Brain tumor is an abnormal growth of the tissue in the brain.
The brain tumors can be mainly divided into two primary brain tumors and secondary/metastatic brain tumor
Edward Fohrman | Anesthetic Considerations in Vascular Neurosurgery Edward Fohrman
Edward Fohrman discusses what to take into consideration during vascular neurosurgery. Dr. Fohrman is the CEO of Fohrman Anesthesia Services & Consulting, Inc., which he founded in 2010.
Visit EdwardFohrman.com for more.
Edward Fohrman | Anesthetic Considerations for Intracranial TumorsEdward Fohrman
This document discusses anesthetic considerations for patients undergoing surgery for intracranial tumors. It covers how brain tumors present clinically, the effects of increased intracranial pressure on cerebral blood flow and herniation, techniques for controlling ICP such as hyperventilation and osmotic diuretics, and special considerations for anesthetic induction and positioning for different types of neurosurgeries.
Edward Fohrman | Neuroanesthesia in NeurotraumaEdward Fohrman
Edward Fohrman, anesthesiologist extraordinaire, describes how to use neuroanesthesia when it comes to neurotrauma in this presentation for one of his lectures.
Visited EdwardFohrman.com for more information!
This document discusses anesthesia considerations for neurosurgery patients. It covers common neurosurgical procedures, intracranial hypertension, cerebral edema, and the goals of anesthesia which include maintaining stable intracranial pressure and hemodynamics. It then focuses on anesthesia management for patients undergoing craniotomy for mass lesions, including preoperative evaluation and preparation, induction, maintenance with goals of optimal surgical conditions and neurological protection, and controlled emergence.
Brain tumor is an abnormal growth of the tissue in the brain.
The brain tumors can be mainly divided into two primary brain tumors and secondary/metastatic brain tumor
Edward Fohrman | Anesthetic Considerations in Vascular Neurosurgery Edward Fohrman
Edward Fohrman discusses what to take into consideration during vascular neurosurgery. Dr. Fohrman is the CEO of Fohrman Anesthesia Services & Consulting, Inc., which he founded in 2010.
Visit EdwardFohrman.com for more.
Edward Fohrman | Anesthetic Considerations for Intracranial TumorsEdward Fohrman
This document discusses anesthetic considerations for patients undergoing surgery for intracranial tumors. It covers how brain tumors present clinically, the effects of increased intracranial pressure on cerebral blood flow and herniation, techniques for controlling ICP such as hyperventilation and osmotic diuretics, and special considerations for anesthetic induction and positioning for different types of neurosurgeries.
Edward Fohrman | Neuroanesthesia in NeurotraumaEdward Fohrman
Edward Fohrman, anesthesiologist extraordinaire, describes how to use neuroanesthesia when it comes to neurotrauma in this presentation for one of his lectures.
Visited EdwardFohrman.com for more information!
This document discusses anesthesia considerations for neurosurgery patients. It covers common neurosurgical procedures, intracranial hypertension, cerebral edema, and the goals of anesthesia which include maintaining stable intracranial pressure and hemodynamics. It then focuses on anesthesia management for patients undergoing craniotomy for mass lesions, including preoperative evaluation and preparation, induction, maintenance with goals of optimal surgical conditions and neurological protection, and controlled emergence.
This document provides information about stroke, including definitions, statistics, risk factors, signs and symptoms, treatments, and the stroke program at PGI, Chd. Some key points:
- Stroke is defined as a sudden loss of brain function caused by an interruption of blood flow to the brain. It is the second most common cause of death worldwide.
- India has a high burden of stroke, with over 5000 new cases reported daily. Risk factors include hypertension, diabetes, smoking, heart disease, prior stroke or TIA, and high cholesterol.
- Signs of stroke include sudden numbness, confusion, vision problems, trouble walking or talking. The acronym FAST is used to help remember common
Supportive management in neurological icuNeurologyKota
This document discusses neurointensive care, which aims to treat and prevent brain injury. It describes the role of the neurointensivist in comprehensively managing neurologic status while integrating knowledge of other organ systems. Various conditions treated in neurointensive care units are listed, along with assessments of neurologic function and scales used to evaluate levels of consciousness, motor response, and brainstem reflexes. Monitoring techniques and their indications are also outlined.
Current strategies for cerebral protection during planned cerebral ischemia include hypothermia, colloidal volume expansion, induced hypertension, and barbiturate coma. Hypothermia between 34-35°C is an effective cerebral protector. Colloidal volume expanders are preferable to crystalloids. Induced hypertension can be achieved short-term with phenylephrine or long-term with dopamine. Barbiturate coma decreases cerebral metabolism and intracranial pressure, but carries risks of hemodynamic and respiratory depression that require intensive monitoring. The goal is to maintain intracranial pressure below 20mmHg and cerebral perfusion pressure above 70mmHg.
This document provides an overview and update on issues in neuroanesthesia. It discusses recurrent issues such as patient positioning, monitoring, fluid management and more that have not changed significantly over time. It also reviews cerebral physiology concepts like blood flow regulation and the effects of anesthetic agents. The document outlines current surgical trends like minimally invasive procedures and equipment trends like intraoperative CT. It concludes by emphasizing the importance of multidisciplinary team training to continually improve neuroanesthesia care.
This document discusses the anaesthetic management considerations for supratentorial brain tumours. It begins with an overview of common brain tumour types and surgeries. Key factors include maintaining cerebral homeostasis, minimizing brain retraction, reducing intracranial pressure, and early postoperative awakening. Specific techniques covered are osmotic agents, steroids, hyperventilation, fluid management, positioning, and hemodynamic control. Close monitoring of vital signs, gases, glucose and electrolytes is emphasized due to the risks of pressure effects, seizures, and other complications.
