This document discusses updated clinical classifications for cerebral palsy. It begins by defining cerebral palsy and describing the typical classifications based on tone, topography, and motor involvement. It then introduces newer classification systems like the Gross Motor Function Classification System (GMFCS) and Manual Ability Classification System (MACS) which focus on functional ability rather than impairments. These systems aim to provide a common language for clinicians, researchers, and families to describe what individuals with CP can do, not just what body structures are involved. The classifications systems can help guide treatment planning and set realistic functional goals.
Alan T. Rasof explains the four major types of Cerebral Palsy and why it is important that we, as a society, understand the differences and affects of each.
Alan T. Rasof explains the four major types of Cerebral Palsy and why it is important that we, as a society, understand the differences and affects of each.
Definition, Medical and Nursing Management of the following Neurological Disorder-Cerebrovascular Disorders, Transient Ischemic Attack, Brain Attack, Cerebral Aneurysm, Subarachnoid Hemorrhage
Learn more about autism and how it impacts an individual's ability to function on a day to day basis.
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Presentation on cerebral palsy (CP), with a focus on CP in Singapore. It examines the characteristics and impacts of CP, the possible educational pathways and assessments available for those with CP. We (my presenter, Camelia and I) believe that those with CP can lead fulfilled lives.
Definition, Medical and Nursing Management of the following Neurological Disorder-Cerebrovascular Disorders, Transient Ischemic Attack, Brain Attack, Cerebral Aneurysm, Subarachnoid Hemorrhage
Learn more about autism and how it impacts an individual's ability to function on a day to day basis.
We would love to read your feedback so please contact us and tell us what you think/feel.
Presentation on cerebral palsy (CP), with a focus on CP in Singapore. It examines the characteristics and impacts of CP, the possible educational pathways and assessments available for those with CP. We (my presenter, Camelia and I) believe that those with CP can lead fulfilled lives.
David parra reyes curriculum vitae actual setiembre 2013David Parra
Posee el Título de Licenciado en Tecnología Médica en la Especialidad de Terapia de Lenguaje por la Universidad Nacional Federico Villarreal (2003) y Grado de Magister en Neurociencias en laFacultad de Medicina de la Universidad Nacional Mayor de San Marcos (2006); estudios concluidosen Doctorado de Ciencias de la Educación en la Universidad Nacional de Educación “EnriqueGuzmán y Valle”. Diplomado en Docencia Superior/Universitaria por la UNE. Título de Especialista enMotricidad Orofacial concedido por CEFAC (Brasil) – CPAL (2007) y en Tecnologías de laEnseñanza del Lenguaje en la UNASAM. Miembro de la INTERNATIONAL ASSOCIATION OFOROFACIAL MYOLOGY (IAOM), y de la Academia Latinoamericana de DisfuncionesEstomatognáticas (ALDE), miembro fundador de la Academia de Neurociencias del Perú (ANP), dela Comunidad de Motricidad Orofacial Latinoamericana (CMOL) y de la Asociación Peruana para elEstudio del Dolor (APED). Actualmente labora en el Seguro Social de Salud – ESSALUD Hospital IV“Guillermo Almenara Irigoyen”. Docente en pre-grado y post-grado de diversos cursos especializadossobre Terapia de Lenguaje en la UNFV, UNMSM, ULADECH y UAP. Con amplia experiencia clínica,investigación científica y de producciones bibliográficas en la especialidad de Terapia de Lenguaje yFonoaudiología, con énfasis en Motricidad Orofacial, Síndromes Neurológicos y Foniatría, actuandoprincipalmente en los temas de: succión, respiración, masticación, deglución y fonoarticulación, contrabajos y artículos presentados y expuestos en cursos, jornadas científicas, congresos nacionales einternacionales. Miembro del cuerpo editorial en la calidad de Editor de la revista científica deMedicina de Rehabilitación del Departamento de Medicina de Rehabilitación del Servicio de otrasDiscapacidades y Minusvalías del HNGAI, y Miembro del comité editorial en la calidad de Editor Asociado de CoDAS (publicación técnico-científica de la Sociedad Brasilera de Fonoaudiología, SãoPaulo). Director y editor de la revista digital de motricidad Orofacial “RevMOf”. Columnista de laRevista Latinoamericana de Ciencia y Cultura “ANRA”. Miembro del Instituto de Investigación de laFTM y del comité consultivo interno como docente investigativo invitado por el Vicerrectorado deInvestigación de la UNFV. Director científico y de investigación de la Comunidad de MotricidadOrofacial Latinoamericana.
Cerebral Palsy (CP) is one of the nervous system impairment that occurs during fetal life in womb, birth or infancy. ‘Cerebral’ comes from the word cerebrum; the two hemisphere of the forebrain and ‘Palsy’ means paralysis accompanied by involuntary tremors.
