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Prepared By: Justin V Sebastian, MSc N, RN, PhD Scholar
Integumentary System
• This system is divided into:


	
1- skin


	
2- hair


	
3- glands


	
4- nails


	
5- nerve endings


	


Skin


	
Skin is an organ because it consists of different
tissues that are joined to perform a specific function.


	


Largest organ of the body in surface area and weight.


	
Dermatology is the medical specialty concerning the
diagnosing and treatment of
	
skin disorders.
Anatomy (structure)


Epidermis (thinner outer layer of skin)


Dermis (thicker connective tissue layer)


Hypodermis (subcutaneous layer or Sub-Q)


Muscle and bone


Physiology (function)


1- Protection


	
	
- physical barrier that protects underlying
tissues from injury, UV light and bacterial
invasion.
2- Regulation of body temperature


	
	
- high temperature or strenuous exercise;
sweat is evaporated from the skin surface to
cool it down.


- vasodilatation (increases blood flow) and
vasoconstriction (decrease in blood flow)
regulates body temp.


3-Sensation


	
	
- nerve endings and receptor cells that
detect stimuli to temp, pain, pressure and touch.
4- Excretion


	
	
- sweat removes water and small amounts of
salt, uric acid and ammonia from the body
surface


5- Blood reservoir


- dermis houses an extensive network of
blood vessels carrying 8-10% of total blood
flow in a resting adult.


6- Synthesis of Vitamin D (cholecalciferol)


-UV rays in sunlight stimulate the production
of Vit. D. Enzymes in the kidney and liver
modify and convert to final form; calcitriol
(most active form of Vit. D.) Calcitriol aids in
absorption of calcium from foods and is
considered a hormone.
Definitions


1. A burn occurs when there is an injury to the
tissues of the body caused by heat, chemicals,
electric current, or radiation


	
	
	
	
	
	
lewis’s
	


	
	
The effects are influenced by


• The temperature of the burning agent


• Duration of contact time


• Type of tissue that injured
2, 	
Burns are a form of traumatic injury caused
by thermal, electrical, chemical, or radioactive
agents.


	
	
	
	
	
	
	
Lippincott


3,
	
Burns are caused by transfer of energy from
a heat source to the body


	
	
	
	
	
	
Brunner and suddarth’s


	
	
The depth of injury depend on:


• Temperature of the burning agent


• Duration of contact with it
Incidence
• Males have greater then twice the chance of
burn injury than females.


• The most frequent age group for contact burns
is between 20 to 40 years of age.


• The national fire protection association reports
4,000 fire and burn deaths each year. Of these,
3500 deaths occur from residential fires and
the remaining 500 from other sources such as
motor vehicle crashes, scalds, or electrical and
chemical sources.
Etiology and types
1. Thermal burns may result from any external heat
source (flame, liquids, solid objects, or gases).


2
,
Radiation burns most commonly result from
prolonged exposure to solar ultraviolet radiation
(sunburn) but may result from prolonged or intense
exposure to other sources of ultraviolet radiation (eg,
tanning beds) or from exposure to sources of x-ray or
other non solar radiation.
3, Chemical burns may result from strong acids, strong
alkalis (e.g, lye, cement), phenols, cresols,
mustard gas, or phosphorus. Skin and deeper
tissue necrosis due to these agents may progress
over several hours.


4, Smoke and inhalation injury


	
Result from the inhalation of hot air or noxious
chemicals and can cause damage to the tissues of
the respiratory tract.


There are three types of inhalation injury


Carbon monoxide poisoning


Inhalation injury above the glottis


Inhalation injury below the glottis
5, Electrical burns result from the electrical
generation of heat; they may cause extensive deep
tissue damage despite minimal apparent cutaneous
injury.


6, Cold injury

	
A general term for an injury induced by low
ambient temperatures ( eg. Frostbit), which may
be accompanied by tissue freezing.


	
Risk groups includes infants, young children,
elderly, alcohol use
Common places and causes of burn injury
❖ Occupational hazards


• Tar


• Chemicals


• Hot metals


• Steam pipes


• Combustible fuels


• Fertilizers/pesticides


• Electricity from power
lines


• Spark from live
electric sources
❖ Home and
recreational hazards


• Pressure cookers


• Microwaved food


• Hot water heaters


• Defective wiring


• Radiators


• Carelessness with
cigarettes or matches


• Storage of flammables


	
( e.g.,kerosene)
Risk Factors
Non Modifiable Risk Factors


•Age: Children under 4 who are
poorly supervised are at particular
risk.


•Gender: Males are more than
twice as likely to suffer burn
injuries.
Modifiable Risk Factor
s

•Careless smoking: Cigarettes are the leading cause of house
fires.
•Absent or nonfunctioning smoke detectors: The presence of a
functioning smoke detector decreases risk of death by fire by 60
percent.
•Use of wood stove
s

•Exposed heating sources or electrical cords
 

•Unsafe storage of flammable or caustic materials
 

•Water heaters set above 120°F
 

•Microwave heated foods and containers
 

•Substandard or older housing
 

•Substance abuse: Use of alcohol and illegal drugs increases risk.
Classification of burn in jury
1. Depth of burn


2. Extent of burn


3. Location of burn
Assessment of depth of Burn Injury
Takes several weeks to heal.


Scarring may occur.


Takes several weeks to heal.


Scarring may occur.


Superficial:


Pink or red; blisters form (vesicles);
weeping, edematous, elastic.


Superficial layers of skin are destroyed;
wound moist and painful.


Deep dermal:


Mottled white and red: edematous
reddened areas blanch on pressure.


May be yellowish but soft and elastic –
may or may not be sensitive to touch;
sensitive to cold air.


Hair does not pull out easily
Second degree


(deep partial-
thickness)
In about 5 days, epidermis peels, heals
spontaneously.


Itching and pink skin persist for about a
week.


No scarring.


Heals spont. If it does not become infected
w/in 10 days - 2 weeks.
Pink to red: slight edema, which
subsides quickly.


