This document discusses tests used to evaluate facial nerve function including:
1. Topographic or topodiagnostic tests evaluate specific branches of the facial nerve including the Schirmer test for lacrimation (geniculate ganglion), stapedial reflex test (stapedius branch), taste testing (chorda tympani nerve), and measuring salivary flow rates and pH (chorda tympani nerve).
2. Electrophysiological tests include nerve excitability testing, electromyography, maximal stimulation testing, and electroneuronography.
3. The Schirmer test evaluates lacrimation mediated by the geniculate ganglion and petrosal nerve. A difference in wetted
The document provides an overview of the anatomy of the eye and orbit. It describes the seven bones that make up the bony orbit, including the frontal, zygomatic, maxillary, ethmoidal, sphenoid, lacrimal and palatine bones. It details the structures forming each wall of the orbit, such as the medial orbital wall formed by the frontal process of maxillary, lacrimal bone, orbital plate of ethmoid and lesser wing of sphenoid. Key orbital foramina and fissures transmitting nerves and vessels are also outlined, along with the blood supply and venous drainage pathways. Sinuses related to the orbit including the frontal, ethmoid, sphenoid and maxillary sinuses
The nose serves several important physiological functions:
- It warms, humidifies, and filters incoming air, protecting the lower airways. Nasal secretions containing enzymes, antibodies, and other proteins help fight infections.
- Airflow through the nose is turbulent due to its irregular shape and variable cross-section. The nasal cycle causes periodic congestion of one side to control airflow.
- Rhinomanometry measures nasal resistance by determining the relationship between pressure and airflow, providing information about nasal patency and function.
The paranasal sinuses are air spaces that develop within the bones of the skull. There are four pairs - maxillary, frontal, ethmoid, and sphenoid. They are rudimentary at birth and enlarge throughout childhood. The maxillary sinus is the largest and the first to develop. Acute sinusitis is usually caused by bacterial infections spreading from the nose. Chronic sinusitis lasts for months or years. Complications can include infections of nearby structures like the orbit or brain. Neoplasms of the sinuses can also occur but are usually benign growths like osteomas.
The facial nerve originates from the pons and has both motor and sensory components. It passes through the internal acoustic meatus, facial canal within the temporal bone, and exits at the stylomastoid foramen. Within the facial canal it has several segments separated by genu. Important surgical landmarks along the facial canal include the geniculate ganglion, cochleariform process, and digastric ridge. The main trunk divides into 5 terminal branches in the parotid gland which innervate muscles of facial expression. Careful knowledge of the anatomy is important for safe middle ear, mastoid, and parotid surgery.
This document discusses techniques for reanimating facial paralysis. It begins by outlining general principles, including reinnervating muscles early, separately reanimating the upper and lower face, and tailoring the procedure to the patient's needs and assessment. Surgical techniques are then described, including neural methods like nerve grafting and transfers, musculofacial transpositions, and static procedures. The timing of different techniques depends on whether the paralysis is acute (<3 weeks), intermediate (3 weeks to 2 years), or chronic (>2 years). Assessment involves evaluating the cause and extent of paralysis along with patient factors. The goal is to restore facial symmetry, competence, protection, and dynamic smile.
Clinical anatomy of facial nerve and facial nerve palsy Ramesh Parajuli
The facial nerve is a mixed nerve that originates in the brainstem and has motor, sensory, and parasympathetic functions. It has several segments as it exits the brainstem and travels through the skull and internal auditory canal before exiting behind the ear. It gives off several branches and terminates in branches that innervate the muscles of facial expression. Facial nerve palsy can result from various causes like Bell's palsy, trauma, infection, tumors, or iatrogenic injuries. Clinical assessment and electrical tests can localize the site of injury which guides management including medications, physical therapy, or surgical interventions like decompression or repair.
The infratemporal fossa is a complex space located deep to the mandible containing neurovascular structures. It has boundaries of the maxilla anteriorly, styloid process posteriorly, and lateral pterygoid plate medially. Contents include the lateral and medial pterygoid muscles, fat pad, buccal lymph node, mandibular nerve and its branches, maxillary artery, and otic ganglion. The fossa communicates superiorly with the cranial cavity and medially with the pterygopalatine fossa. Anatomy of this region is important for spread of infection, tumors, and trauma.
This document discusses tests used to evaluate facial nerve function including:
1. Topographic or topodiagnostic tests evaluate specific branches of the facial nerve including the Schirmer test for lacrimation (geniculate ganglion), stapedial reflex test (stapedius branch), taste testing (chorda tympani nerve), and measuring salivary flow rates and pH (chorda tympani nerve).
2. Electrophysiological tests include nerve excitability testing, electromyography, maximal stimulation testing, and electroneuronography.
3. The Schirmer test evaluates lacrimation mediated by the geniculate ganglion and petrosal nerve. A difference in wetted
The document provides an overview of the anatomy of the eye and orbit. It describes the seven bones that make up the bony orbit, including the frontal, zygomatic, maxillary, ethmoidal, sphenoid, lacrimal and palatine bones. It details the structures forming each wall of the orbit, such as the medial orbital wall formed by the frontal process of maxillary, lacrimal bone, orbital plate of ethmoid and lesser wing of sphenoid. Key orbital foramina and fissures transmitting nerves and vessels are also outlined, along with the blood supply and venous drainage pathways. Sinuses related to the orbit including the frontal, ethmoid, sphenoid and maxillary sinuses
The nose serves several important physiological functions:
- It warms, humidifies, and filters incoming air, protecting the lower airways. Nasal secretions containing enzymes, antibodies, and other proteins help fight infections.
- Airflow through the nose is turbulent due to its irregular shape and variable cross-section. The nasal cycle causes periodic congestion of one side to control airflow.
- Rhinomanometry measures nasal resistance by determining the relationship between pressure and airflow, providing information about nasal patency and function.
