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STROKE
-PURVI SHAH
(B.P.T)
Brain Parts & Function
Circle of willis
ACA/MCA/PCA Supply
Definition
• It is a focal neurological deficit from CVA lasting more than 24
h.
• Brain injury caused by when the blood vessels to the brain cut,
burst, laceration as a result of blood supply & o2 to brain is
decresed.
Types
Ischemic Haemorrhagic
Types
ISCHEMIC
• 80 %
• Block the blood flow as a
result of loss of blood supply
& o2
HAEMORRHAGIC
• 20 %
• When the blood vessels
rupture, causing leakage of
blood in & around brain
Ischemic Stroke
• ETIOLOGY :
1.Atherosclerosis
2.cerebral thrombus
3.cerebral embolism
4.embolism of heart
Atherosclerosis
HAEMORRHAGIC STOKE
• ETIOLOGY :
1.ICH
2.SAH
3.Intracrainal small blood vessels disease
4.Aneurysm
5.Aeteriovenous malformation
Risk Factor
• MODIFIABLE :
Smoking / Alcohol consumption
Obesity
Lack of physical activities
Infection
Psychological factor
Risk Factor
• NON MODIFIABLE :
AGE
Gender
Positive family history
Blood pathology
TIA
HTN & Diabetes
Heart disease
ACA SYNDEOME
• Contralateral hemiparesis in LE> UE, face
• Contralateral hemisensory loss
• Urinary incontinence
• Problem with limitation & bimanual tasks, apraxia
• Abulia, slowness, delay, lack of spontaneity motor action
• Contalateral grasp reflex & sucking reflex can be
asymptomatic
MCA SYNDROME
• Contralateral hemiparesis UE>face, LE
• Contralateral hemisensory
• Broca’s aphasia
• Receptive aphasia
• Global aphasia
• Perceptual deficits
• Limb kinetic apraxia
• Contralateral homonymous hemianopia
• Pure motor hemiplegia
PCA SYNDROME
PERIPHERAL TERRITOEY
• Contralateral & bilateral
homonymous hemianopia
• Visual agnosia
• Prospagnosia
• Dyslexia,agraphia,anomia
• Memory defect
• Topographic disorientation
CENTRAL TERRITORY
• Central post stroke
• Involuntary movement
• Contralateral hemiplegia
• Webber’s syndrome
• Paresis of vertical eye
movement, miosis,ptosis
HEMIPLEGIC GAIT
• CIRCUMDUCTORY GAIT
• Circumduction seen in swing phase to clear ground
• Poor proprioception
• Spasticity
• Weakness
• Flexion contracture
• Abnormal synergy pattern
• Equinus gait, varus deformity (ankle), hammer toe
• Unequal stride & step length
• Insufficient pelvic rotation
Pathophysiology
Plaque formation in the arterial wall
Cerebral blood flow & o2 reduced
Ischemic core tissue die, in longer time surrounding
penumbra survive but without reperfusion it’s die
Neuronal activity reduce & infarct expanded
Damage brain cells
PHYSIOTHERAPY
• In ACTUE stage, low intensity rehabilitation begun.
• Patient is medically stabilized within 72 h.
POSITIONING STRATEGIES
FOR RESPIRATORY FUNCTION
• BREATHING EXERCISE, PD, FET, Chest Expansion
Exercise
IMPROVE FLEXIBILITY & JOINT
INTEGRITY
• AAROM & PROM
• Scapula mobilization
• Safe self ROM activity such as arm cradling, table top
polishing, stretching & splinting
REEDUCATE SENSORY FUNCTION
• Presentation of repeated sensory stimuli like pressure,
vibration, touch, temp
• Weight bearing & joint approximation tech
• PNF
• Use bilateral extremity
SPASTICITY MANAGEMENT
• Ice
• Splinting
• Weight bearing ex
• Neural warmth
• Massage
• Inhibitory tech such as stoking, pressure on tendentious
insertion of muscle
IMPROVE STRENGTH
• Both agonist & antagonist muscles
• For weak muscle <3/5 gravity eliminated plan & for >3/5
against gravity
• Use therabands, weight cuff, dumbells
IMPROVE MOVEMENT CONTROL
• Task oriented exercise
• CIMT
• MCIMT
• NMES
• EMG Biofeedback
• EFS
• Postural control & bed mobility
Rolling, transfer activity, sit to stand
• For improve UL function
Lifting activity, push up, manipulation activity, weight
bearing ex, pick up the objects
• For improve LL function
PNF pattern, improve pelvic/knee & ankle control
FOR PUSHER SYNDROME
• Use visual stimuli, verbal cues
• Use an environmental boundary corner or doorway
• Therapist sit towards the stronger side & ask the patient lean
over me
FOR SHOULDER PAIN
• Two types of pain
1.SPASTIC
2.FLACCID
• Treatment
Proper handling, splinting, perper
positioning, taping & sling, EMG, NMES
IMPROVE BALANCE
• Symmetrical weight bearing on both side
• Wide BOS to Narrow BOS
• Stable surface to dynamic surface
• Postural perturbation
• Later on reaching activity, dual tasks training, divert
attention
LATER ON MORE CHELLENGEBLE ACTIVITY
IMPROVE GAIT
• Initially in parallel bar with help of crutches
• Later on out of parallel bar with or without crutches
• Use hemiplegic wheelchair
Assistive devices: crutches, cane
Orthosis : HKAFO/KAFO/AFO ( depending upon
weakness)
Correct gait pattern with help to verbal cues, visual feedback
& manually by therapist
AEROBIC ACTIVITY
• Functional activity
• Treadmill training
• Cycle ergometer
• Graded exercise training
• Proper & adequate rest
IMPROVE FEEDING & SWALLOWING
• Exercise of jaw, lips, tongue
• Facial exercise
• Rubbing of ice chips around lips
Thank You

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Purvi shah stroke ppt

  • 2. Brain Parts & Function
  • 5. Definition • It is a focal neurological deficit from CVA lasting more than 24 h. • Brain injury caused by when the blood vessels to the brain cut, burst, laceration as a result of blood supply & o2 to brain is decresed.
