This document discusses stroke, its causes, signs and symptoms, diagnosis, risk factors, and complications. It also summarizes the roles of occupational therapists and prosthetists/orthotists in treating stroke patients. Occupational therapists focus on intervention for upper extremities, while prosthetists/orthotists design and fit lower extremity orthoses like ankle-foot orthoses to improve walking. Both work closely with physical therapists to facilitate rehabilitation.
PHYSIOTHERAPY REHABILITATION IN SURGICAL AND NON SURGICAL ONCOLOGY prasad naik
Physiotherapy is a valuable and often underutilized tools in the management of cancer.
Physiotherapy for cancer patients can occur in the home, an outpatient clinic, an inpatient rehabilitation center, or in an acute care hospital.
Physiotherapy can help them regain their previous quality of life, or even improve upon it.
Therapists who understand the complexities of oncology rehabilitative care can have a huge impact on cancer patients’ ability to tolerate such toxic treatments by working with them to maintain their strength and function.
PHYSIOTHERAPY REHABILITATION IN SURGICAL AND NON SURGICAL ONCOLOGY prasad naik
Physiotherapy is a valuable and often underutilized tools in the management of cancer.
Physiotherapy for cancer patients can occur in the home, an outpatient clinic, an inpatient rehabilitation center, or in an acute care hospital.
Physiotherapy can help them regain their previous quality of life, or even improve upon it.
Therapists who understand the complexities of oncology rehabilitative care can have a huge impact on cancer patients’ ability to tolerate such toxic treatments by working with them to maintain their strength and function.
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different soft tissue injuries are the part of curriculum for the undergraduate students at KUSMS.
Prof. Anisuddin Bhatti describes spasticity management in Cerebral Palsy patients. Botulinum Toxin (BOTOX) therapy and its application techniques live demonstration given. lectured delivered on zoom.us on 13th September 2020 for Trainees & trainers at Pakistan. Acknowledged for few text material & pictures taken from google.com and E Blecks book on Cerebral Palsy.
Bryan L. Reuss, M.D., presents "Management of Distal Biceps Injuries in Athletes" at the 2013 9th Annual Cutting Edge Concepts in Orthopaedics & Sports Medicine Seminar presented by Orlando Orthopaedic Center Foundation.
A course Review from James Moore's Sporting Hip and Groin Course - February 2016 (Highly Recommend!). Following my attendance of the course, i performed my own research on 'The Sporting Hip and Groin' and incorporated this into the course review which I presented to the Sports Science and Medicine staff at Wigan Athletic FC. Further references available upon request.
This talk evaluates specific components of the history, physical examination and imaging in patients with gluteal pain to identify a specific diagnosis
In this presentation I summarize research on Endobutton-assisted repair of the distal biceps tendon. Find out more about hand and arm problems at http://www.noelhenley.com
This is the presentation which was delivered to third year Bachelor of Physiotherapy students at Kathmandu University School of Medical Sciences (KUSMS), Dhulikhel, Nepal. Different soft tissue injuries are the part of curriculum for the undergraduate students at KUSMS.
Prof. Anisuddin Bhatti describes spasticity management in Cerebral Palsy patients. Botulinum Toxin (BOTOX) therapy and its application techniques live demonstration given. lectured delivered on zoom.us on 13th September 2020 for Trainees & trainers at Pakistan. Acknowledged for few text material & pictures taken from google.com and E Blecks book on Cerebral Palsy.
Bryan L. Reuss, M.D., presents "Management of Distal Biceps Injuries in Athletes" at the 2013 9th Annual Cutting Edge Concepts in Orthopaedics & Sports Medicine Seminar presented by Orlando Orthopaedic Center Foundation.
A course Review from James Moore's Sporting Hip and Groin Course - February 2016 (Highly Recommend!). Following my attendance of the course, i performed my own research on 'The Sporting Hip and Groin' and incorporated this into the course review which I presented to the Sports Science and Medicine staff at Wigan Athletic FC. Further references available upon request.
