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SEMINAR PRESENTATION
ON
GLOMERULONEPHRITIS
PRESENTED BY-
MISS. DIKSHA SINGH
B.SC.NURSING 3RD YEAR
2017 BATCH
VIVEKANANDA COLLEGE OF
NURSING
GENERAL OBJECTIVE
 To deal with glomerulonephritis-
 Introduction
 Definition
 Incidence rate
 Types
 Etiology and risk factors
 Pathophysiology
 Clinical manifestation
CONT..
 Diagnostic evaluation
 Management
-pharmacological management
-non pharmacological management
-patient education
-surgical management
-nursing management
 Complication
 Outlook
INTRODUCTION
 It is a term used to refers to several kidney disease (both
kidney) characterized by inflammation either of the
glomeruli or of the small blood vessels in the kidney.
 These knots of blood vessels help to filter the blood and
remove excess fluid. If glomeruli are damaged , kidney
will stop working properly and can lead to kidney failure.
 It is sometimes called nephritis.
 Also known as Bright’s disease.
DEFINITION
 Glomerulonephriti
s is a kidney
condition tha
involves
damage/inflammati
on to the glomeruli
and its blood
vessels.
INCIDENCE RATE
 Incidence rate of primary glomerulonephritis vary
between 0.2/100000/year and 2.5/100000/year. This
disease can exist subclinically and is therefore only
detected by chance in some patients.
TYPES OF GLOMERULONEPHRITIS
 There are two types of glomerulonephritis-
1. Acute glomerulonephritis
2. Chronic glomerulonephritis
TYPES
Acute glomerulonephritis
 begins suddenly
 occur after 5-21 days of
streptococcal infection.
 It may be problem with the
immune system.
Chronic glomerulonephritis
 Develops gradually over
several years.
 Occur after acute phase.
 Irreversible damage to
kidney that can lead to
complete kidney failure.
 Shows few symptoms.
 No clear cause.
CAUSES
Cause
 Streptococcal infection
 Abscessed tooth
 SLE
 Goodpasture syndrome
 Amyloidosis
 Granulomatosis with
polyangitis
 Polyarteritis nodosa
Cause
 Hereditary disease
 Immune disease
 Genetic
 History of cancer
 Exposure to some
hydrocarbons
CONT..
Causes
 Vasculitis
 Endocarditis
 High blood pressure
 Diabetes
Risk factor
 Overdose of NSAIDS
SYMPTOMS
Symptoms
 Puffiness in face
 Oliguria
 Dysuria
 Gross hematuria
 High blood pressure
Symptoms
 Proteinuria
 High blood pressure
 Swelling in ankles and face
 Foamy urine
 Abdominal pain
PATHOPHYSIOLOGY
 Due to etiological factor like immune, streptococcal
infection
 Release of antigen subsatnce into the circulation
 In response formation of antiboy occur
 Formation of antigen and antibody complex in the
glomerulus
 Inflammatory response
 Proliferation of epithelial cells lining the glomerulus
 Leukocytes infiltration of the glomerulus
CONT..
 Thickening of the glomerular filtration membrane
 Scarring and loss of glomerular filtration membrane
 Decrease GFR and glomerular plasma flow
 Retention of sodium and water
 Edema and hypertension
DIAGNOSTIC EVALUATION
 History collection
 Physical examination
 Urineanalysis
 Blood test
 Biopsy
 Intravenous pyelogram
 Immunological test
 CT scan
 USG
 Chest X-ray
MANAGEMENT: MEDICAL
MANAGEMENT
 Pharmacological management
 Treatement depend on the cause of disorder , type and
severity of symptoms.
-diuretic-e.g.chlorthalidone, chlorothiazide
-immunosuppresants-e.g.cyclosporine
antiproliferative agent-e.g.azathioprine
-antihypertensive drug
ACE inhibitors-e.g.benazepril, perindopril, enalapril
Angiotensin receptor blocker-e.g.losartan
CONT..
 Corticosteroids-e.g.decadron, cotolone
 Plasmapheresis
 Dialysis if condition worsen
Non pharmacological management
 Sodium and water restriction.
 Potassium, phosphorus, magnesium restriction.
 Limit intake of protein in the diet.
 Take calcium supplements.
 Maintain healthy weight through diet $ excercise.
CONT..
 Measure liquid intake.
Patient education
 Breathing excercise – pursed lip $ diaphragmatic
breathing.
 Endurance excercise such as walking, swimming,
bicycling, aerobic dancing, circulatory excercise.
 This excercise improves blood circulation, accelerate
kidney to discharge waste and toxins.
 To quit smoking.
SURGICAL MANAGEMENT
KIDNEY TRANSPLANT-
 It is done to treat kidney failure when glomerulonephritis
is difficult to treat with medication and other procedure
and there is immediate need to replace the diseased
kidney.
NURSING MANAGEMENT
NURSING DIAGNOSIS-
1. Acute pain related to inflammation of renal cortex as
evidenced by facial expression and patient verbalization.
2. Excess fluid volume related to accumulation of fluid in
the body as evidenced by edema and weight gain.
3. Ineffective breathing pattern related to accumulation of
fluid in the peritoneal cavity as evidenced by respiratory
rate, dyspnea.
COMPLICATIONS
 Nephrotic syndrome
 Acute and chronic renal failure
 Chronic kidney disease
 Electrolyte imbalance
 Chronic UTI
 Congestive heart failure
 Malignant hypertension
 Pulmonary edema
 Increased risk of infection
OUTLOOK
 If glomerulonephritis early caught then acute
glomerulonephritis could be temporary and chronic
glomerulonephritis could be slowed.
 If glomerulonephritis worsen, causes reduced kidney
function which turn into chronic renal failure and then
into end stage renal disease.