While our brain consists of sensory area to receive stimuli and motor area to give respond, a child with CP has a damage motor area of the brain. This will cause them to loss their ability to control their muscle and body coordination (Tortora & Derrickson, 2011, p.630). Among the causative factor of CP include prematurity of baby during delivery, placental insufficiency, anoxia (low oxygen) during birth or other infection of mother, fetus or infant that can affect the central nervous system (Mahan, Stump, Raymond, 2012, p.1033).
According to MyChild™ (n.d), CP is incurable, permanent and chronic. It is irreversible and currently cannot be fixed. Once the brain damage occurs, it does not heal like other cell in the body does. CP is a permanent occasion that neither the injury in the brain undergoes healing process nor worsens during a person life time. As a person is diagnosed with CP, they will have the condition for their entire life.
Despite of being a permanent disorder, CP is a non-progressive disorder. The brain lesion occur is a one-time brain injury and will not cause further degeneration. CP is also a non-communicable disease thus does not spread through human contact. However, environmental factor can increase the risk of CP such as abuse, accident, medical malpractice or bacterial and viral infection. Fortunately, CP is manageable.
Although the person may experience difficulties in their movement, speech and other motor skill, therapy, surgery, medication and assistive technology can help them to be more independence. Plus with the support from their family, they can enhance the quality of life. (MyChild™, n.d)
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Similar to Actualizaciones en la clasificación de Parálisis Cerebral Infantil y su relevancia en el pronóstico. Dra. Deborah Gaebler (20)
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
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MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Actualizaciones en la clasificación de Parálisis Cerebral Infantil y su relevancia en el pronóstico. Dra. Deborah Gaebler
1. UPDATE-CLINICAL
CLASSIFICATIONS FOR
CEREBRAL PALSY Deborah Gaebler-Spira
XIII International ORITEL Conference
Foundational and First General Assembly
of the Latin American Academy on Child
Development and Disability
2. 9/2/11
2 A Neural Interface for Artificial Limbs: Targeted Muscle Reinnervation
REHABILITATION INSTITUTE OF CHICAGO
2
3. OBJECTIVES
CP - descriptors
The context of the ICF
Classifications and relationships
How this moves us forward together
4. LET’S START
What do parents ask about?
• Diagnosis - what does my child have?
• Function - what can my child do?
5. CEREBRAL PALSY-
DEFINITION-BAX-2001
Disorder of movement and posture resulting from a
condition of non-progressive brain damage that occurred
in infancy
Abnormality of tone
Inclusive-many etiologies
Brain lesion is static-musculoskeletal system changes
10. DEFINITION OF CEREBRAL PALSY
Cerebral palsy (CP) describes a group of permanent
disorders of the development of movement and posture,
causing activity limitation, that are attributed to non-
progressive disturbances that occurred in the developing
fetal or infant brain.
The motor disorders of cerebral palsy are often
accompanied by disturbances of sensation, perception,
cognition, communication, and behavior, by epilepsy,
and by secondary musculoskeletal problems.
Rosenbaum, et al. (2007)
11. HOW THAT CHANGES THE PERSPECTIVE
Creates an emphasis on activities, not just
impairments
Creates the inclusion of sensory abnormalities
Attributes co-morbidities as important factors in
prognosis
12. NEW/WHO/ICF
Health Condition
(disorder or disease)
Body Functions &
Structures
Activities Participation
Environmental
Factors
Personal Factors
Interactions between components of the ICF
13. 9/2/11
2 A Neural Interface for Artificial Limbs: Targeted Muscle Reinnervation
GMFC-GROSS MOTOR FUNCTION CLASSIFICATION
14. GMFCS
The Gross Motor Classification System
Developed to classify severity of functional
limitation/disability in children with cerebral palsy.
Ages birth to 12 years
Not to be used as a diagnostic tool- describes gross motor
function with an emphasis on movement initiation, sitting
control and walking.
15. GMFCS
Reliable method of classifying based on function
Inherent meaning to families-therapists-physicans
Usual performance
18. GMFCS LEVELS
Level I: Walks without assistive
device indoors. Climbs stairs
without limitation. Able to run
and jump. Impaired speed,
balance, coordination.
19. GMFCS LEVELS
Level II: Children walk indoors
and climb stairs holding onto
railing. Difficulty with walking
on uneven surfaces and
inclines or within crowds or
confined spaces.
20. GMFCS LEVELS
Level III: Walks with assistive
mobility devices on level
surface. Limitations on uneven
surfaces or inclines. May
propel wheelchair manually.
May use wheelchair for long
distance transport.
21. GMFCS LEVELS
Level IV: Walks for short
distances on a walker.
Wheeled mobility for
outdoors, school and
community.