This talk evaluates specific components of the history, physical examination and imaging in patients with gluteal pain to identify a specific diagnosis
In this presentation I summarize research on Endobutton-assisted repair of the distal biceps tendon. Find out more about hand and arm problems at http://www.noelhenley.com
The term Spinal Cord Injury is used to refer to neurological damage of the spinal cord
Any lesion involving the spinal cord result a syndrome called a “myelopathy”
Spinal cord injuries are defined as complete or incomplete according to the International Standards for the Neurological Classifification of SCI and the American Spinal Injuries Association Impairment Scale (AIS)
Complete lesions are defifined as AIS A, and incomplete lesions are defifined as AIS B, AIS C, AIS D or AIS E (Harvey, 2016)
Steppage gait (High stepping, Neuropathic gait) is a form of gait abnormality characterised by foot drop or ankle equinus due to loss of dorsiflexion. The foot hangs with the toes pointing down, causing the toes to scrape the ground while walking, requiring someone to lift the leg higher than normal when walking
ADVANCED UPPER LIMB ORTHOTIC MANAGEMENT IN STROKE PPT.pptxDibyaRanjanSwain3
In this ppt we have included stroke and its types and causes and advanced orthotic management of stroke for upper extrimity. like shoulder orthosis, elbow orthosis, wrist and hand orthosis and also electrical stimulation. also the biomechanics of shoulder orthosis and elbow and wrist hand orthosis also included.
Post Polio Residual Palsy: Pathophysiology & Principles of RxAnisuddin Bhatti
Prof. Anisuddin Bhatti, Paeds Orthop Surgeon delivered lecture on Post Polio paralysis and deformities Part 1 on Pathophysio and principles of treatment, through Dr. Ziauddin University Hospital Clifton Karachi webinar on googel.meet, on 3rd April 2021. Acknowledge for material taken from Research papers, slideshare and books as referred in reference list.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
2. stroke
ischemic
disruption of blood flow to
a portion of the brain. This
usually stems from a blood
clot in a blood vessel in the
neck or brain causing cell
damage in that area
hemorrhagic
result of
bleeding into
the brain,
causing injury
to brain tissue.
3.
4.
5. Signs and symptoms
• Hemiparesis, monoparesis, or (rarely) quadriparesis
• Hemisensory deficits
• Monocular or binocular visual loss
• Visual field deficits
• Diplopia
• Dysarthria
• Facial droop
• Ataxia
• Vertigo (rarely in isolation)
• Aphasia
• Sudden decrease in the level of consciousness
6. Diagnosis
• CT angiography and CT perfusion
scanning
• Magnetic resonance imaging (MRI)
• Carotid duplex scanning
• Digital subtraction angiography
7. Diagnosis
Laboratory test
• Complete blood count (CBC): A baseline study
that may reveal a cause for the stroke or provide
evidence of concurrent illness (eg, anemia)
• Coagulation studies: May reveal a coagulopathy
and are useful when fibrinolytics or
anticoagulants are to be used
• Toxicology screening: May assist in identifying
intoxicated patients with symptoms/behavior
mimicking stroke syndromes
8. Differential Diagnosis
Stroke mimics commonly confound the clinical diagnosis of stroke.
One study reported that 19% of patients diagnosed with acute
ischemic stroke by neurologists before cranial CT scanning actually
had non-cerebrovascular causes for their symptoms.
