This document provides information on cardiac conduction systems, normal ECG rhythms, cardiac arrhythmias, and arrhythmia management. It defines normal sinus rhythm on ECG and describes the two main types of arrhythmias as bradycardia and tachycardia. Nine common arrhythmias are defined including atrial flutter, atrial fibrillation, junctional rhythm, and various ventricular arrhythmias. Treatment options for arrhythmias include electrical cardioversion, antiarrhythmic medications, and pacemakers.
Heart arrhythmia, also known as irregular heartbeat or cardiac dysrhythmia, is a group of conditions where the heartbeat is irregular, too slow, or too fast. Arrhythmias are broken down into: Slow heartbeat: bradycardia. Fast heartbeat: tachycardia. Irregular heartbeat: flutter or fibrillation.
Heart arrhythmia, also known as irregular heartbeat or cardiac dysrhythmia, is a group of conditions where the heartbeat is irregular, too slow, or too fast. Arrhythmias are broken down into: Slow heartbeat: bradycardia. Fast heartbeat: tachycardia. Irregular heartbeat: flutter or fibrillation.
The rhythm is best analyzed by looking at a rhythm strip.
On a 12 lead ECG this is usually a 10 second recording from Lead II.
Confirm or corroborate any findings in this lead by checking the other leads.
A longer rhythm strip, recorded perhaps recorded at a slower speed, may be helpful.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
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This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
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Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
3. By the end of this presentation learners will be able
to know about
Conduction system
Normal ECG rhythem
Cardiac Arrhythmias
Management of Arrhythmias
Heart block
3
6. P wave should be positive in lead two/ three/AVF (
limbs leads) and negative in AVR
Heart rate should be between 60bpm to 80bpm
QRS complex should be narrow
Every p should followed by QRS complex
P-P R-R P-R should b constant
6
7. An abnormality of the cardiac rhythm is called
cardiac arrhythmia.
Arrhythmias may cause sudden death,
syncope, heart failure, dizziness, palpitations
or no symptoms at all.
There are two main types of arrhythmia:
bradycardia: the heart rate is slow (< 60
bpm).
tachycardia: the heart rate is fast (> 100
bpm).
7
8. 1. ATRIAL FLUTTER
2. ATRIAL FIBRILLATION
3. JUNCTIONAL RHYTHM
4. VENTRICULAR TACHYCARDIAS
5. VENTRICULAR FIBRILLATION
6. VENTRICULAR ASYSTOLE
7. FIRST DEGREE ATRIOVENTRICULAR BLOCK TYPE
1
8. SECOND DEGREE ATRIOVENTRICULAR BLOCK
TYPE 2
9. THIRD DEGREE ATRIOVENTRICULAR BLOCK TYPE
8
9. The two
"shockable" rhythms are
Ventricular fibrillation and
Pulseless ventricular tachycardia
while the two
"non–shockable" rhythms are
Asystole and
Pulseless electrical activity
9
10. Atrial flutter is a common abnormal heart
rhythm that starts in the atrial chambers of the
heart. When it first occurs, it is usually associated
with a fast heart rate and is classified as a type of
supraventricular tachycardia
Occurs in atrium and creates impulses at regular
rate between 250 and 400 times per minute.
Because the atrial rate is faster than the AV node
can conduct, all atrial impulses are not
conducted into the ventricle, causing a
therapeutic block at AV node.
10
12. Ventricula
r and
Atrial rate
Ventricula
r and
atrial
rhythm
QRS P wave PR interval P:QRS
ratio
Atrial rate
range
between
250-
400bpm
Ventricula
r rate
ranges
between
75-
150bpm
Atrial
rhythm is
regular
ventricle
rhythm is
usually
regular
but may
change in
AV
conductio
n
Usually
normal,
may be
abnormal
or may be
absent
Saw-
toothed
shape;
These
waves are
referred
to as F
waves
Multiple F
waves
make it
difficult to
determine
PR
intervals.
2:1
3:1or may
be
4:1
12
14. Atrial fibrillation causes a rapid, disorganized
, and uncoordinated twitching of atrial
musculature.
Can increase your risk of strokes, heart
failure and other heart-related complications.
Can be transient, starting and stopping
suddenly and occurring for short time.
14
16. Ventricula
r and
Atrial rate
Ventricula
r and
atrial
rhythm
QRS P wave PR interval P:QRS
ratio
Atrial rate
300 to
600bpm
Ventricula
r rate
ranges
between
120-
200bpm
Highly
irregular
Usually
normal,
may be
abnormal
No
discernibl
e P wave.
Irregular
undulatin
g waves
are seen
and are
referred
to as
fibrillatory
or F waves
Can not
be
measured
Many:1
16
18. Commonly called flat line ventricular asystole
is characterized by ABSCENT QRS COMPLEX
confirmed in two different leads
Although P waves may be apparent for short
duration.
There is no heart beat, no palpable pulse, and
no respiration.
