Class 9 English The Snake And The Mirror (Part 2)Vista's Learning
The doctor was admiring himself in the mirror and thinking about marrying a wealthy, heavyset woman. As he was lost in thought, a snake suddenly appeared on his chair and then coiled around his arm, coming within inches of his face. Frozen with fear, the doctor realized he should not be arrogant about his looks. The snake then moved towards the mirror and admired its own reflection. The doctor fled and returned to find all his belongings stolen except a dirty vest. He concluded his story by saying he never saw the snake again and assumed it was enticed by its own beauty in the mirror.
This document discusses traumatic hyphema, which is bleeding into the anterior chamber of the eye following an eye injury. It can be caused by blunt trauma, surgery, or spontaneously from conditions like tumors or blood clotting issues. Symptoms include blurred vision, pain and photophobia. Treatment involves protecting the eye, preventing further bleeding with medications, and sometimes surgically removing blood if pressure rises too high. Goals are to prevent secondary glaucoma and damage to vision.
This document summarizes the presentation, evaluation, treatment, and complications of hyphema, which is bleeding into the anterior chamber of the eye. It describes grading systems for hyphemas based on the amount of bleeding. The main treatment is bed rest, patching, and medications to prevent further bleeding like aminocaproic acid. Complications include increased eye pressure, blood staining of the cornea, synechiae formation, and optic atrophy. Hospitalization is usually required for larger hyphemas or those with elevated eye pressure.
Penetrating eye injuries are commonly caused by sharp objects like needles, sticks, or glass. They can cause mechanical damage to eye structures as well as introduce infection. Symptoms include redness, pain, and vision loss. Examination may reveal lacerations or penetration of the sclera or cornea. Treatment depends on the severity but generally involves antibiotics to prevent infection, with suturing of larger wounds. Globe rupture from blunt trauma risks extrusion of intraocular contents and requires careful examination to avoid further damage.
Hyphema refers to blood in the anterior chamber of the eye. It is typically caused by trauma that tears blood vessels in the iris, ciliary body, or trabecular meshwork. Hyphemas are classified based on their etiology (cause), amount of bleeding, and location in the eye. Treatment involves resting the eye, using cycloplegic eye drops to prevent inflammation, and occasionally surgically removing blood if it causes increased pressure or vision loss. The prognosis is generally good if the hyphema involves less than half the anterior chamber, but can be poorer for larger bleeds.
1. Blunt trauma to the eye can cause a variety of injuries depending on the force and location of impact, including direct damage to the eyeball at the point of impact, damage from compression waves transmitted through the eye's fluid contents, and indirect damage from impact with bony eye socket structures.
2. Specific injuries include abrasions, tears or detachments of the cornea, iris, retina, choroid or sclera, hyphema, cataracts, subluxation or dislocation of the lens, vitreous hemorrhages, glaucoma, commotio retinae, retinal tears and detachments, and macular holes.
3. More severe blunt trauma can cause
1. The document discusses various ocular emergencies including traumatic injuries like hyphema, ruptured globe, and chemical burns as well as non-traumatic emergencies like acute angle closure glaucoma and orbital cellulitis.
2. Management strategies are provided for different conditions including irrigation for chemical burns, medications to lower IOP for glaucoma, and IV antibiotics for orbital cellulitis.
3. Immediate referral to an ophthalmologist is recommended for penetrating injuries, ruptured globe, retinal artery occlusion, and conditions requiring surgery.
This document provides an overview of eyelid anatomy and physiology. It describes the gross anatomy and layers of the eyelid, including the skin, subcutaneous tissue, orbicularis oculi muscle, orbital septum, tarsal plates, smooth muscles, and conjunctiva. It also discusses eyelid arterial supply, venous and lymphatic drainage, and nerve supply. The functions of eyelids and different types of eyelid movements such as opening, closing, blinking, winking, and Bell's phenomenon are explained.