The paranasal sinuses are air spaces that develop within the bones of the skull. There are four pairs - maxillary, frontal, ethmoid, and sphenoid. They are rudimentary at birth and enlarge throughout childhood. The maxillary sinus is the largest and the first to develop. Acute sinusitis is usually caused by bacterial infections spreading from the nose. Chronic sinusitis lasts for months or years. Complications can include infections of nearby structures like the orbit or brain. Neoplasms of the sinuses can also occur but are usually benign growths like osteomas.
The facial nerve originates from the pons and has both motor and sensory components. It passes through the internal acoustic meatus, facial canal within the temporal bone, and exits at the stylomastoid foramen. Within the facial canal it has several segments separated by genu. Important surgical landmarks along the facial canal include the geniculate ganglion, cochleariform process, and digastric ridge. The main trunk divides into 5 terminal branches in the parotid gland which innervate muscles of facial expression. Careful knowledge of the anatomy is important for safe middle ear, mastoid, and parotid surgery.
This document discusses techniques for reanimating facial paralysis. It begins by outlining general principles, including reinnervating muscles early, separately reanimating the upper and lower face, and tailoring the procedure to the patient's needs and assessment. Surgical techniques are then described, including neural methods like nerve grafting and transfers, musculofacial transpositions, and static procedures. The timing of different techniques depends on whether the paralysis is acute (<3 weeks), intermediate (3 weeks to 2 years), or chronic (>2 years). Assessment involves evaluating the cause and extent of paralysis along with patient factors. The goal is to restore facial symmetry, competence, protection, and dynamic smile.
Clinical anatomy of facial nerve and facial nerve palsy Ramesh Parajuli
The facial nerve is a mixed nerve that originates in the brainstem and has motor, sensory, and parasympathetic functions. It has several segments as it exits the brainstem and travels through the skull and internal auditory canal before exiting behind the ear. It gives off several branches and terminates in branches that innervate the muscles of facial expression. Facial nerve palsy can result from various causes like Bell's palsy, trauma, infection, tumors, or iatrogenic injuries. Clinical assessment and electrical tests can localize the site of injury which guides management including medications, physical therapy, or surgical interventions like decompression or repair.
The infratemporal fossa is a complex space located deep to the mandible containing neurovascular structures. It has boundaries of the maxilla anteriorly, styloid process posteriorly, and lateral pterygoid plate medially. Contents include the lateral and medial pterygoid muscles, fat pad, buccal lymph node, mandibular nerve and its branches, maxillary artery, and otic ganglion. The fossa communicates superiorly with the cranial cavity and medially with the pterygopalatine fossa. Anatomy of this region is important for spread of infection, tumors, and trauma.
The document provides information on the anatomy of the external and middle ear. It discusses the development of the external ear, auricle, external auditory canal, tympanic membrane, and ossicles from pharyngeal arches. It then describes the structures and walls that make up the middle ear cleft, including the tympanic cavity, eustachian tube, and mastoid air cells. Key structures like the ossicles, nerves, muscles, and mucosal folds within the middle ear are also outlined.
The infratemporal fossa is located below the temporal fossa. It is bounded by the ramus of the mandible laterally, the maxilla anteriorly, and the lateral pterygoid plate medially. The infratemporal fossa contains the mandibular nerve, maxillary artery, pterygoid venous plexus, and the medial and lateral pterygoid muscles. The maxillary artery passes through the infratemporal fossa and gives off several branches including the middle meningeal artery, accessory meningeal artery, inferior alveolar artery, and infraorbital artery. It communicates with surrounding areas through gaps in bones and openings in the skull.
The retropharyngeal space is a potential space located posterior to the pharynx that contains areolar fat and lymph nodes. It allows for movement of the pharynx during swallowing and respiration. Lesions and fluid collections like abscesses or hematomas can develop in this space. Imaging like CT scans are useful for evaluating these conditions. Retropharyngeal abscesses require prompt treatment with antibiotics and drainage to prevent airway complications. Lymph nodes in this region can metastasize early from cancers like nasopharyngeal carcinoma.
The document discusses the anatomy, course, branches and clinical aspects of the facial nerve (cranial nerve VII). Some key points:
- The facial nerve has both motor and sensory components. It innervates the muscles of facial expression and provides parasympathetic innervation to certain glands.
- The course of the nerve can be divided into intracranial, intratemporal and extracranial parts as it exits the brainstem and travels through the temporal bone.
- Common causes of facial nerve palsy include Bell's palsy (idiopathic), herpes zoster infection, fractures of the temporal bone, parotid surgery and tumors in the parot
The facial nerve has three nuclei and contains approximately 10,000 fibers. It exits the brainstem at the pontomedullary junction and travels through the internal acoustic meatus and fallopian canal. It has motor, parasympathetic, and sensory functions. Facial nerve palsy can result from various causes such as Bell's palsy, tumors, fractures, or inflammation. Diagnosis involves evaluating for signs of upper vs. lower motor neuron involvement. Treatment depends on the cause but may include corticosteroids, antivirals, or decompression surgery.
The document discusses the embryology, anatomy, components, causes of injury, grading systems, evaluation, and treatment of the facial nerve. It covers the development of the facial nerve from the embryonic stage through maturity and describes the various parts of the nerve and their functions. The document also outlines different classification systems for nerve injuries, approaches for evaluating facial nerve paralysis, and surgical and non-surgical techniques for treating injuries or reanimating paralysis of the facial nerve.
Cochlear Fluid is the one of the most important fluid not only for hearing sensation but also for the balance of human body. It is very important to know the embryology, anatomy, and physiology of cochlear fluid mechanism to know the various pathological conditions of inner ear.
This document discusses the anatomy, development, classification and diagnosis of microtia, as well as approaches to total auricular reconstruction for microtia. It covers the nerve supply and embryological development of normal ears. It also describes classifications of microtia, associated deformities, timing of surgery, and factors to consider in patient assessment for ear reconstruction, including facial symmetry, skin envelope, vestige skin and hair.
