This document discusses polycystic ovarian disease and its homoeopathic approach. It begins with descriptions of normal ovaries and their attachments. It then covers the histology, stages of development, and menstrual cycle as they relate to the ovaries. The document discusses polycystic ovaries in depth, including epidemiology, history, etiologies, pathophysiology, clinical features, investigations, differential diagnosis, and therapeutic medicines in homeopathy. Key points covered include the Rotterdam criteria for diagnosis of PCOS and common symptoms like menstrual irregularity and hyperandrogenism.
PCOS (Polycystic ovary syndrome), a hormonal disorder causing enlarged ovaries with small cysts, or fluid-filled sacs. It is a condition in which a woman's hormones are out of balance. It's a health problem that affects 1 in 10 women of childbearing age. Over the years, numerous hypothesis have been proposed regarding the proximate physiological origin for PCOS. Difference between PCOD & PCOS is important to know. A common confusion among women, is understanding the difference between having PCOS & having been diagnosed with it.
Various researches have studied the prevalence of PCOS in India (Tamil Nadu, Mumbai, Karnataka & Lucknow). Maintaining a good health is essential to prevent as well as treat hormonal disturbances & conditions. Management of these both at risk for PCOS and those with a confirmed PCOS diagnosis includes education, healthy lifestyle and therapeutic interventions targeting their symptoms.
Pcos by dr alka mukherjee dr apurva mukherjee nagpur m.s.alka mukherjee
Polycystic ovary syndrome (PCOS) is a highly prevalent endocrine-metabolic disorder that implies various severe consequences to female health, including alarming rates of infertility. Although its exact etiology remains elusive, it is known to feature several hormonal disturbances, including hyperandrogenemia, insulin resistance (IR), and hyperinsulinemia. Insulin appears to disrupt all components of the hypothalamus-hypophysis-ovary axis, and ovarian tissue insulin resistance results in impaired metabolic signaling but intact mitogenic and steroidogenic activity, favoring hyperandrogenemia, which appears to be the main culprit of the clinical picture in PCOS. In turn, androgens may lead back to IR by increasing levels of free fatty acids and modifying muscle tissue composition and functionality, perpetuating this IR-hyperinsulinemia-hyperandrogenemia cycle. Nonobese women with PCOS showcase several differential features, with unique biochemical and hormonal profiles. Nevertheless, lean and obese patients have chronic inflammation mediating the long term cardiometabolic complications and comorbidities observed in women with PCOS, including dyslipidemia, metabolic syndrome, type 2 diabetes mellitus, and cardiovascular disease. Given these severe implications, it is important to thoroughly understand the pathophysiologic interconnections underlying PCOS, in order to provide superior therapeutic strategies and warrant improved quality of life to women with this syndrome.
Polycystic Ovarian Syndrome: Etiology, Diagnosis and ManagementAparajeya Shanker
Polycystic Ovarian Syndrome is a complex disease, with a constellation of etiology and symptoms. Much of the diagnosis is dependent on the laboratory tests of hormones. This presentation provides a concise and focused approach towards PCOS diagnosis and management.
PCOS- An insight into polycystic ovary syndrome
Polycystic ovary syndrome (PCOS) is extremely prevalent and probably constitutes the most frequently encountered endocrine (hormone) disorder in women of reproductive age
Mark Perloe, M.D., Reproductive Endocrinologists, Georgia Reproductive Specialists, shares the latest information about PCOS. For more information, visit www.IVF.com/pcostreat.html.
PCOS (Polycystic Ovary Syndrome) is a combined metabolic and hormonal disorder found in women. Incidences of PCOS appear to be rising and it is now being diagnosed more often.It is seen in as many as 25 to 30% of young women.Unfortunately, due to unfavorable lifestyle changes the number of incidences of PCOS and PCOD (Polycystic Ovarian Disorder) are on rise.
This Presentation Includes
1. What is PCOS?
2. Symptoms of PCOS
3. PCOS risk factors
4. Life Style Factors and PCOS
5. Testing PCOS
6. PCOS linked Infertility
7. Managing PCOS
8. Life Style Changes to manage PCOS
Treatment of Polycystic Ovary Syndrom (PCOS)Dr JP Singh
An Invented technique to treat the PCOS, Introduced by Dr JP Singh. PCOS is a leading cause of women infertility. Near about 50% women at the age group of 15-30 in Kolkata, (India) are suffering from PCOS. Polycystic ovary syndrome is a Gynecological problem that can affect woman's: Menstrual cycle, Difficulty to be pregnant, Hormonal imbalances, Skin and hair problems. It may be treated through this technique. More details logon: www.brainstup.com
For more Info visit www.healthlibrary.com "Management of PCOS in Unani System of Medicine" by Dr. Shaikh Nikhat held on 11th June 2016.
