Regular exercise leads to several long-term effects on the cardiovascular and respiratory systems. The heart muscle thickens and increases in size, raising stroke volume and cardiac output. This allows the heart to pump more blood with each beat and a lower resting heart rate. Capilliarization improves oxygen delivery to working muscles. Blood volume and aerobic fitness increase while resting blood pressure and recovery time decrease. The muscular system also adapts through increased myoglobin and mitochondria stores, glycogen and fat storage, tendon and muscle strength, and tolerance to lactic acid buildup.
Effects of Exercise on Cardiovascular SystemAdam Sturm
The benefits of regular exercise include more than just having a well-toned body. Besides serving as a mood intensifier, physical activity has both long term and short term effects on the cardiovascular system. Good blood circulation throughout your body may provide long standing positive effects to your health. Giving your body a temperate workout for 30 minutes at least 5 days a week may help mitigate the chance of developing many heart-related diseases.
Effect of exercise on Cardiovascular system.
introduction.
type of exercise.
a) based on contraction of muscle.
b) based on the type of metabolism.
c) based on the severity of exercise.
effect of exercise on cardio vascular system:-
a) on blood.
b) on blood volume.
c) on heart rate.
d) on cardiac output.
e) on venous return.
f) on blood flow to skeletal muscles.
g) on blood pressure.
Blood pressure after exercise.
vivekanand quotes.
thank you.
Dear all,
This ppt includes the acute and chronic effect of exercise on different body system which includes musculoskeletal systems, cardiovascular systems, respiratory system, endocrive system, psychological effects etc. I hope this is helpful for you.
Thank you
CVS in exercise - SPORTS PHYSIOLOGY
Cardiovascular system and the influence of exercises on it The effects of exercise on cardiovascular system can be determined it by :-
1. The effect on heart size,
2. The effect on plasma volume ,
3. The effect on stroke volume,
4. The effect on heart rate ,
5. The effect on cardiac output ,
6. The effect on oxygen extraction ,
7. The effect on blood flow and distribution
8. The effect on blood pressure
Term 1 How does the body respond to aerobic training?
The basis of aerobic training
Immediate physiological responses to training
Physiological adaptations in response to aerobic training
Effects of Exercise on Cardiovascular SystemAdam Sturm
The benefits of regular exercise include more than just having a well-toned body. Besides serving as a mood intensifier, physical activity has both long term and short term effects on the cardiovascular system. Good blood circulation throughout your body may provide long standing positive effects to your health. Giving your body a temperate workout for 30 minutes at least 5 days a week may help mitigate the chance of developing many heart-related diseases.
Effect of exercise on Cardiovascular system.
introduction.
type of exercise.
a) based on contraction of muscle.
b) based on the type of metabolism.
c) based on the severity of exercise.
effect of exercise on cardio vascular system:-
a) on blood.
b) on blood volume.
c) on heart rate.
d) on cardiac output.
e) on venous return.
f) on blood flow to skeletal muscles.
g) on blood pressure.
Blood pressure after exercise.
vivekanand quotes.
thank you.
Dear all,
This ppt includes the acute and chronic effect of exercise on different body system which includes musculoskeletal systems, cardiovascular systems, respiratory system, endocrive system, psychological effects etc. I hope this is helpful for you.
Thank you
CVS in exercise - SPORTS PHYSIOLOGY
Cardiovascular system and the influence of exercises on it The effects of exercise on cardiovascular system can be determined it by :-
1. The effect on heart size,
2. The effect on plasma volume ,
3. The effect on stroke volume,
4. The effect on heart rate ,
5. The effect on cardiac output ,
6. The effect on oxygen extraction ,
7. The effect on blood flow and distribution
8. The effect on blood pressure
Term 1 How does the body respond to aerobic training?
The basis of aerobic training
Immediate physiological responses to training
Physiological adaptations in response to aerobic training
Building Elite Cardio for Competative Boxingringsiderising
If you train hard for boxing and your cardio still isn't great, there is a reason. Your training regimen isn't taking energy systems into account. If you aren't training your aerobic system to work with your anaerobic alactic and anaerobic lactic systems, then you may as well be training to not be the best. So if you LIKE losing and gassing out, skip this e-book. For all of those people who want to separate themselves from the mediocre fighters with ok conditioning, this e-book will put you on the right track. You're welcome!
The only person you are destined to become is the person you decide to be. The last three or four reps is what makes the muscle grow. This area of pain divides a champion from someone who is not a champion.
