Fractures can be classified in several ways, including by their relationship to the environment (closed vs open), their displacement (translation, angulation, shortening), fracture pattern (transverse, oblique, etc.), and etiology (traumatic vs pathological). Compound fractures involve a break in the overlying skin. Gustilo-Anderson classification grades open fractures based on soft tissue damage. Fracture healing occurs in stages, from hematoma formation and inflammation to callus formation and remodeling. Management includes first aid, closed or open reduction, and immobilization techniques like internal/external fixation, traction, splinting, and casting to align fragments and promote healing. Complications can be early like fat embolism or
classification of soft tissue injuries. gustilo anderson classification, tscheren classification, hanover fracture scale and ao soft tissue grading system, types of wounds. orthopedic open fracture classification for management of soft tissue injuries
Injuries to a ligament are common, especially during athletic activity. Ligaments in the ankle, knee, and wrist are consistently in action during athletic activity and thus are under a lot of stress.
Dr. Bharani Kumar Dayanandam is a prominent Orthopaedic Surgeon providing a wide range of treatments for Shoulder Injuries in Chennai, India
Visit us @ https://www.chennaiorthopaedics.com
classification of soft tissue injuries. gustilo anderson classification, tscheren classification, hanover fracture scale and ao soft tissue grading system, types of wounds. orthopedic open fracture classification for management of soft tissue injuries
Injuries to a ligament are common, especially during athletic activity. Ligaments in the ankle, knee, and wrist are consistently in action during athletic activity and thus are under a lot of stress.
Dr. Bharani Kumar Dayanandam is a prominent Orthopaedic Surgeon providing a wide range of treatments for Shoulder Injuries in Chennai, India
Visit us @ https://www.chennaiorthopaedics.com
fracture is the breakdown in the continutity of the bone alignment this has many types as the fracure this topic include its definition , etiology, pathophysiology, clinical menisfestation, diagnosis and its treatment which can be used by nursing students for taking care of the patient suffering from fracture and for learning for their examination and knowledge purpose
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
2. DEFINATION:
“ BREAK IN THE CONTINUITY OF BONE”.
BREAK DOWN OF BONE IN TWO OR MORE THEN
TWO PARTS.
RESULTS FROM :1} INJURY
2} REPETITIVE STRESS
3} ABNORMAL WEAKNESS
5. OPEN FRACTURES:
A fracture with break in the
overlying skin and soft tissues
,leading to the fracture
communicating with the ext.
environment
6. Gustilo and Anderson open fracture classification:
The Gustillo classification is used to classify open fractures.
Three grades that try to quantify the amount of soft tissue damage associated with
the fracture
I A low energy open fracture with a wound less than 1 cm long and clean
II An open fracture with a laceration more than 1 cm long
without extensive soft tissue damage, flaps or avulsion
III Characterised by high energy injury irrespective of the size
of the wound. Extensive damage to soft tissues, including muscles, skin, and
neurovascular structures, and a high degree of contamination. Multifragmentary and
unstable fractures
7. GRADE 3A
LIMITED STRIPPING OF PERIOSTEUM AND
SOFT TISSUE FROM BONE.
ADEQUATE SOFT TISSUE COVERAGE FOR
BONE, TENDONS AND NEUROVASCULAR
BUNDLE AFTER STABILIZATION.
8. Type 3B
Extensive stripping of soft tissue
and periosteum from bone.
Requires a local flap or free tissue
transfer
Type 3C
A major vascular injury requiring
repair
9.
10. Muller’s Classification:
EACH LONG BONE HAS 3 SEGMENTS
Proximal, Diaphyseal and Distal
DIAPHYSEAL FRACTURES:
Simple
Wedge
Complex
PROXIMAL & DISTAL
Extra-Articular
Partial Articular
Complete Articular
11. AO International classification
It uses an alphanumeric code:
a) First digit specifies the bone
1 = humerus 2= radius/ulna
3 =femur 4 =tibia/fibula
b)Second num relates the
segment:
1= proximal 2=diaphyseal
3= distil 4= malleolar
C) a letter specifies the fracture
pattern:
For diaphysis
A = simple B= wedge
C= complex
For metaphysis
A = extra articular B= partial
articular
C= complete articular
13. Displacement - Translation
Translation is sideways motion of the
fracture - usually described as a
percentage of movement when
compared to the diameter of the bone -
- ----------direction of distal fragment
decides
16. Classification: Based on Pattern
1. Transverse
2. Oblique
3. Spiral
4. Comminuted
5. Segmental
6. Stellate
17. Transverse Fracture
A fracture in
which the line is
perpendicular to
the long axis of the
bone
Oblique Fracture
A fracture in
which the # line is
at oblique angle
to the long axis of
the bone.