  • 7. Types ISCHEMIC • 80 % • Block the blood flow as a result of loss of blood supply & o2 HAEMORRHAGIC • 20 % • When the blood vessels rupture, causing leakage of blood in & around brain
  • 8. Ischemic Stroke • ETIOLOGY : 1.Atherosclerosis 2.cerebral thrombus 3.cerebral embolism 4.embolism of heart
  • 10. HAEMORRHAGIC STOKE • ETIOLOGY : 1.ICH 2.SAH 3.Intracrainal small blood vessels disease 4.Aneurysm 5.Aeteriovenous malformation
  • 11. Risk Factor • MODIFIABLE : Smoking / Alcohol consumption Obesity Lack of physical activities Infection Psychological factor
  • 12. Risk Factor • NON MODIFIABLE : AGE Gender Positive family history Blood pathology TIA HTN & Diabetes Heart disease
  • 13. ACA SYNDEOME • Contralateral hemiparesis in LE> UE, face • Contralateral hemisensory loss • Urinary incontinence • Problem with limitation & bimanual tasks, apraxia • Abulia, slowness, delay, lack of spontaneity motor action • Contalateral grasp reflex & sucking reflex can be asymptomatic
  • 14. MCA SYNDROME • Contralateral hemiparesis UE>face, LE • Contralateral hemisensory • Broca’s aphasia • Receptive aphasia • Global aphasia • Perceptual deficits • Limb kinetic apraxia • Contralateral homonymous hemianopia • Pure motor hemiplegia
  • 15. PCA SYNDROME PERIPHERAL TERRITOEY • Contralateral & bilateral homonymous hemianopia • Visual agnosia • Prospagnosia • Dyslexia,agraphia,anomia • Memory defect • Topographic disorientation CENTRAL TERRITORY • Central post stroke • Involuntary movement • Contralateral hemiplegia • Webber’s syndrome • Paresis of vertical eye movement, miosis,ptosis
  • 16. HEMIPLEGIC GAIT • CIRCUMDUCTORY GAIT • Circumduction seen in swing phase to clear ground • Poor proprioception • Spasticity • Weakness • Flexion contracture • Abnormal synergy pattern • Equinus gait, varus deformity (ankle), hammer toe • Unequal stride & step length • Insufficient pelvic rotation
  • 17.
  • 18. Pathophysiology Plaque formation in the arterial wall Cerebral blood flow & o2 reduced Ischemic core tissue die, in longer time surrounding penumbra survive but without reperfusion it’s die Neuronal activity reduce & infarct expanded Damage brain cells
  • 19. PHYSIOTHERAPY • In ACTUE stage, low intensity rehabilitation begun. • Patient is medically stabilized within 72 h.
  • 21. FOR RESPIRATORY FUNCTION • BREATHING EXERCISE, PD, FET, Chest Expansion Exercise
  • 22. IMPROVE FLEXIBILITY & JOINT INTEGRITY • AAROM & PROM • Scapula mobilization • Safe self ROM activity such as arm cradling, table top polishing, stretching & splinting
  • 23. REEDUCATE SENSORY FUNCTION • Presentation of repeated sensory stimuli like pressure, vibration, touch, temp • Weight bearing & joint approximation tech • PNF • Use bilateral extremity
  • 24. SPASTICITY MANAGEMENT • Ice • Splinting • Weight bearing ex • Neural warmth • Massage • Inhibitory tech such as stoking, pressure on tendentious insertion of muscle
  • 25. IMPROVE STRENGTH • Both agonist & antagonist muscles • For weak muscle <3/5 gravity eliminated plan & for >3/5 against gravity • Use therabands, weight cuff, dumbells
  • 26. IMPROVE MOVEMENT CONTROL • Task oriented exercise • CIMT • MCIMT • NMES • EMG Biofeedback • EFS
  • 27. • Postural control & bed mobility Rolling, transfer activity, sit to stand • For improve UL function Lifting activity, push up, manipulation activity, weight bearing ex, pick up the objects • For improve LL function PNF pattern, improve pelvic/knee & ankle control
  • 28. FOR PUSHER SYNDROME • Use visual stimuli, verbal cues • Use an environmental boundary corner or doorway • Therapist sit towards the stronger side & ask the patient lean over me
  • 29. FOR SHOULDER PAIN • Two types of pain 1.SPASTIC 2.FLACCID • Treatment Proper handling, splinting, perper positioning, taping & sling, EMG, NMES
  • 30. IMPROVE BALANCE • Symmetrical weight bearing on both side • Wide BOS to Narrow BOS • Stable surface to dynamic surface • Postural perturbation • Later on reaching activity, dual tasks training, divert attention LATER ON MORE CHELLENGEBLE ACTIVITY
  • 31.
  • 32. IMPROVE GAIT • Initially in parallel bar with help of crutches • Later on out of parallel bar with or without crutches • Use hemiplegic wheelchair Assistive devices: crutches, cane Orthosis : HKAFO/KAFO/AFO ( depending upon weakness) Correct gait pattern with help to verbal cues, visual feedback & manually by therapist
  • 33. AEROBIC ACTIVITY • Functional activity • Treadmill training • Cycle ergometer • Graded exercise training • Proper & adequate rest
  • 34. IMPROVE FEEDING & SWALLOWING • Exercise of jaw, lips, tongue • Facial exercise • Rubbing of ice chips around lips