This document discusses various types of malnutrition including protein energy malnutrition (PEM) in Nepal. It outlines the immediate, underlying, and basic causes of PEM as inadequate dietary intake and infections which interact in a vicious cycle. Preventive measures proposed include promoting optimal infant and young child feeding practices, vaccination, food fortification, and treating diarrhea and intestinal parasites. The document also discusses iodine deficiency disorders, iron deficiency anemia, vitamin A deficiency and their prevention through salt iodization, food fortification, and supplementation programs.
Malnutrition is the condition that develops when the body does not get the right amount of vitamins, minerals, and other nutrients it needs to maintain healthy tissues and organ function.
Infant and young child feeding ppt describe the nutritional needs of infant and child. Exclusive breastfeeding for six months and complementary feeding for the child. avoid formula feeding for the child and continue breastfeeding for 24 months.
Malnutrition is poor nutrition due to an insufficient, poorly balanced diet, faulty digestion or poor utilization of foods. (This can result in the inability to absorb foods).
Malnutrition is not only insufficient intake of nutrients. It can occur when an individual is getting excessive nutrients as well.
Malnutrition is the condition that develops when the body does not get the right amount of vitamins, minerals, and other nutrients it needs to maintain healthy tissues and organ function.
Infant and young child feeding ppt describe the nutritional needs of infant and child. Exclusive breastfeeding for six months and complementary feeding for the child. avoid formula feeding for the child and continue breastfeeding for 24 months.
Malnutrition is poor nutrition due to an insufficient, poorly balanced diet, faulty digestion or poor utilization of foods. (This can result in the inability to absorb foods).
Malnutrition is not only insufficient intake of nutrients. It can occur when an individual is getting excessive nutrients as well.
Workshop 3: The Agriculture Nutrition Nexus and the Way Forward at The Caribbean-Pacific Agri-Food Forum 2015 (CPAF2015) taking place 2-6 November in Barbados with support from the Intra-ACP Agricultural Policy programme, organized in partnership with the Barbados Agricultural Society (BAS) and the Inter-American Institute for Cooperation on Agriculture (IICA). http://www.cta.int/en/news/caribbean-pacific-agri-food-forum.html
Maternal Anemia within Child Survival Grants: Lessons Learned at Helen Keller...jehill3
Maternal Anemia within Child Survival Grants: Lessons Learned at Helen Keller International
Core Group Spring Meeting Pre-Session, Monday April 26, 2010
This presentation aims at explaining all the components of malnutrition. Such as types, causes, criteria of diagnosis, treatment & Government health initiatives to tackle the problem of malnutrition.
Similar to Malnutrition, Causes,Framework, vicious cycle,Preventive measures,Policy and strategies of Government of Nepal. (20)
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Malnutrition, Causes,Framework, vicious cycle,Preventive measures,Policy and strategies of Government of Nepal.
1. BY : Dhirendra Nath
National Open College
Sanepa lalitpur
Malnutrition
2. Malnutrition
Malnutrition has been defined as "a pathological state
resulting from a relative or absolute deficiency or
excess of one or more essential nutrients".
It comprises four forms –under-nutrition, over-
nutrition, imbalance and the specific deficiency.
3. PEM
as a major health and nutrition problem in Nepal
(weaklings and children in the first years of life)
also to permanent impairment of physical and
possibly, of mental growth of those who survive.
its clinical forms - kwashiorkor and marasmus
two different clinical pictures at opposite poles of a single
continuum.
4. PEM is primarily due to
(a) an inadequate intake of food (food gap) both in
quantity and quality, and
(b) infections, notably diarrhoea, respiratory tract
infections, measles and intestinal worms
increase requirements for calories, protein and other
nutrients,
decreasing their absorption and utilization.