The document discusses anaesthetic management for neurosurgery. It outlines how cerebral blood flow is regulated and the importance of maintaining cerebral perfusion pressure and intracranial pressure. The goals of anaesthesia are to provide optimal surgical conditions while maintaining stable haemodynamics and brain oxygenation levels. Common procedures are described along with considerations for preoperative assessment, induction, maintenance of anaesthesia and fluid management during craniotomy to minimize risks to the patient.
1) Neurological injury is a leading cause of death for patients who experience cardiac arrest and are successfully resuscitated. Only 30% of eligible patients receive post-arrest targeted temperature management (TTM), formerly known as therapeutic hypothermia.
2) TTM involves lowering a patient's body temperature to 32-34°C for 24 hours after resuscitation to reduce neurological injury from global hypoxic insult during cardiac arrest. Proper sedation, electrolyte monitoring, and a slow rewarming period are important aspects of TTM.
3) While TTM is the standard of care, additional neuroprotective strategies are being studied, including pharmacological approaches targeting cell death pathways and modulation of oxygen free radicals
Research guru and PI for the ARISE study, college examiner and semi-professional forrest-based carpenter, Anthony always gives a fascinating talk. This time he gives an intelligent and considered breakdown on the nebulous topic of cerebral protection.
Intracranial hemorrhages account for 8-11% of all acute strokes and have a high mortality rate. The main causes are hypertension, amyloid angiopathy, AVMs, anticoagulation, and tumors. Management involves stabilizing the patient, controlling blood pressure, stabilizing the clot, managing cerebral edema and seizures. Surgery is generally not beneficial except for cerebellar hemorrhages. Clinical trials have found no clear benefit of aggressive blood pressure control or clot evacuation surgery over medical management alone.
This document discusses intracranial pressure and cerebral edema in the neuro-ICU setting. It covers how patients with brain injuries present, mechanisms of primary and secondary brain injury, pathophysiology of increased intracranial pressure and cerebral edema, imaging techniques like CT scans to diagnose brain injuries, and guidelines around monitoring intracranial pressure in severe traumatic brain injury patients.
This document discusses increased intracranial pressure (ICP). It begins by describing the components inside the skull - brain, blood, and cerebrospinal fluid. ICP is the total pressure from these components. Compensatory mechanisms aim to maintain a constant cerebral blood flow. Causes of increased ICP include brain injuries, tumors, and edema. Clinical manifestations include headache, vomiting, and altered vital signs. Diagnostic tests include CT, MRI, and direct ICP measurement. Management focuses on treating the underlying cause, maintaining perfusion, and reducing CSF and blood volume through interventions like osmotic diuretics, hyperventilation, and surgical decompression when needed.
1) Cerebral protection aims to improve neurological outcomes in patients at risk of cerebral ischemia through preemptive therapeutic interventions to prevent further brain damage and reverse existing damage.
2) Various pharmacological and non-pharmacological interventions can be used for cerebral protection, including hypothermia, hypertension, osmotherapy, barbiturate coma, and inhaled anesthetics. These interventions work to maintain cerebral perfusion and oxygenation while minimizing intracranial pressure and secondary brain injury.
3) Careful monitoring of patients is important for cerebral protection, including intracranial pressure, oxygenation, metabolism, hemodynamics, and electrical activity. Targeted interventions are aimed at preventing increases in intracran
Emergency department neurosurgical admissionsSCGH ED CME
This document provides an overview of common emergency neurosurgical presentations and indications for surgical intervention. It discusses the assessment of comatose patients, including the Glasgow Coma Scale. It then covers various neurosurgical topics like cranial trauma, vascular neurosurgery including stroke and subarachnoid hemorrhage, neuro-oncology, hydrocephalus, and spinal surgery. For each topic, it outlines clinical criteria for determining if surgical intervention is required.
Mechanisms of cerebral injury and cerebral protectionDr Kumar
This document discusses mechanisms of cerebral injury and cerebral protection. It provides details on cerebral physiology including metabolism, blood flow, regulation of blood flow, and factors that influence blood flow such as perfusion pressure, autoregulation, respiratory gas tensions, temperature, viscosity, and autonomic influences. It also discusses intracranial pressure, signs of increased ICP, assessment of injury severity, and strategies and principles for cerebral protection including maintaining oxygen supply and reducing increases in ICP, cerebral metabolic rate, and cell damage. The effects of various anesthetic drugs on cerebral blood flow, metabolism, and injury are also summarized.
Anesthetic management of carotid endarterectomy [autosaved] 2Arun Aru
1. The document describes the anesthetic management of a carotid endarterectomy procedure in a 54-year-old male with a history of stroke and risk factors including diabetes and hypertension.
2. Intraoperatively, the patient was induced with thiopental and intubated, and anesthesia was maintained with desflurane. Monitoring included arterial and central lines.
3. The endarterectomy procedure involved clamping of the internal carotid artery and removal of atherosclerotic plaque from the vessel wall via placement of a carotid stent.
This document discusses the management of intracranial pressure and cerebral edema in neurocritical care patients. It covers topics such as how patients typically present with brain injuries, important caveats in neurological examinations, principles of cerebral resuscitation, mechanisms of primary and secondary brain injury, the pathophysiology over time, imaging techniques including CT scans and MRI, monitoring techniques like intracranial pressure monitoring, and treatment approaches like the use of hyperosmolar therapy with mannitol or hypertonic saline. The goal is to prevent secondary brain injury after the initial primary injury occurs.
