This document provides an overview of pressure injuries and other wounds. It summarizes the changes made in 2015 by NPUAP to the pressure injury staging system, replacing Roman numerals with Arabic numerals and changing the term "pressure ulcer" to "pressure injury". Stage 1 is now defined as intact skin with non-blanchable erythema. Stage 2 involves partial skin loss exposing dermis. Stage 3 shows full skin loss exposing subcutaneous fat. Stage 4 penetrates deeper to expose fascia, muscle or bone. An unstageable injury has obscured tissue loss. Deep tissue injuries present as discolored intact skin. Medical device and mucosal membrane injuries are also described. Non-pressure issues like venous, arterial and
Pressure ulcers, also known as decubitus ulcers or bedsores, are localized injuries to the skin and underlying tissue that are usually caused by pressure over bony prominences. They are commonly seen in immobilized or bedridden patients and are associated with increased costs of care and risk of litigation. The Braden Scale is commonly used to assess pressure ulcer risk based on factors like mobility, sensation, moisture, activity, nutrition, and friction/shear. Treatment focuses on pressure redistribution through support surfaces, wound care including debridement and moist dressings, and managing pain and infection. Staging systems classify ulcers by depth of tissue damage from non-blanchable erythema to full thickness tissue loss.
Pressure ulcers, also known as bedsores, develop when skin and underlying tissue is damaged due to prolonged pressure, friction, or moisture. They typically form over bony areas of the body. Factors that increase risk include immobility, incontinence, poor nutrition, aging, and chronic diseases. Pressure ulcers are staged from I to IV based on the depth of tissue damage. Prevention focuses on relieving pressure through repositioning, special beds and cushions, and keeping skin clean and dry. Treatment may include dressings, debridement, and medications to promote healing.
Wound care clinicians: do you need to practice your pressure injury staging skills? View a photo and then click forward to see the correct stage according to the 2016 National Pressure Ulcer Advisory Panel (NPUAP) staging system.
This document provides information on wound classification and assessment. It discusses the different types of wounds including pressure ulcers, vascular wounds, neuropathic wounds, skin tears, and moisture-associated dermatitis. Pressure ulcer staging is explained, with stages ranging from I to IV. Key factors for assessing and documenting wounds are wound size, depth, tissue type, edges, infection signs and undermining/tunneling. Selecting appropriate dressings depends on wound characteristics and patient condition. Comprehensive wound assessment and risk assessment are important for effective care planning.
This document discusses pressure ulcers, including their definition, staging, epidemiology, pathophysiology, presentation, assessment, and management. Pressure ulcers are lesions caused by unrelieved pressure that damage underlying tissue. They are commonly staged from I to IV based on depth of tissue damage. Risk factors include immobility, incontinence, and nutritional deficiencies. Treatment involves reducing pressure, managing infection, debridement, dressing wounds, and surgery in some cases.
The document discusses various topics related to skin and wound care including:
- The layers of skin and types of wounds
- Staging criteria for pressure ulcers which describes the level of tissue damage
- Risk factors for pressure ulcers like limited mobility and incontinence
- Guidelines for preventing pressure ulcers including repositioning, maintaining nutrition, and managing moisture
- Common skin conditions like fungal infections, abrasions, and vascular wounds
This document discusses pressure ulcer prevention and stages. It defines the revised stages of pressure ulcers and identifies evidence-based practices for prevention. There is debate around whether all pressure ulcers are avoidable or if some are due to unavoidable circumstances. The roles of nurses, facilities, and regulators in pressure ulcer prevention and accountability are also examined.
This document provides guidance on preventing and treating pressure ulcers. It begins with the learner objectives and background on skin anatomy. It then discusses early prevention techniques, risk factors, the Braden risk assessment scale, nutrition screening, thorough skin assessment including staging, documentation, and appropriate wound care and support surfaces. The overall message is that diligent skin assessment and individualized prevention plans are necessary to reduce pressure ulcer risk.
Pressure ulcers, also known as decubitus ulcers or bedsores, are localized injuries to the skin and underlying tissue that are usually caused by pressure over bony prominences. They are commonly seen in immobilized or bedridden patients and are associated with increased costs of care and risk of litigation. The Braden Scale is commonly used to assess pressure ulcer risk based on factors like mobility, sensation, moisture, activity, nutrition, and friction/shear. Treatment focuses on pressure redistribution through support surfaces, wound care including debridement and moist dressings, and managing pain and infection. Staging systems classify ulcers by depth of tissue damage from non-blanchable erythema to full thickness tissue loss.
Pressure ulcers, also known as bedsores, develop when skin and underlying tissue is damaged due to prolonged pressure, friction, or moisture. They typically form over bony areas of the body. Factors that increase risk include immobility, incontinence, poor nutrition, aging, and chronic diseases. Pressure ulcers are staged from I to IV based on the depth of tissue damage. Prevention focuses on relieving pressure through repositioning, special beds and cushions, and keeping skin clean and dry. Treatment may include dressings, debridement, and medications to promote healing.
Wound care clinicians: do you need to practice your pressure injury staging skills? View a photo and then click forward to see the correct stage according to the 2016 National Pressure Ulcer Advisory Panel (NPUAP) staging system.
This document provides information on wound classification and assessment. It discusses the different types of wounds including pressure ulcers, vascular wounds, neuropathic wounds, skin tears, and moisture-associated dermatitis. Pressure ulcer staging is explained, with stages ranging from I to IV. Key factors for assessing and documenting wounds are wound size, depth, tissue type, edges, infection signs and undermining/tunneling. Selecting appropriate dressings depends on wound characteristics and patient condition. Comprehensive wound assessment and risk assessment are important for effective care planning.
This document discusses pressure ulcers, including their definition, staging, epidemiology, pathophysiology, presentation, assessment, and management. Pressure ulcers are lesions caused by unrelieved pressure that damage underlying tissue. They are commonly staged from I to IV based on depth of tissue damage. Risk factors include immobility, incontinence, and nutritional deficiencies. Treatment involves reducing pressure, managing infection, debridement, dressing wounds, and surgery in some cases.
The document discusses various topics related to skin and wound care including:
- The layers of skin and types of wounds
- Staging criteria for pressure ulcers which describes the level of tissue damage
- Risk factors for pressure ulcers like limited mobility and incontinence
- Guidelines for preventing pressure ulcers including repositioning, maintaining nutrition, and managing moisture
- Common skin conditions like fungal infections, abrasions, and vascular wounds
This document discusses pressure ulcer prevention and stages. It defines the revised stages of pressure ulcers and identifies evidence-based practices for prevention. There is debate around whether all pressure ulcers are avoidable or if some are due to unavoidable circumstances. The roles of nurses, facilities, and regulators in pressure ulcer prevention and accountability are also examined.
This document provides guidance on preventing and treating pressure ulcers. It begins with the learner objectives and background on skin anatomy. It then discusses early prevention techniques, risk factors, the Braden risk assessment scale, nutrition screening, thorough skin assessment including staging, documentation, and appropriate wound care and support surfaces. The overall message is that diligent skin assessment and individualized prevention plans are necessary to reduce pressure ulcer risk.
Pressure ulcers, also known as decubitus ulcers or bed sores, are localized areas of soft tissue damage that result from prolonged pressure. They are staged from 1 to 4 based on depth of tissue damage. Risk factors include older age, immobility, moisture, and poor nutrition. Treatment focuses on relieving pressure, cleaning and debridement of wounds, dressing changes, and surgery if needed to prevent complications like infection.
Wound healing is a complex, dynamic process involving several phases: inflammatory, proliferative, and remodeling. The inflammatory phase involves hemostasis and inflammation to limit blood loss and seal the wound. The proliferative phase fills the wound gap with granulation tissue through fibroplasia, angiogenesis, and re-epithelialization. The remodeling phase involves regression of vessels and granulation tissue, wound contraction, and collagen remodeling to strengthen the scar. Successful wound healing depends on factors like adequate blood supply, infection control, and avoiding risks such as smoking, which can impair healing.
The document discusses pressure ulcers, including their causes, assessment, stages, treatment, and prevention. Some key points:
- Pressure ulcers are caused by factors like decreased sensation, mobility, nutrition, incontinence, and shear/friction forces.
- Assessment involves visual skin inspection and using a risk assessment scale like the Braden Scale to document factors like moisture, pressure, shear, and friction.
- Pressure ulcers are staged from I to IV based on tissue depth involvement, from non-blanchable redness to full thickness tissue loss with bone/muscle exposure.