18
22. Ventricula
r and
Atrial rate
Ventricula
r and
atrial
rhythm
QRS P wave PR interval P:QRS
ratio
Atrial and
ventricular
rate 40-
60 bpm if
P waves
are
discernibl
e
Regular Usually
normal,
may be
abnormal
May be
absent,
after the
QRS
complex,
or before
QRS; may
be
inverted
specially
in lead 2
If the P
wave is in
front of
QRS, the
PR interval
is less
than 0.12
seconds
1:1 or 0:1
22
23. No pharmacologic therapy is needed for
asymptomatic, otherwise healthy individuals
with junctional rhythms that result from
increased vagal tone. In patients with
complete AV block, high-grade AV block, or
symptomatic sick sinus syndrome (ie, sinus
node dysfunction), a permanent pacemaker
may be needed.
23
24. If P wave can not be identified, the rhythm
may be called supraventricular tachycardia
SVT, or proximal supraventricular tachycardia
(PSVT), if it had an abrupt onset, until the
underlying rhythm and resulting diagnosis is
determined.
SVT and PSVT indicate only that the rhythm is
not ventricular tachycardia (VT) . SVT could
be atrial fibrillation, atrial flutter, or
atrioventricular reentry tachycardia among
others.
24
26. VT is defined as three or more PVCs in a row,
occurring at a rate exceeding 100 bpm.
Usually associated with coronary artery
disease, and may precede ventricular
fibrillations.
Ventricular tachycardia is SHOCKABLE
RHYTHM if pulseless
26
28. Ventricula
r and
Atrial rate
Ventricula
r and
atrial
rhythm
QRS P wave PR interval P:QRS
ratio
Ventricula
r rate is
100-
200bpm
Atrial rate
depends
on the
underlying
rhythm.
Usually
regular
Duration
is 0.12
seconds
or longer;
shape is
more
bizarre
and
abnormal
Difficult to
detect.
If the P
wave are
seen
interval is
very
irregular
Difficult to
determine
If P waves
are
apparent,
there are
usually
more QRS
than P
wave
28
29. Ventricular fibrillation is a rapid, disorganized
ventricular rhythm that causes ineffective
quivering of the ventricle.
No atrial activity is seen on ECG.
This arrhythmia is always characterized by
the absence of an audible heart beat, a
palpable pulse, and respiration, because of
no coordinated cardiac activity.
ITS SHOCKABLE RHYTHM
29
31. Ventricular and Atrial
rate
Ventricular and atrial
rhythm
QRS
Ventricular rate is
greater than 300bpm
Extremely irregular
without any specific
pattern
Irregular waves, without
recognizable QRS
complex
31
32. Treatment for VF
starts with early and effective CPR. Keeping
the brain, heart and other vital organs
perfused is very important in an arrest.
Once the rhythm is identified as ventricular
fibrillation, a shock should be delivered
immediately. After the shock is delivered,
begin CPR again for two minutes.
32
33. Commonly called flat line ventricular asystole
is characterized by ABSCENT QRS COMPLEX
confirmed in two different leads
Although P waves may be apparent for short
duration.
There is no heart beat, no palpable pulse, and
no respiration.
33
34. Ventricular asystole is treated with high quality CPR with
minimum interruption
After the initiation of CPR, intubation and establishment of
IV access are the next recommended actions with no or
minimum interruptions in chest compressions.
After 2 minutes or five cycles of CPR, a bolus of IV
epinephrine is administered and repeated at 3 to 5
minutes intervals.
One does of vasopressin may be administered for the first
or second does of epinephrine.
1 mg bolus of IV atropine
If patient still not respond resuscitation efforts are ended
The code is called
34
35. First degree AV block occurs when all the atrial
impulses are conducted through the AV node
into the Ventricles at a rate slower than normal.
Second degree AV block occur when there is a
repeating pattern in which a series of atrial
impulses are conducted through the AV node
into the ventricles (eg every 4 of 5 impulses are
conducted)
Each atrial impulse takes a longer time for the
conduction than before. Until one impulse is fully
blocked
35
39. O2 inhalation
Maintain i/v line
Continuously cardiac monitoring
Regularly evaluate the patients blood pressure, pulse
rate, and rhythm, and rate and depth of respiration
Ask the patient about episodes of lightheadedness,
dizziness, or fainting.
Obtain 12 lead ECG to track dysrhythmias, and also to
see results of antiarrhythmic medication.
39
40. Assess for the beneficial and adverse effects of each
medication
Nurse may also administer a 6 minutes walk test
Ask patient if any medication is being taken prior,
which can cause dysrhythmia
eg digoxin
Preparedness of equipment for invasive procedure
Access patient for dizziness ,chest pain, sweating any
other symptoms
40
41. DECREASED CARDIAC OUTPUT
ANXIETY RELATED TO FEAR OF UNKNOWN
OR DEATH
DEFICIT KNOWLEDGE ABOUT DYSRHYTHMIAS
AND ITS TREATMENT
41