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INTRO TO EKG
INTERPRETATION
BRIAN LOCKE, MD
PGY2 INTERNAL MEDICINE
UNIVERSITY OF UTAH
UPDATED APR 18, 2018
OBJECTIVES
Understand why ‘the rules’ are what they are.
1. Label the components of an EKG waveform
2. Understand the spatial representation of the different leads
3. Be able to eyeball an estimate of ventricular rate
4. Determine sinus vs. not sinus
5. Determine RBBB vs LBBB vs Neither
6. Identify the morphology of ischemia and infarction
SOURCES
Images from:
ecg.utah.edu
En.ecgpedia.org
https://lifeinthefastlane.com/ecg-library/basics/
REVIEW:
Normal Cardiac conduction
SINGLE LEAD
Name the waves:
QRS NAMING
e.g.
qRs
qR
Qr
rS
Rs
rSr’
APPROACH TO INTERP
Methodological approach, every time
Numerous variations
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RATE
300 / # big boxes
If Irregular: #QRS in 10 seconds (=the strip) * 6
Eg. 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RATE
300 / # small boxes
If Irregular: #QRS in 10 seconds (=the strip) * 6
Eg. 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RATE
300 / # small boxes
If Irregular: #QRS in 10 seconds (=the strip) * 6
Eg. 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RATE
300 / # small boxes
If Irregular: #QRS in 10 seconds (=the strip) * 6
Closer than 3 boxes = tachycardia.
Spaced more than 5 boxes = bradycardia
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular*?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
RHYTHM
P-waves?
Regular?
P  QRS?
Every QRS preceded by P?
*P wave upright in II
Eg 2
Note: If it’s not sinus, what is it? Another talk: Arrhythmias
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
Recall limb leads:
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
LIMB LEADS
Limb lead I:
+ = an impulse moving R to L
- = an impulse moving L to R
How will a vertical impulse look?
LIMB LEADS
Limb leads I, II, and III:
LIMB LEADS
Augmented limb leads:
PRECORDIAL LEADS
V1 = points toward RV
V6 points toward lateral wall
PRECORDIAL LEADS
AXIS
Definition of Normal Axis :
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
Definition of Normal Axis :
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 1
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 2
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 3
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
E.g. 3 LAD
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
What situation would have a NW / extreme
right / “no-man’s land” axis?
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
AXIS
What situation would have a NW / extreme right
/ “no-man’s land” axis?
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
CONDUCTION
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
Check intervals
PR (?AV Block)
QRS duration (?BBB)
QTc
CONDUCTION
AV Block
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
1st Degree Prolonged PR
2nd Degree Type 1
(Wenkebock)
PR lengthens, until a QRS is not
conducted
2nd Degree Type 2 No PR lengthening prior to non
conducted QRS
High Degree So many non-conducted QRS that
you can’t tell (<2:1)
3rd Degree No conducted QRS complexes (both
P-P and QRS-QRS are regular and
different)
CONDUCTION
AV Block: At or below the level of the AV-node = risk of
becoming complete heart block
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
1st Degree Prolonged PR
2nd Degree Type 1
(Wenkebock)
PR lengthens, until a QRS is not
conducted
2nd Degree Type 2 No PR lengthening prior to non
conducted QRS
High Degree So many non-conducted QRS that
you can’t tell (<2:1)
3rd Degree No conducted QRS complexes (both
P-P and QRS-QRS are regular and
different)
CONDUCTION
Bundle Branch (and fascicle) Blocks – wide QRS (120+ms)
Remember the precordial leads
V1 = toward RV, V6 = Toward lateral wall
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
CONDUCTION
Bundle Branch (and fascicle) Blocks
RBBB = delayed positive deflection in V1 1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
CONDUCTION
Bundle Branch (and fascicle) Blocks
LBBB = Positive Deflection in V6
(Remember, LV size > RV size)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
CONDUCTION
Bundle Branch (and fascicle) Blocks
Meets neither criteria?
IVCD (Intraventricular conduction delay)
Ventricular Ectopic Beat (e.g. PVC, VT)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
Check for:
LAA, RAA, RVH / Strain Pattern, LVH, Hypo/hyperkalemia and
ischemia
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
STEMI = Transmural ischemia from occlusion
NSTEMI = Subendocardial ischemia (further from
the blood vessel
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
An Evolving Infarct
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
Ischemia: ST deviation, T-wave inversion
Infarct: q-waves (ST Elevation will be)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
Ischemia: ST deviation, T-wave inversion
Infarct: q-waves (ST Elevation will be)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
Ischemia: ST deviation, T-wave inversion
Infarct: q-waves (ST Elevation will be)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
MORPHOLOGY
Ischemia: ST deviation, T-wave inversion
Infarct: q-waves (ST Elevation will be)
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
EXAMPLE
EXAMPLE
EXAMPLE
MORPHOLOGY
A note about LBBB
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
6. Check Prior
MORPHOLOGY
A note about LBBB:
Always have STE – when is it pathologic? Sgarbossa
1. Rate
2. Rhythm
3. Axis
4. Conduction
5. Morphology
6. Check Prior
PRACTICE
PRACTICE
PRACTICE
PRACTICE
OBJECTIVES
Understand why ‘the rules’ are what they are.
1. Label the components of an EKG waveform
2. Understand the spatial representation of the different leads
3. Be able to eyeball an estimate of ventricular rate
4. Determine sinus vs. not sinus
5. Determine RBBB vs LBBB vs Neither
6. Identify the morphology of ischemia and infarction
NEXT STEPS
Arrythmias
ACS management
Heart Failure
RESOURCES
Tutorials
• http://en.ecgpedia.org
• https://lifeinthefastlane.com/ecg-library/basics/
• Also with links to many resources
Practice EKGs
• https://ecg.bidmc.harvard.edu/
• https://ecg.utah.edu/
• Questions? Brian.locke@hsc.utah.edu