1. The document discusses intracranial pressure (ICP), cerebrospinal fluid (CSF) circulation and compensation mechanisms when ICP increases. It defines normal ICP and the factors that affect it, including the Monro-Kellie doctrine.
2. Symptoms of increased ICP are described, from early signs like headache to late signs like herniation and changes in vital signs. Different types of herniation are explained.
3. Methods for monitoring ICP are summarized, including invasive techniques like intraventricular and subdural monitors and non-invasive options. Indications for ICP monitoring include severe head injuries with abnormal CT scans or certain risk factors.
Hypothyroidism and hyperthyroidism have significant clinical effects. Both should be optimized. Anesthesia providers should be able to diagnose and manage.
This document provides a history of the development of neuroanesthesia. It discusses early discoveries and practices from ancient times through the 19th century. It then summarizes major advances in the 20th century related to anesthetic agents, monitoring, airway management, and techniques to protect the brain during surgery. These include the development of muscle relaxants, inhalational agents, intravenous medications, hypothermia, and methods to prevent increases in intracranial pressure and glucose levels.
Extradural hematoma, also known as epidural hematoma, is a collection of blood between the skull and the dura mater. It typically results from arterial bleeding, usually from the middle meningeal artery. Presentation includes an initial loss of consciousness followed by a lucid interval and then deterioration in consciousness. Treatment involves prompt surgery if signs of mass effect or herniation are present. Surgical evacuation aims to remove the clot and achieve hemostasis to prevent reaccumulation. Outcomes are generally good if diagnosed and treated quickly, but mortality increases with delay in treatment or presence of additional intracranial injuries.
Head injuries top the list of trauma patienrts coming to the casualty. The condition has to be immediately assessed and investigated. Depending upon the findings prompt medical or neurosurgical treatment has to be administered.
This document provides information about stroke, including definitions, statistics, risk factors, signs and symptoms, treatments, and the stroke program at PGI, Chd. Some key points:
- Stroke is defined as a sudden loss of brain function caused by an interruption of blood flow to the brain. It is the second most common cause of death worldwide.
- India has a high burden of stroke, with over 5000 new cases reported daily. Risk factors include hypertension, diabetes, smoking, heart disease, prior stroke or TIA, and high cholesterol.
- Signs of stroke include sudden numbness, confusion, vision problems, trouble walking or talking. The acronym FAST is used to help remember common
Supportive management in neurological icuNeurologyKota
This document discusses neurointensive care, which aims to treat and prevent brain injury. It describes the role of the neurointensivist in comprehensively managing neurologic status while integrating knowledge of other organ systems. Various conditions treated in neurointensive care units are listed, along with assessments of neurologic function and scales used to evaluate levels of consciousness, motor response, and brainstem reflexes. Monitoring techniques and their indications are also outlined.
Current strategies for cerebral protection during planned cerebral ischemia include hypothermia, colloidal volume expansion, induced hypertension, and barbiturate coma. Hypothermia between 34-35°C is an effective cerebral protector. Colloidal volume expanders are preferable to crystalloids. Induced hypertension can be achieved short-term with phenylephrine or long-term with dopamine. Barbiturate coma decreases cerebral metabolism and intracranial pressure, but carries risks of hemodynamic and respiratory depression that require intensive monitoring. The goal is to maintain intracranial pressure below 20mmHg and cerebral perfusion pressure above 70mmHg.
This document provides an overview and update on issues in neuroanesthesia. It discusses recurrent issues such as patient positioning, monitoring, fluid management and more that have not changed significantly over time. It also reviews cerebral physiology concepts like blood flow regulation and the effects of anesthetic agents. The document outlines current surgical trends like minimally invasive procedures and equipment trends like intraoperative CT. It concludes by emphasizing the importance of multidisciplinary team training to continually improve neuroanesthesia care.
This document discusses the anaesthetic management considerations for supratentorial brain tumours. It begins with an overview of common brain tumour types and surgeries. Key factors include maintaining cerebral homeostasis, minimizing brain retraction, reducing intracranial pressure, and early postoperative awakening. Specific techniques covered are osmotic agents, steroids, hyperventilation, fluid management, positioning, and hemodynamic control. Close monitoring of vital signs, gases, glucose and electrolytes is emphasized due to the risks of pressure effects, seizures, and other complications.
The document discusses anaesthetic management for neurosurgery. It outlines how cerebral blood flow is regulated and the importance of maintaining cerebral perfusion pressure and intracranial pressure. The goals of anaesthesia are to provide optimal surgical conditions while maintaining stable haemodynamics and brain oxygenation levels. Common procedures are described along with considerations for preoperative assessment, induction, maintenance of anaesthesia and fluid management during craniotomy to minimize risks to the patient.
1) Neurological injury is a leading cause of death for patients who experience cardiac arrest and are successfully resuscitated. Only 30% of eligible patients receive post-arrest targeted temperature management (TTM), formerly known as therapeutic hypothermia.
2) TTM involves lowering a patient's body temperature to 32-34°C for 24 hours after resuscitation to reduce neurological injury from global hypoxic insult during cardiac arrest. Proper sedation, electrolyte monitoring, and a slow rewarming period are important aspects of TTM.
3) While TTM is the standard of care, additional neuroprotective strategies are being studied, including pharmacological approaches targeting cell death pathways and modulation of oxygen free radicals
Research guru and PI for the ARISE study, college examiner and semi-professional forrest-based carpenter, Anthony always gives a fascinating talk. This time he gives an intelligent and considered breakdown on the nebulous topic of cerebral protection.
