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Medical Concerns
Primary Survey                       Secondary Survey
A                                   Head /Neck   Occipital heamtoma
B                                                C3/4 tenderness
C      HR 125-160 rapid AF                       Epistaxis
       BP 150/85                     Chest         
                                     Abdo          
        warm well perfused           Pelvis         FAST neg
D      GCS 13                        Back         flank abrasion
E      abrasion on back                           T12/L1 tender
       36.6                                       no step deformity
                                     UL/LL         


                   Plan....
                   RSI combative/head injury
                   CT BRAIN/c spine/CXRAY
                   Bloods including ETOH
                   Antiemetic/analgesia
                   Arterial line /IDC
Major Issues
1. HEAD INJURY
Undisplaced # vertex skull
Bilateral subdural haematomas
Extensive Bilateral SAH

Increasing ICP requiring
Neurosurgical intervention

2. Inconsistency in the mechanisms/
history

3. Rapid AF
Rapid AF
(Presumed)
Otherwise healthy 27yo male           140-180bpm
No structural heart
disease, congenital, valvular
pathologies, cardiomegaly
Consider....
Shock (tachycardia)
 -heamorragic/hypovolemic
-myocardail ischemia/infarction
Electrolyte imbalances
Infection
Pain
ETOH
Chest trauma
Head Injuries
TFTs (normal)
Arrhythmias in Head Injuries
        (Neurogenic cardiac dysfunction)
•   Uncommon occurrence (6% SICU)
•   AF, Flutter, PSVT, PVC, PAC, Prolonged QT
•   Mostly associated with ICH, SAH
•   BAD prognostic sign (x 2 higher mortality rate)

Why?
•     Catecholamine storm lasting up to 10 days
? Protective mechanism to maintain CPP in face of a
     rising ICP (CPP =MAP-ICP)
Also other causes...
• Hypoxia, hypercapnia
•     Pulmonary Artery catheter placement, CVC
• NDI – loss of electrolytes (low Mg, Ca, K)

Most occurs within 7days of insult
Majority of these in first 48hrs.
What do we do?
                                                                B Blockers....
Treat the cause!
any AF - Head injury!                                           •   There is discussion regarding the
Surgery!                                                            use of b-blockers in this situation
Most patients who die will because of their brain injury
                                                                    during the peri operative period.
<24hrs a decision should be made regarding anticoagulation vs
obvious bleeding risks
                                                                •   Small number of studies have
Very rarely lead to fatal arrhythmias                               shown a protective effect on
                                                                    mortality and reduces cardiac
Non surgical treatment ie mild SDH, contusions..                    complications during ICU stay.
- Telemetry/ ECG and trops
                                                                •   ? this is due to moderate the
 - Treat if pt heamodynatically unstable                            physiological adenrenergic strain
 - TTE                                                              caused by catecholamine release.
 - Replace electrolytes
                                                                •   No consensus on this am must be
                                                                    balanced with the reduction this
                                                                    could cause in cerebral blood flow
                                                                •   Gregory 2011/Hadjizacharia 2007
Mr O
Back to the patient...
Westmead ICU (27days)               • TFTs normal
• EVD inserted and elevated ICP     • TTE normal
• Active cooling                    • Almost back to baseline
• Hypertonic saline                   level of function
• Digoxin and Mg admin for AF       • Is not likely to require long
• VAP                                 term Rehab (OHS)
• Developed an occlusive R IJ, SC
  Auxiliary and brachial vein
  Thrombus
Real story
Pt was “bonnet surfing”




                            “Bonnet surfing”
References

Trappe HJ. Treating critical supraventricular and ventricular arrhythmias. J Emerg Trauma Shock [serial online] 2010 [cited 2013 Apr
       8];3:143-52. Available from: http://www.onlinejets.org/text.asp?2010/3/2/143/62114

Grunsfeld A, Fletcher JJ, Nathan BR Curr Neurol Neurosci Rep.
Cardiopulmonary complications of brain injury. 2005 Nov;5(6):488-93.

Incidence, Risk Factors, and Outcomes for Atrial Arrhythmias in Trauma Patients
Hadjizacharia, Pantelis;O'Keeffe, Terence;Brown, Carlos V R, MD;Inaba, Kenji;Salim, Ali;Chan, Lin...
The American Surgeon; May 2011; 77, 5; ProQuest Central

Gregory and Smith 2011 “Cardiovascular complications of brain injury” Continuing Education in Anaesthesia, Critical Care & Pain

Clemo HF, Wood MA, Gilligan DM, Ellenbogen KA. Intravenous amiodarone for acute heart rate control in the critically ill patients with
atrial tachyarrhythmias. Am J Cardiol 1998;81:594-8

Overview of Adult Traumatic Brain Injuries. Copyright 2011 Orlando Health, Education & Development

Macha Bourdages, MD, MSc; Jean-Luc Bigras, MD; Catherine A. Farrell, MD; James S. Hutchison, MD;
Jacques Lacroix, MD; 2010 Cardiac arrhythmias associated with severe traumatic brain injury
and hypothermia therapy* Pediatr Crit Care Med Vol. 11, No. 3

