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Tinea Capitis
Mohammed Nabil J AlAli
5th Year Medical Student
At King Faisal University
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Group B (210006209)

Page 1
OUTLINES :
- OVERVIEW
- ETIOLOGY
- CLINICAL FEATURES
- INVESTIGATIONS
- DIAGNOSIS
- COMPLICATIONS
- TREATMENTS
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Page 2
OVERVIEW
• It is a disease caused by superficial fungal
infection of the skin (dermatophyte) of the
scalp, eyebrows, and eyelashes, with a
propensity for attacking hair shafts and
follicles .
• Several synonyms are including ringworm of
the scalp and tinea tonsurans.
• The term tinea meant parasitic infestation of
the skin.
• Tinea capitis is caused by fungi of species of
genera Trichophyton(T) and Microsporum(M)
fungi. Powerpoint Templates
Page 3
• almost always occurs in small children
and less frequently seen in adults
• The incidence of tinea capitis is increasing.

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Page 4
ETIOLOGY
Tinea capitis is classified according to how the
fungus invades the hair shaft.

1- Ectothrix infection

due to infection with 
M. canis, M. audouinii, M. distortum, M. ferrugineum,
M. gypseum, M. nanum, and T. verrucosum.

The fungal branches (hyphae) and spores
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(arthroconidia) cover the outside of the hair.
Page 5
2- Endothrix infection

results from infection with              
T. tonsurans, T. violaceum
and T. soudanense.

The hair shaft is filled with
fungal branches (hyphae) and
spores (arthroconidia).

3- Favus

( tinea favosa )
caused by T.schoenleinii  infection,
which results in a honeycomb
destruction of the hair shaft

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Page 6
CLINICAL FREATURES
-Tinea capitis is most prevalent between 3 and 7
years of age.
- It is slightly more common in boys than girls.
Infection by T. tonsurans may occur in adults.

Tinea capitis may present in several ways :
- Dry scaling – like dandruff but usually with moth-eaten hair loss
- Black dots – the hairs are broken off at the scalp surface,
which is scaly
- Smooth areas of hair loss
- Kerion – very inflamed mass, like an abscess
- Favus – yellow crusts and matted hair
- Carrier state no symptoms and only mild scaling (T. tonsurans)
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Page 7
Tinea capitis can occur in three distinctly
different forms:
gray patch ,black dot and favus .
Gray patch tinea capitis (GPTC)
It begins with an erythematous, scaling,
well-demarcated patch on the scalp that
spreads centrifugally for a few weeks or
months, ceases to spread, and persists
indefinitely, sometimes for years

Round grayish, scaly plaques with
progressive expanding
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Page 8
Black dot tinea capitis (BDTC)
usually begins as an asymptomatic,
erythematous, scaling patch on the
scalp, which slowly enlarges.
Lesions may be single or multiple.
Early lesions are easily overlooked
and the disease is not usually
noticed until areas of alopecia
become evident.

A round patch of alopecia with
overlying scale and "black dots" at the
sites of follicular openings is present.

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Most often caused by Trichophyton tonsurans (endothrix) Page 9
Favus (tinea favosa )
Perifollicular erythema on the
scalp, which progresses to the
characteristic finding

of concave, cup-shaped
yellow crusts called scutula.
Scutula are composed of fungi,
neutrophils, dried serum, and
epidermal cells.
It has unpleasant smell .

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most often due toTrichophyton schoenleinii (endothrix)

Page 10
INVESTIGATIONS
- Ectothrix infections can be
identified by Woods light
(long wave ultraviolet light)
- Endothrix infections do not
fluoresce with Woods light.

- Spores are identifiable by KOH
examination of the hair shaft, not by
KOH scraping of the scale, though
hyphae may be seen on occasion

- Culture on Sabouraud's medium
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Page 11
DIAGNOSIS
- Tinea capitis is suspected if there is a combination
of scale and bald patches.
- Wood's light fluorescence is helpful but not
diagnostic
- The diagnosis of tinea capitis should be
confirmed by microscopy and culture of skin
scrapings and hair pulled out by the roots.
- Sometimes, diagnosis is made on skin biopsy
showing characteristic
histopathological features of tinea capitis.
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Page 12
COMPLICATIONS
-Severe hair loss.
-Scarring alopecia “bald areas”
( Untreated kerion and favus).

- Psychological impact (ridicule,
bullying, isolation, emotional
disturbance,
family
disruption).
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Page 13
TREATMENTS
- Treatment of carriers is necessary to prevent
spread of infection.
- Antifungal shampoo twice weekly for four
weeks may be sufficient but if cultures remain
positive, oral treatment is recommended.
Suitable shampoos include:
- 2.5% selenium sulfide
-1% to 2% zinc pyrithione
- Povidone-iodine
- 2% Ketoconazole
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Page 14
Griseofulvin (20 to 25 mg/kg/day for 6-12 weeks) has a
long history of safety and efficacy in tinea capitis in
children.
Terbinafine tablet treatment schedules are based on
weight :
10 to 20 kg: 62.5 mg daily for two to four weeks
20 to 40 kg: 125 mg daily for two to four weeks
Above 40 kg: 250 mg daily for two to four weeks
Oral itraconazole, and fluconazole are additional therapeutic
options that allow shorter courses of treatment
The duration is 4 to 6 weeks .