The document discusses the use of the Hadad-Bassagasteguy (HB) flap in reconstructing anterior skull base defects after endonasal skull base surgery. The HB flap uses the vascularized nasal septal mucoperiosteum to repair defects. In a study of 53 patients who underwent HB flap reconstruction, only 2 patients (3.8%) experienced post-operative cerebrospinal fluid leaks. The study found the HB flap to be effective at preventing post-operative CSF leaks across a variety of patient profiles and skull base surgery types. The HB flap is becoming a standard technique for reconstructing anterior skull base defects due to its high success rate and versatility.
Eustachian tube final PP ANATOMY,EMBRYOLOGY,FUNCTIONS,DYSFUNCTIONS TREATMENT,...social service
The Eustachian tube connects the middle ear cavity to the nasopharynx. It has important functions like regulating middle ear pressure and ventilation, protecting the middle ear, and facilitating mucociliary clearance and drainage. Dysfunction of the Eustachian tube can lead to negative pressure in the middle ear, retraction of the tympanic membrane, fluid accumulation, and acute otitis media. Tests for Eustachian tube function include the Valsalva maneuver, Toynbee's maneuver, and tympanometry. Treatment options for Eustachian tube dysfunction include medical management with nasal decongestants or steroids, as well as surgical procedures like my
The document provides an overview of the anatomy and embryology of the external and middle ear. It describes how the external ear develops from the first and second pharyngeal arches. It then details the anatomy of the auricle, external acoustic canal, and tympanic membrane. For the middle ear, it discusses the embryological development and describes the structures of the middle ear cleft, tympanic cavity, ossicles, muscles, nerves and blood supply.
Endoscopic anatomy of lateral wall of sphenoid sinusAnkit Choudhary
This document discusses the endoscopic anatomy of the lateral wall of the sphenoid sinus and important relationships. It notes that the sphenoid ostium opens into the sphenoethmoidal recess 1-1.5cm above the roof of the choana. Below the ostium lies the Woodruff's plexus and the septal branch of the sphenopalatine artery runs across the anterior wall. Approaches to the sphenoid sinus include paraseptal, intermediate, and lateral. Precautions are discussed such as dilating the ostium inferiomedially first and taking care with dehiscent structures like the optic nerve or internal carotid artery.
This is a presentation I used for my seminar on 'Phonosurgery' on 4th November, 2015. I hope they are useful to you. Constructive as well as Destructive criticism welcomed.
This document discusses the physiology of phonation, or voice production. It defines phonation as the rapid opening and closing of the vocal cords due to the separation and apposition of the vocal folds, accompanied by breath under lung pressure, which creates vocal sound. It describes the anatomy involved in voice production including the lungs, diaphragm, larynx, throat, mouth and nose. It discusses theories of voice production and covers topics like pitch, volume, quality, vocal registers, vocal disorders, vocal injury, and video stroboscopy.
Surgical anatomy of Infratemporal fossa. by Dr. Aditya TiwariAditya Tiwari
This document provides an overview of the anatomy of the infratemporal fossa. It describes the boundaries, contents, neurovasculature, and communications of the infratemporal fossa. Key structures discussed include the maxillary artery and its branches, the mandibular nerve and its branches, the otic ganglion, and muscles such as the temporalis, lateral pterygoid, and medial pterygoid. Surgical approaches and nerve blocks related to the infratemporal fossa are also summarized.
Facial nerve traumatic injury and repairsarita pandey
The document discusses the anatomy and physiology of the facial nerve, as well as injuries and repair. It covers:
- The gross anatomy and histology of the facial nerve as it traverses the temporal bone.
- Classifications of facial nerve injuries including neuropraxia, axonotmesis, neurotmesis, and transection.
- The degeneration and regeneration processes after injury, including changes in the proximal and distal nerve segments and target muscles.
- Surgical techniques for nerve repair including fascicular repair, grafting, and principles such as achieving a tension-free repair.
- Scales for assessing recovery after compression injuries or surgery, such as the House-B
The document discusses the facial nerve (cranial nerve VII) in three sentences:
It originates in the brainstem and is a mixed nerve that controls facial muscle movement and taste sensation. It exits the skull through the stylomastoid foramen and gives off several branches as it passes through the parotid gland to innervate facial muscles. Disorders of the facial nerve can occur from various causes such as trauma, infections like Bell's palsy, or tumors and result in paralysis of the muscles on the same side of the face.
Pterygopalatine fossa and approaches by Dr.Ashwin MenonDr.Ashwin Menon
The pterygopalatine fossa is a small pyramidal space located between the posterior maxilla and pterygoid processes. It contains the maxillary nerve, pterygopalatine ganglion, vidian nerve and branches of the maxillary artery. The fossa has anterior, posterior, medial, lateral and superior walls. Imaging shows its low density due to contained fat. Conditions involving the fossa include referred otalgia, foramen ovale lesions, and hay fever. Nerve blocks of the maxillary, mandibular and inferior alveolar nerves provide anesthesia to the region. The transantral approach is commonly used to access the fossa during procedures like vidian neurectomy.
This document discusses the anatomy and physiology of the human olfactory system. It describes:
1. The main structures involved in smell including the olfactory epithelium, olfactory bulb, and olfactory cortex.
2. The cell types within the olfactory epithelium including bipolar receptor neurons, supporting cells, and basal stem cells.
3. How odorant molecules bind to receptors on cilia and trigger signal transduction pathways to activate olfactory neurons.
4. The pathways from the olfactory bulb to various parts of the limbic system involved in emotional and memory processing of smells.
This document discusses facial nerve palsy, also known as Bell's palsy. It begins by describing the facial nerve and the muscles it innervates. Facial nerve palsy is characterized by partial or complete loss of function of the facial nerve. The document then covers the causes, types (upper vs lower motor neuron), clinical features, investigations, and management of facial nerve palsy. Both non-surgical and surgical treatment options are described to restore facial symmetry and functions like eye closure and oral competence.