Class 9 English The Snake And The Mirror (Part 2)Vista's Learning
The doctor was admiring himself in the mirror and thinking about marrying a wealthy, heavyset woman. As he was lost in thought, a snake suddenly appeared on his chair and then coiled around his arm, coming within inches of his face. Frozen with fear, the doctor realized he should not be arrogant about his looks. The snake then moved towards the mirror and admired its own reflection. The doctor fled and returned to find all his belongings stolen except a dirty vest. He concluded his story by saying he never saw the snake again and assumed it was enticed by its own beauty in the mirror.
This document discusses traumatic hyphema, which is bleeding into the anterior chamber of the eye following an eye injury. It can be caused by blunt trauma, surgery, or spontaneously from conditions like tumors or blood clotting issues. Symptoms include blurred vision, pain and photophobia. Treatment involves protecting the eye, preventing further bleeding with medications, and sometimes surgically removing blood if pressure rises too high. Goals are to prevent secondary glaucoma and damage to vision.
This document summarizes the presentation, evaluation, treatment, and complications of hyphema, which is bleeding into the anterior chamber of the eye. It describes grading systems for hyphemas based on the amount of bleeding. The main treatment is bed rest, patching, and medications to prevent further bleeding like aminocaproic acid. Complications include increased eye pressure, blood staining of the cornea, synechiae formation, and optic atrophy. Hospitalization is usually required for larger hyphemas or those with elevated eye pressure.
Penetrating eye injuries are commonly caused by sharp objects like needles, sticks, or glass. They can cause mechanical damage to eye structures as well as introduce infection. Symptoms include redness, pain, and vision loss. Examination may reveal lacerations or penetration of the sclera or cornea. Treatment depends on the severity but generally involves antibiotics to prevent infection, with suturing of larger wounds. Globe rupture from blunt trauma risks extrusion of intraocular contents and requires careful examination to avoid further damage.
Hyphema refers to blood in the anterior chamber of the eye. It is typically caused by trauma that tears blood vessels in the iris, ciliary body, or trabecular meshwork. Hyphemas are classified based on their etiology (cause), amount of bleeding, and location in the eye. Treatment involves resting the eye, using cycloplegic eye drops to prevent inflammation, and occasionally surgically removing blood if it causes increased pressure or vision loss. The prognosis is generally good if the hyphema involves less than half the anterior chamber, but can be poorer for larger bleeds.
1. Blunt trauma to the eye can cause a variety of injuries depending on the force and location of impact, including direct damage to the eyeball at the point of impact, damage from compression waves transmitted through the eye's fluid contents, and indirect damage from impact with bony eye socket structures.
2. Specific injuries include abrasions, tears or detachments of the cornea, iris, retina, choroid or sclera, hyphema, cataracts, subluxation or dislocation of the lens, vitreous hemorrhages, glaucoma, commotio retinae, retinal tears and detachments, and macular holes.
3. More severe blunt trauma can cause
1. The document discusses various ocular emergencies including traumatic injuries like hyphema, ruptured globe, and chemical burns as well as non-traumatic emergencies like acute angle closure glaucoma and orbital cellulitis.
2. Management strategies are provided for different conditions including irrigation for chemical burns, medications to lower IOP for glaucoma, and IV antibiotics for orbital cellulitis.
3. Immediate referral to an ophthalmologist is recommended for penetrating injuries, ruptured globe, retinal artery occlusion, and conditions requiring surgery.
This document provides an overview of eyelid anatomy and physiology. It describes the gross anatomy and layers of the eyelid, including the skin, subcutaneous tissue, orbicularis oculi muscle, orbital septum, tarsal plates, smooth muscles, and conjunctiva. It also discusses eyelid arterial supply, venous and lymphatic drainage, and nerve supply. The functions of eyelids and different types of eyelid movements such as opening, closing, blinking, winking, and Bell's phenomenon are explained.
This document provides information about snake bites in KwaZulu-Natal, South Africa. It discusses the different types of venom and clinical syndromes caused by various snakes, including rinkhals, cobras, mambas, boomslangs, berg adders, and Natal black snakes. It outlines the symptoms, management, and treatment of envenomation from these snakes. Key points covered include the use of analgesics, fluids, antivenom, and monitoring for complications such as hypotension, coagulopathy, and respiratory failure. Myths about snake bite treatment are also debunked.
