargement
J point
►Junction between the end of QRS and
beginning of ST segment
►J point loss may be the first ECG sign of
acute MI.
ST segment
►From the end of QRS complex to the
beginning of T wave
►Represent phase 2 of myocyte action
potential
►Normally it is iso electric with the
preceeding TP segment
T wave
► Represent phase 3 of myocyte action potential.
► Due to asynchronous repolarization of subendocardial
and subepicardial myocardium ( Normally
subepicardial repolarization precedes subendocardial
repolarization)
► Usually positive in precordial leads
► May be negative in V1 & V2
► Isolated T inversion in V2 is pathologic.
► Persistent juvenile pattern is T wave inversion in RT
precordial leads especially in blacks
► Asymmetric T wave inversion is pathologic.
► Symmetric T wave inversion is non specific
QT interval
►The interval from beginning of QRS to the
end of T wave.
►Represents the total time of ventricular
electromechanical systole
►Normally up to 440 msec ( or roughly less
than half the corresponding RR interval)
U wave
►A small positive wave may occasionally
follow the T wave
►May represent repolarization of the
posterobasal portion of the LV.
Thank You

Basic Electrocardiography

  • 1.
  • 2.
    What is ECG ►Itis recording of electrical activity of the heart.
  • 5.
    Basic concepts ► Thenormal resting excitable cardiac cells have differential charge distribution with their outside being positive in relation to their inside at rest (Polarized state).
  • 6.
    ► With activationthe charge distribution reverses and this reversal propagates through the cardiac cells producing a propagated action potential( Depolarized state). Basic concepts
  • 7.
    ►The junction betweenthe depolarized and polarized portion on the surface of cardiac myocyte form an electric dipole. Basic concepts
  • 8.
    ►The voltage producedby cardiac dipole can be recorded by applying electrode over the surface of the heart. Basic concepts
  • 9.
    Currents and Voltages ►At rest, Vm is constant ► No current flowing ► Inside of cell is at constant potential ► Outside of cell is at constant potential ++++++++++++++++++ ------------------------------ A piece of cardiac muscle outside inside 0 mV +-
  • 10.
    Currents and Voltages ►During AP upstroke, Vm is NOT constant ► Current IS flowing ► Inside of cell is NOT at constant potential ► Outside of cell is NOT at constant potential ++++------------------------ ------++++++++++++++ A piece of cardiac muscle outside inside Some positive potential +- current AP An action potential propagating toward the positive ECG lead produces a positive signal
  • 11.
    More Currents andVoltages A piece of cardiac muscle outside current +- No voltage +- A negative voltage reading ------++++++++++++++ inside ++++------------------------
  • 12.
    More Currents andVoltages current ------------------------------- A piece of totally depolarized cardiac muscle outside inside +++++++++++++++++++ Vm not changing No current No ECG signal +++++++------------------- A piece of cardiac muscle outside inside ------------+++++++++++ During Repolarization +- Some negative potential Repolarization spreading toward the positive ECG lead produces a negative response
  • 13.
    ►If the waveof depolarization spreads towards the electrode --- positive deflection is recorded. ►If the wave of depolarization spreads away from the electrode --- negative deflection is recorded. ►If the wave of depolarization spreads orthogonal to the electrode --- biphasic deflection is recorded.
  • 14.
    ►If the waveof repolarization spreads towards the electrode --- negative deflection is recorded. ►If the wave of repolarization spreads away from the electrode --- positive deflection is recorded. ►If the wave of repolarization spreads orthogonal to the electrode --- biphasic deflection is recorded.
  • 15.
  • 16.
    ►The cardiac dipolesactivating the individual cardiac myocyte add vectorially and are represented by a single vector.
  • 17.
    ►The direction ofthis vector changes continuously with time giving rise to instantaneous vector .
  • 18.
    Surface Electrocardiographic leads *12standard leads ► More leads can be added for special purposes ► The are subdivided into  Limb leads  Chest leads
  • 19.
    Surface Electrocardiographic leads ►They are of 2 types  Bipolar record voltage or potential difference between 2 sites in the heart as standard limb leads I,II,III)  Unipolar leads directly record the voltage at the site of application ( Augmented limb leads and chest leads) ► Standardization:- ►Paper speed 25 mm/sec always look for ( rapid speed 50 mm/sec may lead to false diagnosis of bradycardia). ►10 mm = 1 milivolt
  • 20.