22. GMFCS LEVELS
Level V: All areas of motor
function are limited. No
independent mobility even
with assistive technology.
25. MANUAL ABILITY CLASSIFICATION-MACS
Children with cerebral palsy use their hands when
handling objects in daily activities
Assesses typical, not optimal performance
Ages 4-18 years
27. MACS
I. Handles objects easily and successfully
II. Handles most objects but with somewhat reduced quality and/or speed of
achievement
III. Handles objects with difficulty; needs help to prepare and/or modify activities. The
performance is slow and achieved with limited success regarding quality and quantity.
Activities are performed independently if they have been set up or adapted.
IV. Handles a limited selection of easily managed objects in adapted situations. Performs
parts of activities with effort and with limited success. Requires continuous support
and assistance and/or adapted equipment, for even partial achievement of the activity.
V. Does not handle objects and has severely limited ability to perform even simple
actions. Requires total assistance
30. VIKING SPEECH SCALE
Speech is not affected by motor disorder.
Speech is imprecise but usually understandable to unfamiliar listeners. Loudness of speech is
adequate for one to one
Conversation. Voice may be breathy or harsh sounding but does not impair intelligibility.
Articulation is imprecise; most consonants are produced, but deterioration is noticeable in longer
utterances. Although difficulties are noticeable, speech is usually understandable to unfamiliar
listeners out of context.
Speech is unclear and not usually understandable to unfamiliar listeners out of context. Difficulties
controlling breathing for speech – can produce one word per utterance and/or speech is
sometimes too loud or too quiet to be understood. Voice may be harsh sounding; pitch may
change suddenly. Speech may be markedly hyper nasal. A very small range of consonants are
produced. The severity of the difficulties makes the speech difficult to understand out of context.
No understandable speech.
31. WHY ARE THEY IMPORTANT
Meant to discriminate and categorize rather than 'assess’ (Damiano et
al.,2006)
Easily applied, simple and quick classifications which may be performed by
a physical therapist, the family or a related person, and provide information
about the functional level of the child with CP (Morris et al., 2004b; Eliasson
et al., 2006, Mutlu et al., 2010)
fulfill each other for a total and whole classification of children with CP
(Morris et al.,2006; Kerem-Gunel et al., 2009)
Universal, translated and studied on many different languages
(www.canchild.ca)
32. EDACS
I - Eats safely and efficiently
II - Eats and drinks safely but have limitations to efficiency
III - Eats and drinks safely but have limitations to efficiency
and safety
IV - Eats and drinks with significant safety issues
V - Unable to eat safely-G tube
33. 9/2/11
2 A Neural Interface for Artificial Limbs: Targeted Muscle Reinnervation
Is the individual
able to swallow
food and drink
without risk of
aspiration?Is the individual able
to bite and chew on
hard lumps of food
without a risk of
choking?
Is the individual
able to eat a meal
in the same time
as peers?
Level I
Eats and drinks
safely and
efficiently
Level IV
Eats and drinks with
significant
limitations to safety.
Level V
Unable to eat or
drink safely – tube
feeding may be
considered to
provide nutrition.
Can risks of
aspiration be
managed to
eliminate harm to
the individual?
No
Yes No
Yes No
Yes
Eating and Drinking Ability Classification System - Algorithm
Yes No
Level II
Eats and drinks
safely but with
some limitations
to efficiency.
Level III
Eats and drinks
with some
limitations to
safety; there maybe
limitations to
efficiency.
34. ICF
Environmental Factors Personal Factors
Body Function & Structure
(Impairment)
Muscle strength (muscle test,
dynamometer)
Spasticity(M.Ashworth, Tardieu)
ROM(Goniometry )
Selective motor control (SCALE-TASC
Tests )Perception, cognition
Postural problems
Activity
(Limitation)
GMFCS,FMS
MACS
,CFCS,EADSC,.
Participation
(Restriction)
Daily Living activities,
Social roles in
community (children,
student, friends,etc.)
WeeFIM
PEDI etc.
35. OPTIMIZES MANAGEMENT
Sharpens aligns focus on function versus impairments
More useful than severity, type and distribution
36. INTERVENTION PLANNING
Assists with realistic goal therapy setting
Children with GMFCS 3 –community wheelchair
GMFCS 3,4-use walker part time
GMFCS 5 limited self mobility
38. THERAPY INTERVENTIONS
Secondary impairments vary with GMFCS level
Endurance, fatigue, weakness –can target better
interventions for groups
Supports evidence based research
39. VARIATIONS IN MEDICAL AND SURGICAL NEEDS
Hip pathology increases with GMFCS level
Use of G-tube and co-morbidities increase with GMFCS
levels
40. IN A VARIABLE DISORDER-ALLOWS-CLINICIANS-
PARENTS
Common language
Common groupings
Common Goals