This document provides a quick refresher on the cranial nerves and performing a neurological examination. It lists the 12 cranial nerves and their functions. It then describes performing a 9 minute neurological screening examination to check for abnormalities like cranial nerve palsies or abnormal eye movements. Specific examples of cranial nerve palsies and abnormal cerebellar findings are demonstrated through videos to illustrate what examiners should look for. The document emphasizes that while a full neurological exam takes much longer, this brief screening exam can help detect potential intracranial abnormalities.
This document discusses the zones of the ECG and how it views the heart in two planes - the coronal and axial planes. It explains the limb leads and precordial leads, and which parts of the heart each views. It notes that when assessing an ECG, you should first look at rhythm, rate, and axis before analyzing the specific zones to locate any abnormalities. It provides examples of interpreting ECGs to locate infarcts in the inferior wall, anteroseptal area, and anterolateral area based on which leads show ST elevations.
This document discusses disturbances of heart rate and how to determine heart rate from an ECG. It provides examples of different types of tachycardias and bradycardias, including sinus tachycardia, sinus bradycardia, supraventricular tachycardia, junctional bradycardia, ventricular tachycardia, third-degree heart block, atrial flutter, atrial fibrillation, and accelerated junctional rhythm. It emphasizes looking at rhythm, rate, and the width of QRS complexes to determine the underlying rhythm disturbance.
This document discusses how to determine a normal sinus rhythm on an ECG. It outlines the four criteria for a normal sinus rhythm: 1) normal P waves, 2) narrow QRS complexes, 3) a QRS following each P wave, and 4) regular grouping of the P wave and QRS complexes. Several example ECG strips are provided and the reader is asked to determine if they show a normal or abnormal rhythm based on these four criteria. The document emphasizes looking for all four criteria when assessing rhythms. It concludes by recommending further reading on different types of abnormal rhythms.
This document provides instructions on how to determine the electrical axis of the heart from an electrocardiogram (ECG). It explains that the electrical axis shows the general direction of electricity in the heart from right to left. It describes the orientation of the limb and augmented leads around the heart and how their deflections relate to the electrical axis, with Lead I typically positive and AVR typically negative in a normal axis. Deviations of the axis to the left or right can be identified by comparing the deflections of Leads I and AVF.
This document provides guidance on evaluating and treating patients presenting with acute chest pain. It emphasizes the importance of obtaining an ECG within the first 20 minutes to classify the patient's risk and guide management. The ECG can indicate conditions like ST-elevation myocardial infarction, non-ST-elevation myocardial infarction, unstable angina, or alternate diagnoses. Proper ECG interpretation and identifying abnormalities is an essential clinical skill for guiding urgent reperfusion or admitting high-risk patients for monitoring.
Based on the history and exam findings, this patient is presenting with features suggestive of occipital neuralgia or cluster headache. Important things to note are:
- Unilateral occipital pain radiating to the neck
- Parasthesias in the same distribution
- Photophobia
- PTOSIS of the eyelid on the same side as the pain
- Pain worsened by posture changes like looking down
- No other focal neurological deficits
Next steps would be to get imaging of the cervical spine to rule out bony/soft tissue causes of nerve root compression. May also consider a short course of oral steroids to see if it provides relief of symptoms, as occipital neuralgia and cluster headaches
An approach to wrist fractures as often seen in emergency rooms
presentation meant mainly to educate jnr drs in looking at x-rays of wrists and how to identify fractures and dislocations
This document discusses the management of head trauma. It outlines the importance of limiting secondary brain injury by maintaining adequate blood pressure and oxygenation. It recommends performing frequent neurologic exams and liberal use of CT scans to identify any brain injuries. Emergent neurosurgical consultation is advised for expanding intracranial masses or deteriorating neurological status. The goal of treatment is to stabilize the patient and arrange for definitive care to prevent further brain damage.
A brief description of facial trauma , reading facial xrays and management of facial trauma for interns and junior mo's covering casualty in a rural/semi-rural setting
The document describes a presentation on managing head trauma and skull fractures. It outlines a case of a 27-year-old male brought in after a steel beam fell on his head at work with an initial GCS of 7/15. The presentation reviews the steps to stabilize the patient, including intubation, application of a hard collar, NG/OG tube placement, blood tests, and secondary survey. Key injuries discussed include suspected cribiform plate injury, haemotympanum, subconjunctival haemorrhage, and signs of basal skull fracture. Next steps outlined are further investigations like CT scans of the head and spine.