Spiral Fracture
A severe form of oblique
fracture in which the #
plane rotates along the long
axis of the bone. These #s
occur secondary to
rotational force.
18. Comminuted
Fracture :
The bone is
broken into many
fragments.
Stellate Fracture:
This # occurs in the
flat bones of the skull
and in the patella,
where the fracture
lines run in various
directions from one
point.
Impacted Fracture:
This # where a vertical
force drives the distal
fragment of the
fracture into the
proximal fragment.
19. Depressed Fracture:
This # occurs in the
skull where a
segment of bone
gets depressed into
the cranium.
Avulsion Fracture:
A chip of bone is avulsed by the sudden and
unexpected contraction of a powerful muscle
from its point of insertion,
Examples
1. ASIS Avulsion
2. JONE’S 5th MT base Avulsion
20. Incomplete fracture
The bone is incompletely divided and the periosteum remains in continuity
Greenstick fracture : bone is buckled or bent
Mainly seen in children, because of their springy bones
Plastically deformed bones
Compressed fracture: crumpled cancellous bone
Seen in adults, mainly in vertebral bodies, calcaneum and tibial plateu
21. Classification: Based on Etiology
1. TRAUMATIC
2. PATHOLOGICAL
Tumors
Bone cysts
Osteomyelitis
Osteoporosis
Osteogenesis imperfecta
Rickets
23. Type I fracture is
when there is a
fracture across the
physis with no
metaphysial or
epiphysial injury
Type III fracture is
when there is a
fracture across the
physis which
extends into the
epiphysis
Type II fracture is
when there is a
fracture across the
physis which extends
into the metaphysis
24. Type IV fracture is
when there is a
fracture through
metaphysis, physis,
and epiphysis
Type V fracture is when
there is a crush injury
to the physis
Type VI fracture is rare
injury consisting of injury
to perichondral structures
by direct trauma e.g. heat
or chemical.
25. BONE HEALING:
PRIMARY FRACTURE HEALING
refers to fractures treated operatively without
callus formation
SECONDARY FRACTURE HEALING
refers to (a) fractures treated non-operatively,
with the formation of callus and no disturbance
of hematoma; (b) fractures operated without
disturbance of hematoma
26. FACTORS AFFECTING BONE HEALING:
(A) Age: Fractures unite faster in children
(B) Type of bone: Faster union in flat and cancellous bone
(C) Pattern of fracture: Spiral # > oblique # > transverse # > comminuted #
(D) Disturbed pathoanatomy: soft tissue interposition and ischaemic # prevent faster healing
(E)Type of reduction: good apposition of
fracture results in faster healing
(F)Immobilisation: depends on the fracture site
eg. Fracture ribs and scapula do not require
immobilisation
(G)Open fractures: often go into delayed union
and non-union
(H)Compression of fracture site: enhances
union(cancellous bone) and primary bone
healing(cortical bone)
27. HEALING BY CALLUS:
STAGE 1: TISSUE DESTRUCTION AND HEMATOMA
FORMATION
- lasts for 7 days
- blood leaks out of torn vessels and forms a
hematoma between and around fracture
- periosteum and local soft tissues are stripped off
- ischaemic necrosis – death of some osteocytes with
sensitization of the remaining precursor cells
28. STAGE 2: INFLAMMATION AND CELLULAR
PROLIFERATION/GRANULATION TISSUE:
-lasts for 2-3 weeks
- precursor cells form cells that differentiate and
organize to provide vessels, fibroblasts, osteoblasts etc
- soft granulation tissue formed between
fracture fragments, providing anchorage to fracture
- hematoma is slowly absorbed and fine new
capillaries grow into the area
29. STAGE 3: CALLUS FORMATION
- lasts for 4-12 weeks
- granulation tissue differentiates and creates
osteoblasts, laying down intercellular matrix
impregnated with calcium salts
- formation of callus/woven bone
- provides good strength to the fracture,
decreasing the movements at the fracture
site and causes union in about 4 weeks
30. STAGE 4: REMODELLING
- takes 1-4 years for the bone to become
strong enough to carry weight
- with continuing osteoclastic and osteoblastic
activities, the woven bone gets transformed into lamellar
bone
- osteoblasts fill in the remaining gap between the new
bone and the fragments to strengthen the bone
31. STAGE 5: MODELLING
- stage where the bone is gradually
strengthened
- shapening of the cortices occurs at the
endosteal and periosteal surfaces
- all these occur when the person starts
resuming his activities ie bearing weight and muscle
forces
- thicker lamellae are laid down where high
stresses are present, unwanted buttresses are
carved away and medullary cavity is reformed
34. MEDICAL /SURGICAL
MANAGEMENT OF FRACTURES:
1. Reduction
Reduction of a fracture (“setting” the bone) refers to restoration of
the fracture fragments to anatomic alignment and rotation.
