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EDEMA
AHAMMED NASEEM
BSc Nursing Student
Introduction
 EDEMA is a normal response of body to inflammation or injury
(For example; a twisted angle, a bee sting or a skin infection this
may result in edema in the involved area)
 In some cases such as infection this may be bacterial. Increased
fluid from the blood vessels allows more infection-fighting with
blood cells to enter the affected area
DEFINITION
 Edema is defined as "the accumulation of excessive
body fluids in the interstitial space or serous body
cavity, which is a pathogenic process caused by
disease rather than a disease entry "
ETIOLOGY
 INCREASED CAPILLARY HYDROSTATIC PRESSURE
• Elevated plasma volume
• increased venous pressure (Eg:- congestive heart failure, Venous thrombosis)
• Arteriolar dilation (Eg:- acute inflammatio)
 REDUCED PLASMA ONCOTIC PRESSURE
Plasma albumin content decreased due to;
• Decreased Protein production (Eg:-hepatic cirrhosis, malnutrition)
• Excess loss of protein (Eg :- Nephrosis)
• Elevated Catabolism of protein (Eg:-Chronic debilitating disease such as malignant
tumors)
 Inflammation
Capillary endothelium injured from;
• Toxins
• Histamine
• Anoxia
• Drugs
 Lymphatic Obstruction
• Blockage by cancer
• Blockage by infection (especially with filariasis)
• Pressure on main lymphatic duct
• Inflammation of lymphatics
 Sodium & Water Retention
 Increased Capillary permeability
• infection
• Burns
• Allergic response
• Trauma
• Anoxia
• Acidosis
• Inflammation
CLASSIFICATION
 Depending upon site of edema;
 Generalized Edema:-
• It is a condition in which notice that swelling, puffiness and water retention
in various body parts including abdomen arms legs face & feel
• Symptoms:- Bloating, tightening of skin & skin that hold an imprint or
looks pitted
• Conditions:- congestive heart failure, Nephrotic syndrome, Cirrhosis of
liver, Systematic infection
 Localised Edema:-
• It is a type of edema confined to a particular part or organ of the body
• Conditions:- Trauma, Infection, Lymphatic and venous obstruction
 Edema can also classified as:
 Putting Edema/Cutaneous edema
• It is a condition in which after a pressure applied on a small area of edema the
indentation persist after release of pressure
• Causes:- water retention, systematic disease, pregnancy, heart failure, vericose
vein, Thrombophlebitis, insect bite & Dermatitis can also cause the pitting
edema
 Non Pitting Edema
• In this condition the indentation doesn't persist
• It is associated with conditions such as lymphedema, lipidemia & myxedema
 Other Major Types of Edema
 Peripheral Edema
• It mainly occurs in the legs, feets, ankles
• This is the most common type of edema it cause swelling in lower extremities
• It may caused by increasing age, hypertension, pregnancy, Congestive Heart
Failure, Kidney problems and other health conditions
• It may also experiance when standing/sitting for extended hours
• Some medications like NSAID's (Ibuprofen, Naproxen), Corticosteroids
(Prednisone, Methyl Prednisolone)can also cure
 Pulmonary Edema
• It is the fluid accumulation in the lungs due to the blockage of pulmonary veins.
