Dr. Md. Monirul Islam
MBBS, BCS, MS (Ortho)
Consultant (Ortho-Surgery)
250 beded Sadar Hospital , Jhenaidah
Presented by :
Definition
Heterogeneous group of disorders
characterized by degree of impairment of
movement and posture due to non
progressive neurological deficit as a result of
an insult to the developing brain.
INCIDENCE
Prevalence rate is about 4% per 1000 live birth
10% of infants born1000gm or less at birth
5%-10% incidence in pre-term
Birth asphyxia as a cause only in3-21%
TYPES/CLASSIFICATIONS
BASED ON MOTOR DEFICIT (MOVEMENT DISORDER)
A. SPASTIC CEREBRAL PALSY :The most common form of cerebral
palsy: about 65-70-%
B. EXTRAPYRAMIDAL CEREBRAL PALSY (DYSKINETIC CP: about 30 %
 Uncontrolled interfere with speaking, feeding, grabbing, reaching and
others skills that require motor skills.
 Caused by cerebral damage due to kernicterus.
C. ATONIC CEREBRAL PALSY: 5% Poor balance and lack of
coordination
D. MIXED FORM : Spastic muscle tone and involuntary movement.
Have damage to the motor cortex and cerebellum
Spastic cerebral palsy
Spastic cerebral palsy is the type of cerebral
palsy characterized by spasticity or high
muscle tone often resulting in stiff, jerky
movements.
Itself an umbrella term encompassing :
1. spastic hemiplegia,
2. spastic diplegia,
3. spastic quadriplegia
4. spastic monoplegia
ACCORDING TO SEVERITY
A. MILD CEREBRAL PALSY (20%): Patients are
ambulatory, fine movement are impaired only
B. MODERATE CP (50%):These children achieve
ambulation by self-help. There is impaired gross
motor, fine motor and speech development.
C. SEVERE CP (30%):The children present with
multiple defects and unable to perform usual
activities of daily living
Prenatal factors/ causes
• 1)Risk factors inherent to the fetus (most
commonly genetic disorders),
• 2)Factors inherent to the mother (seizure
disorders, mental retardation, and previous
pregnancy loss),
• 3)Factors inherent to the pregnancy itself (Rh
incompatibility, polyhydramnios, placental
rupture, and drug or alcohol exposure).
• 4)External factors, such as TORCH (toxoplasmosis,
other agents, rubella, cytomegalovirus, herpes
simplex),
Perinatal Factors
1. Asphyxia or trauma during labor,
2. Oxytocin augmentation,
3. Umbilical cord prolapse,
4. Breech presentation
5. Low Apgar scores
6. Low-birth-weight infants (<1500 g)
Postnatal Causes
I. Trauma
II. Head injury
III. Infections – Septicemia , Meningitis ,
Enchephalitis
IV. Lack of oxygen
V. Stroke in the young age
VI. Tumor, cyst
CLINICAL MANIFESTATIONS EARLY
SIGNS OF CP
i. Asymmetric movement
ii. Restlessness
iii. Irritability
iv. Feeding or swallowing or poor sucking
v. Poor head control
vi. Tongue thrust
vii. Excessive high pitch cry
viii.Slow weight gain
LATE SIGNS OF CP
i. Delayed gross motor development
ii. Persistent infantile reflexes
iii. Weakness
iv. Abnormal postures
v. Drooling
vi. Recurrent infections
vii. Malocclusion of teeth
viii. Constipation
ix. Caries teeth
x. Delayed or defective speech
xi. Evidence of mental retardation
Medical management
Correction or alleviation of specific neuro-motor deficits
or associated disabilities. Symptomatic management
is done.
• Pharmacotherapy
– Diazepam-for spasticity
– Levodopa-for athetosis
– Carbamazepine-for dystonia
– Anticonvulsants-for epilepsy
– Tranquilizers-for behavioral problem
– Muscle relaxants-to improve muscular functions
• Physiotherapy, Development enrichment experiences
Medical management
• Development of vocational and socialization
• Emotional behavioral and social adjustment
• SURGICAL MANAGEMENT:
– Orthopedic correction of:
• Scoliosis
• Contractures
• Dislocations
• Rhizotomyis an attempt to decrease spasticity
Preventing CP During Pregnancy
• Avoiding exposure to infections or viruses known
to impact fetal health, such as German Measles,
Cytomegalovirus or Zika
• Getting vaccinated appropriately
• Controlling underlying health issues, such as
blood pressure, diabetes, etc.
• Avoiding alcohol, cigarettes, recreational drugs,
and prescription drugs known to pose risks
during pregnancy
• Identifying any potential Rh incompatibility
between mother and child
Preventing CP During Birth
• Monitor maternal and fetal heart rate
• Remain as calm as possible during delivery.
Stress-induced trauma to the infant brain can
be reduced by maintaining a quiet, peaceful
delivery room
• Receive regular checkups This is especially
important in the weeks leading up to giving
birth.
Preventing Cerebral Palsy After Birth
a. Making sure child is vaccinated for all
common infant infections
b. Using the correct car seat for child’s weight
and height
c. Using a crib with bed rail
d. Never leaving your child on high countertops
or surfaces unattended
e. Never shake a baby
Thank You All

Cerebral Palsy

  • 1.
