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CASE PRESENTATION
OF
BROCHIESTASIS
Presented by ,
MR JEETENDRA
BANSILAL CHAVAN
(FY.PBBSC(N))
INTITUTE OF NURSING
MUMBAI
Bronchiectasis
INDENTIFICATION DATA OF
PATEINT
NAME -RUPALI UTTAM PARDHI
AGE -9 YEAR 9 MONTH
SEX - FEMALE CHILD
RELIGIOUS –HINDU
DIAGNOSIS –BROCHIESTASIS
PRESSENT HISTORY OF
ILLNESS
PRESENTING COMPLINTS
COLD COUGH
FEVER
DYSPNEA
MALAISE
PERSISTANT COUGH …SINCE 10 DAYS
PRESIPITING FACTORS
IMMUNOSUPRESSIVE
POOR ENVORNMENTAL HYGIENE
PAST HISTORY OF ILLNESS
H/o PULMONARY TUBERCULOSIS
AND EXTENSIVE
BROCHOPNEUMONIA since 5 yr
MEDICATION
She take AKT upto 6 month coarse
Stop AKT as per MEDICAL ADVOICE
FAMILY HISTORY
HAVING A NUCLEUS FAMILY AND 5
PERSON IS THEIR
*GRANDMOTHER
*FATHER
*MOTHER
*BROTHER
IMMUNIZATION
TAKEN
BCG,HEP-B,’0’POLIO …NOT TAKE STAT OR
WHENEVR AT HOSPITALIZATION AT TIME OF
LABOUR …..SHE TAKE AT 21 DAYS IN
ANGANWADI
AFTER OPV1/2/3,DPT1/2/3,HEP-B1/2/3,DPT
BOOSTER ,POLIO BOOSTER,MEASCLE AND
BOOSTER ,VITAMIN 1 AND 2 TAKE AS PER
UNIVERSAL SHEDULE
VITAMIN 3 TO 9 DOSE AND DT 5 YEAR ARE
NOT TAKEN
ECONOMIC STATUS
TOTAL INCOME OF FAMILY IS UPTO
10,000/month FROM LABOUR IN FARM
AND CELLING GOAT,BIRDS.THEY ARE
VERY MUCH POOR AND UNHYGENIC
FAMILY.
EXPENDITURE ON HEALTH
SPEND ONLY 20%,NEARLY 2000 FOR
SHOPING VEGITABLE ,OIL,AND KITCHEN
NECESSORY MATERIAL AND SO ON FROM
WEEKLY MARKET.
THEIR HOUSE IS ‘KACCHA”.
DIET PLAN
THEY TAKE BOTH VEG AND NONVEG DIET.
RUPALI WANT NEARLY 1500 Kcal DAILY IN
HER DIET PLAN
SHE TAKE DIALY
MILK 300ml AT MORNING
TAKE BRAKFAST WITH 1 CHAPATI AND TEA
LUNCH BEFOUR SHOOL 2 CHAPATI ½ CUP
RISE ½ SABJI(dal) OR NONVEG LIKE EGG
LUNCH IN SCHOOL TAKE ½ CUP KHICHADI
DINNER 2 CHAPATI ½ CUP DAL OR
NONVEG ATEMS LIKE BIRDS,FISH,CHIKEN etc
CHILD PERSONAL DATA
PRENATAL HISTORY
MOTHER OF CHILD HAVING TAKE ALL
TRATMENT AS PER GOI WITH RESPECTIVE
NEARER P.H.C
NO.ANY COMPLICATION REGARDING FTND
AT HOME DELIVERY BY TRAIN DAI.
