This document summarizes the key steps and observations in externally examining a fresh dead body. It describes examining the clothes for evidence, skin for defensive wounds or evidence of the killer, eyes for changes indicating time of death, and muscles for signs of rigor mortis. Rigor mortis is important as it gives clues to time and cause of death based on its onset, extent, and disappearance. Other factors like hypostasis, eye changes, and temperature can also provide clues for estimating time of death. The external exam seeks to understand cause and manner of death and detect any post-mortem changes to the body.
this is a powerpoint presentation on external examination at autopsy, presented during pg program.. useful for both undergraduate and postgraduate students
this is a powerpoint presentation on external examination at autopsy, presented during pg program.. useful for both undergraduate and postgraduate students
FORENSIC MEDICINE BOOKS OF
REDDY
GOUTAM BISWAS
MAGENDRAN
OTHERS
TOPICS :-
COLD INJURY
HEAT INJURY
BURN INJURY
SCALDS
ELECTROCUTION
LIGHTENING INJURY
THIS IS ONE OF MY BEST AND FAVORITE PRESENTATIONS. IT WILL SURELY HELP YOU A LOT DURING YOUR EXAMS (PROF/OTHERS). IF YOU FIND IT HELPFUL THEN LIKE IT. MY EMAIL ID IS GIVEN ON THE 2ND PAGE OF THIS PRESENTATION, IF YOU WANT PRESENTATIONS ON OTHER TOPICS (ANY MEDICAL SUBJECTS) THEN MAIL ME. I WILL WORK ON IT LOT AND WILL BE TRYING TO SHARE WITH YOU GUYS...
THANK YOU
Deals with the post-mortem examination (autopsy) particularly the internal examinations of the various organs based on Virchow's technique of organ removal.
Asphyxia which means "Pulselessness" and is cause due to absence of oxygen amount in a body. The death cause by asphyxia is known as asphyxial deaths. They are Hanging, Strangulation, Suffocation and Drowning.
FORENSIC MEDICINE BOOKS OF
REDDY
GOUTAM BISWAS
MAGENDRAN
OTHERS
TOPICS :-
COLD INJURY
HEAT INJURY
BURN INJURY
SCALDS
ELECTROCUTION
LIGHTENING INJURY
THIS IS ONE OF MY BEST AND FAVORITE PRESENTATIONS. IT WILL SURELY HELP YOU A LOT DURING YOUR EXAMS (PROF/OTHERS). IF YOU FIND IT HELPFUL THEN LIKE IT. MY EMAIL ID IS GIVEN ON THE 2ND PAGE OF THIS PRESENTATION, IF YOU WANT PRESENTATIONS ON OTHER TOPICS (ANY MEDICAL SUBJECTS) THEN MAIL ME. I WILL WORK ON IT LOT AND WILL BE TRYING TO SHARE WITH YOU GUYS...
THANK YOU
Deals with the post-mortem examination (autopsy) particularly the internal examinations of the various organs based on Virchow's technique of organ removal.
Asphyxia which means "Pulselessness" and is cause due to absence of oxygen amount in a body. The death cause by asphyxia is known as asphyxial deaths. They are Hanging, Strangulation, Suffocation and Drowning.
Atlas on bethesda system for reporting cervical cytologyAshish Jawarkar
This is an atlas with more nearly 100 images, authentic taken from NCI web atlas. Useful to understand and report pap smears. The subject has been presented in a way which will help students reproduce in exams.
Forensic Medicine (Medico-legal Aspects of Physical Injuries)knip xin
This power point presentation is about Forensic Science that include topics about kinds of death, Medico-legal aspects of physical injuries, types of wounds, and medico-legal aspects of sex crimes.
When a murder takes place and the forensic team comes to investigate it, they get a lot of information from the dead person as if, the corpse speaks to them.
A key factor useful in homicide investigations is timing. Knowing the exact time of the death makes it easier to catch the murderer. This article gives a rough idea of how to determine the exact time of death by examining the dead body. To understand this information, we will first look at when and what changes occur in the dead body after death.
