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Electrolytes Disturbances
Sodium ,[object Object],[object Object]
[object Object],[object Object]
Distribution  ,[object Object],[object Object],[object Object]
Sodium ,[object Object],[object Object],[object Object]
Other sources of Na loss ,[object Object],[object Object],[object Object]
Hyponatraemia ,[object Object],[object Object],[object Object]
GI loss ,[object Object],[object Object],[object Object],[object Object]
Inappropriate ADH secretion ,[object Object],[object Object],[object Object]
Hyponatraemia – C. F. ,[object Object],[object Object],[object Object],[object Object]
. Investigations ,[object Object],[object Object],[object Object],[object Object]
Investigations ,[object Object],[object Object],[object Object],[object Object]
Treatment ,[object Object],[object Object],[object Object]
Hypernatraemia ,[object Object],[object Object],[object Object]
Water loss ,[object Object],[object Object],[object Object]
Hypernatraemia – C.F. ,[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object]
Treatment ,[object Object],[object Object]
Potassium ,[object Object],[object Object]
[object Object],[object Object]
[object Object],[object Object]
Potassium ,[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
Potassium ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object]
[object Object],[object Object],[object Object]
Hypokalaemia ,[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object]
Hypokalaemia . C . F. ,[object Object],[object Object],[object Object]
[object Object],[object Object]
Treatment ,[object Object],[object Object]
Hyperkalaemia ,[object Object],[object Object]
C.F ,[object Object],[object Object]
GI symptoms ,[object Object],[object Object],[object Object],[object Object]
CVS ,[object Object],[object Object],[object Object]
Treatment ,[object Object],[object Object],[object Object]
Magnesium ,[object Object],[object Object],[object Object],[object Object]
Distribution  ,[object Object],[object Object],[object Object]
Magnesium ,[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
Hypomagnesemia ,[object Object],[object Object],[object Object],[object Object],[object Object]
Causes: ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object]
Hypomagnesemia C. F. ,[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object]
Treatment ,[object Object],[object Object]
Trace elements ,[object Object],[object Object],[object Object],[object Object]
Trace elements ,[object Object],[object Object],[object Object]
Replacement of ongoing losses Composition of GI secretions 40 100 5 145 500 Biliary Fistula 120 75 5 140 700 Pancreatic Fistula 15 90 15 60 1500 Gastric Juice (Fasting) 30 10 25 10 1500 Saliva HCO 3 (mg mol/l) Cl (mg mol/l) K (mg mol/l) Na  (mg mol/l) Vol (ml) Locality
Replacement of ongoing losses Composition of GI secretions 45 90 25 120 500- 15000 Diarrhoeal Stool HCO 3 (mg mol/l) Cl (mg mol/l) K (mg mol/l) Na  (mg mol/l) Vol (ml) Locality 30 45 20 80 300 Prox.Colostomy 30 45 8 115 500 Ileostomy

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Electrolytes Disturbances

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  • 50. Replacement of ongoing losses Composition of GI secretions 40 100 5 145 500 Biliary Fistula 120 75 5 140 700 Pancreatic Fistula 15 90 15 60 1500 Gastric Juice (Fasting) 30 10 25 10 1500 Saliva HCO 3 (mg mol/l) Cl (mg mol/l) K (mg mol/l) Na (mg mol/l) Vol (ml) Locality
  • 51. Replacement of ongoing losses Composition of GI secretions 45 90 25 120 500- 15000 Diarrhoeal Stool HCO 3 (mg mol/l) Cl (mg mol/l) K (mg mol/l) Na (mg mol/l) Vol (ml) Locality 30 45 20 80 300 Prox.Colostomy 30 45 8 115 500 Ileostomy