Pain may last up to 48 hours.


Relieved by cooling.


Sunburn is a typical example.
First Degree


(superficial
partial-thickness)
Reparative Process
Assessment of Extent
Extent / Degree
Eschar must be removed. Granulation
tissue forms to nearest epithelium from
wound margins or support graft.


For areas larger than 3-5 cm, grafting is
required.


Expect scarring and loss of skin function.


Area requires debridement for formation of
granulation tissue, and grafting.
Destruction of epithelial cells –
epidermis and dermis destroyed


Reddened areas do not blanch with
pressure.


Not painful; inelastic; coloration varies
from waxy white to brown; leathery
devitalized tissue is called eschar.


Destruction of epithelium, fat, muscles,
and bone.
Third degree


(full- thickness)
Reparative Process
Assessment of Extent
Extent / Degree
Assessment of depth of Burn Injury
AGE AND GENERAL HEALTH


• Mortality rates are higher for children < 4 y.o, particularly those < 1 y.o., and for
clients over the age of 60 years.


• Debilitating disorders, such as cardiac, respiratory, endocrine, and renal,
negatively influence the client’s response to injury and treatment.


• Mortality rate is higher when the client has a preexisting disorder at the time of
the burn injury
Assessment of extent of burn injury
	
Various methods are used to estimate the TBSA
affected by burns; among them are


1.Rule of nines


	
	
Rule of Nines for Establishing Extent of Body Surface
Burned Anatomic Surface


• % of total body surface


• Head and neck
	
	
=9%


• Anterior trunk
	
	
=18%


• Posterior trunk
	
	
=18%


• Arms, including hands
	
=9% each


• Legs, including feet
	
	
=18% each


• Genitalia/Perineum
	
	
	
=1%
9
18
9
18
9
18
1
2, Lund and browder method
3,	
Palmer method
Assessment of location of burn injury
	
	
The severity of the burn injury is
related to location of the burn wound


• Burns to the face and neck and
circumferential burns to the chest/back


• Burns of the hands, feet, joints, and eyes


• Burns to the ears and nose


• Burns to the buttocks and genitalia


• Circumferential burns to the extremities
Zone of burn
Zone of burn
Pathophysiology
Heat causes coagulation necrosis of skin and
subcutaneous tissue


	
	
	
Release of vasoactive peptides


	
	
	
Altered capillary permeability


	
	
	
	
Loss of fluid	
severe hypovolemia
Decreased cardiac
	
decreased myocardial


	
	
output
	
	
	
	
function


	
	
Decreased renal
	
	
oliguria


	
	
blood flow


Altered pulmonary resistant and pulmonary edema


	
	
	
	
Infection


	
	
	
	
	
SIRS
	
	
MODS
Clinical features


❖Cardiovascular alterations


o Hypovolemia


o Decreased CO


o Burn shock


o Increase peripheral resistance


o Anemia


o Increased hematocrit


o Decreased platelets


o Diuresis
❖Fluid and electrolyte alterations


o Edema


o Ischemia


o Hyponatremia


o Hyperkalemia


o Hypokalemia


❖Pulmonary alterations


o Hypoxia


o Loss of ciliary action


o Hyper secretion


o Mucosal edema


o Bronchospasm
o Atelectasis


o Restrictive pulmonary excursion


o Decreased lung compliance


o Decreased arterial O2 level


o Respiratory acidosis


o Pulmonary complications such as airway
obstruction, ulceration, pulmonary edema,
respiratory failure, ARDS and pneumonia can
develop.
❖Renal alterations


o Altered renal function


o Hemoglobin in the urine


o Myoglobin in the urine


o Acute tubular necrosis


o Renal failure


❖Immunological alterations


o Infection and sepsis


❖Thermoregulatory alterations


o Hypothermia
❖Gastrointestinal alterations


o Paralytic ileus


o Curling’s ulcer


o Gastric bleeding


o Infection
Diagnostic
Tests
•A detailed history and
physical examination is the
first step. The physician will
evaluate the type, duration,
and timing of the burn; the
burn location and severity;
and associated dehydration,
disfigurement, and infection.


•Fires in enclosed spaces
should raise the suspicion for
smoke–inhalation injury.
•Routine blood work for a patient with
a burn injury includes a complete blood
count, platelet count, clotting studies,
l i v e r f u n c t i o n s t u d i e s , a n d
carboxyhemoglobin, electrolyte, blood
urea nitrogen, glucose and creatinine
levels.


•Urinalysis may reveal myoglobinuria
and hemoglobinuria.


•If pt. is electric burn, he’ll also need an
electrocardiogram.


•Chest x-rays and arterial blood gas
levels allow the evaluation of alveolar
function. Fiberoptic bronchoscopy shows
the condition of the trachea and bronchi.
Management
Phases of burn care
I. Emergent/resuscitative phase


II.Acute/ intermediate phase


III.Rehabilitation phase
Emergent/resuscitative phase


	
	
From the onset of injury to completion of fluid
resuscitation.
	


Priorities


oFirst aid


oPrevention of shock


oPrevention of respiratory distress


oDetection and treatment of injuries


oWound assessment and initial care
On-the-scene care


oExtinguish the flames


oCool the burn


oRemove restrictive objectives


oCover the wound


oIrrigate chemical burn
ESCHAROTOMY


• A lengthwise incision is made through the burn eschar to relieve
constriction and pressure and to improve circulation


• Performed for circulatory compromise resulting from circumferential
burns


• After escharotomy, assess pulses, color, movement, and sensation of
affected extremity and control any bleeding with pressure


• Apply topical antimicrobial agents as prescribed
Medical management


❖The patient transported to the nearest ED


❖Initial priorities in the ED remain ABC and C spine
control


❖For mild pulmonary injury 100% humidified O2 is
administered


❖The patient is encouraged to cough


❖For more severe situations; administer
bronchodilator and mucolytic agents


❖If edema of airway develops, ET intubation may be
necessary


❖All clothing and jewellery are removed
Medical management ( cont..)