- Treatment focuses on relieving pressure, keeping skin clean and dry, using dressings, and managing in
This document discusses the management of non-healing wounds, which requires a multidisciplinary approach involving thorough assessment and wound bed preparation. It outlines the key factors in assessment including patient history, wound characteristics, and factors affecting healing. Wound bed preparation principles of debridement, control of inflammation and infection, and maintenance of moisture balance are explained. Various debridement methods and appropriate dressings to promote healing are also described.
This document defines pressure ulcers, also known as bedsores or decubitus ulcers, as localized skin injuries that occur over bony prominences due to pressure or pressure combined with shear and friction forces. Pressure ulcers develop when external pressure compresses blood vessels, obstructing blood flow and depriving tissues of oxygen and nutrients. Risk factors include limited mobility, incontinence, poor nutrition, and comorbidities like diabetes. Treatment involves repositioning, special mattresses and dressings, wound cleaning and debridement, nutritional support, and sometimes surgery.
this is presentation talks about basic & updated advanced wounds care,,,,,,,2nd presentation in my internship..i hope you will get benefit from it ......Dr/ Wadie Madi
This document provides information on nursing care for patients with wounds. It begins by defining wounds and describing the wound healing process. It then classifies wounds by intent, whether open or closed, degree of contamination, and depth. Various types of wounds like incisions, contusions, and lacerations are described. Stages of pressure ulcers are defined. The document outlines factors that influence wound healing and potential complications. It discusses assessing wounds by location, appearance, drainage, and for pressure ulcers, the tissue type. Nursing diagnoses, care planning, interventions, and dressing considerations are presented for different wound stages. Cleaning and securing dressings is demonstrated. References are provided.
This document provides an overview of pressure sores including their definition, epidemiology, pathogenesis, risk factors, staging classifications, prevention, and management. Pressure sores, also known as bedsores or decubitus ulcers, are localized skin or tissue injuries caused by unrelieved pressure. They are common in hospital and nursing home patients and costly to healthcare systems. The document outlines the traditional theories behind their pathogenesis as well as intrinsic and extrinsic risk factors. Staging classifications from the National Pressure Ulcer Advisory Panel are presented along with the Braden and PUSH risk assessment tools. Prevention strategies aim to reduce pressure, shearing forces, and moisture on the skin. Management is based on the stage and may involve wound cleaning, dress
The document discusses pressure ulcer prevention and treatment for individuals with spinal cord injuries. It notes that 32-40% of individuals with SCIs develop pressure ulcers during initial hospitalization, with sacral ulcers being the most common. Prevention focuses on frequent repositioning, pressure redistribution, and skin inspections. Treatment involves wound staging, debridement, dressings, and surgery if needed. Factors like nutrition, moisture, shear and friction forces can impact wound healing.
This document provides an overview of wound care including wound healing processes, types of wounds, evaluation and documentation of wounds, and wound management. It discusses the stages of wound healing including hemostasis, inflammation, proliferation, and remodeling. Different types of wounds such as incisions, lacerations, abrasions, and puncture wounds are described along with factors to evaluate like location, size, exudate, signs of infection, and surrounding tissue condition. Methods of documentation including photography and standardized descriptions are covered. The management section addresses wound care plans, cleaning, debridement, exudate management using various dressing materials, and treatment of infections.
This document provides an overview of skin anatomy, wound classification, and wound management. It discusses the objectives of understanding wound care, outlines different topics to be covered including wound healing process and complications. The document defines wounds and classifies them by mechanism of injury, depth and degree of contamination. It describes the three phases of wound healing and different types of healing. Key steps of wound assessment and factors affecting healing are highlighted. Common wound dressings and their properties/indications are also summarized.
Early Detection of Melanoma and Other Skin CancersSummit Health
This lecture provides an overview of skin cancer including risks, early detection, and treatment. Learn to identify the early signs of skin cancer. Melanoma and non-melanoma skin tumors will be discussed and prevention of skin cancer will be emphasized.
This document provides a history and overview of facial chemical peels. It discusses how ancient Egyptians and Turks used exfoliants and thermal methods to treat sun-damaged skin. The history of chemical peels in modern medicine is outlined from the late 19th century to present day. Different types of peels including superficial, medium, and deep peels are described in detail, outlining the techniques, expected outcomes, and risks of each. Key aspects of patient selection and preparation are also summarized.
This document provides an overview of wound management. It discusses wound classification, the wound healing process, factors affecting wound healing, wound assessment, types of wound drainage, complications of wound healing, and various wound dressing techniques. The three main phases of wound healing are the inflammatory phase, proliferative phase, and maturation phase. Wound classification includes factors like the mechanism of injury, degree of contamination, and wound depth. Proper wound management requires assessing these classifications and factors to determine the appropriate treatment approach.
1) The document discusses current trends in wound management, outlining the phases of wound healing and factors that affect it.
2) Key aspects of wound management covered include wound bed preparation through debridement and dressing, as well as various closure methods and the use of negative pressure wound therapy.
3) Emerging treatments like hyperbaric oxygen therapy and various wound covers including skin substitutes are also summarized.
This document discusses wound classification and the phases of wound healing. It defines a wound and classifies wounds based on etiology, Rank and Wakefield classification, and surgical wound classification. The three phases of wound healing are described as the inflammatory phase, proliferative phase, and maturation/remodeling phase. Key cellular and vascular responses are summarized for each phase. Factors affecting wound healing include local factors like infection and ischemia, and systemic factors like nutrition, diabetes, and medications.
The document discusses the history of wound care from ancient times to modern practices. Some key points covered include:
- Ancient civilizations used remedies like honey, grease, and herbs to treat wounds. Hippocrates advocated cleaning wounds with wine or vinegar.
- Different types of wounds are discussed like acute, chronic, pressure ulcers, venous ulcers, and diabetic foot ulcers.
- Modern wound care includes debridement, dressings, antibiotics, growth factors, skin substitutes, and addressing underlying patient issues.
- Wound etiology, staging, and treatments are described for various chronic wounds. Biofilms, infection, and other complex wound factors are also addressed.
The graffiti movement began in Philadelphia in the 1960s and spread to New York, where people would tag their names and gang numbers on subway cars to increase their reputation. Over time, graffiti escalated and spread worldwide. Authorities tried to crack down by cleaning graffiti, but taggers continued in tunnels and other places. While some see graffiti as art, others view it as criminal damage and vandalism due to the costs of cleanup and how it can make areas seem intimidating. There is still debate around whether graffiti constitutes vandalism or is a legitimate art form.
El documento resume las dificultades que enfrentan los productores agropecuarios en Argentina. Explica que los agricultores enfrentan altos costos para cultivar soja y necesitan maquinaria pesada debido a la falta de inversiones en infraestructura rural. Además, argumenta que los productores agropecuarios aportan mucho a la economía pero son tratados injustamente por el gobierno a través de impuestos y retenciones excesivas. Finalmente, advierte que las prácticas insostenibles requeridas para ser rentables están degradando los su
Pressure ulcers, also known as decubitus ulcers or bed sores, are localized areas of soft tissue damage that result from prolonged pressure. They are staged from 1 to 4 based on depth of tissue damage. Risk factors include older age, immobility, moisture, and poor nutrition. Treatment focuses on relieving pressure, cleaning and debridement of wounds, dressing changes, and surgery if needed to prevent complications like infection.
Wound healing is a complex, dynamic process involving several phases: inflammatory, proliferative, and remodeling. The inflammatory phase involves hemostasis and inflammation to limit blood loss and seal the wound. The proliferative phase fills the wound gap with granulation tissue through fibroplasia, angiogenesis, and re-epithelialization. The remodeling phase involves regression of vessels and granulation tissue, wound contraction, and collagen remodeling to strengthen the scar. Successful wound healing depends on factors like adequate blood supply, infection control, and avoiding risks such as smoking, which can impair healing.
The document discusses pressure ulcers, including their causes, assessment, stages, treatment, and prevention. Some key points:
- Pressure ulcers are caused by factors like decreased sensation, mobility, nutrition, incontinence, and shear/friction forces.
- Assessment involves visual skin inspection and using a risk assessment scale like the Braden Scale to document factors like moisture, pressure, shear, and friction.
- Pressure ulcers are staged from I to IV based on tissue depth involvement, from non-blanchable redness to full thickness tissue loss with bone/muscle exposure.