Intracranial hemorrhages account for 8-11% of all acute strokes and have a high mortality rate. The main causes are hypertension, amyloid angiopathy, AVMs, anticoagulation, and tumors. Management involves stabilizing the patient, controlling blood pressure, stabilizing the clot, managing cerebral edema and seizures. Surgery is generally not beneficial except for cerebellar hemorrhages. Clinical trials have found no clear benefit of aggressive blood pressure control or clot evacuation surgery over medical management alone.
This document discusses intracranial pressure and cerebral edema in the neuro-ICU setting. It covers how patients with brain injuries present, mechanisms of primary and secondary brain injury, pathophysiology of increased intracranial pressure and cerebral edema, imaging techniques like CT scans to diagnose brain injuries, and guidelines around monitoring intracranial pressure in severe traumatic brain injury patients.
This document discusses increased intracranial pressure (ICP). It begins by describing the components inside the skull - brain, blood, and cerebrospinal fluid. ICP is the total pressure from these components. Compensatory mechanisms aim to maintain a constant cerebral blood flow. Causes of increased ICP include brain injuries, tumors, and edema. Clinical manifestations include headache, vomiting, and altered vital signs. Diagnostic tests include CT, MRI, and direct ICP measurement. Management focuses on treating the underlying cause, maintaining perfusion, and reducing CSF and blood volume through interventions like osmotic diuretics, hyperventilation, and surgical decompression when needed.
1) Cerebral protection aims to improve neurological outcomes in patients at risk of cerebral ischemia through preemptive therapeutic interventions to prevent further brain damage and reverse existing damage.
2) Various pharmacological and non-pharmacological interventions can be used for cerebral protection, including hypothermia, hypertension, osmotherapy, barbiturate coma, and inhaled anesthetics. These interventions work to maintain cerebral perfusion and oxygenation while minimizing intracranial pressure and secondary brain injury.
3) Careful monitoring of patients is important for cerebral protection, including intracranial pressure, oxygenation, metabolism, hemodynamics, and electrical activity. Targeted interventions are aimed at preventing increases in intracran
Emergency department neurosurgical admissionsSCGH ED CME
This document provides an overview of common emergency neurosurgical presentations and indications for surgical intervention. It discusses the assessment of comatose patients, including the Glasgow Coma Scale. It then covers various neurosurgical topics like cranial trauma, vascular neurosurgery including stroke and subarachnoid hemorrhage, neuro-oncology, hydrocephalus, and spinal surgery. For each topic, it outlines clinical criteria for determining if surgical intervention is required.
Mechanisms of cerebral injury and cerebral protectionDr Kumar
This document discusses mechanisms of cerebral injury and cerebral protection. It provides details on cerebral physiology including metabolism, blood flow, regulation of blood flow, and factors that influence blood flow such as perfusion pressure, autoregulation, respiratory gas tensions, temperature, viscosity, and autonomic influences. It also discusses intracranial pressure, signs of increased ICP, assessment of injury severity, and strategies and principles for cerebral protection including maintaining oxygen supply and reducing increases in ICP, cerebral metabolic rate, and cell damage. The effects of various anesthetic drugs on cerebral blood flow, metabolism, and injury are also summarized.
Anesthetic management of carotid endarterectomy [autosaved] 2Arun Aru
1. The document describes the anesthetic management of a carotid endarterectomy procedure in a 54-year-old male with a history of stroke and risk factors including diabetes and hypertension.
2. Intraoperatively, the patient was induced with thiopental and intubated, and anesthesia was maintained with desflurane. Monitoring included arterial and central lines.
3. The endarterectomy procedure involved clamping of the internal carotid artery and removal of atherosclerotic plaque from the vessel wall via placement of a carotid stent.
This document discusses the management of intracranial pressure and cerebral edema in neurocritical care patients. It covers topics such as how patients typically present with brain injuries, important caveats in neurological examinations, principles of cerebral resuscitation, mechanisms of primary and secondary brain injury, the pathophysiology over time, imaging techniques including CT scans and MRI, monitoring techniques like intracranial pressure monitoring, and treatment approaches like the use of hyperosmolar therapy with mannitol or hypertonic saline. The goal is to prevent secondary brain injury after the initial primary injury occurs.
1. The document discusses intracranial pressure (ICP), cerebrospinal fluid (CSF) circulation and compensation mechanisms when ICP increases. It defines normal ICP and the factors that affect it, including the Monro-Kellie doctrine.
2. Symptoms of increased ICP are described, from early signs like headache to late signs like herniation and changes in vital signs. Different types of herniation are explained.
3. Methods for monitoring ICP are summarized, including invasive techniques like intraventricular and subdural monitors and non-invasive options. Indications for ICP monitoring include severe head injuries with abnormal CT scans or certain risk factors.
Hypothyroidism and hyperthyroidism have significant clinical effects. Both should be optimized. Anesthesia providers should be able to diagnose and manage.
This document provides a history of the development of neuroanesthesia. It discusses early discoveries and practices from ancient times through the 19th century. It then summarizes major advances in the 20th century related to anesthetic agents, monitoring, airway management, and techniques to protect the brain during surgery. These include the development of muscle relaxants, inhalational agents, intravenous medications, hypothermia, and methods to prevent increases in intracranial pressure and glucose levels.
Extradural hematoma, also known as epidural hematoma, is a collection of blood between the skull and the dura mater. It typically results from arterial bleeding, usually from the middle meningeal artery. Presentation includes an initial loss of consciousness followed by a lucid interval and then deterioration in consciousness. Treatment involves prompt surgery if signs of mass effect or herniation are present. Surgical evacuation aims to remove the clot and achieve hemostasis to prevent reaccumulation. Outcomes are generally good if diagnosed and treated quickly, but mortality increases with delay in treatment or presence of additional intracranial injuries.
