This document discusses plantar fasciitis, a painful inflammatory condition of the plantar fascia in the sole of the foot. It defines plantar fasciitis and discusses its common causes such as deterioration or trauma to the plantar fascia. It also outlines the epidemiology, anatomy, biomechanics, pathology, symptoms, diagnostic assessments including imaging, differential diagnosis, and management approaches for plantar fasciitis such as physical therapy, orthotics, night splints, stretching, modalities, and surgery.
Evolution of tunnel placement in ACL reconstructionDhananjaya Sabat
One of my talks at Delhi Arthroscopy Club....... this presentation provides a insight regarding the conceptual evolution in tunnel placement during ACL reconstruction.
Evolution of tunnel placement in ACL reconstructionDhananjaya Sabat
One of my talks at Delhi Arthroscopy Club....... this presentation provides a insight regarding the conceptual evolution in tunnel placement during ACL reconstruction.
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This presentation goes in depth about Primary and Recurrent Patellar dislocation. Its cause, clinical and radiographic evaluation and various modalities of management with update from latest literature.
Acl Reconstruction Surgery In Delhi Dr. Shekhar Srivastav 09971192233DelhiArthroscopy
ACL Reconstruction Surgery in Delhi by Dr. Shekhar Srivastav - Dr. Shekhar Srivastav is an Orthopedic Surgeon attached to Sant Parmanand Hospital, Delhi with special interest in Knee & Shoulder surgery. After obtaining his M.S. Orthopedics degree he has undergone training in various centers in India and Abroad which has helped him in understanding the Orthopedics problems and their Management. He did his AO/ ASIF fellowship at University Hospital, Salzburg, Austria in 2006 and recieved training in Arthroscopy & Sports Medicine at TUM, Munich (Germany) & Rush Orthopedics Centre, Chicago( USA). He has an experience of more than fifteen years of diagnosing and treating Orthopedics & Trauma patients.
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This presentation goes in depth about Primary and Recurrent Patellar dislocation. Its cause, clinical and radiographic evaluation and various modalities of management with update from latest literature.
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Plantar fasciitis'heel pain' is the condition in which pain is occur in the plantar surface of heel...... In this ppt we disscus about condition and physiotherapy management
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
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The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
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Knee anatomy and clinical tests 2024.pdfvimalpl1234
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2. Plantar Fasciitis
It is also referred to as plantar
heel pain syndrome, heel spur
syndrome, or painful heel
syndrome.
It is an overload injury usually associated
with biomechanical abnormalities
3. Definition
It is a painful inflammatory
process of the plantar fascia, the
connective tissue or ligament on
the sole of the foot.
4. Etiology
Deterioration of the plantar fascia.
Mechanical overload of the plantar
fascia
Damaged by direct impact or
repetitive trauma
Damage to other supporting
structures
5. Epidemiology
most common cause of pain in the
inferior heel
Estimated to account for 11 to 15 %
of all foot symptoms requiring
professional care among adults.
affects 15-20% of runners
6. Epidemiology
common among military personnel.
ages of 40 and 60 years in the
general population
Women>Men
65% non-sports demographics are
over-weight
70% unilat. involvement
8. pass
Deep Fascia
Triangular thickening of the
fascia that protects the under
nerves, blood vessels, and
Apex: medial and lateral
the calcaneum.
Base: divides into five
into the toes.
9. Deep Fascia
Have indirect relationship with
Achilles Tendon, if toes are
dorsiflexed plantar fascia tightens
via WINDLASS MECHANISM
12. Biomechanics of Plantar Fascia
It contributes to support of arch of the
foot by acting as a tie-rod, where it
undergoes tension when the foot bears
weight.
It carries as much as 14% of the total
load of the foot.
13. Biomechanics of Plantar fascia
Complete rupture or surgical release of the
plantar fascia
Leads to a ↓ in arch stiffness and a
significant collapse of the longitudinal arch
of the foot.
↑ both stress in the plantar ligaments and
plantar pressures under the metatarsal
heads.
14. Biomechanics of Plantar Fascia
Has an important role in dynamic
function during gait.
Continuously elongated during the
contact phase of gait.
Reaches a maximum of 9% to 12%
elongation between mid-stance and
toe-off.
Plantar fascia behaves like a spring.
15. Biomechanics of Plantar fascia
The plantar fascia has a critical role in
normal mechanical function of the foot,
contributing to the "windlass
mechanism".
17. Pathology
The site of abnormality is typically
near the site of origin of the plantar
fascia at the medial tuberosity of the
calcaneus.
It is more likely caused by
degeneration or weakening of the
tissue.
18. Pathology
This process probably begins with small
tears that occur during activity and that,
in normal circumstances, the body simply
repairs, strengthening the tissue as it
does.
The small tears don’t heal. They
accumulate
19. COMPLAINTS
Pain under the heel
Severe pain &
inability to walk in
morning after
getting from sleep
Same pain in heel
after sitting for
long time and
getting up to walk
Pain gets better
after taking few
steps
Worsening pain
after prolonged
standing/walking
40. Operative Treatment
• Recommended Procedures Include
(1)Elevation of the entire heel pad through a horseshoe incision
around the hindfoot, with release of all soft tissue origins from the
anterior aspect of the calcaneal tuberosity
(2)Neurolysis of a single nerve
(3)Osteotomy of the calcaneus
(4)Excision of the medial inferior tuberosity of the calcaneus
(5)Simple drilling of multiple
41.
42. Resection of small medial portion of plantar fascia
Resect a 2 to 3 × 4-mm rectangle of medial plantar fascia.
An entire plantar fasciotomy may be performed in
some Nonathletic patients who have pain throughout the entire insertion
of the plantar fascia medially and laterally.
43. • If a large spur is present preoperatively and is
thought to contribute to symptoms,
Resect the Spur by gently reflecting the
flexor digitorum brevis off the exostosis.
•Take care not to damage the first branch of the lateral
plantar nerve that lies just superior to the spur.
44. • Endoscopy for Plantar Fascia Release
Based on limited release of the central cord of the fascia.
– An effective procedure with reproducible results, a low
complication rate, and little risk of iatrogenic nerve injury.
45. POSTOPERATIVE CARE
• Non–weight bearing for 2 weeks after surgery.
• The sutures are then removed
• Gradual weight bearing to tolerance is begun.
• Resumption of heel cord stretching and
increased activity are encouraged.