4. DEFINITION:-
it is the condition in which carbohydrates metabolism
disturbed due to insufficient secretion of insulin hormones. It is
characterized by traid features e.g, polyuria, polyphagia.
4
5. Glucose level increases. This is known as
hyperglycaemia.
Normal blood sugar level:
Fasting: 70-110mg/dl
postprandial (PP): 80-150mg/dl
5
12. • CLINICAL MANIFESTATIONS:-
In early stage ( no symptoms)
3P’s Symptoms:-
> polyuria.
> polyphagia.
> polydypsia.
Later symptoms are:-
> drowsiness.
> Blurred vision.
> Weight loss.
> Hypotension.
12
13. • PATHOPHYSIOLOGY:- Destruction of β-cells
Decrease secreation of insulin
Insulin requirement is not sufficient for bod
Disturbance in carbohydrates metabolism
increase blood sugar level (hyperglacemia)
decrease energy patient feels fatigue
weight loss increase appetite
13
14. • DIAGNOSTIC ELAVATUION:-
a) Blood Glucose level
b) Detection of complication
c) By signs & symptoms
d) History collection
14
16. • PREVENTION & CONTROL:-
prevention is depends upon three factors.
1_ Dietary management.
2_ Execrise.
3_ Insulin Therapy.
PATIENT
EXECRISE
MONETERING
NUTRITIONAL
MANAGEMENT
EDUCATION
PHARMA
THERAPY
16
17. • PHARMALOGICAL MANAGEMENT:-
>> Insulin injection. It should given before meal (half an
hours) in three times a day.
>> Hypoglycemic Drugs.
17
18. • NURSING MANAGEMENT:-
* Teach patient abouy following a prescribed meal plan.
* Demontrate & explain thoroughly the procedure for insulin self-
injection.
* Review dosage and time of injc.
18
19. • NURSING DIAGNOSIS:-
* Fluid volume deficit rltd to hyperglycemia nd restricted intake.
* Risk for unstable blood glucose level rltd to deficiency of insulin.
* Self care deficit rltd to disease process.
* Activity intolerance rltd to poor glucose.
*Altered daily living pattern rltd to fatigue and weakness.
19