 Cardiac tamponade is a clinical syndrome caused by
accumulation of fluid in pericardial space, resulting
in reduced ventricular filling and subsequent
haemodynamic compromise.
 The condition is a medical emergency.
 Pericardium is composed of 2 layers
 Thicker outer fibrous layer is parietal
percardium
 Thinner inner serous layer is visceral
pericardium
 Pericardial space normally contains 20 -
50 ml of fluid
 Three phases of haemodynamic changes in
tamponade
 PHASE 1: Accumulation of pericardial fluid
impairs relaxation and filling of ventricles,
requiring a high filling pressure.
 Left and right ventricular filling pressures are
higher than the intrapericardial pressure.
 PHASE 2: Pericardial pressure increases
above the ventricular filling pressure
,resulting in reduced cardiac output.
 PHASE 3: Further decrease in cardiac
output occurs due to equilibration of
pericardial and left ventricular filling
pressures.
 Malignant diseases – most common cause
(30-60%)
 Uremia(10-15%)
 Idiopathic pericarditis(5-15%)
 Infectious diseases(5-10%)
 CTD (2-6%)
 Dressler or post pericardiotomy syndrome(1-
2%)
 Pericarditis caused by
 HIV
 Infections- viral, bacterial(TB),fungal
 Drugs- hydralazine, procainamide, INH, minoxidil
 Post coronary intervention- coronary dissection and
perforation
 Post cardiac percutaneous procedures
 Trauma to chest
 Cardiovascular surgery
 Post MI infarction- free wall ventricular rupture and dressler
syndrome
 CTD- SLE ,RA, Dermatomyositis
 Radiation therapy to chest
 Iatrogenic- pericardiocentesis, central line insertion,
sternal biopsy, TV pacemaker lead implantation
 Uremia
 Anticoagulants
 Pneumopericardium
 Hypothyroidism
 Still disease
 Aortic Dissection
 The signs and symptoms of cardiac tamponade all reflect a
low cardiac output.
 Restlessness
 Agitation
 Drowsiness
 Stupor
 Decreased urine output
 Dyspnea
 Chest discomfort
 Syncope or near syncope
 Weakness
 Anorexia
 Tachycardia
 Tachypnea
 Raised JVP
 Dimnished heart sounds Beck’s Triad
Hypotension
 Hepatomegaly
 Pericardial friction rub
 PULSUS PARADOXUS: It is an exaggeration
>12mmHg or 9% of the normal inspiratory
decrease in systemic blood pressure.
It can be observed in patients with other
conditions such as constrictive pericarditis,
asthma, COPD, pulmonary embolism and right
ventricular infarction with shock.
 KUSSMAUL SIGN: It is paradoxical increase
in jugular venous distention and pressure
during inspiration.
 EWART SIGN/PINS SIGN: It is described as
an area of dullnes with bronchial breath
sounds and bronchophony below the angle
of left scapula.
 Y DESCENT: It is abolished in jugular
venous or right atrial wave form.It is due to
increased intrapericardial pressure
preventing diastolic filling of ventricles.
 With a 12-lead ECG ,the following
findings may be found:
 Sinus tachycardia
 Low-voltage QRS complexes
 Electrical alternans - an electrocardiographic
phenomenon of alternation of QRS complex
usually in a 2:1 ratio. It is caused by movement of
heart in pericardial space.
 PR Segment depression
 CHEST XRAY: may show
 cardiomegaly ,
 water bottle shaped heart,
 pericardial calcification or
 evidence of chest wall trauma.
Compression of coronary sinus is
observerd through CT scanning
as an earlier marker for cardiac
tamponade in 46% of patients.
 Pericardial effusion
Size often correlates w/risk of tamponade but not always.
 Diastolic chambers collapse
 RV diastolic collapse in early diastole (low sensitivity,
high specificity)
 RA diastolic collapse in late diastole (very sensitive
,specificity 82% ).The longer the duration of diastolic
collapse, the more
specific it is for tamponade.
 LA collapse (present in ~ 25%, highly spec)
 Abnormal inspiratory increase of RV dimensions with
abnormal inspiratory decrease of LV dimensions.