❖For chemical burn flushing of the exposed area
should be done


❖Validate an account of the burn scenario provided by
the patient or witnesses


❖A large bore ( 16 or 18 gauge) IV catheter should be
inserted


❖If burn exceeds 20% to 25% TBSA, a NG tube
inserted


❖Clean technique is maintained while assessing and
treating the burn wounds


❖Careful attention is paid to keeping the patient warm
Medical management ( cont..)


❖Photograph may be taken of the burn area initially
and periodically throughout treatment


❖Clean sheets are placed under and over the patient


❖An indwelling urinary catheter is inserted


❖Baseline data should be obtained ( such as height,
weight, ABG, electrolyte, blood alcohol, drug panel,
chest X rays etc..)


❖If the patent has an electrical burn, a baseline ECG


❖Tetanus prophylaxis


❖Attends the patient’s and family’s psychological
needs
Medical management ( cont..)


❖Transfer to a burn centre


Management of fluid loss and shock


Observation


o Generalized dehydration


o Reduction of blood volume


o Decreased urinary output


o Potassium excess


o Sodium deficit


o Metabolic acidosis


o Elevated hematocrit
FLUID REQUIREMENT


• Indications:


	
- Adults with burns involving more than 15% - 20% TBSA


	
- Children with burns involving more than 10-15% TBSA


	
- Patients with electrical injury, the elderly, or those with cardiac
or pulmonary disease and compromised response to burn injury


• The amount of fluid administered depends on how much
intravenous fluid per hour is required to maintain a urinary output
of 30 - 50 ml/hr


• Successful fluid resuscitation is evidenced by:


- Stable vital signs
	
	
- Palpable peripheral pulse
	


- Adequate urine output
	
- Clear sensorium


• Urinary output is the most common and most sensitive assessment
parameter for cardiac output and tissue perfusion


• If the Hgb and Hct levels decrease or if the urinary output exceeds
50ml/hr, the rate of IV fluid administration may be decreased


• Generally, a crystalloid (Ringer’s lactate) solution is used initially.
Colloid is used during the 2nd day (5% albumin, plasmate)
½ in 1st 8 hours


½ in next 16 hours


crystalloid only


(lactated Ringer’s)
PARKLAND (Baxter)


4ml/kg/% BSA for 24hr
period
½ in 1st 8 hours


½ in next 16 hours
crystalloid, colloid


D5W maintenance
BROOKE


2ml/kg/% BSA + 2000ml/
24hr (maintenance)
Infusion Rate
Solution
Formula
Brooke and Parkland (Baxter) Fluid Resuscitation Formulas for 1st 24hrs
after a Burn Injury
PARKLAND FORMULA


Example: Patient’s weight: 70 kg; % TBSA burn: 80%


1st 24 hours:


4ml x 70kg x 80% TBSA = 22,400ml of lactated Ringer’s


• 1st 8 hours = 11,200 ml or 1400ml/hour


• 2nd 16 hours = 11,200 ml or 700ml/hour


BROOKE FORMULA


2nd 24 hours:


0.5ml colloid x weight in kg x TBSA + 2000ml D5W run
concurrently over the 24 hour period


0.5ml x 70kg x 80% = 2800 ml colloid + 2000 ml D5W


= 117 ml colloid/hour + 84 ml D5W/
hour
Acute/ intermediate phase


	
	
From beginning of diuresis to near
completion of wound closure ( 48 – 72
hrs )


❖Observation


oDecreased hematocrit


oIncreased urinary output


oSodium deficit


oPotassium deficit
Medical management (cont..)


❖Bronchial washing


❖Remove ET tube as soon as possible


❖Cautious administration of fluid and
electrolyte


❖Blood components are administered as
needed


❖Central venous, peripheral arterial or
pulmonary artery catheters may be
required
❖Infection prevention


❖Wound cleaning


❖Topical antibacterial therapy


❖Wound dressing


❖Wound debridement


❖Wound grafting


❖Pain management


❖Nutritional support
Medical management (cont..)


❖Infection prevention


➢Early enteral feeding


➢Aseptic technique is used when caring directly for burn
wound


➢Gowns and gloves are worn by all caregivers and
visitors


➢Hand hygiene is used before and after leaving the
patient room


➢Constant monitoring and observation of the wound


➢Tissue specimen may be obtained for culture to
monitor colonization


➢Systemic antibiotics are administered when there is
documented sepsis
Medical management (cont..)


❖Wound cleaning


➢Hydrotherapy


➢The temperature of the water is maintained at 37.8 C


➢During the bath, the patient is encouraged to be active
as possible


➢At the time of wound cleaning, all skin is inspected


➢Intact blisters should be left alone and debrided only if
they rupture or break


➢When nonviable loose skin is removed, aseptic
condition must be established


➢After the wounds are cleaned, they are gently patted
dry, and the prescribed method of wound care is
performed
❖TOPICALANTIMICROBIAL THERAPY


Silver sulfadiazine


• Most widely used agent and least common incidence of side
effects


• May cause transient leukopenia that disappears 2-3 days of
treatment


• Use with either open treatment, light or occlusive dressings


• Applied once or twice daily after thorough wound cleansing


Mafenide acetate 10% cream or 5% solution (Sulfamylon)


• Painful during and for a while after application


• May cause metabolic acidosis, not used if >20% TBSA


• Cream must be reapplied 12 hours to maintain therapeutic
effectiveness


• Solution concentration is maintained with bulky wet dressings,
rewet every 2-4 hours


Silver nitrate (0.5% solution)


• Stains everything including normal skin brown or black


• Monitor electrolyte balance carefully
Medical management (cont..)