    Limb Leads ► Limbleads  Record electrical activity in the frontal plane  Two types ►Nonaugmented Lead I,II,III. ►Augmented aVR, aVL, aVF
  • 21.
    Chest Leads ► Positionof chest leads:-  V1 right fourth intercostal space at sternal edge  V2 left fourth intercostal space at sternal edge  V3 midway between V2 & V4  V4 left fifth intercostal space in midclavicular line  V5 in anterior axillary line on the same horizontal level of V4  V6 in mid axillary line on the same horizontal level of V4 ► They record the electrical activity in the horizontal plane
  • 22.
  • 23.
    Anatomical relations ofthe leads ►Anterior wall of LV ---V1-V4 ►Lateral wall of LV ----- 1,aVL,V5 & V6 ►Inferior wall of LV----- 2,3,aVF ►RA & LV cavity ----- aVR & V1 ►RV ---- V3R & V4R
  • 24.
    How to commenton ECG ►1-Rate ►2-Rhythm ►3-Cardiac axis. ►4- Analysis of individual waves, segments and intervals. ►5- ECG findings specific to disease states.
  • 25.
    1- RATE ► Normally60-100 Bpm ► Less than 60 ---- Bradycardia ► More than 100 --- tachycardia ► How to calculate heart rate from ECG ► 1-Regular:- ► Measure the number of small squares between 2 successive R waves and divide 1500 upon it. ► Measure the number of large squares between 2 successive R waves and divide 300 upon it. ► One small square = 40 msec ► One large square = 200 msec ► 2-Irregular ► Count the number of R waves in 10 seconds and multiply by 6.
  • 27.
    2-Rhythm ►The normal cardiacrhythm is regular ►Irregular rhythm may be present in  Atrial fibrillation  MAT  Atrial flutter with varying conduction  PVCs, PACs  Mobitz type 1 second degree AV block
  • 28.
    3-Cardiac electrical axis ►Theelectric axis is a fundamental and general term ►It can be applied to any wave or segment e.,g( P, QRS,T,U,ST segments) ►Due to 3 dimensional cardiac geometry, the axis can be calculated in the 3 orthogonal planes (Frontal, Sagittal & Horizontal)
  • 29.
    ►The simplest &commonest form of axis calculation is the mean frontal plane QRS.  Normal mean frontal QRS axis (-30 ---- +110)  Left axis deviation< -30  Right axis deviation > 110 3-Cardiac electrical axis
  • 30.
    How to calculatethe QRS axis ►1-The easiest way ► Hold the SINGLE lead ECG strip with your hands ► Put your right hand over lead III ► Put your left hand over lead I ► Look for QRS in lead 1& lead 3  Positive in lead 1 negative in lead 3 ---LAD  Positive in lead 3 negative in lead 1 ---RAD  If both negative ( extreme axis deviation) NO MAN LAND  If both positive the axis is within normal
  • 31.
    ►2- More difficultway ►Choose a lead with a biphasic QRS. ►The axis lie perpendicular to this lead. ►Look for the perpendicular leads or the leads close to the perpendicular leads. ►Normally there is only 2 perpendicular leads for any lead you choose. ►The lead that shows positive QRS, the axis is in that direction
  • 32.
    3-Exact calculation ofmean frontal QRS axis ► 1- Pick up 2 orthogonal leads ( Lead 1 & aVF) ► 2- For lead 1 calculate the net QRS voltage ( - for negative waves Q&S, + sign for positive waves R) ► 3- For lead aVF do the same and multiply the resultant by 2/3 ► 4-Divide net QRS voltage in aVF over net QRS voltage in lead 1. ► 5- The angle the mean frontal QRS vector make with the horizontal axis is given by 2 arctan 3 aVF I QRS QRS        
  • 34.
  • 35.