 IVC dilated & fails to collapse with inspiration at least
50%.
Definitive treatment of cardiac tamponade is achieved by removal of the
pericardial fluid, thereby relieving the elevated intrapericardial pressure
and improving hemodynamic status.
• Oxygen
• Bed rest with leg elevation - This may help increase venous
return
• Inotropic drugs (eg, dobutamine) - These can be useful because
they increase cardiac output without increasing systemic vascular
resistance
• Volume expansion with blood, plasma, dextran, or isotonic
sodium chloride solution, as necessary, to maintain adequate
intravascular volume
• Pericardiocentesis:Most require urgent and emergent
pericardiocentesis.
› PERCUTANEOUS THERAPY:
 Pericardiocentesis allows the rapid drainage
of pericardial fluid.
 Advantages are that it can be performed
quickly, is less invasive than other drainage
methods, and requires minimal preparation.
 Complications include laceration of the heart,
coronary arteries, or lung
 Subxiphoid approach under echo guidance is
most common - minimizes risk & can assess
completeness of fluid removal.
 Apical Approach
 Can alternatively use Fluoroscopic guidance
 Pigtail catheter often left in place
› SURGICAL DRAINAGE (subxiphoid
pericardiectomy, pericardial window, and subtotal
pericardiectomy):
 May be best for loculated effusions, effusions
containing clots or fibrinous material, and/or
effusions that are borderline in size
 Allow creation of a pericardial window for recurrent
effusions
 Surgical drainage is associated with more pain, a
longer recovery time, and more periprocedural
morbidity.
Possible Complications:
 Heart failure
 Pulmonary edema
 Shock
 Death
 Cardiac tamponade is a medical emergency.
If the fluid or blood is not quickly removed
from the pericardium, death can occur
quickly.
 The outcome is often good if the condition is
treated promptly, but tamponade may come
back.
 Tension Pneumothorax
 Constrictive Pericarditis
 Cardiogenic Shock
 Pulmonary Embolism
Cardiac tamponade

Cardiac tamponade

  • 3.
     Cardiac tamponadeis a clinical syndrome caused by accumulation of fluid in pericardial space, resulting in reduced ventricular filling and subsequent haemodynamic compromise.  The condition is a medical emergency.
  • 4.
     Pericardium iscomposed of 2 layers  Thicker outer fibrous layer is parietal percardium  Thinner inner serous layer is visceral pericardium  Pericardial space normally contains 20 - 50 ml of fluid
  • 5.
     Three phasesof haemodynamic changes in tamponade  PHASE 1: Accumulation of pericardial fluid impairs relaxation and filling of ventricles, requiring a high filling pressure.  Left and right ventricular filling pressures are higher than the intrapericardial pressure.
  • 6.
     PHASE 2:Pericardial pressure increases above the ventricular filling pressure ,resulting in reduced cardiac output.  PHASE 3: Further decrease in cardiac output occurs due to equilibration of pericardial and left ventricular filling pressures.
  • 7.
     Malignant diseases– most common cause (30-60%)  Uremia(10-15%)  Idiopathic pericarditis(5-15%)  Infectious diseases(5-10%)  CTD (2-6%)  Dressler or post pericardiotomy syndrome(1- 2%)
  • 8.
     Pericarditis causedby  HIV  Infections- viral, bacterial(TB),fungal  Drugs- hydralazine, procainamide, INH, minoxidil  Post coronary intervention- coronary dissection and perforation  Post cardiac percutaneous procedures  Trauma to chest  Cardiovascular surgery  Post MI infarction- free wall ventricular rupture and dressler syndrome  CTD- SLE ,RA, Dermatomyositis
  • 9.
     Radiation therapyto chest  Iatrogenic- pericardiocentesis, central line insertion, sternal biopsy, TV pacemaker lead implantation  Uremia  Anticoagulants  Pneumopericardium  Hypothyroidism  Still disease  Aortic Dissection
  • 10.