PERINATAL
BABY CRY IMMEGIATLY
BUT WIGHT WAS ONLY 2 KG
NO NEED HOSPITALIZATION
ACTIVE
POSTNATAL
IN NEONATAL AND INFANT PERIOD
PROVIDED EXCLUSIVE BREAST FEEDING
UPTO 6 MONTH AND WEANING FOOD
STARTD FROM THEM
GROWTH AND DEVELOPMENT
S
R
ANTHRO. IN
BOOK
IN
PATIE
NT
INTER
ACTIO
N
1 HEIGHT 133 132 1 cm less
2 WEIGHT 28 18 VERY LESS
3 H.C - - -
4 C.C - - -
Developm
ant
IN BOOK IN PATIENT
Gross
motor
Enjoying all
physical
activity
LESS Enjoying
physical activity
Fights may
Occur
Fights
Occur in patient
Continues to
collection
eg hobbies
Continues to
collection ALSO
eg hobbies
PHYSICAL EXAMINATION
TEMP-99*F
PULSE-110/MIN
RESP- 24/MIN
FACE- FACE WAS DULL APPEARANCE
-WRINCKE ON FACE BY USING O2
MASK
SCLERA-WHITISH IN COLOUR
RESPIRATORY SYSTEM- ON AUSCULTATION
RIGHT SIDE LUNG LOBE RONCHI PRESNT
Bronchiectasis
broncos=airways;
ectasia=dilatation
Bronchial tree
Definition
• Irreversible dilatation of the cartilage
containing airways of the bronchi
and bronchioles (greater than
2mm in diameter)
Pathological
Classification of
Bronchiectasis
High
Resolution CT
Cylindric
al
Saccular/Varico
id
Cysti
c
Cylindrical
bronchiectasis
Varicose
bronchiectasis
Cystic
bronchiectasis
Mild
Tram track
appearance
Moderate
String of beads
Sever
Cluster of grapes
Cylindrical bronchiectasis
Varicose bronchiectasis
Cystic bronchiectasis
ETIOLOGICAL FACTORS
1)IDEOPATHIC CAUSE
2)Bronchiectasis has both
congenital and acquired
causes.
CONGENITAL CAUSES
2Kartagener syndrome
2 primary immunodeficiencies
2 Williams-Campbell syndrome and Marfan’s
syndrome.
2 Patients with alpha 1-antitrypsin deficiency
have been found to be particularly
susceptible to bronchiectasis,
ACQUIRED CAUSES
#Tuberculosis,
#pneumonia,
#inhaled foreign bodies,
#allergic bronchopulmonary aspergillosis and
bronchiol tumours are the major acquired
causes of Bronchiectasis.
INFECTIVE CAUSES ASSOCIATED WITH
BRONCHIECTASIS INCLUDE
2 infections caused by
the Staphylococcus,
Klebsiella, or
Bordetella pertussis,
the causative agent of
whooping cough.
ASPIRATION OF AMMONIA AND OTHER TOXIC
GASES,
2 pulmonary aspiration,
2alcoholism, heroin (drug use),
2 various allergies all appear to be linked
to the development of Bronchiectasis
3Childhood Acquired Immune Deficiency
Syndrome (AIDS), which predisposes patients
to a variety of pulmonary ailments, such as
pneumonia and other opportunistic infections.
3 Inflammatory bowel disease, especially
ulcerative colitis.
3 A Hiatal hernia can cause Bronchiectasis when
the stomach acid that is aspirated into the
lungs causes tissue damage.
CLINICAL MANIFESTATION
The production of large quantities of purulent
and often foul-smelling sputum.
The volume of sputum can be used for
estimating the severity of the disease
2 Mild < 10 mL
2 Moderate 10~150 mL
2 Severe >150 mL
OTHER
2 Some people with bronchiectasis may
produce frequent green/yellow sputum (up to
240ml daily.
2 Bronchiectasis may also present with
hemoptysis
2 Pneumonia
2 Bad breath indicative of active infection.
2 Frequent bronchial infections and
breathlessness are two possible indicators of
2. Chronic cough
3.Hemoptysis:
Frequent
 More commonly in dryvariety
 Usually mild (blood streaking ofpurulent
sputum)
 Massive hemoptysis is usually from
dilated bronchial arteries or bronchial-
pulmonary anastomoses under systemic
pressure
4. Recurrent pneumonia:
same segment
5. Systemic manifestations:
fever, weight loss
PATHOPHYSIOLOGY
2Dilation and distortion of the
bronchi
2Damage of airway epithelium
2Dilation and hyperplasia of
blood capillary
Due to etiological factor
Inflammation of bronchial wall
causing
Loss of supporting structure
Result in
Thick sputum that obstruct the bronchi
The bronchial wall become
permanently dialated and distorted
PATHOLOGY
Airway Injury +
Secretion Stimuli
Secretion Stasis Infection
Airway Destruction +
Airway Dilation
Histologic Changes
in Bronchiectasis
Normal
bronchus
Bronchiectas
is
Inflammato
ry cells
infiltration
Cartilage
destructio
n and
fibrosis
Mucosal
and
mucous
gland
hyperplasia
Increase
d mucus
and
exudates
Chest
Radiograph
Bronchial wall thickening and
widening with parallel
configuration
“Tram track”sign
Chest
Radiograph (2)