- Treatment focuses on relieving pressure, keeping skin clean and dry, using dressings, and managing in
This document discusses the management of non-healing wounds, which requires a multidisciplinary approach involving thorough assessment and wound bed preparation. It outlines the key factors in assessment including patient history, wound characteristics, and factors affecting healing. Wound bed preparation principles of debridement, control of inflammation and infection, and maintenance of moisture balance are explained. Various debridement methods and appropriate dressings to promote healing are also described.
This document defines pressure ulcers, also known as bedsores or decubitus ulcers, as localized skin injuries that occur over bony prominences due to pressure or pressure combined with shear and friction forces. Pressure ulcers develop when external pressure compresses blood vessels, obstructing blood flow and depriving tissues of oxygen and nutrients. Risk factors include limited mobility, incontinence, poor nutrition, and comorbidities like diabetes. Treatment involves repositioning, special mattresses and dressings, wound cleaning and debridement, nutritional support, and sometimes surgery.
this is presentation talks about basic & updated advanced wounds care,,,,,,,2nd presentation in my internship..i hope you will get benefit from it ......Dr/ Wadie Madi
This document provides information on nursing care for patients with wounds. It begins by defining wounds and describing the wound healing process. It then classifies wounds by intent, whether open or closed, degree of contamination, and depth. Various types of wounds like incisions, contusions, and lacerations are described. Stages of pressure ulcers are defined. The document outlines factors that influence wound healing and potential complications. It discusses assessing wounds by location, appearance, drainage, and for pressure ulcers, the tissue type. Nursing diagnoses, care planning, interventions, and dressing considerations are presented for different wound stages. Cleaning and securing dressings is demonstrated. References are provided.
This document provides an overview of pressure sores including their definition, epidemiology, pathogenesis, risk factors, staging classifications, prevention, and management. Pressure sores, also known as bedsores or decubitus ulcers, are localized skin or tissue injuries caused by unrelieved pressure. They are common in hospital and nursing home patients and costly to healthcare systems. The document outlines the traditional theories behind their pathogenesis as well as intrinsic and extrinsic risk factors. Staging classifications from the National Pressure Ulcer Advisory Panel are presented along with the Braden and PUSH risk assessment tools. Prevention strategies aim to reduce pressure, shearing forces, and moisture on the skin. Management is based on the stage and may involve wound cleaning, dress
The document discusses pressure ulcer prevention and treatment for individuals with spinal cord injuries. It notes that 32-40% of individuals with SCIs develop pressure ulcers during initial hospitalization, with sacral ulcers being the most common. Prevention focuses on frequent repositioning, pressure redistribution, and skin inspections. Treatment involves wound staging, debridement, dressings, and surgery if needed. Factors like nutrition, moisture, shear and friction forces can impact wound healing.
This document provides an overview of wound care including wound healing processes, types of wounds, evaluation and documentation of wounds, and wound management. It discusses the stages of wound healing including hemostasis, inflammation, proliferation, and remodeling. Different types of wounds such as incisions, lacerations, abrasions, and puncture wounds are described along with factors to evaluate like location, size, exudate, signs of infection, and surrounding tissue condition. Methods of documentation including photography and standardized descriptions are covered. The management section addresses wound care plans, cleaning, debridement, exudate management using various dressing materials, and treatment of infections.
This document provides an overview of skin anatomy, wound classification, and wound management. It discusses the objectives of understanding wound care, outlines different topics to be covered including wound healing process and complications. The document defines wounds and classifies them by mechanism of injury, depth and degree of contamination. It describes the three phases of wound healing and different types of healing. Key steps of wound assessment and factors affecting healing are highlighted. Common wound dressings and their properties/indications are also summarized.
Early Detection of Melanoma and Other Skin CancersSummit Health
This lecture provides an overview of skin cancer including risks, early detection, and treatment. Learn to identify the early signs of skin cancer. Melanoma and non-melanoma skin tumors will be discussed and prevention of skin cancer will be emphasized.
This document provides a history and overview of facial chemical peels. It discusses how ancient Egyptians and Turks used exfoliants and thermal methods to treat sun-damaged skin. The history of chemical peels in modern medicine is outlined from the late 19th century to present day. Different types of peels including superficial, medium, and deep peels are described in detail, outlining the techniques, expected outcomes, and risks of each. Key aspects of patient selection and preparation are also summarized.
This document provides an overview of wound management. It discusses wound classification, the wound healing process, factors affecting wound healing, wound assessment, types of wound drainage, complications of wound healing, and various wound dressing techniques. The three main phases of wound healing are the inflammatory phase, proliferative phase, and maturation phase. Wound classification includes factors like the mechanism of injury, degree of contamination, and wound depth. Proper wound management requires assessing these classifications and factors to determine the appropriate treatment approach.
1) The document discusses current trends in wound management, outlining the phases of wound healing and factors that affect it.
2) Key aspects of wound management covered include wound bed preparation through debridement and dressing, as well as various closure methods and the use of negative pressure wound therapy.
3) Emerging treatments like hyperbaric oxygen therapy and various wound covers including skin substitutes are also summarized.
This document discusses wound classification and the phases of wound healing. It defines a wound and classifies wounds based on etiology, Rank and Wakefield classification, and surgical wound classification. The three phases of wound healing are described as the inflammatory phase, proliferative phase, and maturation/remodeling phase. Key cellular and vascular responses are summarized for each phase. Factors affecting wound healing include local factors like infection and ischemia, and systemic factors like nutrition, diabetes, and medications.
The document discusses the history of wound care from ancient times to modern practices. Some key points covered include:
- Ancient civilizations used remedies like honey, grease, and herbs to treat wounds. Hippocrates advocated cleaning wounds with wine or vinegar.
- Different types of wounds are discussed like acute, chronic, pressure ulcers, venous ulcers, and diabetic foot ulcers.
- Modern wound care includes debridement, dressings, antibiotics, growth factors, skin substitutes, and addressing underlying patient issues.
- Wound etiology, staging, and treatments are described for various chronic wounds. Biofilms, infection, and other complex wound factors are also addressed.
The graffiti movement began in Philadelphia in the 1960s and spread to New York, where people would tag their names and gang numbers on subway cars to increase their reputation. Over time, graffiti escalated and spread worldwide. Authorities tried to crack down by cleaning graffiti, but taggers continued in tunnels and other places. While some see graffiti as art, others view it as criminal damage and vandalism due to the costs of cleanup and how it can make areas seem intimidating. There is still debate around whether graffiti constitutes vandalism or is a legitimate art form.
El documento resume las dificultades que enfrentan los productores agropecuarios en Argentina. Explica que los agricultores enfrentan altos costos para cultivar soja y necesitan maquinaria pesada debido a la falta de inversiones en infraestructura rural. Además, argumenta que los productores agropecuarios aportan mucho a la economía pero son tratados injustamente por el gobierno a través de impuestos y retenciones excesivas. Finalmente, advierte que las prácticas insostenibles requeridas para ser rentables están degradando los su
Este documento es una prueba diagnóstica de lenguaje para estudiantes de educación especial. Contiene ejercicios de asociación de letras, sílabas y palabras con imágenes, transcripción de letras y palabras en letra manuscrita, lectura de letras, palabras y oraciones, e interpretación gráfica de un texto descriptivo.
El documento presenta información sobre las matemáticas y su presencia en la naturaleza, la arquitectura y la invención. Las matemáticas se encuentran en todas partes, desde la ubicación y movimiento de objetos hasta en figuras geométricas como pentágonos que se ven en diversas flores. La invención de objetos como la rueda y maquinaria moderna se basan en formas matemáticas como círculos, triángulos y conos. El documento propone una actividad para que los estudiantes exploren figuras geomé
El documento describe los conceptos básicos de número que los niños de educación infantil aprenden en la escuela. Explica que aunque los niños tienen experiencias con números en casa, aún no tienen un entendimiento completo del concepto de número. Detalla las fases que los niños pasan para adquirir este concepto, incluyendo la fundamentación lógica, la conservación, y la coordinación cardinal-ordinal. También describe el proceso de contar objetos y las etapas por las que pasan los niños para desarrollar esta habilidad.
The document summarizes a 24-week full stack web development bootcamp program. The program provides rigorous training in both front-end and back-end development skills, including languages like HTML, CSS, JavaScript, jQuery, Node.js, databases like MySQL and MongoDB, and frameworks like React and Spring MVC. Students build a portfolio of projects and gain the skills needed for jobs as full stack web developers. The program includes lectures, in-class exercises, team projects, and one-on-one support to help students learn and build their skills and careers.
