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CATH PRESENTATION
Chairperson
Prof. Dr. M A muqueet
Presenter
Dr. Md.Al-Amin
Phase B Resident, UCC,BSMMU
This week
• Total - 85 cases
• CAG – 51
• PTCA – 31
• TPM -2
• PPM - 1
Particulars of the patient
• Mrs .Moni Mala
• Age:50 year
• Housewife
• Married
• Rayganj, Sirajganj
• Date of admission: 31/12/21
• Date of CAG with PCI: 03/01/22
• He has been diagnosed as a case of
-Chronic Coronary Syndrome (Grade -2)
-Old MI(anteroseptal)
-Systemic Hypertension
-Type-2 DM
On general examination
• Co-operative,
• Decubitus on choice
• Pulse- 88 bpm, regular, normal volume and character
• BP- 120/80mmHg, no postural drop
• Jvp- not raised
• Temp – normal
• Edema - absent
• Other parameters are normal
Precordium
Inspection- No Visible impulse on apical region
No scar mark
Palpation-
-Apex beat is found on the left 5th intercostal space just
medial to midclavicular line
- There is no thrill
Auscultaion:
-First and second heart sounds are audible in all the
auscultatory areas
-No murmur or added sounds
- Clear lung bases
ECG on admission(31.12.21)
Investigation (31.12.21)
CK-MB 6.8 u/l
hs-Troponin -I 67.6 pg/ml
RBS 9.5 mmol/l
S.Craetinine 0.84mg/dl
S. electrolytes Na-143 ,K-3.9 ,Cl- 102
S.SGPT 33 U/l
HbA1c 6.6%
Echocardiography Not done
Investigations (01.01.22)
investigations findings
CBC HB 12 gm/dl
ESR 12 mm in 1st hr
WBC 7x109
Platelet 220x109
BT 3min
CT 6min
RBS 11.1mmol/l
S creatinine 0.82 mg/dl
S electrolytes Normal
HBsAg,Anti-HCV,Anti-HIV Negative
VDRL Non-reactive
Blood group "B" Positive
CAG
Post-PCI ECG(03.01.22)
Left Main Coronary Artery Disease
(LMCA)
Introduction
• Clinically significant LMCA disease is found in 3- 5%
of all patients undergoing CAG and 10- 30% of all
patients undergoing CABG
• LMCA diseas is associated with high morbidity and
mortality
• DES ,PCI for LMCA lesion has become technically
feasible and associated with long term favorable
clinical outcomes
Anatomy and physiology
• LMCA arises from left aortic sinus just below the sino-
tubular junction of the aortic root
• In aproximately, two-thirds of cases,it bifurcates into
LAD and LCX.In one-third cases it trifurcates
intoLAD,LCX and Ramus Intermedius
• LMCA supplies about 75% of the left ventricle
• LMCA has an average lentgh 10.8mm ,diameter 4.9mm
and with an average branches angle 86.7°
• Anatomically LMCA is devided into 3 regions,the
ostium,midshaft and distal bifurcation
• Histologycally,ostial portion resembles to aorta,being
rich in smooth and elastic fibers
• Distal bifurcation part of LMCA is the most common
site of atheromatous plaque formation(low shear flow)
than bifurcation carina
• In minimal LMCA disease,atheromatous plaque type is
intimal thickening
• Significant lesion shows complex plaque,fibroatheroma
with thin cap,surface rupture,fissure and intra-plaque
haemorrhage
Definition of significant LMCA stenosis
• Angiographic LMCA diameter <50%
• Fractional flow reserve (FFR) <0.80
• IVUS LMCA minimal luminal area (MLA)
<4.9mm
PCI of LMCA
• PCI of LM ostial and shaft lesion can
beperformed safely ,and is associated with
excellent short term and long term outcomes
• PCI to LM bifurcation lesion is challenging and is
associated with high rate of adverse clinical
evevents
• Stenting strategy in LM bifurcation lesion
depends on several clinical and anatomical
factors
Multi-Vessel Disease PCI
Cd presentation  (1)
Cd presentation  (1)