Thanatology
Types of transplants
Cause, Mechanism of Death
Manner of death
Anoxia
Signs of Death
Immediate Changes (Somatic Death)
Early Changes (Molecular Death)
Algor Mortis ......
Reference
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
2. In 16th-century Europe, medical practitioners in army and
university settings began to gather information on the cause and
manner of death.
Ambroise Paré, a French army surgeon, systematically
studied the effects of violent death on internal organs.
Two Italian surgeons, Fortunato Fidelis and Paolo Zacchia,
laid the foundation of modern pathologyby studying changes
that occurred in the structure of the body as the result of
disease.[In the late 18th century, writings on these topics
began to appear.
The beginning of
forensic medicine
4. Stuff to be done
1. Not disturbing the crime Scene
2.Take many photos of the place ,the
position of the body and any apparent
wound or evidence as the crime weapon
3.Any sample should be numbered and
labeled and take pic where it was found
5.
6. 1. The door was open or close on first entrance the crime
scene Disturbance in the place like ( furniture )
2. Blood spots at the wall ,floor ,spray pattern
3. Searching for the weapon (knife ,gun or cartridge ,poison
bottle )
4. Searching for finger printing
There was a fight
Circumstantial evidence
9. Clothes
Tearing&Holes Gun powder
Evidence of the killer
HairBlood Finger
printFrom a knife or a bullet or
fighting
Distance of the
firing or even the
deceased had
shot some bullet
himself
All of the sample are sent to the lap to be
analyzed and compared with the database
13. During examining the skin we should use a
Recording method
Any wound is seen should be pointed at including the length and the depth of the wound
14. 1-Defensive evidence
A-Defensive wound
Any evidence on the body that will confirm that the Death was homicidal
Def :-
A wound sustained when a victim places a hand
(often the palms), arm (lateral forearms) or other
body part in harm’s way to prevent or minimize
the impact of a blow or slashing by a sharp
weapon
EX:-
15. B-Evidence of the killer
-Some times when during the fight scratches by the hand and
some hair or skin become attached under the skin
-We takes sample of what is under the skin
It is possible that the killer left a finger print on the skin of the
victim that we can obtain
16. 2-The color
of the skin
-Soon after death the color of the skin start to change as a result of stagnation and
gravitation of the blood at the most depending area not subjected to pressure….
this process is called
(hypostasis)
17. Importance of hypostasis
1-it gives us a good sign of position of the body at the time of death
as the blood gather by gravity if the body in the supine position the blood will be in the
back …..any change to this site will tells us that the body had been moved
In this photo the body was first
lied on the prone position
18. 2-it also gives a good estimated time of the death
The extent of hypostasis
the hypostasis first appear after one hour as a small batches start to get
larger with time to become fixed and complete after 8 hours
3-it can gives why he is died too according to the color of hypostasis
pale blue-------normal death
deep blue--------asphyxia
Red-----------(co-cyanide-cold)
brown--------(K chlorate poisoning—nitrite poison
pale --------- hemorrhage
So in conclusion the hypostasis could give us the
approximate cause of death ,the timing and if there is
tempering with the body after death (position)
19. 3.Laceration ,bruises
,stabbing ,penetrating wound
-It is very important to identify and record all wound and measure the
length and the depth of them and that is very important of identifying the type of the
weapon and the size of the blade and which side the attack happened
Cut & stabbing wound
*Measuring both the length and depth of the wound
20.
21. Gun shot wound
Some important things :-
1-identify the inlet &the exit to extract the bullet later from the body or from the
scene
2-idetify if there is gun powder , tattooing mark that is helpful in measuring
distance of firing
3-diameter &angle of entry ---it help to identify what type of weapon and where
did the assault stand when he was shooting
22.