This document outlines Cameron Duncan's pre-production plans for a graphic novel. It includes a style sheet describing the font and color scheme used for characters. Layout 1 shows the front cover design featuring the gods Zeus, Hades, and Poseidon. Layout 2 depicts a double page scene of gods joining together for an inter-god battle. A schedule is provided detailing the 8-day plan to complete character drawings, locations, the cover, assemble pages, and complete an evaluation. Health and safety precautions are also noted.
1) El documento define las praxias como un sistema de movimientos coordinados con un propósito u objetivo.
2) Se recomienda realizar ejercicios orofaciales para mantener un buen tono muscular aunque no haya dificultades de pronunciación.
3) La terapia miofuncional se utiliza para corregir desequilibrios musculares orofaciales y crear patrones adecuados de deglución y articulación.
Avances en la investigación de la Educación Emocional a través del videojuego...Jorge Guerra-Antequera
El documento analiza el videojuego "El bosque encantado" y cómo promueve la educación emocional en primaria. Describe los objetivos de desarrollar habilidades emocionales a través de videojuegos y analiza cómo el juego se alinea con los estándares de aprendizaje socioemocional de Illinois. Explica cómo el juego ayuda a los estudiantes a mejorar el reconocimiento de emociones, las relaciones interpersonales, y la toma de decisiones responsable.
Baby-Bello-Bella is a boutique that designs and hand-makes dresses for baby girls, including flower girl dresses, little bridesmaid dresses, birthday dresses, formal dresses, and holiday dresses. The boutique offers many colors and styles to match baby girls and more formal options like red, blue, or black. Baby-Bello-Bella wants customers to find a unique and special dress for their little girl's special day.
El documento discute los peligros de seguir a maestros espirituales fraudulentos y ofrece consejos para discernir su autenticidad. Algunos maestros explotan a sus discípulos sexual o económicamente, mientras que otros enseñan de manera coherente y viven de acuerdo a sus enseñanzas. Es importante verificar que un maestro distinga claramente entre los diferentes niveles espirituales, psicológicos y emocionales y no los mezcle para su propio beneficio. Seguir a maestros de tradiciones est
This document appears to be a list of 10 names. It does not provide much context beyond the names listed, which include Lucía Espinosa, Oscar, Noelia Sánchez, Laura Muñoz, Sandra Moreno Gómez, Aitana Muncharaz, Clara Saavedra, Fernando Calderón, and Nacho Alberca.
Avengers: Age of Ultron was filmed in 5 countries and had an enormous budget of $250 million. It starred Robert Downey Jr., Chris Hemsworth, Mark Ruffalo, Chris Evans, Scarlett Johansson, and James Spader as Ultron. The film was a box office success, grossing over $1.4 billion worldwide. Reviews praised the action scenes and villain Ultron, though some felt it didn't live up to the first Avengers film. Extensive marketing including early teaser trailers, promotional events, social media campaigns, and merchandise helped drive its financial success.
1) Avengers: Age of Ultron was released in theaters on April 23, 2015 in the UK and May 1, 2015 in the US. It was produced by Marvel Studios and distributed by Walt Disney Studios Motion Pictures.
2) The film was directed by Joss Whedon and starred Robert Downey Jr., Chris Evans, Mark Ruffalo, Chris Hemsworth, Scarlett Johansson, and Jeremy Renner.
3) It had a production budget of $365 million and grossed over $1.4 billion worldwide, making it one of the highest-grossing films of all time.
Diabetic foot complications are a major source of morbidity and health care costs. They result from a complex interplay of ischemia, ulceration, infection, and Charcot's joint due to diabetes-related changes. Hyperglycemia leads to nonenzymatic glycosylation of proteins and tissues, formation of advanced glycosylation end products, and accelerated atherosclerosis. This causes peripheral vascular disease, neuropathy, and foot deformities which impair sensation and blood flow, making the feet susceptible to infection, ulceration, and gangrene. Charcot's joint is a destructive foot arthropathy caused by loss of sensation from diabetic neuropathy.
Este documento presenta una propuesta para el quinto Foro Mundial del Agua. Propone cinco mesas temáticas para discutir los desafíos del agua: 1) cambios globales y riesgos, 2) desarrollo humano, 3) recursos hídricos y sistemas de suministro, 4) educación y capacidades, y 5) gobierno y gestión. Cada mesa analizará ejes temáticos como adaptación al cambio climático, infraestructura, cooperación transfronteriza y roles público-privados para un manejo
This document discusses the 6 stages of pressure ulcers, from Stage I to Stage IV. Stage I involves intact skin with discoloration over a bony prominence. Stage II is a shallow open ulcer without slough. Stage III involves full thickness tissue loss where subcutaneous fat may be visible. Stage IV exposes bone, tendon or muscle. Unstageable ulcers have slough or eschar covering the wound bed. Deep tissue injuries cause discolored intact skin or blisters. Medical device related or Kennedy terminal ulcers are also discussed.
This document defines and describes the stages of pressure injuries, from Stage 1 (intact skin with non-blanchable erythema) to Stage 4 (full thickness skin and tissue loss with exposed bone, tendon or muscle). It also covers unstageable injuries, deep tissue pressure injuries, and injuries related to medical devices. The stages are defined by the extent of skin and tissue damage, from non-blanchable redness to complete tissue loss. Accurate staging is important for treatment and prevention of further injury.
This document defines and describes the stages of pressure injuries, from Stage 1 (intact skin with non-blanchable erythema) to Stage 4 (full thickness skin and tissue loss with exposed bone, tendon or muscle). It also covers unstageable injuries, deep tissue pressure injuries, and injuries related to medical devices. The stages are defined by the extent of skin and tissue damage, from non-blanchable redness to complete tissue loss. Accurate staging is important for treatment and prevention of further injury.
Bedsores, also known as pressure sores or ulcers, develop from prolonged pressure on skin, especially over bony areas. They are caused by impaired blood flow and damage to skin and tissue beneath. People at highest risk are those with limited mobility or confined to bed. Bedsores range in severity from Stage I (redness) to Stage IV (deep wound exposing bone/muscle). Prevention focuses on frequent repositioning to relieve pressure, good nutrition, and skin care.
Pressure ulcer prevention and care.pptxaneettababu3
This document discusses pressure ulcers, including their definition, risk factors, stages, signs and symptoms, prevention, and treatment. It defines pressure ulcers as injuries to the skin and tissue caused by prolonged pressure. Risk factors include impaired mobility, nutrition, hydration, age, and medical conditions. Prevention focuses on reducing pressure, moisture, friction, and shearing forces on the skin. Treatment involves cleaning wounds, applying dressings, managing pain and infection, dietary interventions, and sometimes surgery.
Pressure ulcers develop as a result of prolonged unrelieved pressure on the skin, usually over bony areas of the body. They are staged from 1 to 4 based on the depth of tissue destruction, with stage 1 involving only the epidermal layer and stage 4 extending into muscle, tendon or bone. Stage 2 ulcers damage the epidermis and dermis, potentially appearing as blisters or abrasions, while stage 3 shows full thickness tissue loss with visible subcutaneous fat but not exposed bone.
Pressure ulcers are localized skin injuries caused by prolonged unrelieved pressure, usually over bony areas. They are staged from 1 to 4 based on the depth of tissue destruction, with stage 1 being non-blanchable erythema and stage 4 extending into muscle, tendon or bone. Stage 2 involves damage to the epidermis and dermis, appearing as a shallow open ulcer. Stage 3 is a full-thickness tissue loss where subcutaneous fat may be visible.
Skin integrity and wound care [autosaved] (2)Nelson Munthali
This document discusses skin integrity and wound care. It describes factors that affect skin health like age, illness, and activity level. Wounds are breaks in skin integrity and can be accidental or surgical. The stages of pressure ulcer development and types of wound healing are explained. Finally, it outlines the three phases of wound healing - defensive, reconstructive, and maturation - and factors that can influence the healing process like nutrition, circulation, and immune function.
This document discusses factors affecting skin integrity and wound healing. It describes the four stages of pressure ulcer development from inflammation and reddening in stage 1 to extensive tissue damage that can expose bone in stage 4. Risk factors for developing pressure ulcers include increased pressure, decreased mobility, moisture, friction, shearing forces, malnutrition, altered mental status, and medical conditions impairing circulation. Nurse must understand wound classification and factors influencing wound healing to properly prevent and manage wounds.