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Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Novas diretrizes da OMS para os cuidados perinatais de mais qualidade
Sport unit 2 p4
1. Assessment Activity 4 – Stepping Up To The Plate…
Unit 2: The Physiology of Fitness
P4 – Describe the Long-Term Effects of Exercise on the Cardiovascular and Respiratory System
2. Cardiovascular System:
Cardiac Hypertrophy – The thickening of the heart muscle which results in a decrease in size of the chamber of the heart, including
the left and right ventricles. A common cause of cardiac hypertrophy is high blood pressure.
Increase In Stroke Volume – Stroke Volume is the amount of blood pumped out per beat. Your stroke volume is increased and your
resting heart is decreased. Increase in stroke volume can lead to increase in cardiac output.
Increase In Cardiac Output – Cardiac Output is the amount of blood pumped around the body per minute. To work out your
maximum heart rate, is 220 – age (in years). Over a long period of time your cardiac output increases because your heart rate and
stroke volume has increased so your heart gets used to the rate of work it has to maintain.
Decrease In Resting Heart Rate – When you exercise your heart gets bigger, which means you can pump more blood which results in
less beats. As your heart rate increases in volume, this means that you can last longer when you are exercising.
Capillarisation – The capillarisation of cardiac and skeletal muscles improves sustained aerobic exercise meaning the amount of
oxygen and energy getting to the muscle in increased. It increases the blood flow to specific areas, depending on what part of the
body you are exercising.
Increase In Blood Volume – Blood Volume is the amount of blood that is in the body, this can be calculated by determining the
volumes of red blood cells and plasma cells. An increase in blood volume, as when there is retention of water and salt in the body
because of renal failure, results in an increase in cardiac output.
Reduction In Resting Blood Pressure – The pressure of the blood in the vessels, especially the arteries, as it circulates through the
body. Blood pressure varies with the strength of the heartbeat, the volume of blood being pumped, and the elasticity of the blood
vessels.
Decreased Recovery Time – The fitter your heart, the quicker it returns to normal after exercise. Fitter individuals generally recover
more rapidly because their cardiovascular system can adapt more quicker to impose demands of exercise.
Increased Aerobic Fitness – This is where a performer has trained over an extensive period of time in long training sessions such as
running or cycling. This helps increases the performers aerobic fitness because it would increase the efficiency and endurance
capability of the heart and lungs.
3. Muscular System:
Cardiac Hypertrophy – This is the thickening of the heart muscle, as the wall of the heart thickens it becomes stronger, less blood is
going into the heart but because the heart is so powerful it does not need to work as hard.
Increase In Tendon Strength – Exercise increases tendon strength by stretching and contracting and putting strain on the tendon. this
makes the tendon active and replace cells with new ones. If you have damaged it before the body will be stimulated into making
stronger cells that before.
Increase In Myoglobin Stores – Myoglobin is a protein within the muscle tissue which acts as an oxygen carrier. As a long term effect
of exercise, the ability of the muscles to store myoglobin is increased. Muscles increase their oxidative capacity through regular
exercise, the myoglobin stores also increase because they get used to the demands of exercise and work placed upon them so
increase stores as they will be needed.
Increased In Number Of Mitochondria – Mitochondria enable cells to produce 15 times more ATP than they could, humans need
large amounts of energy in order to survive. The number of mitochondria present in a cell depends upon the metabolic requirements
of that cell, and may range from a single large mitochondrion to thousands of the organelles.
Increased Storage Of Glycogen And Fat – Glycogen is made primarily by the liver and the muscles, but can also be made by
glycogenesis within the brain and stomach.Liver cells which on demand, are ready to break down their stored glycogen into glucose
and send it through the blood stream as fuel for the brain or muscles.
Fats are stored in the marrow of bones, heart, lungs, liver, spleen, kidneys, intestines, muscles and lipid rich tissues of the central
nervous system.
Increased Muscles Strength – Muscles Strength can be increased by weight, isometric and resistance training which strengthens the
muscles. Strength training can provide benefits and improvements in a performers health and well-being, including increased bone,
muscle, tendon and ligament strength and toughness, improved joint function, reduced potential for injury, increased bone density, a
temporary increase in metabolism and improved cardiac function.
Increased Tolerance To Lactic Acid – Lactic acid is formed when an athlete exercises, if the performer is exercising at a level where
more oxygen is being used than can be replenished to their system. Lactic acid tolerance training will make your body more efficient
at reprocessing the waste products of exercise, transporting oxygen to your blood and allowing you to exercise at your highest point
for longer time.