The document provides information on the anatomy of the external and middle ear. It discusses the development of the external ear, auricle, external auditory canal, tympanic membrane, and ossicles from pharyngeal arches. It then describes the structures and walls that make up the middle ear cleft, including the tympanic cavity, eustachian tube, and mastoid air cells. Key structures like the ossicles, nerves, muscles, and mucosal folds within the middle ear are also outlined.
The infratemporal fossa is located below the temporal fossa. It is bounded by the ramus of the mandible laterally, the maxilla anteriorly, and the lateral pterygoid plate medially. The infratemporal fossa contains the mandibular nerve, maxillary artery, pterygoid venous plexus, and the medial and lateral pterygoid muscles. The maxillary artery passes through the infratemporal fossa and gives off several branches including the middle meningeal artery, accessory meningeal artery, inferior alveolar artery, and infraorbital artery. It communicates with surrounding areas through gaps in bones and openings in the skull.
The retropharyngeal space is a potential space located posterior to the pharynx that contains areolar fat and lymph nodes. It allows for movement of the pharynx during swallowing and respiration. Lesions and fluid collections like abscesses or hematomas can develop in this space. Imaging like CT scans are useful for evaluating these conditions. Retropharyngeal abscesses require prompt treatment with antibiotics and drainage to prevent airway complications. Lymph nodes in this region can metastasize early from cancers like nasopharyngeal carcinoma.
The document discusses the anatomy, course, branches and clinical aspects of the facial nerve (cranial nerve VII). Some key points:
- The facial nerve has both motor and sensory components. It innervates the muscles of facial expression and provides parasympathetic innervation to certain glands.
- The course of the nerve can be divided into intracranial, intratemporal and extracranial parts as it exits the brainstem and travels through the temporal bone.
- Common causes of facial nerve palsy include Bell's palsy (idiopathic), herpes zoster infection, fractures of the temporal bone, parotid surgery and tumors in the parot
The facial nerve has three nuclei and contains approximately 10,000 fibers. It exits the brainstem at the pontomedullary junction and travels through the internal acoustic meatus and fallopian canal. It has motor, parasympathetic, and sensory functions. Facial nerve palsy can result from various causes such as Bell's palsy, tumors, fractures, or inflammation. Diagnosis involves evaluating for signs of upper vs. lower motor neuron involvement. Treatment depends on the cause but may include corticosteroids, antivirals, or decompression surgery.
The document discusses the embryology, anatomy, components, causes of injury, grading systems, evaluation, and treatment of the facial nerve. It covers the development of the facial nerve from the embryonic stage through maturity and describes the various parts of the nerve and their functions. The document also outlines different classification systems for nerve injuries, approaches for evaluating facial nerve paralysis, and surgical and non-surgical techniques for treating injuries or reanimating paralysis of the facial nerve.
Cochlear Fluid is the one of the most important fluid not only for hearing sensation but also for the balance of human body. It is very important to know the embryology, anatomy, and physiology of cochlear fluid mechanism to know the various pathological conditions of inner ear.
This document discusses the anatomy, development, classification and diagnosis of microtia, as well as approaches to total auricular reconstruction for microtia. It covers the nerve supply and embryological development of normal ears. It also describes classifications of microtia, associated deformities, timing of surgery, and factors to consider in patient assessment for ear reconstruction, including facial symmetry, skin envelope, vestige skin and hair.
The document discusses the use of the Hadad-Bassagasteguy (HB) flap in reconstructing anterior skull base defects after endonasal skull base surgery. The HB flap uses the vascularized nasal septal mucoperiosteum to repair defects. In a study of 53 patients who underwent HB flap reconstruction, only 2 patients (3.8%) experienced post-operative cerebrospinal fluid leaks. The study found the HB flap to be effective at preventing post-operative CSF leaks across a variety of patient profiles and skull base surgery types. The HB flap is becoming a standard technique for reconstructing anterior skull base defects due to its high success rate and versatility.
Eustachian tube final PP ANATOMY,EMBRYOLOGY,FUNCTIONS,DYSFUNCTIONS TREATMENT,...social service
The Eustachian tube connects the middle ear cavity to the nasopharynx. It has important functions like regulating middle ear pressure and ventilation, protecting the middle ear, and facilitating mucociliary clearance and drainage. Dysfunction of the Eustachian tube can lead to negative pressure in the middle ear, retraction of the tympanic membrane, fluid accumulation, and acute otitis media. Tests for Eustachian tube function include the Valsalva maneuver, Toynbee's maneuver, and tympanometry. Treatment options for Eustachian tube dysfunction include medical management with nasal decongestants or steroids, as well as surgical procedures like my
The document provides an overview of the anatomy and embryology of the external and middle ear. It describes how the external ear develops from the first and second pharyngeal arches. It then details the anatomy of the auricle, external acoustic canal, and tympanic membrane. For the middle ear, it discusses the embryological development and describes the structures of the middle ear cleft, tympanic cavity, ossicles, muscles, nerves and blood supply.
Endoscopic anatomy of lateral wall of sphenoid sinusAnkit Choudhary
This document discusses the endoscopic anatomy of the lateral wall of the sphenoid sinus and important relationships. It notes that the sphenoid ostium opens into the sphenoethmoidal recess 1-1.5cm above the roof of the choana. Below the ostium lies the Woodruff's plexus and the septal branch of the sphenopalatine artery runs across the anterior wall. Approaches to the sphenoid sinus include paraseptal, intermediate, and lateral. Precautions are discussed such as dilating the ostium inferiomedially first and taking care with dehiscent structures like the optic nerve or internal carotid artery.
This is a presentation I used for my seminar on 'Phonosurgery' on 4th November, 2015. I hope they are useful to you. Constructive as well as Destructive criticism welcomed.
This document discusses the physiology of phonation, or voice production. It defines phonation as the rapid opening and closing of the vocal cords due to the separation and apposition of the vocal folds, accompanied by breath under lung pressure, which creates vocal sound. It describes the anatomy involved in voice production including the lungs, diaphragm, larynx, throat, mouth and nose. It discusses theories of voice production and covers topics like pitch, volume, quality, vocal registers, vocal disorders, vocal injury, and video stroboscopy.