Head injuries top the list of trauma patienrts coming to the casualty. The condition has to be immediately assessed and investigated. Depending upon the findings prompt medical or neurosurgical treatment has to be administered.
This document provides an overview of head injury management in the emergency department. It begins with an introduction on the importance of not neglecting or giving up on head injuries. It then describes a case of a 25-year-old man brought to the ED unconscious after a bike accident while intoxicated. The document reviews head injury classification, mechanisms of injury, diagnostic imaging, medical and surgical management strategies, and goals of preventing secondary brain injury. Key points covered include initial resuscitation, indications for observation versus admission, guidelines for mild, moderate and severe injuries, and timing of surgical interventions.
This document provides an overview of the neurological management of severely injured patients. It discusses the pathophysiology of neurotrauma and emphasizes the importance of preventing secondary brain and spinal cord injuries through careful management of factors like hypotension, hypoxia, intracranial pressure, seizures and temperature. The initial assessment and priorities on scene and in the emergency department are outlined. Medical and surgical management strategies for head injuries, spinal injuries and monitoring of intracranial pressure are summarized, along with guidelines for interventions like osmotherapy, blood transfusion thresholds and timing of surgery. Key evidence-based recommendations are highlighted.
This document provides an outline on head injury (HI). It discusses the pathophysiology of HI, classification based on Glasgow Coma Scale and mechanism of injury. It describes the components of HI including scalp laceration, skull fractures, and traumatic brain injury. For traumatic brain injury, it covers concussion, contusion, diffuse axonal injury, and intracranial hematomas. It outlines the primary and secondary survey approach for patients with HI, including airway management, breathing, circulation, disability assessment, and exposure.
This document provides information on Guillain-Barre syndrome (GBS), craniotomies, and intracranial hypertension. It defines GBS as an acute inflammatory polyradiculoneuropathy causing weakness and diminished reflexes. It describes the pathophysiology and management of GBS, including plasma exchange and IV immunoglobulin treatment. It then discusses different types of craniotomies and pre, intra, and post-operative care. Finally, it examines the pathophysiology and assessment of intracranial hypertension, management to reduce intracranial volume, and herniation syndromes that can occur if intracranial pressure is not controlled.
This patient presented with left-sided weakness and slurred speech. CT scan was normal. After tPA infusion, her blood pressure was elevated. The appropriate next step is to administer nicardipine to lower her blood pressure and prevent intracerebral hemorrhage.
This patient is being followed up after an ischemic stroke. Testing shows a left pontine infarct. The appropriate secondary prevention is to substitute clopidogrel for aspirin given his history of peripheral artery disease.
This patient presented with headache and papilledema. MRI was normal. Magnetic resonance venography is the best next test to evaluate for dural sinus venous thrombosis given her risk factors.
intra-operative acre of patients of aneurysm.pptxAnujaSebastian
An aneurysm is an abnormal dilation of a brain artery that can rupture, causing bleeding in the brain. Ruptured aneurysms have high mortality rates. Symptoms include severe headaches and changes in consciousness. Diagnosis involves CT, MRI, or cerebral angiography. Treatment may involve surgical clipping or endovascular coiling to prevent rebleeding. Postoperative care focuses on monitoring for complications like vasospasm, seizures, hydrocephalus, or rebleeding while restricting activity and managing anxiety.
Cerebrovascular Accident or stroke is defined as an abrupt onset of neurological deficit caused by a focal vascular issue. Stroke is the second leading cause of death worldwide. The clinical manifestations of stroke can vary widely due to the complex anatomy of the brain and vasculature. Imaging such as CT and MRI are used to determine if the cause is ischemia or hemorrhage. Treatment focuses on rapid evaluation, managing risk factors, IV thrombolysis if appropriate, and rehabilitation to prevent complications and encourage recovery.
This document discusses the peri-operative management of traumatic brain injury. It outlines key aspects of management including recognition of mass lesions, intracranial pressure monitoring and control, cerebral perfusion pressure management, oxygenation, and avoidance of hypotension and hypoxia. Structural brain injuries are described along with guidelines for management of hematomas, contusions, diffuse axonal injury, and other injuries.
Head injuries can range from mild to severe based on factors like loss of consciousness and Glasgow Coma Scale. The primary goals of management are to stabilize the patient by protecting the airway and maintaining adequate oxygenation, ventilation, and circulation to prevent secondary brain injury. Imaging with CT scan is important to identify fractures and intracranial bleeding like extradural and subdural hematomas that may require neurosurgery. Ongoing monitoring of things like neurological status and intracranial pressure is also important for managing head injuries.
1. The document discusses traumatic brain injuries and CNS infections, outlining their assessment, management, and surgical treatment.
2. Key points include evaluating patients using the Glasgow Coma Scale, identifying different types of intracranial hemorrhages on imaging and their presentations, and treating brain abscesses medically with antibiotics and surgically via burr hole aspiration or craniotomy for excision.
3. Surgical management of conditions like epidural hematomas and brain abscesses aims to decrease intracranial pressure and obtain samples for culture.