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Locke intro ekg

Editor's Notes

  1. 4 boxes = 75
  2. 14*6 = 84
  3. HR 70, normal sinus, LAD (from LAFB), otherwise normal conduction, no signs of ischemia
  4. *small variation w/ breath
  5. Aflutter w/ 2:1, 3:1 block
  6. Einthoven
  7. Bipolar limb leads (frontal plane): Lead I: RA (-) to LA (+) (Right Left, or lateral) Lead II: RA (-) to LL (+) (Superior Inferior) Lead III: LA (-) to LL (+) (Superior Inferior)
  8. Augmented unipolar limb leads (frontal plane): Lead aVR: RA (+) to [LA & LL] (-) (Rightward) Lead aVL: LA (+) to [RA & LL] (-) (Leftward) Lead aVF: LL (+) to [RA & LA] (-) (Inferior)
  9. Unipolar (+) chest leads (horizontal plane): Leads V1, V2, V3: (Posterior Anterior) Leads V4, V5, V6:(Right Left, or lateral)
  10. Unipolar (+) chest leads (horizontal plane): Leads V1, V2, V3: (Posterior Anterior) Leads V4, V5, V6:(Right Left, or lateral)
  11. Hint: up in both = normal. If Down in I and Up in II => Reaching towards eachother = RAD. Up in I and down in II = leaving = LAD
  12. Normal Axis
  13. Normal Axis
  14. Rightward axis
  15. Rightward axis
  16. Left axis
  17. Left axis
  18. Normal Axis
  19. Normal Axis
  20. Note: structural abnormality detection is not very sensitive or specific – echo is definitive
  21. ST depression can be either upsloping, downsloping, or horizontal (see diagram below). Horizontal or downsloping ST depression ≥ 0.5 mm at the J-point in ≥ 2 contiguous leads indicates myocardial ischaemia (according to the 2007 Task Force Criteria). ST depression ≥ 1 mm is more specific and conveys a worse prognosis. ST depression ≥ 2 mm in ≥ 3 leads is associated with a high probability of NSTEMI and predicts significant mortality (35% mortality at 30 days). Upsloping ST depression is non-specific for myocardial ischaemia  
  22. ST elevation is maximal in the anteroseptal leads (V1-4). Q waves are present in the septal leads (V1-2). There is also some subtle STE in I, aVL and V5, with reciprocal ST depression in lead III. There are hyperacute (peaked ) T waves in V2-4. These features indicate a hyperacute anteroseptal STEMI
  23. Same patient 45 minutes later
  24. Culprit lesion: RCA sinus bradycardia about 55/min normal conduction intermediate (normal) axis normal p wave morphology tall R in V2, otherwise normal QRS morphology ST elevation in II, III, AVF (in III > II). Depression in I, AVL, V2. Conclusion: Inferoposterior MI caused by a RCA occlusion Arguments in favor of RCA occlusion (instead of RCX): ST depression in I, AVL bradycardia ST elevation in III > II ('the highest ST elevation points at the culprit lesion')
  25. 75, Normal sinus, normal axis, normal intervals, no ischemia
  26. LBBB, does not meet sgarbossa
  27. Diffuse subendocardial ischemia (ST-depression
  28. RBBB