Surgical anatomy of Infratemporal fossa. by Dr. Aditya TiwariAditya Tiwari
This document provides an overview of the anatomy of the infratemporal fossa. It describes the boundaries, contents, neurovasculature, and communications of the infratemporal fossa. Key structures discussed include the maxillary artery and its branches, the mandibular nerve and its branches, the otic ganglion, and muscles such as the temporalis, lateral pterygoid, and medial pterygoid. Surgical approaches and nerve blocks related to the infratemporal fossa are also summarized.
Facial nerve traumatic injury and repairsarita pandey
The document discusses the anatomy and physiology of the facial nerve, as well as injuries and repair. It covers:
- The gross anatomy and histology of the facial nerve as it traverses the temporal bone.
- Classifications of facial nerve injuries including neuropraxia, axonotmesis, neurotmesis, and transection.
- The degeneration and regeneration processes after injury, including changes in the proximal and distal nerve segments and target muscles.
- Surgical techniques for nerve repair including fascicular repair, grafting, and principles such as achieving a tension-free repair.
- Scales for assessing recovery after compression injuries or surgery, such as the House-B
The document discusses the facial nerve (cranial nerve VII) in three sentences:
It originates in the brainstem and is a mixed nerve that controls facial muscle movement and taste sensation. It exits the skull through the stylomastoid foramen and gives off several branches as it passes through the parotid gland to innervate facial muscles. Disorders of the facial nerve can occur from various causes such as trauma, infections like Bell's palsy, or tumors and result in paralysis of the muscles on the same side of the face.
Pterygopalatine fossa and approaches by Dr.Ashwin MenonDr.Ashwin Menon
The pterygopalatine fossa is a small pyramidal space located between the posterior maxilla and pterygoid processes. It contains the maxillary nerve, pterygopalatine ganglion, vidian nerve and branches of the maxillary artery. The fossa has anterior, posterior, medial, lateral and superior walls. Imaging shows its low density due to contained fat. Conditions involving the fossa include referred otalgia, foramen ovale lesions, and hay fever. Nerve blocks of the maxillary, mandibular and inferior alveolar nerves provide anesthesia to the region. The transantral approach is commonly used to access the fossa during procedures like vidian neurectomy.
This document discusses the anatomy and physiology of the human olfactory system. It describes:
1. The main structures involved in smell including the olfactory epithelium, olfactory bulb, and olfactory cortex.
2. The cell types within the olfactory epithelium including bipolar receptor neurons, supporting cells, and basal stem cells.
3. How odorant molecules bind to receptors on cilia and trigger signal transduction pathways to activate olfactory neurons.
4. The pathways from the olfactory bulb to various parts of the limbic system involved in emotional and memory processing of smells.
This document discusses facial nerve palsy, also known as Bell's palsy. It begins by describing the facial nerve and the muscles it innervates. Facial nerve palsy is characterized by partial or complete loss of function of the facial nerve. The document then covers the causes, types (upper vs lower motor neuron), clinical features, investigations, and management of facial nerve palsy. Both non-surgical and surgical treatment options are described to restore facial symmetry and functions like eye closure and oral competence.
The document provides information on facial palsy/paralysis, including its causes, symptoms, diagnosis, and treatment options. It discusses how facial palsy can result from various congenital, traumatic, infectious, neoplastic, and metabolic disorders. Diagnostic tests are described that evaluate different branches of the facial nerve, such as the Schirmer test for the lacrimal branch. Management includes medical approaches like corticosteroids as well as surgical options when needed like nerve decompression or microsurgery. Prognosis depends on factors like results from electrophysiological tests measured within the first weeks.
Diseases of Autonomic Nervous System I Autonomic Nervous System II Nervous Sy...HM Learnings
Diseases of Autonomic Nervous System I Autonomic Nervous System II Nervous System Physiology I
This video will cover the following topics:
1. Diseases of Sympathetic Nervous System
2. Horner Syndrome- Pathophysiology, Etiology, Clinical features
3. Raynaud Phenomenon- Pathophysiology, Clinical features
4. Diseases of the Parasympathetic Nervous System
5. Argyll Robertson Pupil- Pathophysiology, Clinical features
6. Adie tonic Pupil- Pathophysiology, Clinical features
You can also watch the same topic on HM Learnings Youtube channel.
You can also follow HM Learnings on facebook, instagram and twitter for daily updates
This document provides information on facial palsy (facial paralysis), including:
1. It discusses the anatomy of the facial nerve and different classifications of nerve injuries.
2. Common causes of facial palsy are also outlined, such as Bell's palsy which is an idiopathic sudden onset paralysis of the facial nerve.
3. Evaluation and assessment of facial nerve function is described, including tests of tear production, taste, saliva flow, and electrical nerve testing to determine the severity and likely prognosis of the palsy.
This document provides information on facial paralysis (palsy) including its causes, types, treatments, and more. It begins with an introduction to facial function and paralysis. It then covers nerve anatomy and classifications of nerve injuries. Specific topics include facial nerve anatomy, types of facial paralysis (central vs peripheral), common causes like Bell's palsy, and surgical treatment options depending on when paralysis occurred (acute, intermediate, or chronic stages). Evaluation methods and the House-Brackmann grading scale for facial function are also summarized.
This document provides information on the facial nerve (cranial nerve VII) including its embryology, anatomy, functions, and various disorders. It discusses the facial nerve's motor and sensory roles. Disorders covered include Bell's palsy, Ramsay Hunt syndrome, Moebius syndrome, and Guillain-Barré syndrome. Classification systems for facial nerve paralysis and nerve injuries are also summarized. The document provides detailed information on evaluating facial nerve disorders.
1) The document discusses a seminar on unconsciousness presented by a nurse. It defines consciousness and different levels of unconsciousness.
2) It reviews the anatomy of the brain and describes various causes of unconsciousness including structural lesions, metabolic disorders, drugs, and psychological factors.