As blood pressure rises inthe blood vessels of the lungs fluid rush into fill the
lungs
• The pleural cavity can also be filled with fluid in such cases pulmonary edema is
said to also present with pleural Effusion
• It can be caused by malfunction of left ventricle, acute respiratory distress
syndrome & also hypersensitivity reaction
 Cerebral Edema
• It is the accumulation of fluid in the intracellular and extracellular spaces of brain
• It can be cause by metabolic abnormalities due to an underlying diseases Or as a
response to O2 deprivation at high altitude
• It is very serious condition & can also leads to unconsciousness and brain
damage
• It is devided into 3 sub types; Vasogenic, Cytogenic & Interstitial ceribral edema
PATHOPHYSIOLOGY
 INCREASED CAPILLARY HYDROSTATIC PRESSURE
Due to the Cause
Increased capillary hydrostatic pressure at the venular end of capillaries to a level more
than plasma Oncotic Pressure
Minimal Or No absorption of fluid at the venular end
Edema
 Reduced plasma oncotic pressure
Due to the cause
Fall in plasma oncotic pressure
decreased net movement of fluid into interstitial tissues
Edema
 INFLAMMATION
Due to the cause
Capillary endothelium get injured
Endothelial gap
Increased Capillary permeability to plasma proteins
decreased plasma oncotic Pressure
Edema
 LYMPHATIC OBSTRUCTION
Due to the cause
Impaired lymphatic drainage
Localised lymphedema
 SODIUM AND WATER RETENTION
Hypovolemia
Renal Vasoconstriction Renin Increased ADH level
Decreased GFR Aldosteron Reabsorption of water
Renal retention of sodium and water
Edema
 INCREASED CAPILLARY PERMEABILITY
Due to the cause
Decreased capillary permeability
filtration of more protein from capillary to interstitium
Decreased plasma colloid osmotic pressure
Increased formation of Interstitial fluid
Edema
Signs & Symptoms
 Increase in the total body water cause weight gain in short period of time
 Peripheral Edema
 Excess Fluid in the Interstitial space
 Distended Neck and peripheral Veins
 Slow emptying peripheral Veins
 CVP over 11cm
 Crackles and wheezes in lungs
 Poly urea (if renal function is normal)
 Ascites
 Pleural Effusion
 Decreased Blood Urea Nitrogen (BUN) Due to plasma dilution
 Bounding Pulse
 Pulmonary Edema (if severe)
DIAGNOSIS
 Health History
 Diagnostic tests like ;
• monitor serum osmolarity
• Hemoglobin & Hematocrit
• Urine specific gravity and Osmolality
• CVP Reading
 In Physical Examination
• Daily weight
• Lung sounds
• Extremity measurement
• Abdominal Measurement
• grading of edema
Grading of Edema
Measurement
of Depression
(In mm)
1. 0mm
2. 2mm
3. 4mm
4. 6mm
5. 8mm
Gradi
ng
1. 0+
2. 1+
3. 2+
4. 3+
5. 4+
Discription
1. Non Pitting Edema
2. Mild Pitting Edema
3. Moderate pitting edema(Depression dis
appear in 10-15 s)
4. moderately severe Pitting
edema(Dipression may last for >1min)
5. Severe Pitting edema (Dipression may
last >2min
COMPLICATIONS
Untreated edema can leads to ;
 Painful Swelling, Pain that gets worse
 Stiffness and difficulties in walking
 Stretched & itchy skin
 Infection in the area of swelling
 Scarring Between layers of tissue
 Poor blood circulation
 Loss of elasticity in Artery, Veins & Joints
 Ulceration on skin
MEDICAL MANAGEMENT
 Treat the underlying Cause
 Treat the sign and symptoms
 Restrict salt intake
 Provide Oxygen in pulmonary edema
PHARMACOLOGICAL
MANAGEMENT
 Analgesics
 Diuretics
 Antibiotics
NURSING MANAGEMENT
 Assess the presence of worsening edema
 Encourage adherence to Sodium Restriction to avoid over counter drugs
 When indicated encourage rest periods
 monitor patients Response to Diuretics, Check the daily weight
 Monitor rate of parental fluids & Client response
 Teach the self monitoring of weight & I/O measurement (in cases such as
CCF, Renal failure, Cirrhosis of liver)
 Turn the position of patient frequently
 Help the patient to Relax & promote oxygenation