Co-Chairs, Val J. Lowe, MD, and Cyrus A. Raji, MD, PhD, prepared useful Practice Aids pertaining to Alzheimer’s disease for this CME/AAPA activity titled “Alzheimer’s Disease Case Conference: Gearing Up for the Expanding Role of Neuroradiology in Diagnosis and Treatment.” For the full presentation, downloadable Practice Aids, and complete CME/AAPA information, and to apply for credit, please visit us at https://bit.ly/3PvVY25. CME/AAPA credit will be available until June 28, 2025.
Cell Therapy Expansion and Challenges in Autoimmune DiseaseHealth Advances
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
These lecture slides, by Dr Sidra Arshad, offer a simplified look into the mechanisms involved in the regulation of respiration:
Learning objectives:
1. Describe the organisation of respiratory center
2. Describe the nervous control of inspiration and respiratory rhythm
3. Describe the functions of the dorsal and respiratory groups of neurons
4. Describe the influences of the Pneumotaxic and Apneustic centers
5. Explain the role of Hering-Breur inflation reflex in regulation of inspiration
6. Explain the role of central chemoreceptors in regulation of respiration
7. Explain the role of peripheral chemoreceptors in regulation of respiration
8. Explain the regulation of respiration during exercise
9. Integrate the respiratory regulatory mechanisms
10. Describe the Cheyne-Stokes breathing
Study Resources:
1. Chapter 42, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 36, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 13, Human Physiology by Lauralee Sherwood, 9th edition
This document provides information about snake bites in KwaZulu-Natal, South Africa. It discusses the different types of venom and clinical syndromes caused by various snakes, including rinkhals, cobras, mambas, boomslangs, berg adders, and Natal black snakes. It outlines the symptoms, management, and treatment of envenomation from these snakes. Key points covered include the use of analgesics, fluids, antivenom, and monitoring for complications such as hypotension, coagulopathy, and respiratory failure. Myths about snake bite treatment are also debunked.
This document provides a quick refresher on the cranial nerves and performing a neurological examination. It lists the 12 cranial nerves and their functions. It then describes performing a 9 minute neurological screening examination to check for abnormalities like cranial nerve palsies or abnormal eye movements. Specific examples of cranial nerve palsies and abnormal cerebellar findings are demonstrated through videos to illustrate what examiners should look for. The document emphasizes that while a full neurological exam takes much longer, this brief screening exam can help detect potential intracranial abnormalities.
This document discusses the zones of the ECG and how it views the heart in two planes - the coronal and axial planes. It explains the limb leads and precordial leads, and which parts of the heart each views. It notes that when assessing an ECG, you should first look at rhythm, rate, and axis before analyzing the specific zones to locate any abnormalities. It provides examples of interpreting ECGs to locate infarcts in the inferior wall, anteroseptal area, and anterolateral area based on which leads show ST elevations.
This document discusses disturbances of heart rate and how to determine heart rate from an ECG. It provides examples of different types of tachycardias and bradycardias, including sinus tachycardia, sinus bradycardia, supraventricular tachycardia, junctional bradycardia, ventricular tachycardia, third-degree heart block, atrial flutter, atrial fibrillation, and accelerated junctional rhythm. It emphasizes looking at rhythm, rate, and the width of QRS complexes to determine the underlying rhythm disturbance.
This document discusses how to determine a normal sinus rhythm on an ECG. It outlines the four criteria for a normal sinus rhythm: 1) normal P waves, 2) narrow QRS complexes, 3) a QRS following each P wave, and 4) regular grouping of the P wave and QRS complexes. Several example ECG strips are provided and the reader is asked to determine if they show a normal or abnormal rhythm based on these four criteria. The document emphasizes looking for all four criteria when assessing rhythms. It concludes by recommending further reading on different types of abnormal rhythms.