❖Wound dressing


➢After wound cleaning, prescribed topical agent is
applied; the wound is then covered with dressings


➢A light dressing is applied over the areas of splint,
joints


➢Circumferential dressing should be applied to distally
to proximally


➢The fingers and toes should be wrapped individually


➢Burns to the face may be left open/a light dressing can
be applied


➢Occlusive dressing may be used over new skin graft to
protect the graft. Ideally, these dressing remain in place
for 3 to 5 days
Medical management (cont..)


❖Wound debridement


Goals


• Removal of tissue contaminated by bacteria and
foreign bodies, thereby protecting the patient from
invasion of bacteria


• Removal of devitalized tissue or burn eschar in
preparation for grafting and wound healing


There are 4 types


➢Natural debridement


➢Mechanical debridement


➢Chemical debridement


➢Surgical debridement
Natural debridement


	
	
The dead tissue separates from the underlying
viable tissue spontaneously.
Mechanical debridement


	
	
Involves the use of surgical scissors,
scalpels, and forceps to separate and remove
the eschar.
Chemical debridement


	
	
Use of topical enzymatic debridement agents to
remove eschar.
Surgical debridement


	
	
It is an operative procedure involving either
primary excision of the full thickness of the skin or
down to the burned skin layers gradually down to
freely bleeding, viable tissue.
❖Wound grafting


The purpose of wound grafting are:


oTo decrease the risk of infection


oTo prevent further loss of protein, fluids and
electrolytes


oTo minimize heat loss through evaporation


	
	
The main areas for skin grafting include the face
( for cosmetic and psychological reasons); functional
areas such as the hands and feet; and areas that
involves joints.


Methods


❖Biologic


❖Biosynthetic and synthetic


❖Autologous (autograft)
❖Biological dressing( homograft and
heterograft)


➢It provide temporary wound coverage and protect the
granulation tissue until auto-grafting is possible


➢Once the biological dressing appears to be adhering to the
granulating surface with minimal underlying exudation , the
patient is ready for an autologous skin graft


➢They decreases pain by protecting the nerve endings and are
effective barrier against water loss and entry of bacteria


➢These dressing often require fewer dressing changes


➢Homograft are skin obtained from living or deceased
humans


➢Heterograft consist of skin taken from animals ( usually
pigs)
❖Biosynthetic and synthetic dressings


➢A widely used synthetic dressing is Biobrane


➢It is semitransparent and sterile


➢Like biological dressing, Biobrane protects the
wound from fluid loss and bacterial invasion


➢When the Biobrane dressing is adheres to the
wound, the wound remain stable


➢It can remain in place until spontaneous
epithelialisation and wound healing occur


➢It can be laid on top of a wide meshed
autograft
Biosynthetic and synthetic dressings
(cont..)


Skin substitutes


	
	
It is believed that skin substitutes
enhance the healing process of an open
wound when autologous skin is
unavailable or limited for use
Biosynthetic and synthetic dressings (cont..)


Integra Artificial Skin


➢It is composed of two main layers


➢The epidermal layer consist of silicone, act as a bacterial
barrier and prevents water loss from the dermis


➢The dermal layer consist of animal collagen. It allows
migration of fibroblast and capillaries in to the material
❖Auto graft


	
	
Autograft are the ideal means of
covering burn wound because the graft are
the patient’s own skin and therefore are not
rejected by the patients immune system


They can be


❖Split-thickness


❖Full thickness


❖Pedicle flaps


❖Epithelial graft
❖Split thickness autograft


	
	
In this skin can be expanded by meshing so
that they cover 1.5 to 9 times more than a
given donor site area


❖Full thickness autograft


	
	
It consist of full thickness of the skin, with
little or none of the subcutaneous tissue


❖Pedicle flaps


	
	
Consisting of full thickness of the skin and
the subcutaneous tissue, attached by tissue
through which it receives its blood supply
❖Epithelial graft


	
	
Transplantation of epithelial tissue


Cultured epithelial autograft (CEA)


➢Provides permanent coverage of large wound


➢This involves biopsy of the patient’s skin in a
unburned area. keratinocytes are isolated, an
epithelial cells are cultured in a laboratory.


Patient have longer hospital stay and higher
hospital cost


Therefore, CEA is very limited for patients whose
donor sites are limited
PAIN MANAGEMENT


• Administer morphine sulfate or meperidine (Demerol), as
prescribed, by the IV route.


• Avoid IM or SC routes because absorption through the soft tissue
is unreliable when hypovolemia and large fluid shifts are
occurring.


• Avoid administering medication by the oral route, because of the
possibility of GI dysfunction.


• Medicate the client prior to painful procedures.


NUTRITIONAL SUPPORT


• Essential to promote wound healing and prevent infection.


• Maintain nothing by mouth (NPO) status until the bowel sounds
are heard; then advance to clear liquids as prescribed.


• Nutrition may be provided via enteral tube feeding, or peripheral
parenteral nutrition.


• Provide a diet high in protein, carbohydrates, fats and vitamins.
Rehabilitation phase


	
	
Rehabilitation begins immediately after the
burn has occurred and often extends for years
after injury


oPsychological support


Complications in rehabilitation phase


oNeuropathies


Interventions


• Assess peripheral pulses and sensation


• Prevent edema and pressure by elevation


• Assess splints


• Consult OT and PT
o Heterotopic ossification


Intervention


• Perform gentle range of motion exercise


o Hypertrophic scaring


Intervention


• Keep the skin soft


• Massage


o Contractures


Intervention


• Perform gentle range of motion exercise


• Consult OT and PT
oWound breakdown


Interventions


• Teach patient about importance of nutrition


• Protect wound from pressure and forces


oComplex regional pain syndrome


Interventions


• Provide adequate pain management


• Consult OT and PT


• Promote gentle motion of affected
extremities
Actual:
•Impaired gas exchange


•Ineffective tissue perfusion
Potential:
• Risk for Infection


•Risk for Post traumatic stress
	
disorder (PTSD)
Interventions
BURN INTERVENTIONS
• MAINTAIN AIRWAY


• FLUID RESUSCITATION


• RELIEVE PAIN


• PREVENT INFECTION


• PROVIDE NUTRITION


• PREVENT STRESS ULCERATION


• PROVIDE PSYCHOLOGIC SUPPORT


• PREVENT CONTRACTURE
ASSIGNMENT ON NURSING PROCESS
:

• Care of the Patient During the Emergent/
Resuscitative Phase of Burn Injury
 

• CARE OF THE PATIENT DURING THE
ACUTE PHASE
 

• CARE OF THE PATIENT DURING THE
REHABILITATION PHASE
The END
• Questions?