It is a vicious circle - infection contributing to
malnutrition and malnutrition contributing to
infection, both acting synergistically
6. Other contributory factors in the web of causation, viz.
poor environmental conditions,
large family size,
poor maternal health,
failure of lactation,
premature termination of breast feeding, and
adverse cultural practices relating to child rearing and
weaning such as
the use of over-diluted cow's milk and
discarding cooking water from cereals and
delayed supplementary feeding
7. Impact of malnutrition
High Morbidity and mortality
survival
Academic performance of children
Socio-economic
National Devlopment
Drug action
Health and Devlopment
13. Causes of Malnutrition and Death
Manifestations
Immediate Causes
Underlying Causes
Basic Causes
Malnutrition and Death
Inadequate
Dietary Intake
Disease
Insufficient
Household
Food Security
Inadequate
Maternal and
Child Care
Insufficient Health
Services & Unhealthy
Environment
Formal and Non-
Formal Institutions
Potential
Resources
UNICEF
Framework
14. Preventive measures
There is no simple solution to the problem of
PEM.
Many types of actions are necessary.
The following is adapted from the 8th FAO/WHO
Expert Committee on Nutrition for the prevention
of PEM in the community
15. Early detection of PEM
The first indicator of PEM is under-weight for age
The most practical method to detect this, is to
maintain growth charts
These charts indicate at a glance whether the child is
gaining or losing weight.
16. (a) Health promotion
1. Measures directed to pregnant and lactating women
(education, distribution of supplements)
2. Promotion of breast feeding.
3. Development of low cost weaning foods : the child
should be made to eat more food at frequent intervals
4. Measures to improve family diet
17. Cont……
5. Nutrition education - Promotion of correct feeding
practices.
6. Home economics
7. Family planning and spacing of births
8. Family environment
18. (b) Specific protection
1. The child's diet must contain protein and energy-
rich foods. Milk, eggs, fresh fruits should be given if
possible.
2. Immunization
3. Food fortification
19. Early diagnosis and treatment
1. Periodic surveillance
2. Early diagnosis of any lag in growth
3. Early diagnosis and treatment of diarrhoea
4. Development of programmes for early rehydration
of children with diarrhoea.
5. Development of supplementary feeding
programmes during epidemics.
6. De-worming of heavily infested children
21. Policy:
To reduce PEM in under 5 years and reproductive
aged women
Strategies:
Protect, promote and support optimal feeding
practice for infants and young children (capacity
building on IYCF)
Increase coverage of GM
BCC for changing dietary practices
Strengthen Nutrition Rehabilitation Homes
Community Based Management of Acute
Malnutrition
Improve maternal nutrition and low birth weight of
baby through improved maternal nutrition practices
22. IDD
IDDs are spectrum of the disorders that occur
due to deficiency of iodine & associated
hypothyroidism, commencing from intrauterine life
& extending through infancy, childhood,
adolescent to adult life with serious implications
23. Prevention and Control
Strengthen the implementation of Iodized Salt
Act, 2055 for regulation and monitoring of Iodized
salt trade to ensure that all edible salt is iodized
Increase the accessibility and market share of
iodized packet salt with ‘two‐child’ logo
24. Create awareness about the importance of use
of iodized salt for the control of IDD through
social marketing campaign
Celebrate month of February as an iodine month
25. Government action on IDD:
Universal Iodization of Salt, 50 ppm at the point of
production to 30 ppm at retail and 15 ppm at
household level
Distribution of iodized salt in remote area at
subsidized rates
Monitoring of the iodized salt at the entry point and in
the market
Iodized salt warehouse construction in various part of
the country
Increase accessibility of 2- child logo iodized salt
Social marketing of 2-child logo iodized salt
BCC for use of iodized salt
26. Iron Deficiency Anemia
It is a condition that results a lowering of
hemoglobin levels below which is considered to
be normal for a specific demographic group
It occurs at all stages of the lifecycle but is more
prevalent in women especially pregnant women
and young children
This is frequently worsened by infectious
diseases i.e. malaria, TB, hookworm infestations
27. Cause of IDA
Some underlying causes
Inadequate BF
Incorrect or incomplete complementary feeding
Low level of family education
Poor intra family distribution of food at H/H level
Poor cooking/processing, storage, preservation at
H/H level
Food fad(trend) and faulty food habits
28. Cause of IDA
Poor health and agricultural services
Lack of institutional capacity/trained manpower
Low production of iron rich foods
Lack of H/H gardening
Insufficient marketing infrastructures
Poorly developed commercial food processing
industry
29. Prevention and Control
Health Promotion
Adequate nutrition, household gardening
Nutrition education to improve dietary habits
Health education specially to pregnant mothers
about hazards of anemia and their prevention
Periodical deforming specially among children and
at least once during second trimester of pregnancy
30. Prevention and Control
Specific Protection
Food fortification with iron
Salt, sugar, wheat flour & other foods for fortifications
Supplementation with iron and other nutrients:
Early Diagnosis and Prompt Treatment
ED:
By history of headache, giddiness, fatigue, loss of appetite
etc
By clinical signs
By lab investigations
31. Prevention and Control
Disability Limitation
It can be done by giving intensive treatment in
hospital by blood transfusion
If severe anemia is associated with cardiac
failure(high output failure), packed cell transfusion is
given under the umbrella of digoxin, lasix &
potassium salts
Rehabilitation
If treatment is given correctly & completely, the
person will not become handicapped
32. Objective
To reduce the prevalence of anemia among women and
children.