Xavier Wittebole, et al 2005. Electrocardiographic changes after head trauma. Journal of Electrocardiology 38; 77–81

The Brain Trauma Foundation https://www.braintrauma.org/

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Preso

  • 1. Medical Concerns Primary Survey Secondary Survey A  Head /Neck Occipital heamtoma B  C3/4 tenderness C HR 125-160 rapid AF  Epistaxis BP 150/85 Chest  Abdo  warm well perfused Pelvis  FAST neg D GCS 13 Back flank abrasion E abrasion on back T12/L1 tender 36.6 no step deformity UL/LL  Plan.... RSI combative/head injury CT BRAIN/c spine/CXRAY Bloods including ETOH Antiemetic/analgesia Arterial line /IDC
  • 2. Major Issues 1. HEAD INJURY Undisplaced # vertex skull Bilateral subdural haematomas Extensive Bilateral SAH Increasing ICP requiring Neurosurgical intervention 2. Inconsistency in the mechanisms/ history 3. Rapid AF
  • 3. Rapid AF (Presumed) Otherwise healthy 27yo male 140-180bpm No structural heart disease, congenital, valvular pathologies, cardiomegaly Consider.... Shock (tachycardia) -heamorragic/hypovolemic -myocardail ischemia/infarction Electrolyte imbalances Infection Pain ETOH Chest trauma Head Injuries TFTs (normal)
  • 4. Arrhythmias in Head Injuries (Neurogenic cardiac dysfunction) • Uncommon occurrence (6% SICU) • AF, Flutter, PSVT, PVC, PAC, Prolonged QT • Mostly associated with ICH, SAH • BAD prognostic sign (x 2 higher mortality rate) Why? • Catecholamine storm lasting up to 10 days ? Protective mechanism to maintain CPP in face of a rising ICP (CPP =MAP-ICP) Also other causes... • Hypoxia, hypercapnia • Pulmonary Artery catheter placement, CVC • NDI – loss of electrolytes (low Mg, Ca, K) Most occurs within 7days of insult Majority of these in first 48hrs.
  • 5. What do we do? B Blockers.... Treat the cause! any AF - Head injury! • There is discussion regarding the Surgery! use of b-blockers in this situation Most patients who die will because of their brain injury during the peri operative period. <24hrs a decision should be made regarding anticoagulation vs obvious bleeding risks • Small number of studies have Very rarely lead to fatal arrhythmias shown a protective effect on mortality and reduces cardiac Non surgical treatment ie mild SDH, contusions.. complications during ICU stay. - Telemetry/ ECG and trops • ? this is due to moderate the - Treat if pt heamodynatically unstable physiological adenrenergic strain - TTE caused by catecholamine release. - Replace electrolytes • No consensus on this am must be balanced with the reduction this could cause in cerebral blood flow • Gregory 2011/Hadjizacharia 2007
  • 6. Mr O Back to the patient... Westmead ICU (27days) • TFTs normal • EVD inserted and elevated ICP • TTE normal • Active cooling • Almost back to baseline • Hypertonic saline level of function • Digoxin and Mg admin for AF • Is not likely to require long • VAP term Rehab (OHS) • Developed an occlusive R IJ, SC Auxiliary and brachial vein Thrombus
  • 7. Real story Pt was “bonnet surfing” “Bonnet surfing”
  • 8. References Trappe HJ. Treating critical supraventricular and ventricular arrhythmias. J Emerg Trauma Shock [serial online] 2010 [cited 2013 Apr 8];3:143-52. Available from: http://www.onlinejets.org/text.asp?2010/3/2/143/62114 Grunsfeld A, Fletcher JJ, Nathan BR Curr Neurol Neurosci Rep. Cardiopulmonary complications of brain injury. 2005 Nov;5(6):488-93. Incidence, Risk Factors, and Outcomes for Atrial Arrhythmias in Trauma Patients Hadjizacharia, Pantelis;O'Keeffe, Terence;Brown, Carlos V R, MD;Inaba, Kenji;Salim, Ali;Chan, Lin... The American Surgeon; May 2011; 77, 5; ProQuest Central Gregory and Smith 2011 “Cardiovascular complications of brain injury” Continuing Education in Anaesthesia, Critical Care & Pain Clemo HF, Wood MA, Gilligan DM, Ellenbogen KA. Intravenous amiodarone for acute heart rate control in the critically ill patients with atrial tachyarrhythmias. Am J Cardiol 1998;81:594-8 Overview of Adult Traumatic Brain Injuries. Copyright 2011 Orlando Health, Education & Development Macha Bourdages, MD, MSc; Jean-Luc Bigras, MD; Catherine A. Farrell, MD; James S. Hutchison, MD; Jacques Lacroix, MD; 2010 Cardiac arrhythmias associated with severe traumatic brain injury and hypothermia therapy* Pediatr Crit Care Med Vol. 11, No. 3 Xavier Wittebole, et al 2005. Electrocardiographic changes after head trauma. Journal of Electrocardiology 38; 77–81 The Brain Trauma Foundation https://www.braintrauma.org/

Editor's Notes

  1. SYMP surge -&gt;stimulate b1 adrenoboreceptorsrelease excitory NT Glutamate, causes NA into the cells - depolarozing effect.