The most frequent stroke mimics include the following:
• Seizure (17%)
• Systemic infection (17%)
• Brain tumor (15%)
• Toxic-metabolic disorders, such as hyponatremia and
hypoglycemia (13%)
• Positional vertigo (6%)
• Conversion disorder
9. Nonmodifiable:
• Age
• Race
• Sex
• Ethnicity
• History of migraine
headaches
• Fibromuscular dysplasia
• Heredity: Family history of
stroke or transient
ischemic attacks (TIAs)
Modifiable:
• Hypertension
• Diabetes mellitus
• Cardiac disease
• Hypercholesterolemia
• TIAs
• Lifestyle issues
• Obesity
• Oral contraceptive
use/postmenopausal
hormone use
• Sickle cell disease
10. • Paralysis or loss of muscle movement
– usually on one side of body(HEMIPLGIA)
– loss of sensation on one side of body
– lose control of certain muscles
• Difficulty talking or swallowing
– difficulty with language (aphasia), including speaking or
understanding speech, reading, or writing
– less control over the way the muscles in mouth and throat
move
• Memory and concentration difficulties
– experience some memory loss
– Others may have difficulty thinking, making judgments,
reasoning and understanding concepts.
11. • Emotional problems
– more difficulty controlling their emotions
– psychological problems such as anxiety or depression
• Extreme tiredness and sleep problems
• Problems with vision, such as double vision or
partial blindness
• Difficulty controlling bladder and bowel
movements (incontinence or constipation)
• Changes in personality, behaviour and self care
ability
– more withdrawn
– less social or more impulsive
– need help with grooming and daily chores.
12. • Pain
– pain, numbness or other strange sensations in parts of the bodies
affected by stroke
– For example: if a stroke causes lose feeling in left arm, itcan
develop an uncomfortable tingling sensation in that arm.
– sensitive to temperature changes, especially extreme cold
(central stroke pain or central pain syndrome)
• Dynamic balance and gait symmetry
– The former has been linked to fall risk, whereas the later has
been associated with both fall risk and poor balance.
– drop foot which is a lack of dorsiflexion during the swing phase of
gait and equinovarus deformity.
– lack of knee and hip stability (an incorrect ankle position during
their gait cycle).
– toe contact at the initial stance phase of gait.
– unaffected side is always turned forward
– equinovarus deformity of the foot and ankle often accompanied
by a hyperextension or recurvatum at the knee joint
13. • Bed sores (pressure ulcers)
• Deep vein thrombosis (DVT)
• Pneumonia
• Contractures (altered position of your
hands, feet, arms or legs because of muscle
tightness)
14.
15. • Prevent secondary impairments
• Restore performance skills
• Modify activity demands and the contexts
in which activities are performed
• Promote a healthy and satisfying lifestyle
• Maintain performance and health
16.
17. Intervention to Prevent Secondary
Impairments
• Abnormal changes in postural alignment (postural deformities)
– using available motor control in the affected and nonaffected limbs to begin a
self-exercise program designed to stretch muscles gently throughout the body
• Pain associated with immobility or abnormal joint alignment
• Learned nonuse
– Therapists use every opportunity to teach the stroke survivor to be aware of
and to use the paretic limbs to the limits of current available motor function.
• Injury due to falls
– develop strategies for adjusting to shifts in their body's center of mass to
enhance their balance skill and efficacy
• Aspiration during feeding, eating, and swallowing
– use techniques to improve sensation, strength, and muscle tone of oral
structures to maximize the potential for safe of independent eating.
• Depression following stroke
– by promoting independence, autonomy, participation
18.
19. Intervention to Restore
Performance Skills
• To generalize their new skills to enhanced performance of
activities in their daily lives
• Cognitive skills include the abilities to attend to
environmental stimuli; remember relevant information;
plan, organize, and sequence activity performance; and
assess actions.
• Perceptual skills include the abilities to interpret sensory
information and navigate the spatial environment.
• Emotional coping skills include a core of effective strategies
that stroke survivors must develop to negotiate their
interactions with others and return to full participation in
their communities
20.
21. Intervention to Modify Activity Demands
and the Contexts in Which Activities Are
Performed
• Environmental Modifications
– Depend on each client's ambulation status and
capacity to use the paretic arm
• Adaptive Equipment
– Equipment selection is highly individualized and is
based on the constellation of factors assessed in
the occupational therapy evaluation
22.
