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MD SHAHID
 Ataxia is a neurological sign consisting of
lack of voluntary coordination of muscle
movements that includes gait abnormality.
Ataxia is a non-specific clinical manifestation
implying dysfunction of the parts of the
nervous system that coordinate movement,
such as the cerebellum.
 Age
 Sex
 Presenting complaints
 History of presenting illness
 Past history
 Family history
 Personal history
 Drug intake
Age of onset:
Childhood- Congenital, Metabolic, Infectious,
Hereditary ataxias
Adult- Sporadic ataxia, hereditary ataxia.
Course of illness:
Acute- Metabolic, toxic, infectious, inflammatory
Chronic- Genetic, degenerative, tumour,
Family history- Consanguinity
Personal history- Alcohol and drug
abuse, smoking.
Drug intake- Phenytoin, barbiturates,
lithium,immunosuppressants etc.
Look for the signs of cerebellar lesion:
 Involvement of anterior
lobe(paleocerebellum)
 Flail joints: lack of stability of joint.
 Pendular knee jerk: tapping of patellar
tendon produces oscillating movements of
leg.
 Ataxia: Inability in maintaining balance while
walking.
 Involvement of posterior lobe(neocerebellum)
Dysmetria –Inability to measure the
distance for reaching the intended
target.
Finger nose test
Finger finger test
Intensional tremor
Dysdiadochokinesia- Inability to do
alternate opposite movement rapidly like
supination and pronation.
Dysarthria/ scanning of speech-
speech is prolonged and with pause.
Rebound phenomenon- patient is unable
to check the action of agonist by corresponding
antagonist muscle.
 Involvement of flocculo-nodular
lobe(Archiocerebellum)
 Trunkal ataxia- Patient is unsteady while standing
and staggering while walking.
Test- Romberg test
 Nystagmus- Involuntary to and fro oscillatory
movement of eyeball while looking on either side.
 Ataxia is generally not curable but we just
ease the symptoms to make the patients life
better.
 Treat the underlying cause.
Ataxia

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Ataxia

  • 2.  Ataxia is a neurological sign consisting of lack of voluntary coordination of muscle movements that includes gait abnormality. Ataxia is a non-specific clinical manifestation implying dysfunction of the parts of the nervous system that coordinate movement, such as the cerebellum.
  • 3.
  • 4.  Age  Sex  Presenting complaints  History of presenting illness  Past history  Family history  Personal history  Drug intake
  • 5. Age of onset: Childhood- Congenital, Metabolic, Infectious, Hereditary ataxias Adult- Sporadic ataxia, hereditary ataxia. Course of illness: Acute- Metabolic, toxic, infectious, inflammatory Chronic- Genetic, degenerative, tumour,
  • 6. Family history- Consanguinity Personal history- Alcohol and drug abuse, smoking. Drug intake- Phenytoin, barbiturates, lithium,immunosuppressants etc.
  • 7. Look for the signs of cerebellar lesion:  Involvement of anterior lobe(paleocerebellum)  Flail joints: lack of stability of joint.  Pendular knee jerk: tapping of patellar tendon produces oscillating movements of leg.  Ataxia: Inability in maintaining balance while walking.
  • 8.  Involvement of posterior lobe(neocerebellum) Dysmetria –Inability to measure the distance for reaching the intended target. Finger nose test Finger finger test Intensional tremor
  • 9. Dysdiadochokinesia- Inability to do alternate opposite movement rapidly like supination and pronation. Dysarthria/ scanning of speech- speech is prolonged and with pause. Rebound phenomenon- patient is unable to check the action of agonist by corresponding antagonist muscle.
  • 10.  Involvement of flocculo-nodular lobe(Archiocerebellum)  Trunkal ataxia- Patient is unsteady while standing and staggering while walking. Test- Romberg test  Nystagmus- Involuntary to and fro oscillatory movement of eyeball while looking on either side.
  • 11.
  • 12.
  • 13.
  • 14.
  • 15.
  • 16.
  • 17.
  • 18.  Ataxia is generally not curable but we just ease the symptoms to make the patients life better.  Treat the underlying cause.