Conglomerating cysts of varying
size and wall thickness
“Honeycomb” sign
INVESTIGATION
3 History and physical examination
3 Chest x-ray
3 CT (computerised tomography) scan
3 Blood tests
3 Testing of the mucus to identify any bacteria
present
3 Checking oxygen levels in the blood
3 Lung function tests (spirometry).
Spirometry and Arterial
Blood Gas Analysis
Supportive evidence of airway disease (Obstructive
defect) Severity assessment (Severe impairment =
poor prognosis) Bronchodilator therapy (Positive
bronchodilator response) Oxygen therapy
(Hypoxemia)
Dilated bronchus
BRONCHIECTASIS
Signet ring sign
broncho-arterial ratio > 1
Multiple air fluid levels
INVESTIGATION
BLOOD GAS(ABG) REPORT
BLOOD PH-7.298(7.35-7.45)
PCO2 -47.9mmhg(35.0-45.0)
Po2 -31.8mmhg(80.0-100.0)
COUNT
WBC COUNT -11500(4000-10000)
TREATMENT
Therapy has several major goals:
(1)Treatment of infection, particularly during acute
exacerbations
(2) Improved clearance of tracheobronchial secretions
(3) Reduction of inflammation
(4) Treatment of an identifiable underlying problem
TREATMENT
2 Treatment of bronchiectasis includes
2 controlling infections and bronchial
secretions,
2 relieving airway obstructions,
2 removal of affected portions of lung by
surgical removal or artery embolization
2 preventing complications.
TREATMENT
 Medical management
1.Improving the drainage of airway
1) expectorant
2) bronchodilators
3) postural drainage
2. Antibiotic
 The choice of antibiotics should be
accurately by the results of sputum
culture and drug sensitivity test.
 Empirical therapy ---
antipseudomonal antibiotics.
ANTIBIOTICSARETHE OF BRONCHIECTASIS
MANAGEMENT
2 Antibiotics are used only during acute
episodes
2 Choice of an antibiotic should be guided by
gram's stain and culture of sputum
2 Empiric coverage (amoxicillin, co-
trimoxazole,levofloxacin) is often given
initially
BRONCHODILATER
 Bronchodilators to improve
obstruction and aid clearance
of secretions are useful in
patients with airway
hyperreactivity and reversible
airflow obstruction
TRATMENT
INJ MONOCEF
TAB PCM
TAB CITRAZINE
TAB SEPTRON
TAB ASTHALINE
TAB MVBC
TAB CAL-LACTATE
SYP PROMITHAZINE
NEBULAZATION GENTA,ASTHALINE
,BRUDECORT
O2 (2-3 lit/min)
 Surgical management is indicated
1. Recurrent and refractory clinical
symptoms are due to a focal area
of disease involvement.
2. Massive hemoptysis
 Management of hemoptysis
NURSING MANAGEMENT:
2 History and physical examination
2 Obtain history regarding amount
and characteristics of sputum
produced, including haemoptysis.
2 Auscultate lungs for diffuse rhonchi
COMPLICATIONS
2 Progressive suppuration.
2 Haemoptysis, major pulmonary
haemorrhage.
2 COPD,
2 emphysema,
2chronic respiratory insufficiency
COMPLICATION
Local complication
 Recurrent pneumonia
 Lung abcess
 Empyema
 Hemoptysis
 Pulmonary hypertension
Bronchiectasis: Summary
Abnormal irreversibly dilated and often
thick-walledbronchi
Pathogenesis related to one or more
defects ofmucociliary clearance, cellular
and immunity defense mechanism or
presence of associated conditions
“The vicious cycle”and P aeruginosa
contributes progression and severity of
disease
Imaging greatly helps in diagnosis: Tram
line, honeycombing, cystic, signet ring sign
Additional test may be required in specific
clinical settings
Microbiology of the diseased
airway may aidproper
antimicrobial therapy
BRONCHIESTASIS PRESENTATION

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