Glomerulonephritis
Glomerulonephritis
Glomerulonephritis
Glomerulonephritis

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Glomerulonephritis

  • 1.
  • 2. SEMINAR PRESENTATION ON GLOMERULONEPHRITIS PRESENTED BY- MISS. DIKSHA SINGH B.SC.NURSING 3RD YEAR 2017 BATCH VIVEKANANDA COLLEGE OF NURSING
  • 3. GENERAL OBJECTIVE  To deal with glomerulonephritis-  Introduction  Definition  Incidence rate  Types  Etiology and risk factors  Pathophysiology  Clinical manifestation
  • 4. CONT..  Diagnostic evaluation  Management -pharmacological management -non pharmacological management -patient education -surgical management -nursing management  Complication  Outlook
  • 5. INTRODUCTION  It is a term used to refers to several kidney disease (both kidney) characterized by inflammation either of the glomeruli or of the small blood vessels in the kidney.  These knots of blood vessels help to filter the blood and remove excess fluid. If glomeruli are damaged , kidney will stop working properly and can lead to kidney failure.  It is sometimes called nephritis.  Also known as Bright’s disease.
  • 6. DEFINITION  Glomerulonephriti s is a kidney condition tha involves damage/inflammati on to the glomeruli and its blood vessels.
  • 7. INCIDENCE RATE  Incidence rate of primary glomerulonephritis vary between 0.2/100000/year and 2.5/100000/year. This disease can exist subclinically and is therefore only detected by chance in some patients.
  • 8. TYPES OF GLOMERULONEPHRITIS  There are two types of glomerulonephritis- 1. Acute glomerulonephritis 2. Chronic glomerulonephritis
  • 9. TYPES Acute glomerulonephritis  begins suddenly  occur after 5-21 days of streptococcal infection.  It may be problem with the immune system. Chronic glomerulonephritis  Develops gradually over several years.  Occur after acute phase.  Irreversible damage to kidney that can lead to complete kidney failure.  Shows few symptoms.  No clear cause.
  • 10. CAUSES Cause  Streptococcal infection  Abscessed tooth  SLE  Goodpasture syndrome  Amyloidosis  Granulomatosis with polyangitis  Polyarteritis nodosa Cause  Hereditary disease  Immune disease  Genetic  History of cancer  Exposure to some hydrocarbons
  • 11. CONT.. Causes  Vasculitis  Endocarditis  High blood pressure  Diabetes Risk factor  Overdose of NSAIDS
  • 12. SYMPTOMS Symptoms  Puffiness in face  Oliguria  Dysuria  Gross hematuria  High blood pressure Symptoms  Proteinuria  High blood pressure  Swelling in ankles and face  Foamy urine  Abdominal pain
  • 13. PATHOPHYSIOLOGY  Due to etiological factor like immune, streptococcal infection  Release of antigen subsatnce into the circulation  In response formation of antiboy occur  Formation of antigen and antibody complex in the glomerulus  Inflammatory response  Proliferation of epithelial cells lining the glomerulus  Leukocytes infiltration of the glomerulus
  • 14. CONT..  Thickening of the glomerular filtration membrane  Scarring and loss of glomerular filtration membrane  Decrease GFR and glomerular plasma flow  Retention of sodium and water  Edema and hypertension
  • 15. DIAGNOSTIC EVALUATION  History collection  Physical examination  Urineanalysis  Blood test  Biopsy  Intravenous pyelogram  Immunological test  CT scan  USG  Chest X-ray
  • 16. MANAGEMENT: MEDICAL MANAGEMENT  Pharmacological management  Treatement depend on the cause of disorder , type and severity of symptoms. -diuretic-e.g.chlorthalidone, chlorothiazide -immunosuppresants-e.g.cyclosporine antiproliferative agent-e.g.azathioprine -antihypertensive drug ACE inhibitors-e.g.benazepril, perindopril, enalapril Angiotensin receptor blocker-e.g.losartan
  • 17. CONT..  Corticosteroids-e.g.decadron, cotolone  Plasmapheresis  Dialysis if condition worsen Non pharmacological management  Sodium and water restriction.  Potassium, phosphorus, magnesium restriction.  Limit intake of protein in the diet.  Take calcium supplements.  Maintain healthy weight through diet $ excercise.
  • 18. CONT..  Measure liquid intake. Patient education  Breathing excercise – pursed lip $ diaphragmatic breathing.  Endurance excercise such as walking, swimming, bicycling, aerobic dancing, circulatory excercise.  This excercise improves blood circulation, accelerate kidney to discharge waste and toxins.  To quit smoking.
  • 19. SURGICAL MANAGEMENT KIDNEY TRANSPLANT-  It is done to treat kidney failure when glomerulonephritis is difficult to treat with medication and other procedure and there is immediate need to replace the diseased kidney.
  • 20. NURSING MANAGEMENT NURSING DIAGNOSIS- 1. Acute pain related to inflammation of renal cortex as evidenced by facial expression and patient verbalization. 2. Excess fluid volume related to accumulation of fluid in the body as evidenced by edema and weight gain. 3. Ineffective breathing pattern related to accumulation of fluid in the peritoneal cavity as evidenced by respiratory rate, dyspnea.
  • 21. COMPLICATIONS  Nephrotic syndrome  Acute and chronic renal failure  Chronic kidney disease  Electrolyte imbalance  Chronic UTI  Congestive heart failure  Malignant hypertension  Pulmonary edema  Increased risk of infection
  • 22. OUTLOOK  If glomerulonephritis early caught then acute glomerulonephritis could be temporary and chronic glomerulonephritis could be slowed.  If glomerulonephritis worsen, causes reduced kidney function which turn into chronic renal failure and then into end stage renal disease.