• Comments


• Criticisms


• Slide Remarks


• If not thank You

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Burn

  • 1. Prepared By: Justin V Sebastian, MSc N, RN, PhD Scholar
  • 3. • This system is divided into: 1- skin 2- hair 3- glands 4- nails 5- nerve endings Skin Skin is an organ because it consists of different tissues that are joined to perform a specific function. Largest organ of the body in surface area and weight. Dermatology is the medical specialty concerning the diagnosing and treatment of skin disorders.
  • 4.
  • 5. Anatomy (structure) Epidermis (thinner outer layer of skin) Dermis (thicker connective tissue layer) Hypodermis (subcutaneous layer or Sub-Q) Muscle and bone Physiology (function) 1- Protection - physical barrier that protects underlying tissues from injury, UV light and bacterial invasion.
  • 6. 2- Regulation of body temperature - high temperature or strenuous exercise; sweat is evaporated from the skin surface to cool it down. - vasodilatation (increases blood flow) and vasoconstriction (decrease in blood flow) regulates body temp. 3-Sensation - nerve endings and receptor cells that detect stimuli to temp, pain, pressure and touch.
  • 7. 4- Excretion - sweat removes water and small amounts of salt, uric acid and ammonia from the body surface 5- Blood reservoir - dermis houses an extensive network of blood vessels carrying 8-10% of total blood flow in a resting adult. 6- Synthesis of Vitamin D (cholecalciferol) -UV rays in sunlight stimulate the production of Vit. D. Enzymes in the kidney and liver modify and convert to final form; calcitriol (most active form of Vit. D.) Calcitriol aids in absorption of calcium from foods and is considered a hormone.
  • 8. Definitions 
 1. A burn occurs when there is an injury to the tissues of the body caused by heat, chemicals, electric current, or radiation lewis’s The effects are influenced by • The temperature of the burning agent • Duration of contact time • Type of tissue that injured
  • 9. 2, Burns are a form of traumatic injury caused by thermal, electrical, chemical, or radioactive agents. Lippincott 3, Burns are caused by transfer of energy from a heat source to the body Brunner and suddarth’s The depth of injury depend on: • Temperature of the burning agent • Duration of contact with it
  • 10. Incidence • Males have greater then twice the chance of burn injury than females. • The most frequent age group for contact burns is between 20 to 40 years of age. • The national fire protection association reports 4,000 fire and burn deaths each year. Of these, 3500 deaths occur from residential fires and the remaining 500 from other sources such as motor vehicle crashes, scalds, or electrical and chemical sources.
  • 11. Etiology and types 1. Thermal burns may result from any external heat source (flame, liquids, solid objects, or gases). 2 , Radiation burns most commonly result from prolonged exposure to solar ultraviolet radiation (sunburn) but may result from prolonged or intense exposure to other sources of ultraviolet radiation (eg, tanning beds) or from exposure to sources of x-ray or other non solar radiation.
  • 12. 3, Chemical burns may result from strong acids, strong alkalis (e.g, lye, cement), phenols, cresols, mustard gas, or phosphorus. Skin and deeper tissue necrosis due to these agents may progress over several hours. 4, Smoke and inhalation injury Result from the inhalation of hot air or noxious chemicals and can cause damage to the tissues of the respiratory tract. There are three types of inhalation injury Carbon monoxide poisoning Inhalation injury above the glottis Inhalation injury below the glottis
  • 13. 5, Electrical burns result from the electrical generation of heat; they may cause extensive deep tissue damage despite minimal apparent cutaneous injury. 6, Cold injury A general term for an injury induced by low ambient temperatures ( eg. Frostbit), which may be accompanied by tissue freezing. Risk groups includes infants, young children, elderly, alcohol use
  • 14. Common places and causes of burn injury ❖ Occupational hazards • Tar • Chemicals • Hot metals • Steam pipes • Combustible fuels • Fertilizers/pesticides • Electricity from power lines • Spark from live electric sources ❖ Home and recreational hazards • Pressure cookers • Microwaved food • Hot water heaters • Defective wiring • Radiators • Carelessness with cigarettes or matches • Storage of flammables ( e.g.,kerosene)
  • 16. Non Modifiable Risk Factors •Age: Children under 4 who are poorly supervised are at particular risk. 
 •Gender: Males are more than twice as likely to suffer burn injuries.
  • 17. Modifiable Risk Factor s •Careless smoking: Cigarettes are the leading cause of house fires. •Absent or nonfunctioning smoke detectors: The presence of a functioning smoke detector decreases risk of death by fire by 60 percent. •Use of wood stove s •Exposed heating sources or electrical cords •Unsafe storage of flammable or caustic materials •Water heaters set above 120°F •Microwave heated foods and containers •Substandard or older housing •Substance abuse: Use of alcohol and illegal drugs increases risk.
  • 18. Classification of burn in jury 1. Depth of burn 2. Extent of burn 3. Location of burn
  • 19. Assessment of depth of Burn Injury Takes several weeks to heal. Scarring may occur. Takes several weeks to heal. Scarring may occur. Superficial: Pink or red; blisters form (vesicles); weeping, edematous, elastic. Superficial layers of skin are destroyed; wound moist and painful. Deep dermal: Mottled white and red: edematous reddened areas blanch on pressure. May be yellowish but soft and elastic – may or may not be sensitive to touch; sensitive to cold air. Hair does not pull out easily Second degree (deep partial- thickness) In about 5 days, epidermis peels, heals spontaneously. Itching and pink skin persist for about a week. No scarring. Heals spont. If it does not become infected w/in 10 days - 2 weeks. Pink to red: slight edema, which subsides quickly. Pain may last up to 48 hours. Relieved by cooling. Sunburn is a typical example. First Degree (superficial partial-thickness) Reparative Process Assessment of Extent Extent / Degree
