The data is Quantitative as there are numbers , and its an observational study, where they studies
about the H.pylori in the cause of ulceration.
2. From table -II, we see that among the 22 patients having Ulcer, 18 have infection of H.pylori.
Out of 16, 8 were with infection.
Gastritis and Duodenitis.
Out of 22, 18 patients with gastric ulcers had bacteria, and four had both the ulcers, and all were
Helicobacter positive. With gastric ulcer 77% had bacteria, and with duodenal ulcer 100% had
bacteria. 4 patients with ulcers became negative for bacteria is significant as they had taken
NSAIS drugs which are known to cause ulcers.
Although gastric colonization with H.pylori, induces histologic gastritis in all infected
individuals, only a small portion of them develop any significant sign and symptom for this
colonization. It sestimated that H. pylori, infected patients have a 10-20% lifetime risk of
developing ulcer disease. The risk of development of these diseases in the presence of H.pylori,
depends on a variety of bacteria, host, and environmental factors, that relate to the pattern and
severity of gastritis. Thus a patient can show normal examination.
Solution
The data is Quantitative as there are numbers , and its an observational study, where they studies
about the H.pylori in the cause of ulceration.
2. From table -II, we see that among the 22 patients having Ulcer, 18 have infection of H.pylori.
Out of 16, 8 were with infection.
Gastritis and Duodenitis.
Out of 22, 18 patients with gastric ulcers had bacteria, and four had both the ulcers, and all were
Helicobacter positive. With gastric ulcer 77% had bacteria, and with duodenal ulcer 100% had
bacteria. 4 patients with ulcers became negative for bacteria is significant as they had taken
NSAIS drugs which are known to cause ulcers.
Although gastric colonization with H.pylori, induces histologic gastritis in all infected
individuals, only a small portion of them develop any significant sign and symptom for this
colonization. It sestimated that H. pylori, infected patients have a 10-20% lifetime risk of
developing ulcer disease. The risk of development of these diseases in the presence of H.pylori,
depends on a variety of bacteria, host, and environmental factors, that relate to the pattern and
severity of gastritis. Thus a patient can show normal examination..
The data is Quantitative as there are numbers , and its an observati.pdf
1. The data is Quantitative as there are numbers , and its an observational study, where they studies
about the H.pylori in the cause of ulceration.
2. From table -II, we see that among the 22 patients having Ulcer, 18 have infection of H.pylori.
Out of 16, 8 were with infection.
Gastritis and Duodenitis.
Out of 22, 18 patients with gastric ulcers had bacteria, and four had both the ulcers, and all were
Helicobacter positive. With gastric ulcer 77% had bacteria, and with duodenal ulcer 100% had
bacteria. 4 patients with ulcers became negative for bacteria is significant as they had taken
NSAIS drugs which are known to cause ulcers.
Although gastric colonization with H.pylori, induces histologic gastritis in all infected
individuals, only a small portion of them develop any significant sign and symptom for this
colonization. It sestimated that H. pylori, infected patients have a 10-20% lifetime risk of
developing ulcer disease. The risk of development of these diseases in the presence of H.pylori,
depends on a variety of bacteria, host, and environmental factors, that relate to the pattern and
severity of gastritis. Thus a patient can show normal examination.
Solution
The data is Quantitative as there are numbers , and its an observational study, where they studies
about the H.pylori in the cause of ulceration.
2. From table -II, we see that among the 22 patients having Ulcer, 18 have infection of H.pylori.
Out of 16, 8 were with infection.
Gastritis and Duodenitis.
Out of 22, 18 patients with gastric ulcers had bacteria, and four had both the ulcers, and all were
Helicobacter positive. With gastric ulcer 77% had bacteria, and with duodenal ulcer 100% had
bacteria. 4 patients with ulcers became negative for bacteria is significant as they had taken
NSAIS drugs which are known to cause ulcers.
Although gastric colonization with H.pylori, induces histologic gastritis in all infected
individuals, only a small portion of them develop any significant sign and symptom for this
colonization. It sestimated that H. pylori, infected patients have a 10-20% lifetime risk of
developing ulcer disease. The risk of development of these diseases in the presence of H.pylori,
depends on a variety of bacteria, host, and environmental factors, that relate to the pattern and
severity of gastritis. Thus a patient can show normal examination.