3) Clinical manifestations involving different body systems are outlined. Assessment tools like the Glasgow Coma Scale and brainstem reflexes are also discussed.
a case of lower motor neuron facial nerve palsySamten Dorji
A 27-year-old female monk presented with weakness on the left side of her face for 2 weeks. On examination, she had signs of a left lower motor neuron 7th nerve palsy including inability to close her left eye. Dilated fundus exam showed vessel sheathing and healed scars in the left eye. She was diagnosed with House-Brackmann grade 3 left facial nerve palsy of unknown/idiopathic cause. She was treated with oral corticosteroids, facial physiotherapy, and eye lubricants.
This document provides an overview of the anatomy and embryology of the facial nerve (cranial nerve VII). It discusses the nuclei of origin, functional components, course through the skull and branches/distribution. Key points include that the facial nerve has motor, secretomotor and sensory fibers and exits the skull via the stylomastoid foramen. It describes associated ganglia like the geniculate ganglion and presents variations, disorders like Bell's palsy, and evaluation methods involving tests of motor/sensory function.
This document provides information on physiotherapy treatment for Bell's palsy. It begins with an overview of Bell's palsy, including its causes, symptoms, and grading scales. It then discusses specific assessments, including cranial nerve and facial muscle testing. Treatment approaches covered include corticosteroids, antiviral medications, eye care, facial exercises, electrical stimulation, massage, and rarely, surgery. Outcome measures used to evaluate recovery are also outlined.
This document discusses disorders of the facial nerve. It begins by describing the anatomy and course of the facial nerve, including its motor and sensory functions. It then discusses specific disorders like Bell's palsy, Ramsay Hunt syndrome, and injuries from fractures or ear/parotid surgery. Bell's palsy is described as the most common cause of facial paralysis, affecting both sexes equally and possibly resulting from viral infection or vascular issues. Diagnosis involves ruling out other causes through examination and tests. Treatment focuses on steroids, antivirals, and physiotherapy. Outcomes range from full to incomplete recovery. Other topics covered include localization of facial lesions, topodiagnostic tests, complications, and surgical procedures.
The cranial nerves VII-XII control facial expression and innervate muscles of the face and neck. The facial nerve is commonly damaged and can cause Bell's palsy with paralysis of facial muscles on one side. Damage to different branches causes specific symptoms like taste loss or ear problems. Most Bell's palsy cases recover on their own but steroids may speed recovery. The facial nerve exits the skull and splits into branches innervating individual facial muscles. Central facial palsy spares forehead muscles while peripheral lesions weaken all facial muscles on one side.
This document discusses disorders and topodiagnostic tests of the facial nerve. It covers several infections that can cause facial nerve disorders like Herpes zoster oticus and various types of ear infections. Trauma from temporal bone fractures, iatrogenic injury from ear/mastoid/parotid surgery, or facial trauma can also cause facial nerve disorders. Topodiagnostic tests like the Schirmer test, stapedial reflex test, taste test, and submandibular salivary flow test can help locate the site of lesion or injury to determine the etiology and appropriate surgical treatment.
This document discusses the anatomy and function of the facial nerve (cranial nerve VII). It begins by describing the supranuclear and infranuclear pathways of the facial nerve from the brain to the muscles of facial expression. It then discusses the facial nucleus and branches of the facial nerve. The document outlines the muscles innervated by branches of the facial nerve and clinical examination of the facial nerve's motor, sensory and secretory functions. Finally, it describes various disorders of facial nerve function including Bell's palsy, central facial palsy, and other causes of peripheral facial paralysis.
The term facial palsy generally refers to weakness of the facial muscles, mainly resulting from temporary or permanent damage to the facial nerve
Facial palsy not only cause a paresis of the target muscles, but as the nerve is responsible for a range of facial expressions, it causes serious disturbances in social life, facial expression being so important in transferring emotion.
The facial nerve is the seventh cranial nerve, or simply CN VII. It emerges from the pons of the brainstem, controls the muscles of facial expression, and functions in the conveyance of taste sensations from the anterior two-thirds of the tongue.
The document summarizes the anatomy and clinical applications of the facial nerve. It begins with the nuclear origin and functional components of the facial nerve. It then describes the intra cranial and extra cranial course of the nerve, its branches including the greater petrosal, chorda tympani, and terminal branches. Applications including facial nerve palsy, Bell's palsy, and preventing injury during dental procedures are discussed. Clinical testing and special tests of facial nerve function are also outlined.
- Video recording of this lecture in English language: https://youtu.be/Pt1nA32sdHQ
- Video recording of this lecture in Arabic language: https://youtu.be/uFdc9F0rlP0
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdfOsvaldo Bernardo Muchanga
GASTROINTESTINAL INFECTIONS AND GASTRITIS
Osvaldo Bernardo Muchanga
Gastrointestinal Infections
GASTROINTESTINAL INFECTIONS result from the ingestion of pathogens that cause infections at the level of this tract, generally being transmitted by food, water and hands contaminated by microorganisms such as E. coli, Salmonella, Shigella, Vibrio cholerae, Campylobacter, Staphylococcus, Rotavirus among others that are generally contained in feces, thus configuring a FECAL-ORAL type of transmission.
Among the factors that lead to the occurrence of gastrointestinal infections are the hygienic and sanitary deficiencies that characterize our markets and other places where raw or cooked food is sold, poor environmental sanitation in communities, deficiencies in water treatment (or in the process of its plumbing), risky hygienic-sanitary habits (not washing hands after major and/or minor needs), among others.
These are generally consequences (signs and symptoms) resulting from gastrointestinal infections: diarrhea, vomiting, fever and malaise, among others.
The treatment consists of replacing lost liquids and electrolytes (drinking drinking water and other recommended liquids, including consumption of juicy fruits such as papayas, apples, pears, among others that contain water in their composition).
To prevent this, it is necessary to promote health education, improve the hygienic-sanitary conditions of markets and communities in general as a way of promoting, preserving and prolonging PUBLIC HEALTH.