  • 20. Eschar must be removed. Granulation tissue forms to nearest epithelium from wound margins or support graft. For areas larger than 3-5 cm, grafting is required. Expect scarring and loss of skin function. Area requires debridement for formation of granulation tissue, and grafting. Destruction of epithelial cells – epidermis and dermis destroyed Reddened areas do not blanch with pressure. Not painful; inelastic; coloration varies from waxy white to brown; leathery devitalized tissue is called eschar. Destruction of epithelium, fat, muscles, and bone. Third degree (full- thickness) Reparative Process Assessment of Extent Extent / Degree Assessment of depth of Burn Injury AGE AND GENERAL HEALTH • Mortality rates are higher for children < 4 y.o, particularly those < 1 y.o., and for clients over the age of 60 years. • Debilitating disorders, such as cardiac, respiratory, endocrine, and renal, negatively influence the client’s response to injury and treatment. • Mortality rate is higher when the client has a preexisting disorder at the time of the burn injury
  • 21.
  • 22. Assessment of extent of burn injury Various methods are used to estimate the TBSA affected by burns; among them are 1.Rule of nines Rule of Nines for Establishing Extent of Body Surface Burned Anatomic Surface • % of total body surface • Head and neck =9% • Anterior trunk =18% • Posterior trunk =18% • Arms, including hands =9% each • Legs, including feet =18% each • Genitalia/Perineum =1% 9 18 9 18 9 18 1
  • 23. 2, Lund and browder method
  • 25. Assessment of location of burn injury The severity of the burn injury is related to location of the burn wound • Burns to the face and neck and circumferential burns to the chest/back • Burns of the hands, feet, joints, and eyes • Burns to the ears and nose • Burns to the buttocks and genitalia • Circumferential burns to the extremities
  • 29.
  • 30. Heat causes coagulation necrosis of skin and subcutaneous tissue Release of vasoactive peptides Altered capillary permeability Loss of fluid severe hypovolemia
  • 31. Decreased cardiac decreased myocardial output function Decreased renal oliguria blood flow Altered pulmonary resistant and pulmonary edema Infection SIRS MODS
  • 32.
  • 33. Clinical features ❖Cardiovascular alterations o Hypovolemia o Decreased CO o Burn shock o Increase peripheral resistance o Anemia o Increased hematocrit o Decreased platelets o Diuresis
  • 34. ❖Fluid and electrolyte alterations o Edema o Ischemia o Hyponatremia o Hyperkalemia o Hypokalemia ❖Pulmonary alterations o Hypoxia o Loss of ciliary action o Hyper secretion o Mucosal edema o Bronchospasm
  • 35. o Atelectasis o Restrictive pulmonary excursion o Decreased lung compliance o Decreased arterial O2 level o Respiratory acidosis o Pulmonary complications such as airway obstruction, ulceration, pulmonary edema, respiratory failure, ARDS and pneumonia can develop.
  • 36. ❖Renal alterations o Altered renal function o Hemoglobin in the urine o Myoglobin in the urine o Acute tubular necrosis o Renal failure ❖Immunological alterations o Infection and sepsis ❖Thermoregulatory alterations o Hypothermia
  • 37. ❖Gastrointestinal alterations o Paralytic ileus o Curling’s ulcer o Gastric bleeding o Infection
  • 39. •A detailed history and physical examination is the first step. The physician will evaluate the type, duration, and timing of the burn; the burn location and severity; and associated dehydration, disfigurement, and infection. •Fires in enclosed spaces should raise the suspicion for smoke–inhalation injury.
  • 40. •Routine blood work for a patient with a burn injury includes a complete blood count, platelet count, clotting studies, l i v e r f u n c t i o n s t u d i e s , a n d carboxyhemoglobin, electrolyte, blood urea nitrogen, glucose and creatinine levels. •Urinalysis may reveal myoglobinuria and hemoglobinuria. •If pt. is electric burn, he’ll also need an electrocardiogram. •Chest x-rays and arterial blood gas levels allow the evaluation of alveolar function. Fiberoptic bronchoscopy shows the condition of the trachea and bronchi.
  • 42. Phases of burn care I. Emergent/resuscitative phase II.Acute/ intermediate phase III.Rehabilitation phase
  • 43. Emergent/resuscitative phase 
 From the onset of injury to completion of fluid resuscitation. Priorities oFirst aid oPrevention of shock oPrevention of respiratory distress oDetection and treatment of injuries oWound assessment and initial care
  • 44. On-the-scene care oExtinguish the flames oCool the burn oRemove restrictive objectives oCover the wound oIrrigate chemical burn
  • 45. ESCHAROTOMY • A lengthwise incision is made through the burn eschar to relieve constriction and pressure and to improve circulation • Performed for circulatory compromise resulting from circumferential burns • After escharotomy, assess pulses, color, movement, and sensation of affected extremity and control any bleeding with pressure • Apply topical antimicrobial agents as prescribed
  • 46. Medical management ❖The patient transported to the nearest ED ❖Initial priorities in the ED remain ABC and C spine control ❖For mild pulmonary injury 100% humidified O2 is administered ❖The patient is encouraged to cough ❖For more severe situations; administer bronchodilator and mucolytic agents ❖If edema of airway develops, ET intubation may be necessary ❖All clothing and jewellery are removed
  • 47. Medical management ( cont..) ❖For chemical burn flushing of the exposed area should be done ❖Validate an account of the burn scenario provided by the patient or witnesses ❖A large bore ( 16 or 18 gauge) IV catheter should be inserted ❖If burn exceeds 20% to 25% TBSA, a NG tube inserted ❖Clean technique is maintained while assessing and treating the burn wounds ❖Careful attention is paid to keeping the patient warm