This document provides instructions on how to determine the electrical axis of the heart from an electrocardiogram (ECG). It explains that the electrical axis shows the general direction of electricity in the heart from right to left. It describes the orientation of the limb and augmented leads around the heart and how their deflections relate to the electrical axis, with Lead I typically positive and AVR typically negative in a normal axis. Deviations of the axis to the left or right can be identified by comparing the deflections of Leads I and AVF.
This document provides guidance on evaluating and treating patients presenting with acute chest pain. It emphasizes the importance of obtaining an ECG within the first 20 minutes to classify the patient's risk and guide management. The ECG can indicate conditions like ST-elevation myocardial infarction, non-ST-elevation myocardial infarction, unstable angina, or alternate diagnoses. Proper ECG interpretation and identifying abnormalities is an essential clinical skill for guiding urgent reperfusion or admitting high-risk patients for monitoring.
Based on the history and exam findings, this patient is presenting with features suggestive of occipital neuralgia or cluster headache. Important things to note are:
- Unilateral occipital pain radiating to the neck
- Parasthesias in the same distribution
- Photophobia
- PTOSIS of the eyelid on the same side as the pain
- Pain worsened by posture changes like looking down
- No other focal neurological deficits
Next steps would be to get imaging of the cervical spine to rule out bony/soft tissue causes of nerve root compression. May also consider a short course of oral steroids to see if it provides relief of symptoms, as occipital neuralgia and cluster headaches
An approach to wrist fractures as often seen in emergency rooms
presentation meant mainly to educate jnr drs in looking at x-rays of wrists and how to identify fractures and dislocations
This document discusses the management of head trauma. It outlines the importance of limiting secondary brain injury by maintaining adequate blood pressure and oxygenation. It recommends performing frequent neurologic exams and liberal use of CT scans to identify any brain injuries. Emergent neurosurgical consultation is advised for expanding intracranial masses or deteriorating neurological status. The goal of treatment is to stabilize the patient and arrange for definitive care to prevent further brain damage.
A brief description of facial trauma , reading facial xrays and management of facial trauma for interns and junior mo's covering casualty in a rural/semi-rural setting
The document describes a presentation on managing head trauma and skull fractures. It outlines a case of a 27-year-old male brought in after a steel beam fell on his head at work with an initial GCS of 7/15. The presentation reviews the steps to stabilize the patient, including intubation, application of a hard collar, NG/OG tube placement, blood tests, and secondary survey. Key injuries discussed include suspected cribiform plate injury, haemotympanum, subconjunctival haemorrhage, and signs of basal skull fracture. Next steps outlined are further investigations like CT scans of the head and spine.
Co-Chairs, Val J. Lowe, MD, and Cyrus A. Raji, MD, PhD, prepared useful Practice Aids pertaining to Alzheimer’s disease for this CME/AAPA activity titled “Alzheimer’s Disease Case Conference: Gearing Up for the Expanding Role of Neuroradiology in Diagnosis and Treatment.” For the full presentation, downloadable Practice Aids, and complete CME/AAPA information, and to apply for credit, please visit us at https://bit.ly/3PvVY25. CME/AAPA credit will be available until June 28, 2025.
Cell Therapy Expansion and Challenges in Autoimmune DiseaseHealth Advances
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
These lecture slides, by Dr Sidra Arshad, offer a simplified look into the mechanisms involved in the regulation of respiration:
Learning objectives:
1. Describe the organisation of respiratory center
2. Describe the nervous control of inspiration and respiratory rhythm
3. Describe the functions of the dorsal and respiratory groups of neurons
4. Describe the influences of the Pneumotaxic and Apneustic centers
5. Explain the role of Hering-Breur inflation reflex in regulation of inspiration
6. Explain the role of central chemoreceptors in regulation of respiration
7. Explain the role of peripheral chemoreceptors in regulation of respiration
8. Explain the regulation of respiration during exercise
9. Integrate the respiratory regulatory mechanisms
10. Describe the Cheyne-Stokes breathing
Study Resources:
1. Chapter 42, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 36, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 13, Human Physiology by Lauralee Sherwood, 9th edition
Travel vaccination in Manchester offers comprehensive immunization services for individuals planning international trips. Expert healthcare providers administer vaccines tailored to your destination, ensuring you stay protected against various diseases. Conveniently located clinics and flexible appointment options make it easy to get the necessary shots before your journey. Stay healthy and travel with confidence by getting vaccinated in Manchester. Visit us: www.nxhealthcare.co.uk
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdfOsvaldo Bernardo Muchanga
GASTROINTESTINAL INFECTIONS AND GASTRITIS
Osvaldo Bernardo Muchanga
Gastrointestinal Infections
GASTROINTESTINAL INFECTIONS result from the ingestion of pathogens that cause infections at the level of this tract, generally being transmitted by food, water and hands contaminated by microorganisms such as E. coli, Salmonella, Shigella, Vibrio cholerae, Campylobacter, Staphylococcus, Rotavirus among others that are generally contained in feces, thus configuring a FECAL-ORAL type of transmission.