23. Intervention to Promote a Healthy
and Satisfying Lifestyle
• Help stroke survivors establish performance
patterns in:
– Medication routine
– Appropriate diet
– Appropriate levels of physical activity
– Satisfying levels of engagement in social
relationships and activities
24.
25. Intervention to Maintain Performance
and Health
• Education of clients, family, and caregivers to
maintain performance and health after
services have ended
• Establish active, healthy daily routines:
– Maintaining the performance capacities
– Preventing an avoidable decline toward inactivity,
loss of social roles, and emotional depression.
27. PROSTHETIST & ORTHOTIST
• The role are
– To assess patient condition.
– To produce (prescribe and design) suitable
devices for patient.
– To fit and help patient learn to wear the
devices.
– To educate regarding the use and care of an
appropriate orthosis/prosthesis that serves
an individual’s requirements.
28. LOWER EXTREMITY
• In general ankle foot orthosis(AFO) is given to
improves stroke patient’s stability, safety, and
efficiency of walking.
• Types of orthosis given based on
– patient condition.
– muscle strength in ankle, calf, and lower leg.
• In order to improve biomechanical correction of
deformity, the footplate will be put on the base
of polypropylene AFO.
29. AFO candidates
Patient with drop
foot or unable to
lift their leg to
swing
To prevent foot
dragging on the
ground when walk
Patient with knee
recurvatum
Aid in managing
and controlling
recurvatum of the
knee.
31. • Knee ankle foot orthosis (KAFO) not a suitable
choice for stroke patient.
• Reason:
– Heavy
– No strength to lift it to walk properly.
– Can control the problem at the knee by correctly
addressing the problem at the foot and ankle.
32. UPPER EXTREMITY
• The majority of orthotic prescriptions for upper
extremity stroke patients are for the wrist, hand or
both.
Preserve functional
position of hand
and wrist , keeping
it in a neutral
position
No
moveable
parts
Static
splinting
33. Splint
• Sling
Resting splint
Patient with low
muscle tone.
Anti-spastic
splint
Patient with high
muscle tone.
- Support the arm when sublaxation occur.
- Used to hold joint and assist in long term
stability
34. Intervention
of CPO and
OT in stroke
patient Lower
extremity
Upper
extremity
35. • Historically, OT has primarily been involved in
the provision of upper extremity orthotic.
• In upper extremity orthotic practice for stroke
patient, OT typically design, fabricate, fit and
supervise functional training.
• OT manage every stage of the upper extremity
orthotic delivery process therefore able to
adapt each step to individual need in stroke
patient
36. • In contrast, OT are not direct providers of
lower extremity orthotic care.
• Orthotist design, fabricate and fit lower
extremity and PT provide functional gait
training with the orthotics.
• OT collaborates in the delivery of the lower
extremity orthotic services to ensure that the
orthosis is designed to facilitate occupational
performance at each stage of development.
37. • The orthosis may address a biomechanical goal
such as providing a stable base of support and a
functional gait training goal such as increasing the
ankle dorsiflexion during toe-off.
• However, if the orthosis does not address the
occupational performance goal (such as donning
and doffing the device independently) the person
may discard the orthosis.
• Thus, OT plays important role to anticipate such
patient’s performance issues and initiate effective
interventions before design and fabrication
decisions have been completed by the orthotist.
38. Designing the orthosis
• The OT and orthotist works closely during the design phase.
• Considerations must be made based on:
Patient’s strength
• the weight and force required to use the device
Fine motor strength and coordination
• Design and material strapping system
Skin integrity and sensation
• Material selection
Status of activities of daily living and functional capabilities
• Donning and doffing
39. • Although the orthotics devices is made by the
orthotist, the occupational therapist is
typically the one who teaches the user how to
perform daily activities while wearing it.