  • 48. Medical management ( cont..) ❖Photograph may be taken of the burn area initially and periodically throughout treatment ❖Clean sheets are placed under and over the patient ❖An indwelling urinary catheter is inserted ❖Baseline data should be obtained ( such as height, weight, ABG, electrolyte, blood alcohol, drug panel, chest X rays etc..) ❖If the patent has an electrical burn, a baseline ECG ❖Tetanus prophylaxis ❖Attends the patient’s and family’s psychological needs
  • 49. Medical management ( cont..) ❖Transfer to a burn centre Management of fluid loss and shock Observation o Generalized dehydration o Reduction of blood volume o Decreased urinary output o Potassium excess o Sodium deficit o Metabolic acidosis o Elevated hematocrit
  • 50. FLUID REQUIREMENT • Indications: - Adults with burns involving more than 15% - 20% TBSA - Children with burns involving more than 10-15% TBSA - Patients with electrical injury, the elderly, or those with cardiac or pulmonary disease and compromised response to burn injury • The amount of fluid administered depends on how much intravenous fluid per hour is required to maintain a urinary output of 30 - 50 ml/hr • Successful fluid resuscitation is evidenced by: - Stable vital signs - Palpable peripheral pulse - Adequate urine output - Clear sensorium • Urinary output is the most common and most sensitive assessment parameter for cardiac output and tissue perfusion • If the Hgb and Hct levels decrease or if the urinary output exceeds 50ml/hr, the rate of IV fluid administration may be decreased • Generally, a crystalloid (Ringer’s lactate) solution is used initially. Colloid is used during the 2nd day (5% albumin, plasmate)
  • 51. ½ in 1st 8 hours ½ in next 16 hours crystalloid only (lactated Ringer’s) PARKLAND (Baxter) 4ml/kg/% BSA for 24hr period ½ in 1st 8 hours ½ in next 16 hours crystalloid, colloid D5W maintenance BROOKE 2ml/kg/% BSA + 2000ml/ 24hr (maintenance) Infusion Rate Solution Formula Brooke and Parkland (Baxter) Fluid Resuscitation Formulas for 1st 24hrs after a Burn Injury
  • 52. PARKLAND FORMULA Example: Patient’s weight: 70 kg; % TBSA burn: 80% 1st 24 hours: 4ml x 70kg x 80% TBSA = 22,400ml of lactated Ringer’s • 1st 8 hours = 11,200 ml or 1400ml/hour • 2nd 16 hours = 11,200 ml or 700ml/hour BROOKE FORMULA 2nd 24 hours: 0.5ml colloid x weight in kg x TBSA + 2000ml D5W run concurrently over the 24 hour period 0.5ml x 70kg x 80% = 2800 ml colloid + 2000 ml D5W = 117 ml colloid/hour + 84 ml D5W/ hour
  • 53. Acute/ intermediate phase From beginning of diuresis to near completion of wound closure ( 48 – 72 hrs ) ❖Observation oDecreased hematocrit oIncreased urinary output oSodium deficit oPotassium deficit
  • 54. Medical management (cont..) ❖Bronchial washing ❖Remove ET tube as soon as possible ❖Cautious administration of fluid and electrolyte ❖Blood components are administered as needed ❖Central venous, peripheral arterial or pulmonary artery catheters may be required
  • 55. ❖Infection prevention ❖Wound cleaning ❖Topical antibacterial therapy ❖Wound dressing ❖Wound debridement ❖Wound grafting ❖Pain management ❖Nutritional support
  • 56. Medical management (cont..) ❖Infection prevention ➢Early enteral feeding ➢Aseptic technique is used when caring directly for burn wound ➢Gowns and gloves are worn by all caregivers and visitors ➢Hand hygiene is used before and after leaving the patient room ➢Constant monitoring and observation of the wound ➢Tissue specimen may be obtained for culture to monitor colonization ➢Systemic antibiotics are administered when there is documented sepsis
  • 57. Medical management (cont..) ❖Wound cleaning ➢Hydrotherapy ➢The temperature of the water is maintained at 37.8 C ➢During the bath, the patient is encouraged to be active as possible ➢At the time of wound cleaning, all skin is inspected ➢Intact blisters should be left alone and debrided only if they rupture or break ➢When nonviable loose skin is removed, aseptic condition must be established ➢After the wounds are cleaned, they are gently patted dry, and the prescribed method of wound care is performed
  • 58. ❖TOPICALANTIMICROBIAL THERAPY Silver sulfadiazine • Most widely used agent and least common incidence of side effects • May cause transient leukopenia that disappears 2-3 days of treatment • Use with either open treatment, light or occlusive dressings • Applied once or twice daily after thorough wound cleansing Mafenide acetate 10% cream or 5% solution (Sulfamylon) • Painful during and for a while after application • May cause metabolic acidosis, not used if >20% TBSA • Cream must be reapplied 12 hours to maintain therapeutic effectiveness • Solution concentration is maintained with bulky wet dressings, rewet every 2-4 hours Silver nitrate (0.5% solution) • Stains everything including normal skin brown or black • Monitor electrolyte balance carefully
  • 59. Medical management (cont..) ❖Wound dressing ➢After wound cleaning, prescribed topical agent is applied; the wound is then covered with dressings ➢A light dressing is applied over the areas of splint, joints ➢Circumferential dressing should be applied to distally to proximally ➢The fingers and toes should be wrapped individually ➢Burns to the face may be left open/a light dressing can be applied ➢Occlusive dressing may be used over new skin graft to protect the graft. Ideally, these dressing remain in place for 3 to 5 days
  • 60.
  • 61. Medical management (cont..) ❖Wound debridement Goals • Removal of tissue contaminated by bacteria and foreign bodies, thereby protecting the patient from invasion of bacteria • Removal of devitalized tissue or burn eschar in preparation for grafting and wound healing There are 4 types ➢Natural debridement ➢Mechanical debridement ➢Chemical debridement ➢Surgical debridement
