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Fractures – Complications &
Management
Complication of Fractures
 Immediate Complication (at the time of
fracture)
 Early complication (initial few days)
 Late Complication
Immediate Complications
Systemic
 Hypovolaemic Shock
Local
 Injury to major vessels
 Injury to muscles & tendons
 Injury to joints
 Injury to viscera
Early complications
Systemic
 Hypovolaemic shock
 ARDS
 Fat Embolism Syndrome
 DVT & Pulmonary embolism
 Aseptic traumatic fever
 Septicemia (Open fractures)
 Crush syndrome
Early Complications
Local
 Infection
 Compartmental Syndrome
Late complications
Bone Union related
 Delayed-Union
 Non-union
 Mal-union
 Cross-union
Late complications
Others
 Avascular Necrosis
 Shortening
 Joint Stiffness
 Sudeck’s dystrophy
(Reflex Sympathetic
dystrophy)
 Osteomyelitis
 Ischaemic
contracture
 Myossitis Ossificans
 Osteoarthritis
Hypovolaemic Shock
 Commonest cause for death
 Pelvis(2 lts) & Long Bone(1.5lts) #’s
 External Hemorrhage
eg: Open fracture, vascular Injury
 Internal Hemorrhage
eg: Chest/Abdominal bleeding
Hypovolaemic shock-Management
 Follow the BLS/ATLS protocols
Eg: No 14 IV cannula + 2lts of
crystalloids/colloids/blood, Localize the site of
bleeding, needle aspiration, Inv - X-ray,
Ultrasound
 Avoid movements at the Fracture
 Stabilize the fracture eg : External
Fixator for Pelvis, Splints for long bones
ARDS –Adult Respiratory Distress
Syndrome
 Cause – Trauma & Shock
 Release of Inflammatory mediators
 Disruption of Pulmonary
microvasculature
 Onset in 24 hrs
 Tachypnoea & laboured breathing
ARDS - Management
 Chest X-ray: diffuse Pulm infiltrate
 Arterial PO2 <50
 100% Oxygen
 Assisted ventilation
 Chest clears in 4 to 7 days
 Not treated – CardioPulmonary failure -
Death
Fat Embolism
 Occlusion of Small vessels by fat globules
 Bone Marrow/Adipose tissue
 Polytrauma of long major bones
 Release of free fatty acids(Lipases action) –
toxic vasculitis – Platelet fibrin thrombosis
 Obstruction of Pulm vessels by fat
Fat Embolism – clinical features
 Develops in 24 to 72hrs
 Cerebral type – drowsy, restless,
disoriented, coma
 Pulmonary type – tachypnoea, tachycardia
 Patechial rash- neck, axillary fold, chest,
conjunctiva
 Respiratory failure - Death
Fat Embolism - Diagnosis
 Strong suspicion
 Retinal artery emboli (Striate haemorrhages)
 Fat globules in sputum & Urine
 X-ray – Patchy pulm Infiltrate (snow storm)
 Blood PO2 <50
Fat Embolism - treatment
 Respiratory support
 Heparinisation, IV Low molecular wt
dextran, corticosteriods
Deep Vein Thrombosis (DVT)
& Pulmonary Embolism (PE)
 Lower limb & Spinal Injuries
 Cause: Immobilization – venous stasis –
thrombosis of veins
 DVT proximal to knee is Dangerous
 DVT in 48 hrs – PE in 4 to 5 days
DVT & PE – Clinical Feature
 High index of suspicion
 Elderly & Obese pts
 Leg swelling & calf tenderness
 Calf tenderness on passive dorsiflexion
of Ankle ( Homan’s sign)
 Venography/Doppler Ultrasound
 PE – tachypnoea, dyspnoea, chest
pain, hemoptysis
DVT & PE - treatment
 DVT : Elevation of limb, Elastic
bandage, active mobilization after early
fracture stabilization, anticoagulation
 PE : Respiratory support,
Anticoagulation therapy
Crush syndrome
 Massive crushing of Muscles
 Release of Myohaemoglobin
 Precipitates in Renal tubules
 Acute renal tubular necrosis
 Treated as for Acute renal failure
Injury to the blood vessels
 Femoral - # L/3rd
Femur
 Popliteal – Supracondylar # Femur(commenest)
 Post Tibial – Dislocation Knee, # Tibia
 Subclavian – Clavicle #
 Axillary – Fracture dislocation shoulder
 Brachial – Supracondylar # Humerus
Injury to the blood vessels
 No effect – collateral circulation is good
 Exercise Ischemia – vascular
claudiction
 Ischemic contracture – Ischemic muscle
necrosis – contracture/fibrosis –
volkmann’s ischemic contracture
 Gangrene
Injury to the blood vessels
 Pain – cramp like
 Pulse – absent
 Pallor
 Paraesthesia
 Paralysis
Doppler study / Angiogram
Repair of vessel
Injury to Nerves