This document defines and describes pressure ulcers and their prevention and treatment. It discusses that pressure ulcers are areas of damaged skin and tissue caused by unrelieved pressure, and grades them from 1 to 4 based on severity of tissue damage. It provides tips for prevention, including changing positions regularly, using proper mattresses and cushions, regular skin assessment, self care, diet, and choosing appropriate dressings and treatments based on the ulcer grade and characteristics.
Wounds can be classified in several ways, including by depth of tissue involvement. Superficial wounds only affect the epidermis, partial-thickness wounds also affect part of the dermis, and full-thickness wounds involve the epidermis and dermis with potential damage to underlying tissues. Wound healing involves inflammation, proliferation, and maturation phases. Factors like age, dehydration, infection, and poor circulation can affect wound healing. A thorough wound assessment should document characteristics of the wound bed, surrounding skin, and underlying factors.
This document discusses skin integrity and wound healing. It begins by outlining learning objectives related to factors affecting skin health, pressure ulcer risk and stages, and wound healing phases. The skin's functions are described, along with factors influencing integrity like age, health conditions, and activity level. Wound types include accidental injuries and surgical wounds, which are classified. Pressure ulcer stages and risk factors are then defined in detail.
MDR and bed sores injuries - Read-Only.pptxIbnSaad1
MDR injuries, medical devices related injuries are a common happening issues affecting, elderly, bed ridden patients...etc
In this data show we are discussing the definition, causes, degrees and management of this medical problem
This document defines pressure ulcers, discusses their pathogenesis and risk factors, and outlines their classification and management. Key points:
- Pressure ulcers are localized skin injuries caused by pressure that disrupts blood flow, often over bony prominences. The elderly are especially at risk.
- Risk factors include immobility, sensory impairment, malnutrition, moisture, shear and friction forces on the skin. Common sites are the sacrum and heels.
- Pressure ulcers are classified in stages from I to IV based on tissue damage depth. Prevention focuses on pressure reduction through repositioning, support surfaces, and skin care. Treatment involves dressing, debridement and wound healing promotion.
This document discusses bedsores, also known as pressure sores or ulcers. Bedsores develop from prolonged pressure on skin, especially over bony areas, and people at highest risk are those confined to beds or wheelchairs. Bedsores are staged from I to IV based on severity, from changes in skin color to deep wounds exposing tissue below. Common sites for bedsores in those using wheelchairs are tailbones, shoulders, and limbs, while bedridden patients often develop them on heads, ears, hips and heels. Treatment focuses on reducing pressure through repositioning and support surfaces, and cleaning and dressing wounds.
The presentation talks about BED sore, most common complication of spinal Cord Injury.
Definition
cause
Prevention
and Treatment
---------------------------------------
Video Links:
https://youtu.be/56sHJ3g_-Bw
https://youtu.be/wOq_4X2M_gY
https://youtu.be/tG3_1xvLo8I
https://youtu.be/pW88OX5mAqc
https://youtu.be/UGmwYCJiyz4
This document provides information on pressure ulcers/bed sores, including definitions, anatomy of the skin, risk factors, stages of pressure ulcers, prevention, and treatment. It defines a pressure ulcer as localized injury to the skin from prolonged unrelieved pressure, discusses the three layers of skin (epidermis, dermis, hypodermis), lists common sites of pressure ulcers, and identifies intrinsic and extrinsic risk factors. The stages of pressure ulcers from 1 to 4 are described based on depth of tissue damage. Prevention focuses on position changes, skin inspection, nutrition, lifestyle changes, and pressure-relieving devices. Treatment includes changing position, support surfaces, cleaning, controlling incontinence, debridement
This document provides guidance for nurses on skin assessment and pressure ulcer prevention. It outlines the purpose of assessing skin and identifying risk factors for pressure ulcers. Key points include identifying pressure ulcer stages from 1 to 4, as well as unstageable and deep tissue injuries. Common risk factors discussed are moisture, friction, shear forces, nutritional status, and mobility level. The document emphasizes the importance of regular skin inspection and position changes every 2 hours to relieve pressure on bony prominences and prevent skin breakdown.
This document provides an overview of pressure ulcer prevention and wound care. It defines the skin layers and their functions. Common causes of pressure ulcers are identified as prolonged pressure, shear forces, friction, and excessive moisture. Key pressure points are listed. The stages of pressure ulcer development are described. Factors that impact wound healing like nutrition, infection and moisture are discussed.
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Handout with scan codes to articles for providing culturally sensitive care at end of life for 6 patient populations
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Understand the definitions of professional boundaries • Explain how Maslow’s hierarchy is applicable to identify caregiver versus patient needs • Discuss challenges and possible scenarios encountered • Review the impact of social media on caregiving • Identify warning signs of crossed boundaries • Explore strategies to maintain healthy boundaries
Speaking points are in note section once downloaded
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Discusses bed bugs,scabies,lice,fleas,ticks and mosquitos, the identification, potential disease and eradication. Created for both long term care and home patients of a hospice. Notes are seen in note section once downloaded.
PP slides to accompany Teepa Snow's youtube video about her Gems Approach
https://www.youtube.com/watch?v=UXzJRZCNiRU&list=PLeu1xlHHkFCU_k85X1xBwjzayVIzNB12R&index=20
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At Apollo Hospital, Lucknow, U.P., we provide specialized care for children experiencing dehydration and other symptoms. We also offer NICU & PICU Ambulance Facility Services. Consult our expert today for the best pediatric emergency care.
For More Details:
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Name: Apollo Hospital
Address: Singar Nagar, LDA Colony, Lucknow, Uttar Pradesh 226012
Phone: 08429021957
Opening Hours: 24X7
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Joker Wigs has been a one-stop-shop for hair products for over 26 years. We provide high-quality hair wigs, hair extensions, hair toppers, hair patch, and more for both men and women.
2024 HIPAA Compliance Training Guide to the Compliance OfficersConference Panel
Join us for a comprehensive 90-minute lesson designed specifically for Compliance Officers and Practice/Business Managers. This 2024 HIPAA Training session will guide you through the critical steps needed to ensure your practice is fully prepared for upcoming audits. Key updates and significant changes under the Omnibus Rule will be covered, along with the latest applicable updates for 2024.
Key Areas Covered:
Texting and Email Communication: Understand the compliance requirements for electronic communication.
Encryption Standards: Learn what is necessary and what is overhyped.
Medical Messaging and Voice Data: Ensure secure handling of sensitive information.
IT Risk Factors: Identify and mitigate risks related to your IT infrastructure.
Why Attend:
Expert Instructor: Brian Tuttle, with over 20 years in Health IT and Compliance Consulting, brings invaluable experience and knowledge, including insights from over 1000 risk assessments and direct dealings with Office of Civil Rights HIPAA auditors.
Actionable Insights: Receive practical advice on preparing for audits and avoiding common mistakes.
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Ensure your compliance strategy is up-to-date and effective. Enroll now and be prepared for the 2024 HIPAA audits.
Enroll Now to secure your spot in this crucial training session and ensure your HIPAA compliance is robust and audit-ready.
https://conferencepanel.com/conference/hipaa-training-for-the-compliance-officer-2024-updates
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TEST BANK For Accounting Information Systems, 3rd Edition by Vernon Richardson, Verified Chapters 1 - 18, Complete Newest Version
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Here is summary of hypertension -
Hypertension, also known as high blood pressure, is a serious medical condition that occurs when blood pressure in the body's arteries is consistently too high. Blood pressure is the force of blood pushing against the walls of blood vessels as the heart pumps it. Hypertension can increase the risk of heart disease, brain disease, kidney disease, and premature death.
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Michigan HealthTech Market Map 2024. Includes 7 categories: Policy Makers, Academic Innovation Centers, Digital Health Providers, Healthcare Providers, Payers / Insurance, Device Companies, Life Science Companies, Innovation Accelerators. Developed by the Michigan-Israel Business Accelerator
3. NPUAP Formed Task Force January
2015
damaged soft tissue now labeled as
“pressure injury” replacing “pressure ulcer.”
because the term “injury” was more inclusive of
all 6 stages.
Change from Roman to Arabic numerals
Example: Stage ii is now Stage 2
Medical Device Related Pressure Injury
Mucosal Membrane Pressure Injury
What’s New?
4. localized damage to the
skin and underlying soft
tissue usually over a bony
prominence or related to a
medical or other device.
