Acute herpetic gingivostomatitis
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UjmaAcute herpetic gingivostomatitis (AHG) is a viral infection of the oral mucosa caused by the herpes simplex virus type 1. It occurs most frequently in infants and children under 6 years old and presents with diffuse erythema and edema of the gums, as well as clusters of vesicles dispersed throughout the mouth, lips, palate, and tongue. The symptoms include generalized oral soreness and fever, and the full course of the disease is typically 7-10 days. After the primary infection subsides, the virus can remain latent in nerve tissues and potentially reactivate to cause cold sores.
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syphilissssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssyphilissssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssyphilissssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssyphilissssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssyphilissssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssyphilisssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssssss
Orofacial viral infections
This document discusses orofacial viral infections. It begins by introducing common viral infections of the orofacial region, including herpes viruses, HIV, HPV, coxsackle virus, and mumps virus. It then focuses on herpes viruses, describing the three subfamilies (alpha, beta, gamma) and some examples within each. Specific sections discuss infections caused by alpha herpes viruses like HSV-1 and VZV, beta herpes viruses like CMV and HHV-6, and gamma herpes viruses like EBV. Clinical features, pathogenesis, diagnosis and treatment are described for various infections like infectious mononucleosis caused by EBV. Complications of infections are also mentioned.
Hsv , mazin malik
The document summarizes information about herpes simplex viruses types 1 and 2 (HSV-1 and HSV-2), including that they cause a variety of illnesses depending on site of infection, host immunity, and whether it is a primary or recurrent infection. Primary infections tend to be more severe while recurrent infections are generally milder. HSV establishes latent infections in sensory ganglia. Infections range from mild, self-limiting oral-facial infections to potentially life-threatening infections in immunocompromised individuals or newborns. Antiviral medications can shorten symptoms of recurrent infections but do not cure the infection.
Oral viral infections
- Herpes simplex virus types 1 and 2 are common oral viral infections, usually transmitted via saliva or direct contact. Both viruses establish latency in ganglia. HSV-1 typically causes oral lesions while HSV-2 typically causes genital lesions.
- Varicella zoster virus causes chickenpox in children and shingles in adults via reactivation. It establishes latency in ganglia.
- Epstein-Barr virus causes infectious mononucleosis, commonly known as "mono" or "kissing disease". It is transmitted through saliva and causes fever, lymphadenopathy and pharyngitis.
Vesiculo bullous diseases
vesicobullous diseases
inludes Viral & imune mediated disease
i hope these slides ill help retain this topic
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- 1. ACUTE HERPETIC GINGIVOSTOMATITIS Uzma Jan 3rd year B.D.S
- 2. AHG is a viral infection of the oral mucous membrane caused by herpes simplex virus-I(HSV-I). Occurs most frequently in infants & Children younger than 6 years .
- 3. CLINICAL FEATURES ORAL SIGNS : Appears as a diffuse shiny erythematous edema & gingival bleeding. Discrete spherical clusters of vesicle dispersed in different areas (labial buccal mucosa hard palate pharynx and tongue) Course of disease is 7-10 days SYMPTOMS: Generalized soreness of oral cavity Ruptured vesicles sensitive t0 touch thermal changes and food
- 4. EXTRA ORAL & SYSTEMIC SIGN & SYMPTOMS 1-3 days of fever , loss of appetite and myalgia Cervical lymphadenopathy is present.
- 5. After the Primary infection the virus remains latent in the nerve tissues. If reactivation occurs it causes herpes labialis (cold sore).
- 6. HISTOPATHOLOGY The virus targets the epithelial cells which show “ballooning degeneration” These cells are called Tzanck cells. Infected cells fuse , forming multinucleated cells and IC edema leads to formation of an intraepithelial vesicles that rupture and develops a sec. inflammatory response with a fibro purulent exudate Discrete ulceration have a central portion of acute inflammation with exudate surrounded by engorged blood vessels.
- 7. DIAGNOSIS Established from pts history & clinical findings. HSV isolation by cell culture is the gold standard. Tzanck smear
- 8. D/D Necrotizing Ulcerative Gingivitis Erythema multiforme Stevens-Johnson Syndrome Apthous Stomatitis (canker sores)
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