 Place the patient in high Fowler's position to enhance lungs
expansion
 Administer O2 As prescribed
 Assess patients condition frequently
 Auscultate Lung field for breath sounds & be alert for crackles
 Watch for complications of treatment such as electrolyte
depletion
 Monitor ABG results to identify Hypoxemia & hypercapnia

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Edema Pathophysiology and Management

  • 2. Introduction  EDEMA is a normal response of body to inflammation or injury (For example; a twisted angle, a bee sting or a skin infection this may result in edema in the involved area)  In some cases such as infection this may be bacterial. Increased fluid from the blood vessels allows more infection-fighting with blood cells to enter the affected area
  • 3. DEFINITION  Edema is defined as "the accumulation of excessive body fluids in the interstitial space or serous body cavity, which is a pathogenic process caused by disease rather than a disease entry "
  • 4. ETIOLOGY  INCREASED CAPILLARY HYDROSTATIC PRESSURE • Elevated plasma volume • increased venous pressure (Eg:- congestive heart failure, Venous thrombosis) • Arteriolar dilation (Eg:- acute inflammatio)  REDUCED PLASMA ONCOTIC PRESSURE Plasma albumin content decreased due to; • Decreased Protein production (Eg:-hepatic cirrhosis, malnutrition) • Excess loss of protein (Eg :- Nephrosis) • Elevated Catabolism of protein (Eg:-Chronic debilitating disease such as malignant tumors)
  • 5.  Inflammation Capillary endothelium injured from; • Toxins • Histamine • Anoxia • Drugs  Lymphatic Obstruction • Blockage by cancer • Blockage by infection (especially with filariasis) • Pressure on main lymphatic duct • Inflammation of lymphatics  Sodium & Water Retention
  • 6.  Increased Capillary permeability • infection • Burns • Allergic response • Trauma • Anoxia • Acidosis • Inflammation
  • 7. CLASSIFICATION  Depending upon site of edema;  Generalized Edema:- • It is a condition in which notice that swelling, puffiness and water retention in various body parts including abdomen arms legs face & feel • Symptoms:- Bloating, tightening of skin & skin that hold an imprint or looks pitted • Conditions:- congestive heart failure, Nephrotic syndrome, Cirrhosis of liver, Systematic infection  Localised Edema:- • It is a type of edema confined to a particular part or organ of the body • Conditions:- Trauma, Infection, Lymphatic and venous obstruction
  • 8.  Edema can also classified as:  Putting Edema/Cutaneous edema • It is a condition in which after a pressure applied on a small area of edema the indentation persist after release of pressure • Causes:- water retention, systematic disease, pregnancy, heart failure, vericose vein, Thrombophlebitis, insect bite & Dermatitis can also cause the pitting edema  Non Pitting Edema • In this condition the indentation doesn't persist • It is associated with conditions such as lymphedema, lipidemia & myxedema
  • 9.  Other Major Types of Edema  Peripheral Edema • It mainly occurs in the legs, feets, ankles • This is the most common type of edema it cause swelling in lower extremities • It may caused by increasing age, hypertension, pregnancy, Congestive Heart Failure, Kidney problems and other health conditions • It may also experiance when standing/sitting for extended hours • Some medications like NSAID's (Ibuprofen, Naproxen), Corticosteroids (Prednisone, Methyl Prednisolone)can also cure
  • 10.  Pulmonary Edema • It is the fluid accumulation in the lungs due to the blockage of pulmonary veins. As blood pressure rises inthe blood vessels of the lungs fluid rush into fill the lungs • The pleural cavity can also be filled with fluid in such cases pulmonary edema is said to also present with pleural Effusion • It can be caused by malfunction of left ventricle, acute respiratory distress syndrome & also hypersensitivity reaction  Cerebral Edema • It is the accumulation of fluid in the intracellular and extracellular spaces of brain • It can be cause by metabolic abnormalities due to an underlying diseases Or as a response to O2 deprivation at high altitude • It is very serious condition & can also leads to unconsciousness and brain damage • It is devided into 3 sub types; Vasogenic, Cytogenic & Interstitial ceribral edema