Can present as intact skin
or an open ulcer and may
be painful.
What is a Pressure Injury ?
5. a result of intense and/or prolonged pressure or
pressure in combination with shear.
What is a Pressure Injury ?
9. Stage 1 Definition :tage 1 Definition : OldOld vs.vs. NewNew
Old Stage I Pressure Ulcer :
Non-blanchable erythema
Intact skin with non-blanchable
redness of a localized area usually
over a bony prominence.
Darkly pigmented skin may not
have visible blanching; its color may
differ from the surrounding area.
The area may be painful, firm, soft,
warmer or cooler as compared to
adjacent tissue.
Stage 1 may be difficult to detect in
individuals with dark skin tones
New Stage 1 Pressure Injury:
Non-blanchable erythema of intact
skin
Intact skin with a localized area of
non-blanchable erythema, which
may appear differently in darkly
pigmented skin.
Presence of blanchable erythema
or changes in sensation,
temperature, or firmness may
precede visual changes.
Color changes do not include
purple or maroon
discoloration; these may indicate
deep tissue pressure injury.
10. Updated Stage 1 Definition :tage 1 Definition :
Intact skin with a localized area of non-
blanchable erythema, which may appear
differently in darkly pigmented skin.
Presence of blanchable erythema or changes
in sensation, temperature, or firmness may
precede visual changes.
Color changes do not include purple or maroon
discoloration; these may indicate deep tissue
pressure injury.
11.
12. Stage 1 pressure areas can
develop in as short as 30
minutes
Boggy
He
Cocc
yx
13.
14. Stage 2 Definition :Stage 2 Definition : OldOld vs.vs. NewNew
Old Stage II Pressure Ulcer
•Partial thickness loss of dermis
presenting as a shallow open ulcer
with a red pink wound bed, without
slough.
May also present as an intact or
open/ruptured serum-filled or sero-
sanguineous filled blister.
Presents as a shiny or dry shallow
ulcer without slough or bruising*.
This category should not be used to
describe skin tears, tape burns,
incontinence associated dermatitis,
maceration or excoriation.
*Bruising indicates deep tissue
injury
Stage 2 Pressure Injury:
Partial-thickness loss of skin with
exposed dermis. The wound bed is
viable, pink or red, moist, and may
also present as an intact or ruptured
serum-filled blister.
Adipose (fat) is not visible and
deeper tissues are not visible.
Granulation tissue, slough and
eschar are not present. These
injuries commonly result from
adverse microclimate and shear in
the skin over the pelvis and shear in
the heel.
This stage should not be used to
describe moisture associated skin
damage (MASD) or traumatic
wounds(skin tears, burns, abrasions).
15. Updated Stage 2 Definition :Updated Stage 2 Definition :
Stage 2 Pressure Injury:
Partial-thickness loss of skin with exposed dermis.
The wound bed is viable, pink or red, moist, and may
also present as an intact or ruptured serum-filled
blister.
Adipose (fat) is not visible and deeper tissues are not
visible. Granulation tissue, slough and eschar are not
present. These injuries commonly result from adverse
microclimate and shear in the skin over the pelvis and
shear in the heel.
This stage should not be used to describe moisture
associated skin damage (MASD) or traumatic
wounds(skin tears, burns, abrasions).
21. Stage 3 Definition :Stage 3 Definition : OldOld vs.vs. NewNew
Old Stage III Pressure Ulcer
Full thickness tissue loss.
Subcutaneous fat may be visible
but bone, tendon or muscle are not
exposed. Slough may be present but
does not obscure the depth of
tissue loss. May include
undermining and tunneling. The
depth of a Stage III pressure ulcer
varies by anatomical location. The
bridge of the nose, ear, occiput and
malleolus do not have (adipose)
subcutaneous tissue and Stage III
ulcers can be shallow. In contrast,
areas of significant adiposity can
develop extremely deep Stage III
pressure ulcers. Bone/tendon is not
visible or directly palpable.
Stage 3 Pressure Injury:
Full-thickness loss of skin, in which
adipose(fat) is visible in the ulcer and
granulation tissue and epibole(rolled
wound edges) are often present.
Slough and/or eschar may be visible.
The depth of tissue damage varies
by anatomical location; areas of
significant adiposity can develop
deep wounds. Undermining and
tunneling may occur. Fascia, muscle,
tendon, ligament, cartilage and/or
bone are not exposed. If slough or
eschar obscures the extent of tissue
loss this is an Unstageable pressure
Injury.
22. Updated Stage 3 Definition :Updated Stage 3 Definition :
Stage 3 Pressure Injury:
Full-thickness loss of skin, in which adipose(fat) is
visible in the ulcer and granulation tissue and
epibole(rolled wound edges) are often present. Slough
and/or eschar may be visible. The depth of tissue
damage varies by anatomical location; areas of
significant adiposity can develop deep wounds.
Undermining and tunneling may occur. Fascia,
muscle, tendon, ligament, cartilage and/or bone are
not exposed. If slough or eschar obscures the extent
of tissue loss this is an Unstageable pressure Injury.
26. Stage 4 Definition :Stage 4 Definition : OldOld vs.vs. NewNew
Old Stage Iv Pressure Ulcer
Full thickness tissue loss with
exposed bone, tendon or muscle.
Slough or eschar maybe present.
Often includes undermining and
tunneling. The depth of a Stage IV
pressure ulcer varies by anatomical
location. The bridge of the nose,
ear, occiput and malleolus do not
have (adipose) subcutaneous tissue
and these ulcers can be shallow.
Stage IV ulcers can extend into
muscle and/or supporting structures
(e.g., fascia, tendon or joint
capsule) making osteomyelitis or
osteitis likely to occur. Exposed
bone/muscle is visible or directly
palpable.
Stage 4 Pressure Injury:
Full-thickness skin and tissue loss
with exposed or directly palpable
fascia, muscle, tendon, ligament,
cartilage or bone in the ulcer. Slough
and/or eschar may be visible.
Epibole(rolled edges),undermining
and/or tunneling often occur. Depth
varies by anatomical location. If
slough or eschar obscures the extent
of tissue loss this is an
Unstageable Pressure Injury.
27. Updated Stage 4 DefinitionUpdated Stage 4 Definition
Stage 4 Pressure Injury:
Full-thickness skin and tissue loss with exposed or
directly palpable fascia, muscle, tendon, ligament,
cartilage or bone in the ulcer. Slough and/or eschar
may be visible. Epibole (rolled edges),undermining
and/or tunneling often occur. Depth varies by
anatomical location. If slough or eschar obscures the
extent of tissue loss this is an Unstageable Pressure
Injury.
32. If you can seeIf you can see
or feelor feel anyany
cartilagecartilage in thein the
wound,wound,
it isit is a Stage 4a Stage 4
pressure injury.pressure injury.
33.
34. Unstageable Definition :Unstageable Definition : OldOld vs.vs. NewNew
Old Unstageable Pressure
Ulcer: •Full thickness tissue loss in
which actual depth of the ulcer is
completely obscured by slough
(yellow, tan, gray, green or brown)
and/or eschar(tan, brown or black)
in the wound bed. Until enough
slough and/or eschar are removed
to expose the base of the wound,
the true depth cannot be
determined; but it will be either a
Stage III or IV. Stable (dry,
adherent, intact without erythema or
fluctuance) eschar on the heels
serves as “the body’s natural
(biological) cover” and should not
be removed.
Unstageable Full-Thickness
Pressure Injury:
Full-thickness skin and tissue loss in
which the extent of tissue damage
within the ulcer cannot be confirmed
because it is obscured by slough or
eschar. If slough or eschar is s
removed, a Stage 3 or Stage 4
pressure injury will be revealed.
Stable eschar( i.e. dry, adherent,
intact without erythema or fluctuance)
on an ischemic limb or the
heel(s)should not be softened or
removed.
35. Updated Unstageable Definition :Updated Unstageable Definition :
Unstageable Full-Thickness Pressure Injury:
Full-thickness skin and tissue loss in which the
extent of tissue damage within the ulcer cannot be
confirmed because it is obscured by slough or
eschar.
If slough or eschar is s removed, a Stage 3 or Stage
4 pressure injury will be revealed. Stable eschar( i.e.
dry, adherent, intact without erythema or fluctuance)
on an ischemic limb or the heel(s)should not be
softened or removed.