This document discusses considerations for anesthesia management of supratentorial brain tumors. It begins by describing the anatomy of the supratentorial and infratentorial compartments. Common tumor types in the supratentorial compartment include gliomas, meningiomas, pituitary adenomas and metastases. Key goals for anesthesia include maintaining adequate brain perfusion and oxygenation, facilitating tumor resection, and allowing for rapid emergence. Monitoring includes standard ASA monitors plus ICP monitoring if elevated preoperatively. Positioning can affect ventilation and ICP, so padding pressure points is important. Induction aims to avoid ICP elevations while maintaining cerebral perfusion pressure. Maintenance involves propofol, opioids and muscle relaxation to prevent movements
This document provides an overview of cerebrovascular accidents or strokes. It defines a stroke as the sudden death of brain cells due to lack of oxygen from a blocked or ruptured artery in the brain. Risk factors include hypertension, heart disease, smoking, obesity, and age. Strokes are classified as ischemic or hemorrhagic and treatment involves medications to break up clots, surgery, and rehabilitation to regain functions. Nursing care focuses on airway maintenance, communication, mobilization, and psychological support during recovery.
Patho physiology and mechanism of head injuries .pptxVignesh283945
Trauma can be defined as an injury to any part of the human body as a result of energy transfer from an inflicting source.
Trauma management is based on the principles of Advanced Trauma Life Support(ATLS) guidelines to rapidly identify and treat life threatening injuries during primary survey.
1. Supratentorial surgeries require careful anesthetic management to maintain adequate cerebral perfusion and oxygenation while optimizing conditions for tumor resection.
2. Key goals include preventing increases in intracranial pressure through careful induction, positioning, ventilation, and emergence from anesthesia.
3. Emergence should be smooth to avoid straining or bucking which can abruptly increase intracranial pressure and risk hemorrhage or herniation.
Neuraxial anesthesia involves injecting anesthetic medication into the epidural space surrounding the spinal nerve roots or into the cerebrospinal fluid surrounding the spinal cord. This numbs the patient from the abdomen to the toes and often eliminates the need for general anesthesia. There are several types of neuraxial anesthesia including spinal, epidural and caudal blocks. Potential complications include hypotension, neurological issues, infection and post-dural puncture headache. Proper patient positioning, drug selection and dosage are important factors for achieving the desired level and density of the block with the fewest complications.
Edward Fohrman | Anesthetic Considerations for Carotid EndarterectomyEdward Fohrman
Edward Fohrman, an experienced anesthesiologist in CA, discusses some considerations to take into consideration when it comes to carotid endarterectomy and anesthesia.
This document discusses the care of patients with head injuries. It begins by defining head injuries and noting that over 30% of serious head injuries are fatal before hospital arrival due to injuries. It then discusses the clinical manifestations such as skull fractures and concussions. The major goals of care for severely head injured patients are to treat hypoxia, acid-base disorders, control increasing intracranial pressure, and stabilize other conditions. Surgical management may be needed for hematomas, depressed fractures, or foreign bodies. Rehabilitation is also important for returning patients to maximal functioning.
This document discusses oral medication administration and the nurse's responsibilities. It defines oral medication as drugs that are swallowed through the oral cavity. Nurses must follow the 10 rights of medication administration, which include the right patient, medication, dose, route, time and documentation. The nurse's responsibilities include identifying the patient, selecting the correct medication, administering it properly, educating the patient, documenting appropriately, and evaluating the effects of the medication. Safe oral drug administration requires thorough assessment, accurate documentation and monitoring of the patient.
This document discusses suicide risk assessment and prevention from a nursing perspective. It begins by defining key terms like suicide, suicidal ideation, suicide attempt, and parasuicide. It then covers risk factors for suicide like gender, ethnicity, marital status, mental illness, and prior attempts. The document outlines a mental status examination and risk assessment approach. It proposes the SAD PERSONS scale for risk assessment. Finally, it details nursing management of suicidal patients, which includes close observation, environmental safety precautions, developing a care plan, and multidisciplinary involvement. The overall goal is vigilance and a collaborative approach to treatment and prevention of suicide.
The specific knowledge, skills, and attitudes required, as well as provide educational practices under supervision. Has a direct bearing on students’ ability to integrate theory to practice.
This document provides an overview of blood cancers including definitions, types, symptoms, diagnostic tests, and treatment. The three major types of blood cancer are leukemia, lymphoma, and myeloma. Leukemia occurs in the bone marrow and causes abnormal blood cells to enter the bloodstream. Lymphomas are cancers of the immune system cells. Diagnostic tests include physical exams, blood tests, bone marrow biopsies, and scans. Treatment depends on the type and severity but may include chemotherapy, radiation, stem cell transplants, and other therapies. Nursing care focuses on managing risks of infection, acute pain, and activity intolerance through comfort measures, strict protocols, and encouraging rest.
NURSING MANAGEMENT OF PATIENT WITH EMPHYSEMA .PPTblessyjannu21
Prepared by Prof. BLESSY THOMAS, VICE PRINCIPAL, FNCON, SPN.
Emphysema is a disease condition of respiratory system.
Emphysema is an abnormal permanent enlargement of the air spaces distal to terminal bronchioles, accompanied by destruction of their walls and without obvious fibrosis.
Emphysema of lung is defined as hyper inflation of the lung ais spaces due to obstruction of non respiratory bronchioles as due to loss of elasticity of alveoli.
It is a type of chronic obstructive
pulmonary disease.
It is a progressive disease of lungs.
nursing management of patient with Empyema pptblessyjannu21
prepared by Prof. BLESSY THOMAS, SPN
Empyema is a disease of respiratory system It is defines as the accumulation of thick, purulent fluid within the pleural space, often with fibrin development.
Empyema is also called pyothorax or purulent pleuritis.
It’s a condition in which pus gathers in the area between the lungs and the inner surface of the chest wall. This area is known as the pleural space.