Among the factors that lead to the occurrence of gastrointestinal infections are the hygienic and sanitary deficiencies that characterize our markets and other places where raw or cooked food is sold, poor environmental sanitation in communities, deficiencies in water treatment (or in the process of its plumbing), risky hygienic-sanitary habits (not washing hands after major and/or minor needs), among others.
These are generally consequences (signs and symptoms) resulting from gastrointestinal infections: diarrhea, vomiting, fever and malaise, among others.
The treatment consists of replacing lost liquids and electrolytes (drinking drinking water and other recommended liquids, including consumption of juicy fruits such as papayas, apples, pears, among others that contain water in their composition).
To prevent this, it is necessary to promote health education, improve the hygienic-sanitary conditions of markets and communities in general as a way of promoting, preserving and prolonging PUBLIC HEALTH.
Gastritis and Gastric Health
Gastric Health is one of the most relevant concerns in human health, with gastrointestinal infections being among the main illnesses that affect humans.
Among gastric problems, we have GASTRITIS AND GASTRIC ULCERS as the main public health problems. Gastritis and gastric ulcers normally result from inflammation and corrosion of the walls of the stomach (gastric mucosa) and are generally associated (caused) by the bacterium Helicobacter pylor, which, according to the literature, this bacterium settles on these walls (of the stomach) and starts to release urease that ends up altering the normal pH of the stomach (acid), which leads to inflammation and corrosion of the mucous membranes and consequent gastritis or ulcers, respectively.
In addition to bacterial infections, gastritis and gastric ulcers are associated with several factors, with emphasis on prolonged fasting, chemical substances including drugs, alcohol, foods with strong seasonings including chilli, which ends up causing inflammation of the stomach walls and/or corrosion. of the same, resulting in the appearance of wounds and consequent gastritis or ulcers, respectively.
Among patients with gastritis and/or ulcers, one of the dilemmas is associated with the foods to consume in order to minimize the sensation of pain and discomfort.
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotesPsychoTech Services
A proprietary approach developed by bringing together the best of learning theories from Psychology, design principles from the world of visualization, and pedagogical methods from over a decade of training experience, that enables you to: Learn better, faster!
low birth weight presentation. Low birth weight (LBW) infant is defined as the one whose birth weight is less than 2500g irrespective of their gestational age. Premature birth and low birth weight(LBW) is still a serious problem in newborn. Causing high morbidity and mortality rate worldwide. The nursing care provide to low birth weight babies is crucial in promoting their overall health and development. Through careful assessment, diagnosis,, planning, and evaluation plays a vital role in ensuring these vulnerable infants receive the specialize care they need. In India every third of the infant weight less than 2500g.