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Cd presentation (1)

  • 1. CATH PRESENTATION Chairperson Prof. Dr. M A muqueet Presenter Dr. Md.Al-Amin Phase B Resident, UCC,BSMMU
  • 2. This week • Total - 85 cases • CAG – 51 • PTCA – 31 • TPM -2 • PPM - 1
  • 3. Particulars of the patient • Mrs .Moni Mala • Age:50 year • Housewife • Married • Rayganj, Sirajganj • Date of admission: 31/12/21 • Date of CAG with PCI: 03/01/22
  • 4. • He has been diagnosed as a case of -Chronic Coronary Syndrome (Grade -2) -Old MI(anteroseptal) -Systemic Hypertension -Type-2 DM
  • 5. On general examination • Co-operative, • Decubitus on choice • Pulse- 88 bpm, regular, normal volume and character • BP- 120/80mmHg, no postural drop • Jvp- not raised • Temp – normal • Edema - absent • Other parameters are normal
  • 6. Precordium Inspection- No Visible impulse on apical region No scar mark Palpation- -Apex beat is found on the left 5th intercostal space just medial to midclavicular line - There is no thrill Auscultaion: -First and second heart sounds are audible in all the auscultatory areas -No murmur or added sounds - Clear lung bases
  • 8. Investigation (31.12.21) CK-MB 6.8 u/l hs-Troponin -I 67.6 pg/ml RBS 9.5 mmol/l S.Craetinine 0.84mg/dl S. electrolytes Na-143 ,K-3.9 ,Cl- 102 S.SGPT 33 U/l HbA1c 6.6% Echocardiography Not done
  • 9. Investigations (01.01.22) investigations findings CBC HB 12 gm/dl ESR 12 mm in 1st hr WBC 7x109 Platelet 220x109 BT 3min CT 6min RBS 11.1mmol/l S creatinine 0.82 mg/dl S electrolytes Normal HBsAg,Anti-HCV,Anti-HIV Negative VDRL Non-reactive Blood group "B" Positive
  • 10. CAG
  • 12. Left Main Coronary Artery Disease (LMCA)
  • 13. Introduction • Clinically significant LMCA disease is found in 3- 5% of all patients undergoing CAG and 10- 30% of all patients undergoing CABG • LMCA diseas is associated with high morbidity and mortality • DES ,PCI for LMCA lesion has become technically feasible and associated with long term favorable clinical outcomes
  • 14. Anatomy and physiology • LMCA arises from left aortic sinus just below the sino- tubular junction of the aortic root • In aproximately, two-thirds of cases,it bifurcates into LAD and LCX.In one-third cases it trifurcates intoLAD,LCX and Ramus Intermedius • LMCA supplies about 75% of the left ventricle • LMCA has an average lentgh 10.8mm ,diameter 4.9mm and with an average branches angle 86.7° • Anatomically LMCA is devided into 3 regions,the ostium,midshaft and distal bifurcation
  • 15. • Histologycally,ostial portion resembles to aorta,being rich in smooth and elastic fibers • Distal bifurcation part of LMCA is the most common site of atheromatous plaque formation(low shear flow) than bifurcation carina • In minimal LMCA disease,atheromatous plaque type is intimal thickening • Significant lesion shows complex plaque,fibroatheroma with thin cap,surface rupture,fissure and intra-plaque haemorrhage
  • 16.
  • 17. Definition of significant LMCA stenosis • Angiographic LMCA diameter <50% • Fractional flow reserve (FFR) <0.80 • IVUS LMCA minimal luminal area (MLA) <4.9mm
  • 18.
  • 19.
  • 20.
  • 21.
  • 22. PCI of LMCA • PCI of LM ostial and shaft lesion can beperformed safely ,and is associated with excellent short term and long term outcomes • PCI to LM bifurcation lesion is challenging and is associated with high rate of adverse clinical evevents • Stenting strategy in LM bifurcation lesion depends on several clinical and anatomical factors
  • 23.
  • 24.