38. Deep Tissue Injury Definition :Deep Tissue Injury Definition :OldOld vs.vs. NewNew
Old DTI Ulcer: Purple or maroon
localized area of discolored intact
skin or blood-filled blister due to
damage of underlying soft tissue
from pressure and/or shear.
The area may be preceded by
tissue that is painful, firm, mushy,
boggy, warmer or cooler as
compared to adjacent tissue.
Evolution may include a thin blister
over a dark wound bed. The wound
may further evolve and become
covered by thin eschar. Evolution
may be rapid exposing additional
layers of tissue even with
optimal treatment
New Deep Tissue Pressure
Injury:
Intact or non-intact skin with localized
area of persistent non-blanchable
deep red, maroon, purple
discoloration or epidermal separation
revealing a dark wound bed or blood
filled blister. Pain and temperature
change
often precede skin color changes.
Discoloration may appear differently
in darkly pigmented skin. This injury
results from intense and/or prolonged
pressure and shear forces at the
bone-muscle interface. The wound
may evolve rapidly to reveal the
actual extent of tissue injury, or may
resolve without tissue loss.
41. Medical Device Related Definition:Medical Device Related Definition:OldOld vs.vs. NewNew
Old medical Devise
related pressure Ulcer:
Medical device
related pressure ulcers are
pressure ulcers that result
from the use of devices
designed and applied for
diagnostic or therapeutic
purposes. The resultant
pressure ulcer generally
closely conforms to the
pattern or shape of the
device.
New medical device related
pressure injury:
This describes an etiology.
Medical device related
pressure injuries result from
the use of devices designed
and applied for diagnostic or
therapeutic purposes. The
resultant pressure injury
generally conforms to the
pattern or shape of the device.
The injury should be staged
using the staging system.
42.
43. Mucosal Pressure Definition:Mucosal Pressure Definition:OldOld vs.vs. NewNew
Old Mucosal pressure
Ulcer:
Mucosal Pressure Ulcers are
pressure ulcers found on
mucous membranes with a
history of a medical device in
use at the location of the
ulcer.
New Mucousal Membrane
pressure injury:
Mucosal membrane pressure
injury is found on mucous
membranes with a history of a
medical device in use at the
location of the injury. Due to
the anatomy of the tissue these
injuries cannot be staged
47. New skin that is light
pink and shiny
In Stage 2 pressure ulcers,
epithelial tissue is seen in
the center and edges of the
ulcer.
In full thickness Stage 3 and
4 pressure ulcers, epithelial
tissue advances from the
edges of the wound.
Tissue Type:Tissue Type: EpithelialEpithelial
49. Non-viable yellow, tan, gray,
green or brown tissue;
usually moist, can be soft, stringy
and mucinous in texture.
Slough may be adherent to the
base of the wound or present in
clumps throughout the wound bed.
If slough is present in a pressure
injury-it is at a stage 3 or higher
Tissue Type:Tissue Type: SloughSlough
50.
51. Dead or devitalized tissue
that is hard or soft in
texture
usually black, brown, or tan in
color, and may appear scab
like.
usually firmly adherent to the
base of the wound and often
the sides/ edges of the wound.
Presence of eschar is a stage 4
or higher
Tissue Type-Tissue Type-EscharEschar
54. Due to venous insufficiency
Medial Aspect of the leg
Beefy Red
Jagged
Painless
may start with some kind of
minor trauma
Does not typically occur over
a bony prominence
VenousVenous Ulcer CharacteristicsUlcer Characteristics
56. Ischemia is the etiology but
pressure or trauma may be a
factor
frequently seen on the dorsum
(top) of the foot
Small, punctuated ulcers that are
usually well circumscribed
painful
Delayed capillary refill/pulse
Hairless extremities
skin is shiny, thin, dry, cold
ArterialArterial Ulcer CharacteristicsUlcer Characteristics
57. Usually occur on the bottom of the
foot.
May have discoloration in feet:
black, blue, or red
They precede over 80% of leg
amputations in the US
Diabetic Foot UlcersDiabetic Foot Ulcers
60. usually starts on the sacrum
May start as a Stage 2 and
rapidly become a Stage 3
or 4
may be shaped like a pear,
butterfly or horseshoe with
irregular borders
colors of red, yellow, black
or purple.
The life expectancy of the
sudden onset presentation
can be within 8-24 hours.
KennedyKennedy Ulcers-CharacteristicsUlcers-Characteristics
70. Contributing Factors::Contributing Factors::
Skin CareSkin Care
Manage moistureManage moisture
keep skin clean/ drykeep skin clean/ dry
pH balanced cleanserpH balanced cleanser
manage incontinencemanage incontinence
protect from urine/stool/perspirationprotect from urine/stool/perspiration
Minimize friction and shearMinimize friction and shear
Lift sheet/devicesLift sheet/devices
LIFT –do not drag/pull up in bedLIFT –do not drag/pull up in bed
71. Contributing Factors:Contributing Factors: PositioningPositioning
Frequent TurningFrequent Turning
avoid positioning on area of erythemaavoid positioning on area of erythema
Use foam wedges for 30 degree lateralUse foam wedges for 30 degree lateral
positioningpositioning
Maintain sheets without creasesMaintain sheets without creases
Avoid multiple layers of incontinence paddingAvoid multiple layers of incontinence padding
Protect/offload heelsProtect/offload heels
Pressure Reducing Support Surface for bedPressure Reducing Support Surface for bed
or chairor chair
72. Preventing Heel UlcersPreventing Heel Ulcers
National Pressure Ulcer Advisory Panel (NPUAP) directive:
“Ensure that the heels are free of the surface of the bed.
Heel-protection devices should elevate the heel completely (offload them)
in such a way as to distribute the weight of the leg along the calf."
“Total heel offloading is the only effective method for heel ulcer
prevention."
“Heel ulcers are the most common facility-acquired pressure ulcer in long-
term acute care facilities and second most common pressure ulcer overall."
73. Patients at low risk for heel pressure
ulcers/skin breakdown (These devices do
NOT provide pressure relief!)
74. Patients at moderate to highPatients at moderate to high
risk for heel pressurerisk for heel pressure
79. TreatmentTreatment
1. Optimize the host response by:
evaluating nutritional status/deficits;
stabilizing glycemic control
improving arterial blood flow
reducing immunosuppressant therapy if
possible.
2. Prevent contamination of the pressure ulcer.
3. Reduce bacterial load and biofilm
4. Use non-toxic topical antiseptics for a limited
time period
80. WarningsWarningsHydrogen peroxide is highly toxic to tissues even at low
concentrations14, 15 and should not be used as a preferred topical
antiseptic. Its use should be totally avoided in cavity wounds due to
the risk of surgical emphysema and gas embolus.
Iodine products should be avoided in patients with impaired renal
failure, history of thyroid disorders or known iodine sensitivity.
Sodium hypochlorite (Dakin’s solution) is cytotoxic at all
concentrations and should be used with caution, at concentrations no
greater than 0.025%, for short periods only when no other appropriate
option is available.
There is a risk of acidosis when acetic acid is used for extended
periods over large wound surface areas.
81. TreatmentTreatment 5. Consider the use of
medical-grade honey in
heavily contaminated or
infected pressure ulcers until
definitive debridement is
accomplished.
•Caution: Before applying a honey
dressing, ensure the individual is
not allergic to honey. Individuals
who have bee or bee stings allergies
are usually able to use properly
irradiated honey products.
82. TreatmentTreatment 6. Limit the use of topical
antibiotics on infected
pressure ulcers, except in
special situations where the
benefit to the patient
outweighs the risk of
antibiotic side effects and
resistance.
•In general, topical
antibiotics are not
recommended for treating
pressure ulcers.
83. TreatmentTreatment
7. Use systemic antibiotics for
individuals with clinical evidence of
systemic infection, such as positive
blood cultures, cellulitis, fasciitis,
osteomyelitis, systemic
inflammatory response syndrome
(SIRS), or sepsis.
84. Selecting Wound DressingsSelecting Wound Dressings
Select a wound dressing based on the:
1. ability to keep the wound bed moist
2. need to address bacterial bio burden
3. nature and volume of wound exudate
4. condition of the tissue in the ulcer bed
5. condition of periulcer skin
6. ulcer size, depth and location
7. presence of tunneling and/or undermining
8. goals of the individual with the ulcer
87. Deep Tissue InjuryDeep Tissue Injury
Intact or non-intact skin with localized area of
persistent non-blanchable deep red, maroon,
purple discoloration or epidermal separation
revealing a dark wound bed or blood filled blister.