Target:
To reduce the prevalence of iron deficiency anemia to
less than 40% by the year 2017.
33. Control of Anemia
Increase coverage and compliance of iron/folic
supplementation for pregnant and breast feeding
women
Reduce the burden of parasitic infestations
(Helminthes, Malaria and Kala‐azar)
Identify and implement food fortification to
increase the dietary iron intake focusing on
commercial as well as small‐scale community
based fortification initiatives
34. Control of Anemia
Promote dietary diversification to improve the
quality of food consumed with an emphasis on
bio‐available iron
Promote maternal care practices and services to
improve health and nutritional status of mother
and babies
35. Control of Anemia
Identify and implement the effective modalities to
address iron deficiency in adolescents and
non‐pregnant women of reproductive age
Advocate for equity among genders in access
and control over household foods
36. Vitamin A Deficiency
When there is deficiency of Vitamin A in the body,
several complications results in the body which is
called as VAD
37. Causes of VAD
Some Immediate Causes
Low intake of Vit A rich foods
High incidence (of measles, diarrhea, ARI)
Interference with the absorption of Vit A in the small
intestine(obs jaundice, giardiasis, ascariasis)
Conversion of caretenoids to vitamin A in liver(in
certain diseases like chronic pancreatitis)
Interference in transport(in liver disease)
38. VAD Control
Objective
To virtually eliminate Vitamin A
deficiency disorder and sustain the
elimination
Target
To virtually eliminate Vitamin A
deficiency disorder by the year 2017
39. VAD Control
The Nepal vitamin A supplementation (VAS)
programme for young children is held up as a
global success story
It was started in 1993 in 8 districts following a
meeting in Kathmandu that considered three
major research projects in Nepal in the late
1980s which all concluded that periodic
dosing of children 6‐60 months with high dose
vitamin A resulted in significant reductions in
child mortality
40. By the end of 1997 the programme was
implemented in 32 districts and by 2002 it had
expanded to all 75 districts
FCHVs distribute the capsules to young children
twice a year through a ‘campaign‐style’ activity
41. Government action on VAD:
Bi-annually mass supplementation of Vitamin A to under
five children, 100000 IU for 6-12 months and 200000 IU
for 12-60 months children
Vitamin A supplementation to postpartum mothers, 200000
IU within 6 weeks of delivery
Treatment of clinical cases (eye problems, malnutrition,
measles, chronic diarrhea etc.)
BCC for changing dietary practices
Promote vitamin A fortification
Nutrition education
Support for home gardening to increase production of
Vitamin A rich fruits and vegetables
Training on improved methods for production, preparation
and preservation of Vitamin A rich foods
42. Recommendation
Proper monitoring of activities.
Implementation plan should be prepared properly.
Staff should be increased for education program .
Train FCHV about BCC.
Proper monitoring and evaluation of BFHI program.
43. Cont….
Lack access of iron and folic acid fortified food in
community level ,increase access and
awareness.