23. 4-temprature
of the body
Soon after death the body temp start to decline from the normal body temp until it
reaches the room temp because there are no metabolic process and the cold external
temp
*There are 3 area to measure the temp
from by chemical thermometer
1-Rectal
2-Vaginal
3-Abdominal
24. 2.5c/hrs. 1st 6 hrs.
1.5-2c/hrs. 1st 12hrs.
• So in moderate the body loses 1c /hr. ..the
body needs 18hrs. To reach the room
temp
*Temp of 21c is a sure sign of death
But it is rarely to be used as is affected by many factor
as the room temp and it is hard to know the exact temp
of the body at time of death
Rate of cooling (Simpson's formula)
26. Concerning the eye we will talk about four things
1. The cornea
2. Eye pressure
3. The sclera
4. The pupil
1-The cornea :-
clouding and accumulation of dust on cell debris and there is no
reflexes
27. 2-Eye pressure :-
there is decrease in the IOP soon after death until it reaches Zero
it decline to ½ the normal pressure after half an hour,1/8 after two hours
and after 3 hours its becomes zero
3-The sclera :-
black spots start to appear after 3 hours due to desiccation of
tissue & accumulation of dust and debris (tacheuoire spots )
4-The pupil:- * loses all reflexes
a- becomes fixed dilated after death resembling(1ry flaccidity)
b- distortion of the pupillary margin
c- muscle tissue of the iris losses tone
29. If we are going we talk about the
muscle we mustn't forget any of the
following
*Immediately after death the CNS stop sending signals to the
muscles which lead to ( primary flaccidity )
*After two hours the voluntary &the involuntary muscle
become rigid and stiffness And this condition is called (Rigor
Mortis)
*And after 18 hours start to appear flaccidity in all of the
muscles (2ry flaccidity )
30. Rigor mortis
The cause of this
condition is :-
Decrease ATP
production
Dehydration
Dehydrated complex of actin
&myosin
It happens in a descending order starting for the voluntary muscle from the
eye lid muscle ,TMJ and also to the involuntary muscles as(eye constrictor
pupil then Erector pilae “goose skin “and the heart )
*The rigor mortis start at 2 hrs. after death and
reaches it peak at 6hrs. and takes 12 hrs.to
disappears
31. Rigor mortis is so
important as
*Gives us the time of death from
Extent &Rate of disappearance
*Gives us the cause of death from
Time of onset &disappearance as
some factors increase the rate of
disappearance and some decrease it
*Gives us where he was dead as a sure sign of
death and no one can change the body position
in this situation
Goose skin
32. Factors affecting Rigor mortis
Any thing that cause increase ATP resource
decrease the Rigor mortis and other wise
ATP
Athletes
Cold temp
ATP
Child
Asphyxia
Electrocution
Hot temp
33. Secondary flaccidity
It happens as a result of disintegration and autolysis of the
muscular protein
It starts after 18 hours of death
And the muscle doesn't respond to
electrical stimulation as the primary
flaccidity
34. Cases Resembling (Rigor mortis)
1-Cadaveric Spasm :-
Contraction of one group of muscle (voluntary )the hand &the jaw
in most cases usually associated with violent deaths happening
under extremely physical circumstances with intense emotion
*It can be mistaken diagnosis with Rigor mortis but in
this case the spasm stay until the putrefaction
Importance :- the deceased may be holding in his
hand some of the killer belonging (button –hair –
piece of cloth)
35. 2-heat stiffness
It is a state resulting from exposure to fire and high temp which
lead to coagulation of the muscle protein
Contracture of all the flexor muscle in the body this called
(Boxer sign )
36. Finally
The cause of death could be known or assumed from
1-hypostasis (toxic –hemorrhage- asphyxia)
2-Rigor mortis (electrocuted-convulsion)
The time of death could be known from
1-Extent of hypostasis 3-Eye changes
2-Extent of Rigor mortis 4-Temp measuring and Samson's formula
We can also known if there is any change of the body position
after death
1-Hypostasis 2-Rigor Mortis