88.
89. Stage 2 Pressure Injury
Partial-thickness loss of skin with exposed
dermis. The wound bed is viable, pink or red,
moist, and may also present as an intact or
ruptured serum-filled blister.
Adipose (fat) is not visible
and deeper tissues
are not visible.
Granulation tissue,
slough and eschar
are not present.
90.
91. Unstageble Full Thickness PressureUnstageble Full Thickness Pressure
InjuryInjury
Full-thickness skin and tissue loss in which the
extent of tissue damage within the ulcer cannot
be confirmed because it is obscured by slough
or eschar. If slough or
eschar is removed, a
Stage 3 or Stage 4
pressure injury
will be revealed.
92.
93. Stage 3 Pressure InjuryStage 3 Pressure Injury
Full-thickness loss of skin, in which adipose(fat)
is visible in the ulcer and granulation tissue and
epibole (rolled wound edges) are often present.
Slough and/or eschar may be visible.
94.
95. Deep Tissue Pressure InjuryDeep Tissue Pressure Injury
Intact or non-intact skin with localized area of
persistent non-blanchable deep red, maroon,
purple discoloration or epidermal separation
revealing a dark wound bed or blood filled
blister.
96.
97. Stage 4 Pressure InjuryStage 4 Pressure Injury
Full-thickness skin and tissue loss with exposed
or directly palpable fascia, muscle, tendon,
ligament, cartilage or bone in the ulcer. Slough
and/or eschar may be visible..
98.
99. Stage 1 Pressure InjuryStage 1 Pressure Injury
Non-blanchable erythema of intact skin
Intact skin with a localized area of non-
blanchable erythema, which may appear
differently in darkly pigmented skin.
101. ResourcesResources
National Pressure Ulcer Advisory Panel
www.nupap.org
•2014 Prevention and Treatment of Pressure Ulcers:
Clinical Practice Guideline (Quick Reference Guide
available as FREE download)
Association for the Advancement of Wound
Care
•http://aawconline.org/
•http://bradenscale.com/
Wound, Ostomy, Continence Nurses
Societywww.wocn.org
Editor's Notes
The National Pressure Ulcer Advisory Panel redefined the definition of a pressure injuries during the NPUAP 2016 Staging Consensus Conference that was held April 8-9, 2016 in Rosemont (Chicago), IL.
The updated staging definitions were presented at a meeting of over 400 professionals. Using a consensus format, Dr. Mikel Gray from the University of Virginia adeptly guided the Staging Task Force and meeting participants to consensus on the updated definitions through an interactive discussion and voting process. During the meeting, the participants also validated the new terminology using photographs.
The updated staging system includes the following definitions:
NPUAP Formed Task Force January 2015
damaged soft tissue now labeled as “pressure injury” replacing “pressure ulcer.”
The term “injury” was more inclusive of all 6 stages, as
Stage 1 is present as intact skin, as is Deep Tissue Pressure Injury, which has always used "injury" in the nomenclature
Medical Device Related Pressure Injury:
•This describes an etiology. Medical device related pressure injuries result from the use of devices designed and applied for diagnostic or therapeutic purposes. The resultant pressure injury generally conforms to the pattern or shape of the device. The injury should be staged using the staging system.
Mucosal Membrane Pressure Injury:
•Mucosal membrane pressure injury is found on mucous membranes with a history of a medical device in use at the location of the injury. Due to the anatomy of the tissue these injuries cannot be staged.
The National Pressure Ulcer Advisory Panel redefined the definition of a pressure injuries during the NPUAP 2016 Staging Consensus Conference that was held April 8-9, 2016 in Rosemont (Chicago), IL.
The updated staging definitions were presented at a meeting of over 400 professionals. Using a consensus format, Dr. Mikel Gray from the University of Virginia adeptly guided the Staging Task Force and meeting participants to consensus on the updated definitions through an interactive discussion and voting process. During the meeting, the participants also validated the new terminology using photographs.
The updated staging system includes the following definitions:
Pressure Injury:A pressure injury is localized damage to the skin and underlying soft tissue usually over a bony prominence or related to a medical or other device. The injury can present as intact skin or an open ulcer and may be painful.
The injury occurs as a result of intense and/or prolonged pressure or pressure in combination with shear. The tolerance of soft tissue for pressure and shear may also be affected by microclimate, nutrition, perfusion, co-morbidities and condition of the soft tissue.
Considered pre injury
Here we go..
Full-thickness skin and tissue loss with exposed or directly palpable fascia, muscle, tendon, ligament, cartilage or bone in the ulcer.
If necrotic tissue, subcutaneous tissue, granulation tissue, fascia, muscle or other underlying structures are visible, this indicates a full thickness pressure injury (Unstageable, Stage 3 or Stage 4). Do not use DTPI to describe vascular, traumatic, neuropathic, or dermatologic conditions.
On tongue from ng tube
Inflammation- (2-3 days)
consists of a vascular and cellular response
Proliferation –( 2-3 weeks)
Begins at the time of injury
Rebuilding & revascularization begins
Maturation /remodeling Stage- (2-3 yrs)
Depositing of scar tissue
The body attempts to contract/close the wound
http://www.shieldhealthcare.com/community/wp-content/uploads/2015/07/Stages-of-Healing_image.jpg
Pink and shiny in dark skin as well
The wound may start with some kind of minor trauma, such as hitting the leg on a wheelchair.
The wound does not typically occur over a bony prominence, and pressure forces play virtually no role in the development of the ulcer
Ischemia is the major etiology of these ulcers, but pressure may be a factor
The wound may also start with some kind of minor trauma, such as hitting the leg on a wheelchair
Doppler, waveform, Ankle Brachial Indices (ABI) and Transcutaneous Oxygen Pressure measurements (TCPO2) to aid in your diagnosis. Duplex scanning and arteriograms may also be performed if indicated.
Diabetes can damage the nerves of the legs and feet so that they may not feel a blister or sore when it begins to appear. If undetected, the sore may become larger and infected
Diabetic foot ulcers are sores that occur on the feet of people with Type 1 or Type 2 Diabetes
Up to 25% of people with diabetes develop foot problems.
Diabetic foot ulcers usually occur on the bottom of the foot.
They precede over 80% of leg amputations in the US.
May have discoloration in feet: black, blue, or red
It usually starts out as a blister or a Stage II and can rapidly progresses to a Stage III or a Stage IV.
In the beginning it can look much like an abrasion as if someone took the patient and drug his or her bottom along a black top driveway.
It can become deeper and starts to turn colors.
The colors can start out as a red/purple area then turn to yellow and then black.
The life expectancy of the sudden onset presentation can be within 8-24 hours.
The two statements you hear most are:
1. “Oh, my gosh, that was not there the other day.”2. “I worked Friday, it was not there then, I was off the weekend and when I came back on Monday there it was.
Urine
Stool - especially liquid stool
Perspiration
Primarily consisting of water, perspiration also contains urea, glucose, sodium, and chloride.
Chronic perspiration most often results in MASD when in a skin fold, where evaporation is minimized.
Effluent from an ostomy
Wound Exudate
Consider applying a polyurethane foam dressing to bony prominences (e.g., heels, sacrum) for the prevention of pressure ulcers in anatomical areas frequently subjected to friction and shear.
Prophylactic dressings differ in their qualities; therefore it is important to select a dressing that is appropriate to the individual and the clinical use.
http://www.molnlycke.sg/PageFiles/69800/pressure-distribution.png
Microclimate Control
The use of specialized surfaces that come into contact with the skin may be able to alter the microclimate by changing the rate of evaporation of moisture and the rate at which heat dissipates from the skin.
http://www.arjohuntleigh.co.uk/PageFiles/1366/SKINIQ.jpg
Electrical Stimulation of the Muscles
•There is emerging evidence that electrical stimulation (ES) induces intermittent tetanic muscle contractions and reduces the risk of pressure ulcer development in at risk body parts
Many systemic factors contribute to the development of pressure ulcers. If these same factors can be improved, the individual’s intrinsic ability to fight infection can usually also be improved3. Reduce bacterial load and biofilm in the pressure ulcer as outlined in the Wound Care: Cleansing and Wound Care: Debridement sections
Consider the use of tissue appropriate strength, non-toxic topical antiseptics for a limited time period to control bacterial bio burden
Judicious use of systemic antibiotics remains an important consideration