    Dr. Md. MonirulIslam MBBS, BCS, MS (Ortho) Consultant (Ortho-Surgery) 250 beded Sadar Hospital , Jhenaidah Presented by :
  • 2.
    Definition Heterogeneous group ofdisorders characterized by degree of impairment of movement and posture due to non progressive neurological deficit as a result of an insult to the developing brain.
  • 3.
    INCIDENCE Prevalence rate isabout 4% per 1000 live birth 10% of infants born1000gm or less at birth 5%-10% incidence in pre-term Birth asphyxia as a cause only in3-21%
  • 4.
    TYPES/CLASSIFICATIONS BASED ON MOTORDEFICIT (MOVEMENT DISORDER) A. SPASTIC CEREBRAL PALSY :The most common form of cerebral palsy: about 65-70-% B. EXTRAPYRAMIDAL CEREBRAL PALSY (DYSKINETIC CP: about 30 %  Uncontrolled interfere with speaking, feeding, grabbing, reaching and others skills that require motor skills.  Caused by cerebral damage due to kernicterus. C. ATONIC CEREBRAL PALSY: 5% Poor balance and lack of coordination D. MIXED FORM : Spastic muscle tone and involuntary movement. Have damage to the motor cortex and cerebellum
  • 5.
    Spastic cerebral palsy Spasticcerebral palsy is the type of cerebral palsy characterized by spasticity or high muscle tone often resulting in stiff, jerky movements. Itself an umbrella term encompassing : 1. spastic hemiplegia, 2. spastic diplegia, 3. spastic quadriplegia 4. spastic monoplegia
  • 6.
    ACCORDING TO SEVERITY A.MILD CEREBRAL PALSY (20%): Patients are ambulatory, fine movement are impaired only B. MODERATE CP (50%):These children achieve ambulation by self-help. There is impaired gross motor, fine motor and speech development. C. SEVERE CP (30%):The children present with multiple defects and unable to perform usual activities of daily living
  • 7.
    Prenatal factors/ causes •1)Risk factors inherent to the fetus (most commonly genetic disorders), • 2)Factors inherent to the mother (seizure disorders, mental retardation, and previous pregnancy loss), • 3)Factors inherent to the pregnancy itself (Rh incompatibility, polyhydramnios, placental rupture, and drug or alcohol exposure). • 4)External factors, such as TORCH (toxoplasmosis, other agents, rubella, cytomegalovirus, herpes simplex),
  • 8.
    Perinatal Factors 1. Asphyxiaor trauma during labor, 2. Oxytocin augmentation, 3. Umbilical cord prolapse, 4. Breech presentation 5. Low Apgar scores 6. Low-birth-weight infants (<1500 g)
  • 9.
    Postnatal Causes I. Trauma II.Head injury III. Infections – Septicemia , Meningitis , Enchephalitis IV. Lack of oxygen V. Stroke in the young age VI. Tumor, cyst
  • 10.
    CLINICAL MANIFESTATIONS EARLY SIGNSOF CP i. Asymmetric movement ii. Restlessness iii. Irritability iv. Feeding or swallowing or poor sucking v. Poor head control vi. Tongue thrust vii. Excessive high pitch cry viii.Slow weight gain
  • 11.
    LATE SIGNS OFCP i. Delayed gross motor development ii. Persistent infantile reflexes iii. Weakness iv. Abnormal postures v. Drooling vi. Recurrent infections vii. Malocclusion of teeth viii. Constipation ix. Caries teeth x. Delayed or defective speech xi. Evidence of mental retardation
  • 12.
    Medical management Correction oralleviation of specific neuro-motor deficits or associated disabilities. Symptomatic management is done. • Pharmacotherapy – Diazepam-for spasticity – Levodopa-for athetosis – Carbamazepine-for dystonia – Anticonvulsants-for epilepsy – Tranquilizers-for behavioral problem – Muscle relaxants-to improve muscular functions • Physiotherapy, Development enrichment experiences
  • 13.
    Medical management • Developmentof vocational and socialization • Emotional behavioral and social adjustment • SURGICAL MANAGEMENT: – Orthopedic correction of: • Scoliosis • Contractures • Dislocations • Rhizotomyis an attempt to decrease spasticity
  • 15.
    Preventing CP DuringPregnancy • Avoiding exposure to infections or viruses known to impact fetal health, such as German Measles, Cytomegalovirus or Zika • Getting vaccinated appropriately • Controlling underlying health issues, such as blood pressure, diabetes, etc. • Avoiding alcohol, cigarettes, recreational drugs, and prescription drugs known to pose risks during pregnancy • Identifying any potential Rh incompatibility between mother and child
  • 16.
    Preventing CP DuringBirth • Monitor maternal and fetal heart rate • Remain as calm as possible during delivery. Stress-induced trauma to the infant brain can be reduced by maintaining a quiet, peaceful delivery room • Receive regular checkups This is especially important in the weeks leading up to giving birth.
  • 17.
    Preventing Cerebral PalsyAfter Birth a. Making sure child is vaccinated for all common infant infections b. Using the correct car seat for child’s weight and height c. Using a crib with bed rail d. Never leaving your child on high countertops or surfaces unattended e. Never shake a baby
  • 18.