It’s a surgical approach, the fracture fragments are reduced.
External/Internal fixation devices (metallic pins, wires, screws, plates,
nails, or rods) may be used to hold the bone fragments in
position until solid bone healing occurs.
36. External fixation :
“External Fixator is a device uses for stabilization and
immobilization of long bone open fractures.
Types
Type -1 Unilateral Uniplanar
Type -2 Uniplanar Bilateral.
Type -3 ◦ Classical Bilateral Biplanar.
◦ Delta Unilateral Biplanar
According to Planes:
◦ Planner: Hoffman’s, orthofix etc.
◦ Circular: Ilizarov
39. Closed reduction :
closed reduction is accomplished by bringing the bone fragments
into apposition (ie, placing the ends in contact) through
manipulation and manual traction.
Extremity is held in the desired position while the physician applies a
cast, splint, or other device.
X - rays are obtained to verify that the bone fragments are correctly
aligned.
Traction (skin or skeletal) may be used to effect fracture reduction
and immobilization.
40. Traction
Traction is the use of weights, ropes and pulleys to apply
force to tissues surrounding a broken bone.
41. Traction:
1. Skin traction-
Bucks traction used for knee,hip bone fracture
Weight usually 5-7 pounds attach to skin
2. Skeletal traction –
Needs invasive procedure
Weight is upto 10 kg attached to bone
42. Splinting:
Splinting is the most common procedure for immobilizing an injury.
The splint should go beyond the joints above and below the fractured or
dislocated bone to prevent these from moving
43. Complication of fracture :
Early complications
Shock
fat embolism
compartment syndrome
deep vein thrombosis
disseminated intravascular coagulopathy
Infection
Delayed complications
delayed union and nonunion
avascular necrosis of bone
reaction to internal fixation devices
45. Compartment Syndrome
Occurs with fracture of elbow, forearm bones, proximal third of
tibia, hands or foot ;
Crush injuries and circumferential burns
Increase of pressure within the osseofascial compartment
Due to bleeding , oedema or inflammation
46. Confirmation of diagnosis:
Measuring the intracompartmental pressures
Introduced a split catheter into the compartment
Pressure measured close to level of the fracture
Differential pressure (difference between the diastolic pressure and
compartment pressure ) is <30mmHg – immediate decompression
Management:
Remove any casts, bandages and dressings
Differential pressure < 30mmHg – immediate fasciotomy
47. LATE COMPLICATIONS
DELAYED UNION
When a fracture takes more than usual time to unite, it is said
to have gone in ‘delayed union’.
NON-UNION
Delayed union gradually turns into non-union – that is it
becomes apparent that the fracture will never unite without
intervention
Movement can be elicited at the fracture site and pain
diminishes; the fracture gap becomes a type of
pseudoarthrosis
48. • Malunion:
• the fragments join in an unsatisfactory position
(unacceptable angulation, rotation or shortening)
• Causes are :
failure to reduce a fracture adequately
failure to hold reduction while healing proceeds
gradual collapse of comminuted or osteoporotic bone.