Birth period, socioeconomical status, nutritional and intrauterine environment are the factors influencing low birth weight
“Psychiatry and the Humanities”: An Innovative Course at the University of Mo...Université de Montréal
“Psychiatry and the Humanities”: An Innovative Course at the University of Montreal Expanding the medical model to embrace the humanities. Link: https://www.psychiatrictimes.com/view/-psychiatry-and-the-humanities-an-innovative-course-at-the-university-of-montreal
Lecture 6 -- Memory 2015.pptlearning occurs when a stimulus (unconditioned st...AyushGadhvi1
learning occurs when a stimulus (unconditioned stimulus) eliciting a response (unconditioned response) • is paired with another stimulus (conditioned stimulus)
3. CORNEAL
ABRASION
• DIFFICULT TO SEE
• BE SUSPICIOUS WHEN PAIN DOES
NOT MATCH WHAT YOU CAN SEE
• NOTICE THE INJECTION OF VESSELS
• IF YOU’RE NOT SURE IF ITS SERIOUS
RATHER REFER TO
OPHTHALMOLOGISTS
4. BLUE LIGHT AND STAINING
THE SAME EYE
THIS TIME IT STAINED WITH
FLUORESCEIN
MULTIPLE LINEAR
ABRASIONS
ABRASION MUCH EASIER
TO SEE NOW
5. REMEMBER TO CHECK UNDER THE EYELID
❖ CAREFUL EXAMINATION
WILL OFTEN SAVE YOU
FROM UNNECESSARY
BLUSHES LATER ON
❖ IF A FOREIGN BODY IS
EMBEDDED DO NOT
REMOVE IT YOURSELF BUT
REFER
7. THIS IS A
SUBCONJUNCTIVAL
HAEMMORHAGE
❖ NOTICE THE FLAT NAURE
❖ BRIGHT RED
❖ OFTEN DUE TO TRIVIAL INJURY
(COUGH , SNEEZE, MINOR
TRAUMA)
❖ NO TX NEEDED
❖ REASSURE PT
8. THIS IS A BLOODY CHEMOSIS
❖ NOTICE THE SWELLING OF
THE SCLERA
❖ THIS IS NORMALLY DUE TO
TRAUMA
❖ HIGHLY SUSPICIOUS OF
PENETRATING GLOBE
TRAUMA
❖ IF NOT FROM TRAUMA MAY
BE SIGN OF A
COAGULOPATHY
❖ REQUIRES REFERRAL
9. FOREIGN BODIES
❖ MECHANISM OF INJURY
VERY IMPORTANT
❖ IF YOU SUSPECT
PENETRATING INJURY
THEN YOU………..
❖ DONT FORGET YOUR
TETANUS PROPHYLAXIS
❖ DONT TRY AND REMOVE
RUST RINGS
RUST RING
FROM
FB
10. HYPHEMA
BLOOD IN ANTERIOR
CHAMBER
❖ YOU KNOW WHAT I’M GONNA
SAY………………………………….
❖ REFER REFER REFER!!!!
❖ THIS IS A RELATIVE EMERGENCY,
SHOULD BE SEEN WITHIN 24 HRS
8 BALL HYPHEMA
11. DESCRIBE WHAT YOU SEE
WHATS GOING
ON HERE?
❖ THIS PT WAS WORKING
WITH AN ANGLE
GRINDER
12. INTRA-OCULAR
FB
❖ MECHANISM OF INJURY
IMPORTANT
❖ PAIN IS OFTEN NOT THAT
SEVERE
❖ YOU NEED TO HAVE A HIGH
INDEX OF SUSPICION
❖ AND THEN YOU……….
NOTICE THE FB!
NOTICE THE
TEAR DROP
PUPIL
14. VERY EASY TO MISS !!!
LENS
DISLOCATION
❖ ALWAYS DO A CAREFUL
EXAMINATION
❖ CURSORY GLANCES AT THE EYE
WONT REVEAL MUCH
❖ PT WILL COMPLAIN OF VISUAL
DISTURBANCES
❖ LOOKS LIKE YOU SHOULD MEMORIZE
THE EYE CLINIC SPEED DIAL!!!!
LENS
15. WHAT DO WE DO IN CASUALTY?
IRIODIALYSIS
❖ THE IRIS IS PULLED AWAY
FROM THE CILLIARY
BODY
❖ PT WILL COMPLAIN OF
VISUAL DISTURBANCES
AND A DOUBLE PUPIL
22. SO WHAT DO YOU DO WITH
THESE INJURIES IN CASUALTY?
23. WE REFER BECAUSE
WE MAY DO MORE
HARM THAN GOOD
THIS REPAIR LOOKS REASONABLE
BUT THE PT SUFFERED LONG TERM
CONTRACTURES, EYELID RETRACTION
LAGOPHTALMOS