Gastritis and Gastric Health
Gastric Health is one of the most relevant concerns in human health, with gastrointestinal infections being among the main illnesses that affect humans.
Among gastric problems, we have GASTRITIS AND GASTRIC ULCERS as the main public health problems. Gastritis and gastric ulcers normally result from inflammation and corrosion of the walls of the stomach (gastric mucosa) and are generally associated (caused) by the bacterium Helicobacter pylor, which, according to the literature, this bacterium settles on these walls (of the stomach) and starts to release urease that ends up altering the normal pH of the stomach (acid), which leads to inflammation and corrosion of the mucous membranes and consequent gastritis or ulcers, respectively.
In addition to bacterial infections, gastritis and gastric ulcers are associated with several factors, with emphasis on prolonged fasting, chemical substances including drugs, alcohol, foods with strong seasonings including chilli, which ends up causing inflammation of the stomach walls and/or corrosion. of the same, resulting in the appearance of wounds and consequent gastritis or ulcers, respectively.
Among patients with gastritis and/or ulcers, one of the dilemmas is associated with the foods to consume in order to minimize the sensation of pain and discomfort.
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)MuskanShingari
Statistics- Statistics is the science of collecting, organizing, presenting, analyzing and interpreting numerical data to assist in making more effective decisions.
A statistics is a measure which is used to estimate the population parameter
Parameters-It is used to describe the properties of an entire population.
Examples-Measures of central tendency Dispersion, Variance, Standard Deviation (SD), Absolute Error, Mean Absolute Error (MAE), Eigen Value
The skin is the largest organ and its health plays a vital role among the other sense organs. The skin concerns like acne breakout, psoriasis, or anything similar along the lines, finding a qualified and experienced dermatologist becomes paramount.
How to Control Your Asthma Tips by gokuldas hospital.Gokuldas Hospital
Respiratory issues like asthma are the most sensitive issue that is affecting millions worldwide. It hampers the daily activities leaving the body tired and breathless.
The key to a good grip on asthma is proper knowledge and management strategies. Understanding the patient-specific symptoms and carving out an effective treatment likewise is the best way to keep asthma under control.
Breast cancer: Post menopausal endocrine therapyDr. Sumit KUMAR
Breast cancer in postmenopausal women with hormone receptor-positive (HR+) status is a common and complex condition that necessitates a multifaceted approach to management. HR+ breast cancer means that the cancer cells grow in response to hormones such as estrogen and progesterone. This subtype is prevalent among postmenopausal women and typically exhibits a more indolent course compared to other forms of breast cancer, which allows for a variety of treatment options.
Diagnosis and Staging
The diagnosis of HR+ breast cancer begins with clinical evaluation, imaging, and biopsy. Imaging modalities such as mammography, ultrasound, and MRI help in assessing the extent of the disease. Histopathological examination and immunohistochemical staining of the biopsy sample confirm the diagnosis and hormone receptor status by identifying the presence of estrogen receptors (ER) and progesterone receptors (PR) on the tumor cells.
Staging involves determining the size of the tumor (T), the involvement of regional lymph nodes (N), and the presence of distant metastasis (M). The American Joint Committee on Cancer (AJCC) staging system is commonly used. Accurate staging is critical as it guides treatment decisions.
Treatment Options
Endocrine Therapy
Endocrine therapy is the cornerstone of treatment for HR+ breast cancer in postmenopausal women. The primary goal is to reduce the levels of estrogen or block its effects on cancer cells. Commonly used agents include:
Selective Estrogen Receptor Modulators (SERMs): Tamoxifen is a SERM that binds to estrogen receptors, blocking estrogen from stimulating breast cancer cells. It is effective but may have side effects such as increased risk of endometrial cancer and thromboembolic events.
Aromatase Inhibitors (AIs): These drugs, including anastrozole, letrozole, and exemestane, lower estrogen levels by inhibiting the aromatase enzyme, which converts androgens to estrogen in peripheral tissues. AIs are generally preferred in postmenopausal women due to their efficacy and safety profile compared to tamoxifen.
Selective Estrogen Receptor Downregulators (SERDs): Fulvestrant is a SERD that degrades estrogen receptors and is used in cases where resistance to other endocrine therapies develops.
Combination Therapies
Combining endocrine therapy with other treatments enhances efficacy. Examples include:
Endocrine Therapy with CDK4/6 Inhibitors: Palbociclib, ribociclib, and abemaciclib are CDK4/6 inhibitors that, when combined with endocrine therapy, significantly improve progression-free survival in advanced HR+ breast cancer.
Endocrine Therapy with mTOR Inhibitors: Everolimus, an mTOR inhibitor, can be added to endocrine therapy for patients who have developed resistance to aromatase inhibitors.
Chemotherapy
Chemotherapy is generally reserved for patients with high-risk features, such as large tumor size, high-grade histology, or extensive lymph node involvement. Regimens often include anthracyclines and taxanes.
Nano-gold for Cancer Therapy chemistry investigatory projectSIVAVINAYAKPK
chemistry investigatory project
The development of nanogold-based cancer therapy could revolutionize oncology by providing a more targeted, less invasive treatment option. This project contributes to the growing body of research aimed at harnessing nanotechnology for medical applications, paving the way for future clinical trials and potential commercial applications.
Cancer remains one of the leading causes of death worldwide, prompting the need for innovative treatment methods. Nanotechnology offers promising new approaches, including the use of gold nanoparticles (nanogold) for targeted cancer therapy. Nanogold particles possess unique physical and chemical properties that make them suitable for drug delivery, imaging, and photothermal therapy.
Know the difference between Endodontics and Orthodontics.Gokuldas Hospital
Your smile is beautiful.
Let’s be honest. Maintaining that beautiful smile is not an easy task. It is more than brushing and flossing. Sometimes, you might encounter dental issues that need special dental care. These issues can range anywhere from misalignment of the jaw to pain in the root of teeth.
5-hydroxytryptamine or 5-HT or Serotonin is a neurotransmitter that serves a range of roles in the human body. It is sometimes referred to as the happy chemical since it promotes overall well-being and happiness.