     The signsand symptoms of cardiac tamponade all reflect a low cardiac output.  Restlessness  Agitation  Drowsiness  Stupor  Decreased urine output  Dyspnea  Chest discomfort  Syncope or near syncope  Weakness  Anorexia
  • 11.
     Tachycardia  Tachypnea Raised JVP  Dimnished heart sounds Beck’s Triad Hypotension  Hepatomegaly  Pericardial friction rub
  • 12.
     PULSUS PARADOXUS:It is an exaggeration >12mmHg or 9% of the normal inspiratory decrease in systemic blood pressure. It can be observed in patients with other conditions such as constrictive pericarditis, asthma, COPD, pulmonary embolism and right ventricular infarction with shock.
  • 14.
     KUSSMAUL SIGN:It is paradoxical increase in jugular venous distention and pressure during inspiration.  EWART SIGN/PINS SIGN: It is described as an area of dullnes with bronchial breath sounds and bronchophony below the angle of left scapula.  Y DESCENT: It is abolished in jugular venous or right atrial wave form.It is due to increased intrapericardial pressure preventing diastolic filling of ventricles.
  • 15.
     With a12-lead ECG ,the following findings may be found:  Sinus tachycardia  Low-voltage QRS complexes  Electrical alternans - an electrocardiographic phenomenon of alternation of QRS complex usually in a 2:1 ratio. It is caused by movement of heart in pericardial space.  PR Segment depression
  • 17.
     CHEST XRAY:may show  cardiomegaly ,  water bottle shaped heart,  pericardial calcification or  evidence of chest wall trauma.
  • 19.
    Compression of coronarysinus is observerd through CT scanning as an earlier marker for cardiac tamponade in 46% of patients.
  • 21.
     Pericardial effusion Sizeoften correlates w/risk of tamponade but not always.  Diastolic chambers collapse  RV diastolic collapse in early diastole (low sensitivity, high specificity)  RA diastolic collapse in late diastole (very sensitive ,specificity 82% ).The longer the duration of diastolic collapse, the more specific it is for tamponade.  LA collapse (present in ~ 25%, highly spec)  Abnormal inspiratory increase of RV dimensions with abnormal inspiratory decrease of LV dimensions.  IVC dilated & fails to collapse with inspiration at least 50%.
  • 30.
    Definitive treatment ofcardiac tamponade is achieved by removal of the pericardial fluid, thereby relieving the elevated intrapericardial pressure and improving hemodynamic status. • Oxygen • Bed rest with leg elevation - This may help increase venous return • Inotropic drugs (eg, dobutamine) - These can be useful because they increase cardiac output without increasing systemic vascular resistance • Volume expansion with blood, plasma, dextran, or isotonic sodium chloride solution, as necessary, to maintain adequate intravascular volume • Pericardiocentesis:Most require urgent and emergent pericardiocentesis.
  • 31.
    › PERCUTANEOUS THERAPY: Pericardiocentesis allows the rapid drainage of pericardial fluid.  Advantages are that it can be performed quickly, is less invasive than other drainage methods, and requires minimal preparation.  Complications include laceration of the heart, coronary arteries, or lung  Subxiphoid approach under echo guidance is most common - minimizes risk & can assess completeness of fluid removal.  Apical Approach  Can alternatively use Fluoroscopic guidance  Pigtail catheter often left in place
  • 32.
    › SURGICAL DRAINAGE(subxiphoid pericardiectomy, pericardial window, and subtotal pericardiectomy):  May be best for loculated effusions, effusions containing clots or fibrinous material, and/or effusions that are borderline in size  Allow creation of a pericardial window for recurrent effusions  Surgical drainage is associated with more pain, a longer recovery time, and more periprocedural morbidity.
  • 36.
    Possible Complications:  Heartfailure  Pulmonary edema  Shock  Death
  • 37.
     Cardiac tamponadeis a medical emergency. If the fluid or blood is not quickly removed from the pericardium, death can occur quickly.  The outcome is often good if the condition is treated promptly, but tamponade may come back.
  • 38.
     Tension Pneumothorax Constrictive Pericarditis  Cardiogenic Shock  Pulmonary Embolism