Management of PCOS in Unani System of Medicine - Unani system have the holistic approach to treat the condition like PCOD / PCOS.
PCOS (Polycystic ovary syndrome), a hormonal disorder causing enlarged ovaries with small cysts, or fluid-filled sacs. It is a condition in which a woman's hormones are out of balance. It's a health problem that affects 1 in 10 women of childbearing age. Over the years, numerous hypothesis have been proposed regarding the proximate physiological origin for PCOS. Difference between PCOD & PCOS is important to know. A common confusion among women, is understanding the difference between having PCOS & having been diagnosed with it.
Various researches have studied the prevalence of PCOS in India (Tamil Nadu, Mumbai, Karnataka & Lucknow). Maintaining a good health is essential to prevent as well as treat hormonal disturbances & conditions. Management of these both at risk for PCOS and those with a confirmed PCOS diagnosis includes education, healthy lifestyle and therapeutic interventions targeting their symptoms.
Pcos by dr alka mukherjee dr apurva mukherjee nagpur m.s.alka mukherjee
Polycystic ovary syndrome (PCOS) is a highly prevalent endocrine-metabolic disorder that implies various severe consequences to female health, including alarming rates of infertility. Although its exact etiology remains elusive, it is known to feature several hormonal disturbances, including hyperandrogenemia, insulin resistance (IR), and hyperinsulinemia. Insulin appears to disrupt all components of the hypothalamus-hypophysis-ovary axis, and ovarian tissue insulin resistance results in impaired metabolic signaling but intact mitogenic and steroidogenic activity, favoring hyperandrogenemia, which appears to be the main culprit of the clinical picture in PCOS. In turn, androgens may lead back to IR by increasing levels of free fatty acids and modifying muscle tissue composition and functionality, perpetuating this IR-hyperinsulinemia-hyperandrogenemia cycle. Nonobese women with PCOS showcase several differential features, with unique biochemical and hormonal profiles. Nevertheless, lean and obese patients have chronic inflammation mediating the long term cardiometabolic complications and comorbidities observed in women with PCOS, including dyslipidemia, metabolic syndrome, type 2 diabetes mellitus, and cardiovascular disease. Given these severe implications, it is important to thoroughly understand the pathophysiologic interconnections underlying PCOS, in order to provide superior therapeutic strategies and warrant improved quality of life to women with this syndrome.
Polycystic Ovarian Syndrome: Etiology, Diagnosis and ManagementAparajeya Shanker
Polycystic Ovarian Syndrome is a complex disease, with a constellation of etiology and symptoms. Much of the diagnosis is dependent on the laboratory tests of hormones. This presentation provides a concise and focused approach towards PCOS diagnosis and management.
PCOS- An insight into polycystic ovary syndrome
Polycystic ovary syndrome (PCOS) is extremely prevalent and probably constitutes the most frequently encountered endocrine (hormone) disorder in women of reproductive age
Mark Perloe, M.D., Reproductive Endocrinologists, Georgia Reproductive Specialists, shares the latest information about PCOS. For more information, visit www.IVF.com/pcostreat.html.
PCOS (Polycystic Ovary Syndrome) is a combined metabolic and hormonal disorder found in women. Incidences of PCOS appear to be rising and it is now being diagnosed more often.It is seen in as many as 25 to 30% of young women.Unfortunately, due to unfavorable lifestyle changes the number of incidences of PCOS and PCOD (Polycystic Ovarian Disorder) are on rise.
This Presentation Includes
1. What is PCOS?
2. Symptoms of PCOS
3. PCOS risk factors
4. Life Style Factors and PCOS
5. Testing PCOS
6. PCOS linked Infertility
7. Managing PCOS
8. Life Style Changes to manage PCOS
Treatment of Polycystic Ovary Syndrom (PCOS)Dr JP Singh
An Invented technique to treat the PCOS, Introduced by Dr JP Singh. PCOS is a leading cause of women infertility. Near about 50% women at the age group of 15-30 in Kolkata, (India) are suffering from PCOS. Polycystic ovary syndrome is a Gynecological problem that can affect woman's: Menstrual cycle, Difficulty to be pregnant, Hormonal imbalances, Skin and hair problems. It may be treated through this technique. More details logon: www.brainstup.com
For more Info visit www.healthlibrary.com "Management of PCOS in Unani System of Medicine" by Dr. Shaikh Nikhat held on 11th June 2016.