Injury to Tendons
Injury to Joints
 Subluxation or Dislocation
 Early reduction & stabilization
 Early Joint movement
 Secondary Osteoarthritis/stiffness
Infection - Osteomyelitis
 Open #’s (Common)
Compartment syndrome
 Rise in Pressure in closed compartment
of the limb
 Jeopardize the Muscle & nerve blood
supply
Injury & oedma to muscles
Fracture hematoma
Ischemia leading to muscle oedema
Compartment syndrome- Vicious cycle
swelling
Blood supply Muscle Ischemia
Injury
Compartment syndrome - Results
 Ischemic Muscle necrosis
 Muscle fibrosis – Contractures
 Nerve damage
 Motor sensory loss
 Gangrene
Compartmental syndrome -
Diagnosis
 Excessive Pain
 High risk injuries
eg:Supracondylar # humerus,
Forearm bones #,
Closed Tibial #,
Crush injuries to leg & forearm
Compartmental syndrome -
Diagnosis
 Stretch test – earliest sign
 Tense compartment
 Hypoaesthesia of involved nerves
 Muscle weakness
 Compartmental pressure of >40 mm of
H2O
Compartmental syndrome - Treatment
 Early prevention – limb elevation, active
finger mobilization
 Early surgical decompression eg:
fasciotomy
Delayed & Non-union
 More than the usual time to unite
 Fracture healing has stopped (not
before six months)
Delayed & Non-union
Causes related to the patients
 Age – common in old age
 Asso Systemic illness eg:Malignancy,
Osteomalacia
Delayed & Non-union
Causes related to fracture
 Distraction at fracture site
Muscle pulling eg:Patella & Olecranon #
Gravity eg: # shaft of humerus
 Soft tissue interposition eg: # shaft
humerus, femur
 Bone loss at the # site
Non-union
Delayed & Non-union
Causes related to fracture
 Infection from open #
 Damage/Poor blood supply eg:# neck
of femur, L/3rd
Tibia
 Pathological #
Delayed & Non-union
Causes related to Treatment
 Inadequate redution
 Inadequate immobilization
 Distration during treatment
Types of Nonunion
 Atrophic
 Hypertrophic
Common sites
Neck of femur, Scaphoid, L/3rd
Tibia,
Lateral condyle Humerus
Delayed & Non-union : C/f
 Persistant Pain
 Pain on stressing the #
 Mobility (Nonunion)
 Increasing deformity
Delayed & Non-union – X-ray
 Fracture line is visible
 Inadequate bridging callus (Delayed)
 No bridging callus (Non-union)
Tomogram
Medullary venography
Infected Nonunion
Delayed & Non-union- Treatment
 Bone Grafting
 BG + Internal Fixation
 Excision of fragment eg: # neck of
femur in elderly(hemiarthroplasty)
 Ilizarov’s method
Malunion
 # unites in improper position
 Disability of clinical significance eg:
deformity, shortening, limitation of motion
 Improper treatment
 Commonest eg: colles #, Clavicle #
Malunion
Malunion - Treatment
 Osteoclasis
 Corrective osteotomy
 No treatment – Remodelling
Children,
5 to 10 deg of Angulation,
Angulation in the plane of movement,
#’s near joints
Avascular Necrosis
 Blood supply is jeopardized
 Head of Femur eg: # neck of femur,
dislocation hip
 Proximal pole of scaphoid, Body of
Talus
Avascular Necrosis
 Sclerosis of necrotic bone
 Deformity due to collapse
 Osteoarthritis
Avascular Necrosis-
Treatment
 Delayed wt bearing
 Vascularised bone graft
 Excision of avascular segment
 Excision & Arthroplasty
Reflex sympathetic Dystrophy
(sudeck’s Dystrophy)
 Following Trauma
 Pain
 Hyperaesthesia
 Tenderness & Swelling
 Skin is red, shiny, warm
 Atrophy of skin, muscle, nail
 Joint deformity & stiffness
Reflex sympathetic Dystrophy
(sudeck’s Dystrophy)
 X-ray – Spotty rarefaction
 Physiotherapy
 Avoid surgery or forceful mobilization
 Sympathetic blocks
Myossitis Ossificans
(post-traumatic ossification)
 Ossification of the hematoma
 Joint stiffness
 Head injury
 Children around elbow
 Massage
Myossitis Ossificans
(post-traumatic ossification)
 X-ray-active myositis – margins are
fluffy
 Mature myositis – bone trabeculated,
well defined margins
Myossitis Ossificans
(post-traumatic ossification)
 Avoid Massage
 Rest to the limb
 Surgical excision of mature myositis
Thankyou
Dr Jai Thilak
Orthopaedics

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Fracture compli & mx