However, these medications are not always successful
and it may be necessary to try another agent.
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Page 15
Any Question ?
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Page 16
REFERENCES
- http://www.uptodate.com
- http://dermnetnz.org/fungal/tinea-capitis.html
- http://emedicine.medscape.com/article/10913
51-overview
- http://www.patient.co.uk/doctor/tinea-capitis
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Page 17
Thank you
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Page 18

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Tinea capitis

  • 1. Tinea Capitis Mohammed Nabil J AlAli 5th Year Medical Student At King Faisal University Powerpoint Templates Group B (210006209) Page 1
  • 2. OUTLINES : - OVERVIEW - ETIOLOGY - CLINICAL FEATURES - INVESTIGATIONS - DIAGNOSIS - COMPLICATIONS - TREATMENTS Powerpoint Templates Page 2
  • 3. OVERVIEW • It is a disease caused by superficial fungal infection of the skin (dermatophyte) of the scalp, eyebrows, and eyelashes, with a propensity for attacking hair shafts and follicles . • Several synonyms are including ringworm of the scalp and tinea tonsurans. • The term tinea meant parasitic infestation of the skin. • Tinea capitis is caused by fungi of species of genera Trichophyton(T) and Microsporum(M) fungi. Powerpoint Templates Page 3
  • 4. • almost always occurs in small children and less frequently seen in adults • The incidence of tinea capitis is increasing. Powerpoint Templates Page 4
  • 5. ETIOLOGY Tinea capitis is classified according to how the fungus invades the hair shaft. 1- Ectothrix infection due to infection with  M. canis, M. audouinii, M. distortum, M. ferrugineum, M. gypseum, M. nanum, and T. verrucosum. The fungal branches (hyphae) and spores Powerpoint Templates (arthroconidia) cover the outside of the hair. Page 5
  • 6. 2- Endothrix infection results from infection with               T. tonsurans, T. violaceum and T. soudanense. The hair shaft is filled with fungal branches (hyphae) and spores (arthroconidia). 3- Favus ( tinea favosa ) caused by T.schoenleinii  infection, which results in a honeycomb destruction of the hair shaft Powerpoint Templates Page 6
  • 7. CLINICAL FREATURES -Tinea capitis is most prevalent between 3 and 7 years of age. - It is slightly more common in boys than girls. Infection by T. tonsurans may occur in adults. Tinea capitis may present in several ways : - Dry scaling – like dandruff but usually with moth-eaten hair loss - Black dots – the hairs are broken off at the scalp surface, which is scaly - Smooth areas of hair loss - Kerion – very inflamed mass, like an abscess - Favus – yellow crusts and matted hair - Carrier state no symptoms and only mild scaling (T. tonsurans) Powerpoint Templates Page 7
  • 8. Tinea capitis can occur in three distinctly different forms: gray patch ,black dot and favus . Gray patch tinea capitis (GPTC) It begins with an erythematous, scaling, well-demarcated patch on the scalp that spreads centrifugally for a few weeks or months, ceases to spread, and persists indefinitely, sometimes for years Round grayish, scaly plaques with progressive expanding Powerpoint Templates Page 8
  • 9. Black dot tinea capitis (BDTC) usually begins as an asymptomatic, erythematous, scaling patch on the scalp, which slowly enlarges. Lesions may be single or multiple. Early lesions are easily overlooked and the disease is not usually noticed until areas of alopecia become evident. A round patch of alopecia with overlying scale and "black dots" at the sites of follicular openings is present. Powerpoint Templates Most often caused by Trichophyton tonsurans (endothrix) Page 9
  • 10. Favus (tinea favosa ) Perifollicular erythema on the scalp, which progresses to the characteristic finding of concave, cup-shaped yellow crusts called scutula. Scutula are composed of fungi, neutrophils, dried serum, and epidermal cells. It has unpleasant smell . Powerpoint Templates most often due toTrichophyton schoenleinii (endothrix) Page 10
  • 11. INVESTIGATIONS - Ectothrix infections can be identified by Woods light (long wave ultraviolet light) - Endothrix infections do not fluoresce with Woods light. - Spores are identifiable by KOH examination of the hair shaft, not by KOH scraping of the scale, though hyphae may be seen on occasion - Culture on Sabouraud's medium Powerpoint Templates Page 11
  • 12. DIAGNOSIS - Tinea capitis is suspected if there is a combination of scale and bald patches. - Wood's light fluorescence is helpful but not diagnostic - The diagnosis of tinea capitis should be confirmed by microscopy and culture of skin scrapings and hair pulled out by the roots. - Sometimes, diagnosis is made on skin biopsy showing characteristic histopathological features of tinea capitis. Powerpoint Templates Page 12
  • 13. COMPLICATIONS -Severe hair loss. -Scarring alopecia “bald areas” ( Untreated kerion and favus). - Psychological impact (ridicule, bullying, isolation, emotional disturbance, family disruption). Powerpoint Templates Page 13
  • 14. TREATMENTS - Treatment of carriers is necessary to prevent spread of infection. - Antifungal shampoo twice weekly for four weeks may be sufficient but if cultures remain positive, oral treatment is recommended. Suitable shampoos include: - 2.5% selenium sulfide -1% to 2% zinc pyrithione - Povidone-iodine - 2% Ketoconazole Powerpoint Templates Page 14
  • 15. Griseofulvin (20 to 25 mg/kg/day for 6-12 weeks) has a long history of safety and efficacy in tinea capitis in children. Terbinafine tablet treatment schedules are based on weight : 10 to 20 kg: 62.5 mg daily for two to four weeks 20 to 40 kg: 125 mg daily for two to four weeks Above 40 kg: 250 mg daily for two to four weeks Oral itraconazole, and fluconazole are additional therapeutic options that allow shorter courses of treatment The duration is 4 to 6 weeks . However, these medications are not always successful and it may be necessary to try another agent. Powerpoint Templates Page 15
  • 16. Any Question ? Powerpoint Templates Page 16
  • 17. REFERENCES - http://www.uptodate.com - http://dermnetnz.org/fungal/tinea-capitis.html - http://emedicine.medscape.com/article/10913 51-overview - http://www.patient.co.uk/doctor/tinea-capitis Powerpoint Templates Page 17