Management of PCOS in Unani System of Medicine - Unani system have the holistic approach to treat the condition like PCOD / PCOS.
Polycystic ovarian syndrome (PCOS) is a condition
of unexplained hyperandrogenic chronic anovulation
that most likely represents a heterogenous disorder.
About 10% of women in the reproductive age group
suffer from this disorder.
This presentation briefly discuss the polycystic ovary syndrome in terms of pathogenesis, features and management. Then, It moves on to discuss the various guidelines laid down by Endocrine Society in 2013 for the management of patients with polycystic ovary syndrome.
La elaboración de PLE es una herramienta que ayuda a identificar aspectos que intervienen en el proceso de enseñanza-aprendizaje, como la búsqueda de información, su modificación y el compartirla.
What is Polycystic Ovarian Syndrome? Hormonal evaluation, diagnosis and treatment and its relation to infertility. How does one manage PCOS in an infertility setting?
Đối với phụ nữ, nội tiết tố nữ được ví như nhựa sống cho cơ thể. Khi tình trạng mất cân bằng nội tiết tố xuất hiện có thể gây ra những ảnh hưởng lớn đối với cơ thể. Để hiểu rõ về nội tiết tố là gì, tác dụng ra sao, nguyên nhân gây mất cân bằng cách điều trị hiệu quả hãy cùng tham khảo bài viết sau.
Nguồn: Trích https://venusglobal.com.vn/noi-tiet-to-nu/
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Ovulation was initially monitored by conventional methods like BBT, mid luteal serum progesterone and urinary LH.
Nowadays, USG is used for follicular monitoring for both natural and stimulated cycles.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
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2 Case Reports of Gastric Ultrasound
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Polycystic ovarian disease by Dr.Shuchita Chattree
1. Polycystic Ovarian Disease
And Its Homoeopathic Approach
By: Dr. Shuchita Chattree
M.D. (PGR)
Department of Materia Medica
Homoeopathy University, Jaipur
Email: shuchita.chattree@gmail.com
11/09/14 1
3. NNoorrmmaall OOvvaarriieess
Normal size: 5 x 3 x 3cm
:Variation in dimensions can result from
Endogenous hormonal production varies with age and –
(menstrual cycle
Exogenous substances, including GnRH agonists, or –
.ovulation-inducing medication, may affect size
3 11/09/14
5. OOvvaarriiaann AAttttaacchhmmeennttss
5
• Several ligaments hold each ovary in position.
• The largest is called the broad ligament and is attached to the
uterine tubes and uterus.
• The suspensory ligament holds the ovary at the upper end.
• The ovarian ligament is a rounded, cord-like thickening of the
broad ligament.
11/09/14
10. SSttaaggeess ooff DDeevveellooppmmeenntt
• Ovarian follicles – in cortex and consist of oocytes in various stages
of development. Surrounding cells nourish developing oocyte and
secrete estrogens as follicle grows.
• Mature (graafian) follicle – large, fluid-filled follicle ready to expel
secondary oocyte during ovulation.
• Corpus luteum – remnants of mature follicle after ovulation
Produces progesterone, estrogens, relaxin and inhibin untill it
degenerates into corpus albicans.
11/09/14 10
22. CCoorrppuuss LLuutteeuumm
After ovulation, the remaining wall
of the graafian follicle transforms
.into the corpus luteum
The wall of the corpus luteum is
folded and contains granulosa
lutein cells derived from granulosa
.cells which secrete progesterone
22 11/09/14
23. CCoorrppuuss AAllbbeeccaannss
In the absence of
fertilization the corpus
luteum degenerates,
decreases in size and form
the corpus albicans which
consists of dense connective
tissue
23 11/09/14
24. FFoorrmmaattiioonn OOff CCYYSSTT
In female reproductive cycle during follicular phase water starts
accumulating around the egg cell, size increases as more water
.accumulate
Because of accumulation of water Follicle comes to the periphery and
.Release of ovum ovulation occurs. If not fertilized, Menstruation occurs
In case of ovarian cyst this collection of fluid remain, surrounded by a
. very thin wall, within an ovary
Any ovarian follicle that is larger than about two centimeters is termed
.an ovarian cyst
24 11/09/14
27. NNoonn NNeeooppllaassttiicc OOvvaarriiaann EEnnllaarrggeemmeenntt
Follicular Cyst
Corpus Luteum Cyst
Theca Lutein And Granulosa Lutien Cyst
Polycystic Ovarian Syndrome
Endometrial Cyst
Except the last all are functional cysts of the Ovary
and are loosely called CYSTIC OVARY
27 11/09/14
28. PPoollyyccyyssttiicc OOvvaarriieess
:Rotterdam criteria defines
PCO solely on total follicle no. Presence of ≥ 12 follicles
measuring 2-9 mm in diameter Epidemiology
and/or increased ovarian
.volume >10 mL in at least one ovary
28 11/09/14
29. EEppiiddeemmiioollooggyy
Very prevalent disease affecting
between 6.5 and 8% of women
overall
It is prevalent in Young Reproductive
Age group (20-30%)
Prevalence much higher in obese
women (28% versus 5.5%).