It is mostly found in the brain, intestines, and blood platelets.
5-HT is utilised to transport messages between nerve cells, is known to be involved in smooth muscle contraction, and adds to overall well-being and pleasure, among other benefits. 5-HT regulates the body's sleep-wake cycles and internal clock by acting as a precursor to melatonin.
It is hypothesised to regulate hunger, emotions, motor, cognitive, and autonomic processes.
7. Facial Nerve Disorders/Causes
Central
1. Brain Abscess
2. Pontine gliomas
Intracranial Part
1. Acaustic Neuroma
2. Meningioma
3. Congenital
Cholisteatoma
Extracranial Part
1. Malignency of Parotid
Gland
2. Surgery of Parotid Gland
3. Neonatal Facial Injury
Systemic Diseases
1. Diabetes Mellitus
2. Hypothyrodism
3. Uremia
4. Polyarthritis Nodosa
8.
9. Types of Lesion
• A supranuclear lesion (i.e., in
hemiplegia) spares upper part of the
face because nuclear fibres
supplying muscles of upper part of
the face are innervated by
corticonuclear fibers of both
cerebral hemispheres.
• Only lower half of face on opposite
side is paralyzed.
• All infranuclear lesions involve
whole of face on same side.
10. Localisation of the Lesion
• If the Abducent & Facial nerve are not functioning
this suggests a lesion in Pons
• If Vestibulocochlear Nerve and Facial Nerve are not
functioning this suggests lesion in Internal Acoustic
Meatus
• Hyperacusis in one ear will occur if lesion involves
nerve to Stapedius in the Facial Canal
11. • Loss of taste over anterior 1/3rd of tongue suggests
lesion proximal to the point where it gives off Chorda
Tympani
• Swelling in the Parotid Gland associated with
impaired function of Facial Nerve suggests Cancer of
Parotid Gland
12. Bell’s Palsy
• Dysfunction of the facial
nerve within the facial canal
• Ipsilateral (Unilateral)
• The swelling of the nerve
fibres within the bony canal
results in temporary loss of
function producing lower
motor neuron type injury
13. Symptoms of Bells Plasy
1. Loss of wrinkles
2. Inability to close the eyes
3. Drooping of tears (Epiphora)
4. Loss of Nasolabial fold
5. Sagging of angle of mouth
6. Drooping of Saliva
7. Leaking of Air between lips
8. Trapping of food
9. Healthy side pulling the affected side
14. Crocodile Tears Syndrome
• Clinical condition
characterized by paroxysmal
lacrimation during eating.
• Results in the facial nerve
lesion proximal to the
geniculate ganglion
• Regenerating preganglionic
fibres meant to provide
secretomotor supply to the
submandibular and
sublingular salivary glands are
misdirected to Lacrimal Gland
15. Ramsay Hunt syndrome
• Occurs due to involvement of geniculate ganglion
in herpes zoster infection.
• Clinically it presents with
1. Herpetic vesicles on the auricle.
2. Hyperacusis.
3. Loss of lacrimation.
4. Loss of taste sensations in the anterior two-third
of the tongue.
5. Complete ipsilateral facial palsy (Bell’s palsy).
16. Diagnostic Tests
Minimal Nerve Excitability Test
• Nerve is stimulated at steadily
increasing intensity till facial
twitch is noticeable.
• This is compared with normal side.
• When difference between two
sides increase 3.5 miliampere,
test is positive for degeneration
17. • Maximal Stimulation Test
• Similar to minimal nerve exitibality test but instead
of measuring threshold of stimulation, the current
level which gives maximum facial moment is
determined and compared with normal side.
• Reduced or absent response with maximal
stimulation indicates degeneration
18. • Electromyography
• This tests the motor activity
of facial muscles by insertion
of needle electrodes into
orbicularis oris and oculi and
recordings are made
• I normal resting muscle,
biphasic or triphasic
potentials are seen every
30-50 milliseconds
• In denervated muscle
spontaneous activity
Fibrillations are seen
19. Topodiagnostic Tests of Lesions
Schiemers Test
• It compares lacrimation of
two sides
• A strip of filter paper is
hooked in the lower fornix of
each eye and amount of
wetting of strip
• Decreased lacrimation
indicates lesion proximal to
geniculate ganglion
20. Stapedial Reflex
• Lost in lesions above the nerve
to stapedius
• Tested by Tympanometry
• Stapedial reflex in Bells Palsy
Gives better prognosis
21. Taste Test
• Its measured by dropping salt
or sugar solution on one side
of the protruded tongue or by
electrogustrometry
• Impairment of taste indicates
lesion above chorda tympani
22. Submandibular Salivary Flow
• Measures function of
Chorda Tympani
• Polythene tubes are passed
into Wartons ducts and
drops of saliva are counted
during one minute
• Decreased salivation shows
injury above chorda
tympani
24. MCQs
• Q.1) If a patient comes in clinical setting with
weakness of one side of face & there is
hyperacusis, then which cranial nerves injury is
involved?
A) 6th & 7th
B) 7th & 8th
C) 5th & 7th
D) 7th & 9th
Ans. B
25. MCQs
• Q.2) In intracranial course facial nerve gives
its branch to which muscle?
• A) Stapedius
• B) Tensor tympani
• C) Temporalis
• D) Zygomaticus major
• Ans. A
26. MCQs
• Q.3)More chances of nerve regeneration are
present in which type of nerve injury?
A) Neurotemesis > Neuroprexia> Axontemesis
B) Neuroprexia> Neurptemesis>Axontemesis
C) Neuroprexia>Axontemesis>Neurotemesis
D) Axontemesis>Neurotemesis>Neuroprexia
• Ans.C
27. MCQs
• Q.4) If there is difficulting in lateral eyeball
movement associated with facial nerve injury
symptoms , then at which level facial nerve is
affected?
A) At pons level
B) At pontomedullary junction
C) In internal ear
D) At parotid gland
• Ans. A