Pus is a fluid that’s filled with immune cells, dead cells, and bacteria.
Pus in the pleural space can’t be coughed out. Instead, it needs to be drained by a needle or surgery.
Empyema usually develops after pneumonia, which is an infection of the lung tissue. it is mainly caused due in infectious micro-organisms. It can be treated with medications and other measures.
Enhancing Hip and Knee Arthroplasty Precision with Preoperative CT and MRI Im...Pristyn Care Reviews
Precision becomes a byword, most especially in such procedures as hip and knee arthroplasty. The success of these surgeries is not just dependent on the skill and experience of the surgeons but is extremely dependent on preoperative planning. Recognizing this important need, Pristyn Care commits itself to the integration of advanced imaging technologies like CT (Computed Tomography) and MRI (Magnetic Resonance Imaging) into the surgical planning process.
English Drug and Alcohol Commissioners June 2024.pptxMatSouthwell1
Presentation made by Mat Southwell to the Harm Reduction Working Group of the English Drug and Alcohol Commissioners. Discuss stimulants, OAMT, NSP coverage and community-led approach to DCRs. Focussing on active drug user perspectives and interests
R3 Stem Cell Therapy: A New Hope for Women with Ovarian FailureR3 Stem Cell
Discover the groundbreaking advancements in stem cell therapy by R3 Stem Cell, offering new hope for women with ovarian failure. This innovative treatment aims to restore ovarian function, improve fertility, and enhance overall well-being, revolutionizing reproductive health for women worldwide.
At Malayali Kerala Spa Ajman we providing the top quality massage services for our customers.
Our massage center prioritizes efficiency to ensure a quality massage experience for our clients at Malayali Kerala Spa Ajman. We offer a convenient appointment system and precise massage services.
Reach us at Villa No 7, Near Ammar Bin Yasir Street Al Rashidiya 2 - Ajman - United Arab Emirates.
Phone : +971 529818279
The Importance of Black Women Understanding the Chemicals in Their Personal C...bkling
Certain chemicals, such as phthalates and parabens, can disrupt the body's hormones and have significant effects on health. According to data, hormone-related health issues such as uterine fibroids, infertility, early puberty and more aggressive forms of breast and endometrial cancers disproportionately affect Black women. Our guest speaker, Jasmine A. McDonald, PhD, an Assistant Professor in the Department of Epidemiology at Columbia University in New York City, discusses the scientific reasons why Black women should pay attention to specific chemicals in their personal care products, like hair care, and ways to minimize their exposure.
Test bank clinical nursing skills a concept based approach 4e pearson educati...rightmanforbloodline
Test bank clinical nursing skills a concept based approach 4e pearson education
Test bank clinical nursing skills a concept based approach 4e pearson education
Test bank clinical nursing skills a concept based approach 4e pearson education
Test bank clinical nursing skills a concept based approach 4e pearson educati...
Stroke Care of Patient With Post Decompressive Craniectomy
1. STROKE CARE OF PATIENT
WITH POST DECOMPRESSIVE
CRANIECTOMY
By Wong Pei Yin (Charissa)
BSN, SRN
2. STOKE CARE OF PATIENT IS
IMPORTANT???
“We treat human as a whole,
not treating the disease itself”
3. blocked by a clot
or bursts/ ruptures
blood vessel that
carries oxygen and
nutrients to the brain
brain cells die
STROKE
4. ISCHEMIC STROKE (CLOTS)
• Majority 87%
of all strokes.
• A blood vessel
supplying
blood to the
brain is
obstructed.
5. HEMORRHAGIC STROKE (BLEEDS)
• Occurs when a
weakened blood vessel
(aneurysms and
arteriovenous
malformations AVMs)
ruptures.
• The most common
cause of hemorrhagic
stroke is uncontrolled
high blood pressure.
6. • used in urgent or emergent conditions where the brain
swelling from bleeding, stroke or infection, the pressure
in the brain can build inside the skull and causing
further damage.
DECOMPRESSIVE CRANIECTOMY?
• The bone flap is surgically removed but is not returned
to the skull after surgery.
7. Role of Decompressive Craniectomy
for stroke
1. Increases Buffering Capacity Of Cranium.
• Allows outward herniation of brain tissue.
- preventing compression of brainstem structures.
2. Intracranial Pressure
- (ICP) reduction 15-85% depending on size of
bone removed.
3. Improves neurological status and quality of life
- Decompressive craniectomy performed within
24hours may improve overall mortality and functional
outcomes.
8. Monroe-Kellie doctorine
• The cranium is a rigid structure that contains 3
main components: brain, cerebrospinal fluid,
and blood.
• An increase in the volume of one component
will result in the decrease of volume in 1 or 2
of the other components.
9. The figure above showed measurements of ICP before,
during, and after the operation, indicating a significant
alleviating effect of DC on intracranial hypertension.
10. Complications Associated With
Decompressive Craniectomy
Early
•Hemorrhage (hematoma expansion)
•External cerebral herniation
•Wound complications
•Seizures/epilepsy
Late or delayed
•Hydrocephalus
•Syndrome of the Trephined
14. Surgical Site Infection
Dehiscence
• Defined as a diastase of facing flap borders
occurring along the line of suture, with
different degrees of exposure of underlying
tissues
15. Ulcer
• Defined as a loss of substance occurring inside
the skin flap, usually distant from the line of
suture, constantly presenting with underlying
tissues exposure
18. Syndrome of the Trephined
Sinking skin flap occurs
following removal of a
large skull bone flap
19. NURSES SHOULD KNOW ABOUT CARE
FOR PATIENT POST OPERATIVE
DECOMPRESSIVE CRANIECTOMY
• The NICEPOD (2011) report found that
postoperative patients are at risk of clinical
deterioration.