11/09/14 29
30. HHiissttoorryy
Originally described by Stein and Leventhal in 1935, first
known as the “Stein-Leventhal syndrome”.
They saw in 7 women with amenorrhoea, hirsutism, and
obesity found to have a polycystic appearance to the
ovaries.
Insulin resistance described later by Burghen (1980)
30 11/09/14
31. EETTIIOOLLOOGGIIEESS
No one is quite sure what causes PCOS, and it is likely to
:be the result of
( Genetic (inherited(1
. Environmental factors(2
(Metabolic disorder (IR(3
31 11/09/14
32. Pathophysiology
Hypothalamic – pituitary abnormalities( 1
that result in gonadotropin – releasing hormone and
.leutinizing hormone dysfunction
A primary enzymatic defect in ovarian or combined ( 2
.ovarian and adrenal steroidogenesis
A metabolic disorder characterized by resistance in ( 3
conjunction with compensatory hyperinsulinaemia that
exert adverse effects on the hypothalamus, pituitary,
.o3v2 aries, and possibly the adrenal glands 11/09/14
33. PPAATTHHOOGGEENNEESSIISS
The ovaries are stimulated to produce excessive amounts of
androgens, particularly testosterone, by either one or a
combination of the following (almost certainly combined with
(.genetic susceptibility
.The release of excessive LH by the anterior pituitary gland
Through hyperinsulinaemia in women whose ovaries are sensitive
.to this stimulus
Alternatively or as well, reduced levels of sex-hormone binding
.globulin(SHBG) can result in increased free androgens 33 11/09/14
35. ABNORMALITIES OF PCOS OVARY
• Increase activity in chromosome CYP17 region leads to
increased p450c17 enzyme and hence increased
androgen synthesis.
• Decrease in chromosomal region CYP19 activity
decreases aromatase enzyme activity and conversion
of androgens to E2 (Estradiol) is reduced.
• This loss of aromatase and E2 biosynthesis has been
proposed to involve dysregulation of signaling within
the follicle leading to follicular arrest. 11/09/14 35
43. Diagnostic Criteria
ASRM/ESHRE, 2003 Criteria
2 out of 3 required
•1. Menstrual Irregularity
•2. Hyperandrogenism (Clinical or Biochemical)
•3. USG – Polycystic ovary
•Exclusion of other etiologies
11/09/14 43
44. Androgen Excess & PCOS society Criteria 2006
•Menstrual irregularity +/- USG - Polycystic ovary.
•Hyperandrogenism.
•Exclusion of other etiologies
44 11/09/14
45. IInnvveessttiiggaattiioonnss::
• History-taking, specifically for menstrual pattern, obesity, hirsutism,
and the absence of breast development.
• BBT (basal body temperature)
• Ultrasonography.
• Serum (blood) levels of androgens (male hormones), including
androstenedione and testosterone may be elevated.
• Serum values of Luteinizing Hormone (LH)
• levels or the ratio between LH : FSH is > 3 : 1
• Laproscopic view
11/09/14 45
59. Hedera helix (common lvy)
• Cystic ovaritis, especially on the
left side.
• Amenorrhea in young girls.
Infrequent menses.
• Menses late, shorter and less
copious. Pre-menstrual
leucorrhea.
• (Murphy)
11/09/14 59
60. Cobaltum nitricum (nitrate of cobalt)
• Lack of libido.
Metrorrhagia. Secondary
amenorrhea.
• Cystic inflammation of
the ovary. Sterility
11/09/14 60
61. Hirudo medicinalis (leech)
Left-sided ovarian pain like
being stabbed.
Brownish leucorrhea two days
before menses.
Menses: too early or late,
heavy or light, painful or less
painful than usual.