  • 11. PATHOPHYSIOLOGY  INCREASED CAPILLARY HYDROSTATIC PRESSURE Due to the Cause Increased capillary hydrostatic pressure at the venular end of capillaries to a level more than plasma Oncotic Pressure Minimal Or No absorption of fluid at the venular end Edema
  • 12.  Reduced plasma oncotic pressure Due to the cause Fall in plasma oncotic pressure decreased net movement of fluid into interstitial tissues Edema
  • 13.  INFLAMMATION Due to the cause Capillary endothelium get injured Endothelial gap Increased Capillary permeability to plasma proteins decreased plasma oncotic Pressure Edema
  • 14.  LYMPHATIC OBSTRUCTION Due to the cause Impaired lymphatic drainage Localised lymphedema
  • 15.  SODIUM AND WATER RETENTION Hypovolemia Renal Vasoconstriction Renin Increased ADH level Decreased GFR Aldosteron Reabsorption of water Renal retention of sodium and water Edema
  • 16.  INCREASED CAPILLARY PERMEABILITY Due to the cause Decreased capillary permeability filtration of more protein from capillary to interstitium Decreased plasma colloid osmotic pressure Increased formation of Interstitial fluid Edema
  • 17. Signs & Symptoms  Increase in the total body water cause weight gain in short period of time  Peripheral Edema  Excess Fluid in the Interstitial space  Distended Neck and peripheral Veins  Slow emptying peripheral Veins  CVP over 11cm  Crackles and wheezes in lungs  Poly urea (if renal function is normal)  Ascites  Pleural Effusion  Decreased Blood Urea Nitrogen (BUN) Due to plasma dilution  Bounding Pulse  Pulmonary Edema (if severe)
  • 18. DIAGNOSIS  Health History  Diagnostic tests like ; • monitor serum osmolarity • Hemoglobin & Hematocrit • Urine specific gravity and Osmolality • CVP Reading  In Physical Examination • Daily weight • Lung sounds • Extremity measurement • Abdominal Measurement • grading of edema
  • 19. Grading of Edema Measurement of Depression (In mm) 1. 0mm 2. 2mm 3. 4mm 4. 6mm 5. 8mm Gradi ng 1. 0+ 2. 1+ 3. 2+ 4. 3+ 5. 4+ Discription 1. Non Pitting Edema 2. Mild Pitting Edema 3. Moderate pitting edema(Depression dis appear in 10-15 s) 4. moderately severe Pitting edema(Dipression may last for >1min) 5. Severe Pitting edema (Dipression may last >2min
  • 20. COMPLICATIONS Untreated edema can leads to ;  Painful Swelling, Pain that gets worse  Stiffness and difficulties in walking  Stretched & itchy skin  Infection in the area of swelling  Scarring Between layers of tissue  Poor blood circulation  Loss of elasticity in Artery, Veins & Joints  Ulceration on skin
  • 21. MEDICAL MANAGEMENT  Treat the underlying Cause  Treat the sign and symptoms  Restrict salt intake  Provide Oxygen in pulmonary edema
  • 23. NURSING MANAGEMENT  Assess the presence of worsening edema  Encourage adherence to Sodium Restriction to avoid over counter drugs  When indicated encourage rest periods  monitor patients Response to Diuretics, Check the daily weight  Monitor rate of parental fluids & Client response  Teach the self monitoring of weight & I/O measurement (in cases such as CCF, Renal failure, Cirrhosis of liver)  Turn the position of patient frequently  Help the patient to Relax & promote oxygenation
  • 24.  Place the patient in high Fowler's position to enhance lungs expansion  Administer O2 As prescribed  Assess patients condition frequently  Auscultate Lung field for breath sounds & be alert for crackles  Watch for complications of treatment such as electrolyte depletion  Monitor ABG results to identify Hypoxemia & hypercapnia