  • 2. Complication of Fractures  Immediate Complication (at the time of fracture)  Early complication (initial few days)  Late Complication
  • 3. Immediate Complications Systemic  Hypovolaemic Shock Local  Injury to major vessels  Injury to muscles & tendons  Injury to joints  Injury to viscera
  • 4. Early complications Systemic  Hypovolaemic shock  ARDS  Fat Embolism Syndrome  DVT & Pulmonary embolism  Aseptic traumatic fever  Septicemia (Open fractures)  Crush syndrome
  • 6. Late complications Bone Union related  Delayed-Union  Non-union  Mal-union  Cross-union
  • 7. Late complications Others  Avascular Necrosis  Shortening  Joint Stiffness  Sudeck’s dystrophy (Reflex Sympathetic dystrophy)  Osteomyelitis  Ischaemic contracture  Myossitis Ossificans  Osteoarthritis
  • 8. Hypovolaemic Shock  Commonest cause for death  Pelvis(2 lts) & Long Bone(1.5lts) #’s  External Hemorrhage eg: Open fracture, vascular Injury  Internal Hemorrhage eg: Chest/Abdominal bleeding
  • 9. Hypovolaemic shock-Management  Follow the BLS/ATLS protocols Eg: No 14 IV cannula + 2lts of crystalloids/colloids/blood, Localize the site of bleeding, needle aspiration, Inv - X-ray, Ultrasound  Avoid movements at the Fracture  Stabilize the fracture eg : External Fixator for Pelvis, Splints for long bones
  • 10. ARDS –Adult Respiratory Distress Syndrome  Cause – Trauma & Shock  Release of Inflammatory mediators  Disruption of Pulmonary microvasculature  Onset in 24 hrs  Tachypnoea & laboured breathing
  • 11. ARDS - Management  Chest X-ray: diffuse Pulm infiltrate  Arterial PO2 <50  100% Oxygen  Assisted ventilation  Chest clears in 4 to 7 days  Not treated – CardioPulmonary failure - Death
  • 12. Fat Embolism  Occlusion of Small vessels by fat globules  Bone Marrow/Adipose tissue  Polytrauma of long major bones  Release of free fatty acids(Lipases action) – toxic vasculitis – Platelet fibrin thrombosis  Obstruction of Pulm vessels by fat
  • 13. Fat Embolism – clinical features  Develops in 24 to 72hrs  Cerebral type – drowsy, restless, disoriented, coma  Pulmonary type – tachypnoea, tachycardia  Patechial rash- neck, axillary fold, chest, conjunctiva  Respiratory failure - Death
  • 14. Fat Embolism - Diagnosis  Strong suspicion  Retinal artery emboli (Striate haemorrhages)  Fat globules in sputum & Urine  X-ray – Patchy pulm Infiltrate (snow storm)  Blood PO2 <50
  • 15. Fat Embolism - treatment  Respiratory support  Heparinisation, IV Low molecular wt dextran, corticosteriods
  • 16. Deep Vein Thrombosis (DVT) & Pulmonary Embolism (PE)  Lower limb & Spinal Injuries  Cause: Immobilization – venous stasis – thrombosis of veins  DVT proximal to knee is Dangerous  DVT in 48 hrs – PE in 4 to 5 days
  • 17. DVT & PE – Clinical Feature  High index of suspicion  Elderly & Obese pts  Leg swelling & calf tenderness  Calf tenderness on passive dorsiflexion of Ankle ( Homan’s sign)  Venography/Doppler Ultrasound  PE – tachypnoea, dyspnoea, chest pain, hemoptysis
  • 18. DVT & PE - treatment  DVT : Elevation of limb, Elastic bandage, active mobilization after early fracture stabilization, anticoagulation  PE : Respiratory support, Anticoagulation therapy
  • 19. Crush syndrome  Massive crushing of Muscles  Release of Myohaemoglobin  Precipitates in Renal tubules  Acute renal tubular necrosis  Treated as for Acute renal failure
  • 20. Injury to the blood vessels  Femoral - # L/3rd Femur  Popliteal – Supracondylar # Femur(commenest)  Post Tibial – Dislocation Knee, # Tibia  Subclavian – Clavicle #  Axillary – Fracture dislocation shoulder  Brachial – Supracondylar # Humerus
  • 21. Injury to the blood vessels  No effect – collateral circulation is good  Exercise Ischemia – vascular claudiction  Ischemic contracture – Ischemic muscle necrosis – contracture/fibrosis – volkmann’s ischemic contracture  Gangrene
  • 22. Injury to the blood vessels  Pain – cramp like  Pulse – absent  Pallor  Paraesthesia  Paralysis Doppler study / Angiogram Repair of vessel