• Knowledge and understanding of the key
areas and local policies will help reduce
potential problems (National Patient Safety
Agency, 2007)
20. POST OPERATIVE
NURSING INTERVENTION
Provide the following care after admission to critical care
unit or acute care unit:
a. Close Observation And Monitoring
Continue for 24 hours after procedure.
Sign of impending brain herniation, Cushing response:
irregular respirations, hypertension and bradycardia
High blood pressure causes reflex bradycardia and
brain stem compromise affecting respiration.
21. B. Assessment Neurological Status
Assess mental status (GCS) hourly and deterioration in
conscious level usually is sign of rising ICP.
Pupillary changes such as pupillary dilatation, bilateral
ptosis, impaired upgaze often seen in association with
raised ICP.
c. Risk Of Develop Seizure
Monitor seizure activity, increase the risk of postoperative
seizure activity especially in first 24 hours after surgery.
Fluctuation of neurologic status may indicate subclinical
seizures.
Administer anticonvulsants as prescribed.
Provide seizure precautions (available for airway, pads on
the bed rails, and suctioning equipment set up)
22. d. Position
Keeping neck in midline neutral position and head of the bed
at 30 degrees that help venous drainage from the brain and
lowers ICP.
Avoid extreme rotation of the neck and extreme hip flexion
which may further raise ICP by increasing intrathoracic
pressure and obstructing cerebral venous outflow
e. Monitor Respiratory Status
Maintain mechanical ventilation and slight hyperventilation
for first 24 to 48hours as prescribed to prevent increased ICP.
Respiratory difficulty due to decreased level of consciousness
and inability to protect airway.
Maintain Spo2> 95%, obtaining ABG as needed.
23. f. Drains
Know the location of each drain and label
them clearly.
Requires monitoring and measurement of the
drainage.
Inform the physician if drainage is more than
the normal amount of 30-50ml per shift.
24. g. Incision Care
Inspect the incision on the head to ensure edges remain
well approximated, and staples/ sutures are intact.
Monitor for redness around the incision, discharge, and
other signs of infection.
h. Pain Control
Assess pain via pain score (scale 1–10)
Administer specific pain management medications as
prescribed by doctor to prevent elevate of blood pressure
result in vasoconstriction.
Short acting agents, such as fentanyl are preferred
especially because reversal agents are available, making
certain that a neurologic exam is possible.
25. i. Monitor intake and output of fluid
• Common signs and symptoms of
hypovolemic shock results from
general fluid loss, tachycardia,
decrease BP, shallow and rapid
respirations, decreased urine output
(10-25ml/hour).
• Fluid resuscitation is directed toward
maintaining normovolemia. Isotonic
and hypertonic saline are generally
used.
• IV fluid with dextrose should be
avoided due to risk of increasing
blood glucose and potential
worsening of cerebral oedema.
26. j. Blood glucose
Hyperglycemia potentially disrupts the blood-
brain barrier and increases cerebral edema.
Maintain normoglycemia, 4-8mmol/l.
k. Adequate nutrition
nutrition guaranteed by using high protein
enteral or parenteral solutions.
nutritional needs necessary to help facilitate
appropriate wound healing.
27. Example Case
• 59 years old, male
• U/L DM, HTN, IHD done bypass 2001, AF
• Found by family member patient was lying down
flat on floor at 10am 21/9/19
• +Vomitus
• +Less responsive
• +Facial asymmetry, unsure fitting episode prior
fall.
• No headache
28. • Patient on arrival ED 11.30am 21/9/19,
• GCS 10/15 E4V1M5, not obey command. Pupil
2mm/2mm reactive.
• BP: 192/67mmhg, HR: 84/min, Temp: 37◦c,
• Spo2: 92%-93% under HFM 15L/min
• Blood glucose: 18.4mmol/l
• Cardiac monitoring: AF rate controlled
• Vomited x2 at ED
• Left Facial Palsy, Left Gaze Preference, Right sided
body weakness, Gag Reflex Absent
• Decided for intubation, airway protection 12.30pm
29. • 1st CT brain: MCA densed sign, no midline
shift, ASPECT 5
• CTA brain: no co-lateral, not for mechanical
thrombectomy.
• 2nd CT brain: mass effect noted.
• Diagnose: Left MCA infarct
30. Post Operative Management
• Left decompressive craniectomy done 11.30am 22/9/19
• Post operation admitted to neuromedical acute care unit
for close monitoring.
• Wean off sedation and aim for extubation.
• IV Zinacef 750mg TDS for 3doses then off (prophylaxis).
• Drain charting- full vacuum.
• IV drip maintenance 5 pints
• Start feeding 6hours post op and taper drips as
tolerating.
• Repeat blood investigation, to optimize if Hb low or
coagulopathy.
• Repeat CT brain earlier if indicated.
• For wound inspection on D3, and STO D7.
31. Post Operative Result day 1-day 3
• GCS 11/15, E4VtM6, open eyes spontaneously
and obey command.
• Able to move left sided.
• BP: 150-180/80-100 under low dose antiHPT
supported
• Spo2: 100% under ventilator supported
• Blood glucose: 8-11mmol/L under IV infusion
insulin supported
• Intake/Output chart: Positive 300-600ml/day
under IV drips normal saline maintenance
• Drain chart: below 60ml/day
• Repeated CT brain: No hemorrhage, No midline
shift