  • 62. Natural debridement The dead tissue separates from the underlying viable tissue spontaneously.
  • 63. Mechanical debridement Involves the use of surgical scissors, scalpels, and forceps to separate and remove the eschar.
  • 64. Chemical debridement Use of topical enzymatic debridement agents to remove eschar.
  • 65.
  • 66. Surgical debridement It is an operative procedure involving either primary excision of the full thickness of the skin or down to the burned skin layers gradually down to freely bleeding, viable tissue.
  • 67. ❖Wound grafting The purpose of wound grafting are: oTo decrease the risk of infection oTo prevent further loss of protein, fluids and electrolytes oTo minimize heat loss through evaporation The main areas for skin grafting include the face ( for cosmetic and psychological reasons); functional areas such as the hands and feet; and areas that involves joints. Methods ❖Biologic ❖Biosynthetic and synthetic ❖Autologous (autograft)
  • 68. ❖Biological dressing( homograft and heterograft) ➢It provide temporary wound coverage and protect the granulation tissue until auto-grafting is possible ➢Once the biological dressing appears to be adhering to the granulating surface with minimal underlying exudation , the patient is ready for an autologous skin graft ➢They decreases pain by protecting the nerve endings and are effective barrier against water loss and entry of bacteria ➢These dressing often require fewer dressing changes ➢Homograft are skin obtained from living or deceased humans ➢Heterograft consist of skin taken from animals ( usually pigs)
  • 69. ❖Biosynthetic and synthetic dressings ➢A widely used synthetic dressing is Biobrane ➢It is semitransparent and sterile ➢Like biological dressing, Biobrane protects the wound from fluid loss and bacterial invasion ➢When the Biobrane dressing is adheres to the wound, the wound remain stable ➢It can remain in place until spontaneous epithelialisation and wound healing occur ➢It can be laid on top of a wide meshed autograft
  • 70.
  • 71. Biosynthetic and synthetic dressings (cont..) Skin substitutes It is believed that skin substitutes enhance the healing process of an open wound when autologous skin is unavailable or limited for use
  • 72. Biosynthetic and synthetic dressings (cont..) Integra Artificial Skin ➢It is composed of two main layers ➢The epidermal layer consist of silicone, act as a bacterial barrier and prevents water loss from the dermis ➢The dermal layer consist of animal collagen. It allows migration of fibroblast and capillaries in to the material
  • 73. ❖Auto graft Autograft are the ideal means of covering burn wound because the graft are the patient’s own skin and therefore are not rejected by the patients immune system They can be ❖Split-thickness ❖Full thickness ❖Pedicle flaps ❖Epithelial graft
  • 74. ❖Split thickness autograft In this skin can be expanded by meshing so that they cover 1.5 to 9 times more than a given donor site area ❖Full thickness autograft It consist of full thickness of the skin, with little or none of the subcutaneous tissue ❖Pedicle flaps Consisting of full thickness of the skin and the subcutaneous tissue, attached by tissue through which it receives its blood supply
  • 75. ❖Epithelial graft Transplantation of epithelial tissue Cultured epithelial autograft (CEA) ➢Provides permanent coverage of large wound ➢This involves biopsy of the patient’s skin in a unburned area. keratinocytes are isolated, an epithelial cells are cultured in a laboratory. Patient have longer hospital stay and higher hospital cost Therefore, CEA is very limited for patients whose donor sites are limited
  • 76.
  • 77. PAIN MANAGEMENT • Administer morphine sulfate or meperidine (Demerol), as prescribed, by the IV route. • Avoid IM or SC routes because absorption through the soft tissue is unreliable when hypovolemia and large fluid shifts are occurring. • Avoid administering medication by the oral route, because of the possibility of GI dysfunction. • Medicate the client prior to painful procedures. NUTRITIONAL SUPPORT • Essential to promote wound healing and prevent infection. • Maintain nothing by mouth (NPO) status until the bowel sounds are heard; then advance to clear liquids as prescribed. • Nutrition may be provided via enteral tube feeding, or peripheral parenteral nutrition. • Provide a diet high in protein, carbohydrates, fats and vitamins.
  • 78. Rehabilitation phase Rehabilitation begins immediately after the burn has occurred and often extends for years after injury oPsychological support Complications in rehabilitation phase oNeuropathies Interventions • Assess peripheral pulses and sensation • Prevent edema and pressure by elevation • Assess splints • Consult OT and PT
  • 79. o Heterotopic ossification Intervention • Perform gentle range of motion exercise o Hypertrophic scaring Intervention • Keep the skin soft • Massage o Contractures Intervention • Perform gentle range of motion exercise • Consult OT and PT
  • 80. oWound breakdown Interventions • Teach patient about importance of nutrition • Protect wound from pressure and forces oComplex regional pain syndrome Interventions • Provide adequate pain management • Consult OT and PT • Promote gentle motion of affected extremities
  • 81.
  • 83. Potential: • Risk for Infection •Risk for Post traumatic stress disorder (PTSD)
  • 85. BURN INTERVENTIONS • MAINTAIN AIRWAY • FLUID RESUSCITATION • RELIEVE PAIN • PREVENT INFECTION • PROVIDE NUTRITION • PREVENT STRESS ULCERATION • PROVIDE PSYCHOLOGIC SUPPORT • PREVENT CONTRACTURE
  • 86. ASSIGNMENT ON NURSING PROCESS : • Care of the Patient During the Emergent/ Resuscitative Phase of Burn Injury • CARE OF THE PATIENT DURING THE ACUTE PHASE • CARE OF THE PATIENT DURING THE REHABILITATION PHASE
  • 87. The END • Questions? • Comments • Criticisms • Slide Remarks • If not thank You