    Abnormal QRS axis ►Left Axis Deviation (LAD): > -30o (i.e., lead II is mostly 'negative')  Left Anterior Fascicular Block (LAFB): rS complex in leads II, III, aVF, small q in leads I and/or aVL, and axis -45o to -90o  Some cases of inferior MI with Qr complex in lead II (making lead II 'negative')  Inferior MI + LAFB in same patient (QS or qrS complex in lead II)  Some cases of LVH ( hypertrophy mainly involves the posterobasal part of LV)  Some cases of LBBB  Ostium primum ASD and other endocardial cushion defects  Some cases of WPW syndrome (large negative delta wave in lead II)
  • 36.
    ► Right AxisDeviation (RAD): > +90o (i.e., lead I is mostly 'negative')  Left Posterior Fascicular Block (LPFB): rS complex in lead I, qR in leads II, III, aVF (however, must first exclude, on clinical basis, causes of right heart overload; these will also give same ECG picture of LPFB)  Many causes of right heart overload and pulmonary hypertension  High lateral wall MI with Qr or QS complex in leads I and aVL  Some cases of RBBB  Some cases of WPW syndrome  Children, teenagers, and some young adults  Bizarre QRS axis: +150o to -90o (i.e., lead I and lead II are both negative) (No man land) ► Consider limb lead error (usually right and left arm reversal) ► Dextrocardia ► Some cases of complex congenital heart disease (e.g., transposition) ► Some cases of ventricular tachycardia
  • 37.
  • 39.
    P wave ► Representsatrial depolarization ► Criteria of normal P wave  Upright in lead II & avF  Negative in lead avR  Variable in lead aVL  Biphasic in V1 with initial positive deflection and late negative deflection  Not more than 0.25 mv in amplitude  Not more than 120 msec in duration  2.5 x 2.5 small squares  If notched look for the distance between 2 notches – normally less than or equal to one small square
  • 40.
    P-R interval ► Representstime taken by the impulse to travel via the AV node, His bundle, Bundle branches and Purkinje fibers. ► Measured from beginning of P wave to the beginning of QRS complex ► Normally 120-220 msec ( 3-5.5 small squares) ► Prolonged in AV block (see later) ► Shortened in preexcitation syndromes (see later)
  • 41.
    QRS ►Q wave:- Anyinitial negative deflection ►R wave any initial positive deflection ►S wave negative wave following the R wave. ►Small letters are used if the amplitude of the wave is less than 5 mv and capital letters are used if the amplitude is more than 5 mm.
  • 42.
    QRS ►Sequence of ventricularactivation  1- Septal depolarization ►Initial vector directed across the interventricular septum from left to right ►Produces small r wave in V1 and small q in V5,V6  2-Ventricular free wall depolarization ►Simultaneous depolarization of LV & RV. ►Because normally the LV is thicker than the RV its electrical forces predominate ►This lead to large S wave in V1 & large R wave in lead V6
  • 43.
    QRS ►Transitional Zone  Zoneat which there is change from predominantly negative QRS to predominantly positive QRS  Usually between V3-V4  Delayed transitional zone– shifted to V5&V6--- clockwise rotation in RV enlargement  Early transitional zone– shifted to V1&V2--- counterclockwise rotation in LV enlargement
  • 44.
    J point ►Junction betweenthe end of QRS and beginning of ST segment ►J point loss may be the first ECG sign of acute MI.
  • 45.
    ST segment ►From theend of QRS complex to the beginning of T wave ►Represent phase 2 of myocyte action potential ►Normally it is iso electric with the preceeding TP segment
  • 46.
    T wave ► Representphase 3 of myocyte action potential. ► Due to asynchronous repolarization of subendocardial and subepicardial myocardium ( Normally subepicardial repolarization precedes subendocardial repolarization) ► Usually positive in precordial leads ► May be negative in V1 & V2 ► Isolated T inversion in V2 is pathologic. ► Persistent juvenile pattern is T wave inversion in RT precordial leads especially in blacks ► Asymmetric T wave inversion is pathologic. ► Symmetric T wave inversion is non specific
  • 47.
    QT interval ►The intervalfrom beginning of QRS to the end of T wave. ►Represents the total time of ventricular electromechanical systole ►Normally up to 440 msec ( or roughly less than half the corresponding RR interval)
  • 48.
    U wave ►A smallpositive wave may occasionally follow the T wave ►May represent repolarization of the posterobasal portion of the LV.
  • 49.