  • 24. Injury to Joints  Subluxation or Dislocation  Early reduction & stabilization  Early Joint movement  Secondary Osteoarthritis/stiffness
  • 25. Infection - Osteomyelitis  Open #’s (Common)
  • 26. Compartment syndrome  Rise in Pressure in closed compartment of the limb  Jeopardize the Muscle & nerve blood supply Injury & oedma to muscles Fracture hematoma Ischemia leading to muscle oedema
  • 27. Compartment syndrome- Vicious cycle swelling Blood supply Muscle Ischemia Injury
  • 28. Compartment syndrome - Results  Ischemic Muscle necrosis  Muscle fibrosis – Contractures  Nerve damage  Motor sensory loss  Gangrene
  • 29. Compartmental syndrome - Diagnosis  Excessive Pain  High risk injuries eg:Supracondylar # humerus, Forearm bones #, Closed Tibial #, Crush injuries to leg & forearm
  • 30. Compartmental syndrome - Diagnosis  Stretch test – earliest sign  Tense compartment  Hypoaesthesia of involved nerves  Muscle weakness  Compartmental pressure of >40 mm of H2O
  • 31. Compartmental syndrome - Treatment  Early prevention – limb elevation, active finger mobilization  Early surgical decompression eg: fasciotomy
  • 32. Delayed & Non-union  More than the usual time to unite  Fracture healing has stopped (not before six months)
  • 33. Delayed & Non-union Causes related to the patients  Age – common in old age  Asso Systemic illness eg:Malignancy, Osteomalacia
  • 34. Delayed & Non-union Causes related to fracture  Distraction at fracture site Muscle pulling eg:Patella & Olecranon # Gravity eg: # shaft of humerus  Soft tissue interposition eg: # shaft humerus, femur  Bone loss at the # site
  • 36. Delayed & Non-union Causes related to fracture  Infection from open #  Damage/Poor blood supply eg:# neck of femur, L/3rd Tibia  Pathological #
  • 37. Delayed & Non-union Causes related to Treatment  Inadequate redution  Inadequate immobilization  Distration during treatment
  • 38. Types of Nonunion  Atrophic  Hypertrophic Common sites Neck of femur, Scaphoid, L/3rd Tibia, Lateral condyle Humerus
  • 39. Delayed & Non-union : C/f  Persistant Pain  Pain on stressing the #  Mobility (Nonunion)  Increasing deformity
  • 40. Delayed & Non-union – X-ray  Fracture line is visible  Inadequate bridging callus (Delayed)  No bridging callus (Non-union) Tomogram Medullary venography
  • 42. Delayed & Non-union- Treatment  Bone Grafting  BG + Internal Fixation  Excision of fragment eg: # neck of femur in elderly(hemiarthroplasty)  Ilizarov’s method
  • 43. Malunion  # unites in improper position  Disability of clinical significance eg: deformity, shortening, limitation of motion  Improper treatment  Commonest eg: colles #, Clavicle #
  • 45. Malunion - Treatment  Osteoclasis  Corrective osteotomy  No treatment – Remodelling Children, 5 to 10 deg of Angulation, Angulation in the plane of movement, #’s near joints
  • 46. Avascular Necrosis  Blood supply is jeopardized  Head of Femur eg: # neck of femur, dislocation hip  Proximal pole of scaphoid, Body of Talus
  • 47. Avascular Necrosis  Sclerosis of necrotic bone  Deformity due to collapse  Osteoarthritis
  • 48. Avascular Necrosis- Treatment  Delayed wt bearing  Vascularised bone graft  Excision of avascular segment  Excision & Arthroplasty
  • 49. Reflex sympathetic Dystrophy (sudeck’s Dystrophy)  Following Trauma  Pain  Hyperaesthesia  Tenderness & Swelling  Skin is red, shiny, warm  Atrophy of skin, muscle, nail  Joint deformity & stiffness
  • 50. Reflex sympathetic Dystrophy (sudeck’s Dystrophy)  X-ray – Spotty rarefaction  Physiotherapy  Avoid surgery or forceful mobilization  Sympathetic blocks
  • 51. Myossitis Ossificans (post-traumatic ossification)  Ossification of the hematoma  Joint stiffness  Head injury  Children around elbow  Massage
  • 52. Myossitis Ossificans (post-traumatic ossification)  X-ray-active myositis – margins are fluffy  Mature myositis – bone trabeculated, well defined margins
  • 53. Myossitis Ossificans (post-traumatic ossification)  Avoid Massage  Rest to the limb  Surgical excision of mature myositis