Feeling in the pelvis as if
menses would come on two
weeks before due.
Ovarian cysts
11/09/14 61
62. Rhododendron chrysanthum
• Pain in ovaries; agg. in
change of weather.
• Caused rupture of cyst in
right ovary.
• (CLARKE J. H., Dictionary of
Practical Materia Medica)
11/09/14 62
64. MMuurreexx ppuurrppuurreeaa
• Large cyst, supposed to be connected with left ovary,
occupied space between rectum, uterus and vagina, so as
to obliterate posterior cul de sac and almost occlude
vagina; abdomen somewhat distended; confined to her
room and bed for more than a year.
• (HERING C., Guiding Symptoms of our Materia Medica)
11/09/14 64
65. The Important Common Homoeopathic drugs indicated for
:Ovarian cysts are
Bovista
Apis mellifica
Platina
Lycopodium
Thuja
Lachesis
65 11/09/14
66. BBOOVVIISSTTAA
Mind -Enlarged sensation. [Arg.n.]
Awkward; everything falls from
. hands.Sensitive
. Diarrhoea before and during menses
Menses too early and profuse; worse at
night. Voluptuous sensation.
Leucorrhoea acrid, thick, tough,
greenish, follows menses. Soreness of
pubes during menses. Metrorrhagia;
6.6 Parovarian cysts 11/09/14
67. APIS MELLIFICA
Mind -Apathy and indifference. Awkward;
Listless; cannot think clearly. Jealous, fidgety,
. hard to please
Tearfulness. Jealously, fright, rage, vexation,
grief. Cannot concentrate mind when
. attempting to read or study
Ovaritis; worse in right ovary. Menses
suppressed, with cerebral and head
symptoms, especially in young girls.
. Dysmenorrhoea, with severe ovarian pains
67 11/09/14
68. Colocynthis
.Boring pain in ovary
Must draw up double, with great
.restlessness
Round, small, cystic tumous in ovaries
.or broad ligaments
Bearing-down cramps, causing her to
.bend double
68 11/09/14
69. LACHESIS MUTUS (lach.)
Menses too short, too feeble; pains all relieved
by the flow. [Eupion.]
Left ovary very painful and swollen, indurated.
Acts especially well at beginning and close of
menstruation.
Ill effects of suppressed discharges.
Mind.-Great loquacity. Jealous. [Hyos.] Mental
labor best performed at night.
Suspicious; nightly delusion of fire.
11/09/14 69
70. Platina
.Parts hypersensitive
Ovaries sensitive and burn; vaginismus, nymphomania, pruritus
.vulva, ovaritis with sterility
Menses too early, too profuse, dark clotted with spasms and
.painful bearing down and sensitiveness of the parts
Mental troubles associated with suppressed menses
Self exaltation
70 11/09/14
71. THUJA
. Left-sided and chilly
Mind.-Fixed ideas, Emotional sensitiveness; music causes weeping and
.trembling
.[Female.-Vagina very sensitive. [Berb.; Kreos.; Lyssin
Warty excrescences on vulva and perineum. Profuse leucorrhoea; thick,
.greenish
Severe pain in left ovary and left inguinal region. Menses scanty,
; retarded. Polypi
.Ovaritis; worse left side, at every menstrual period
. Profuse perspiration before menses 71 11/09/14
72. BBUUFFOO
Burning heat and pain in the ovaries which extends down the thigh.
Dysmenorrhoea with cysts and hydatids about ovaries.
IIOODDUUMM
Congestion and dropsy of right ovary with dwindling of the mammae.
Dull pressing pain extending to the uterus.
Wedge like pain in the right ovarian region.
LLIILLIIUUMM TTIIGG
Ovarian neuralgia.
Burning pains from ovary up into abdomen and down into thighs.
Shooting pain from left ovary across the pubes or upto the mammary
gland.
11/09/14 72
73. CCOONNIIUUMM MMAACCUULLAATTUUMM
Ovary enlarged, indurated, lancinating pain.
Breast enlarge and become painful before and during menses.
Menses delayed and scanty.
Dysmenorrhoea, with drawing down thigh.
Mammae lax and shrunken, hard painful to touch.
Ill effects of repressed sexual desire or suppressed menses.
KKAALLII BBRROOMMAATTUUMM
Ovarian neuralgia with great nervous uneasiness.
Cystic tumours of ovaries.
Exaggerated sexual desire.
Vomiting with intense thirst after each meal
Fidgety of hands, jerking and twitching o muscles.
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