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Dr Sumit Sinha
Additional Professor
Department of Neurosurgery,
JPNATC, AIIMS, New Delhi
HEAD INJURY IN ER
ER Management of Neurotrauma
Initial Management
A (with C1) – B – C
Initial Neuro-Assessment
D - Glasgow Coma Scale
Pupils
Immediate Neurosurgical Management
CT Scan - when?
Neurosurgical Consult - when?
Recognition and treatment of Herniation
Other Considerations
C-Spine, Bleeding, Extremity #, Rest of
Spine
Pitfalls
Concept of ICP & CPP
CPP = MAP – ICP
CPP- Critical : < 50 mmHg
ICP-
When will ICP ?? -↑
 Mass Lesions- Bleeding EDH, SDH, ICH
 Cerebral Edema Cytotoxic, Vasogenic
10 mm Hg = Normal
>20 mm Hg = Abnormal
>40 mm Hg = Severe
Monro-Kellie Doctrine
Classification of Head Injuries
A. Blunt or Penetrating
B. Based on GCS-
Mild 14-15
Moderate 9-13
Severe 3-8
C. Morphology (Fracture and Intracranial)
MANAGEMENT
A- Airway with C-Spine
PATENT
↓
OKAY
Don’t Intubate until
patient needs
sedation for
some other
procedure
THREATENED
↓
Remains
Unresponsive
GCS 8≦
(CANNOT PROTECT AIRWAY)
Oral bleeding
Base of Skull
bleeding
OBSTRUCTED
↓
Massive
Maxillofacial
Trauma
Associated Neck
Injury
* If C-Spine Injury is suspected, intubation should be performed
by the most experienced person available.
MANAGEMENT
B- Breathing
HYPOXIA HYPERCARBIA
Maintain PaO2 > 60 mmHg Maintain Normocarbia
Maintain SpO2 > 95% PaCO2 - 35-40 mmHg
MANAGEMENT
C- Circulation
TREAT HYPOTENSION
Maintain MAP > 90 mm Hg
USE
Crystalloids – RL/ NS
Ionotrope Infusion if needed
Primary Aim of ER management
Prevent Secondary Brain damage at all costs
TREAT AGGRESSIVELY
Hypotension (MAP > 90mmHg)
Hypoxia (PaO2 < 60mmHg)
Hypoglycemia
Fever
Raised Intra-cranial Pressure
Seizures
Early episodes of hypotension or hypoxia greatly increase
morbidity and mortality from severe head injury
MANAGEMENT
D- Initial Neuro-Assessment
 Key History
 Mechanism of Injury
 Response at scene → Neuro-exam at scene →
Change in status
 Note Glasgow Coma Scale (GCS 3-15)
 Note Pupils : Size / Shape / Reaction
 Note the symmetry of motor response-
LATERALIZING SIGNS
Head Injury Management
High risk Moderate Risk
● GCS score still < 15 two
hours after injury
● Neurologic deficit
● Open skull fracture
● Sign of basal skull fracture
● Extremes of age
● “Dangerous mechanism”
● Retrograde amnesia > 30
minutes in duration
● Severe headache
● Vomiting > 2 episodes
Indications for CT
Head Injury Management
Indications for admission
All Moderate and Severe HI
Mild HI-
 Unlikely to be carefully/ periodically checked at home
 Other significant injuries
 Child
 PTA > 1hr.
Head Injury Management
Indications for Repeat CT
 Patients failing to show expected recovery
 Discordant clinical and initial CT findings
 Deterioration
 New deficits
Initial Neurosurgical Management
Abnormal CT Scan
Medical Management
 Controlled ventilation
 Intubate if GCS<8 (Orotracheal)
 Mannitol- 0.25-1.0 gm/Kg IV over 15 min (Not in
Hypotensives)
 Monitor Urine Output
 Spine Cleared – Elevate the Head to 30°
 Sedation/ Paralysis- Neurological examination before
prolonged sedation / paralysis
Other Considerations
Seizure Focus/Post Traumatic Seizures (2-5%)
 Load Phenytion ≈ 11-15mg/Kg IV slow with cardiac monitoring
C-Spine
 5%-20% of patients with severe HI will have C-spine injury
 5%-10% with one spine # will have another one too
 Therefore
 C-Spine motion restriction and log-rolling till full spine cleared
Control of Bleeding
Immobilize other extremity fractures
Surgical Management
● Scalp Wounds
● Possible site of major blood loss
● Direct pressure to control bleeding
● Occasional temporary closure
Surgical Management
Intracranial Mass Lesion
● Can be life-threatening if expanding
rapidly
● Immediate neurosurgical consult
● Hyperventilation / mannitol
● Damage control craniotomy: transfer to
neurosurgeon (rural / austere areas)
Pitfalls
 Alteration in consciousness is hallmark of head injury.
Never attribute neurological abnormality to alcohol /
drugs.
 Assume spinal Injury till ruled out
 No naso-gastric / naso-tracheal tube if base skull #
suspected
 Treat other life threatening bleeding first
 Systolic pressure < 90 mmHg will lead to secondary
brain Injury
 Poor Ventilation and Oxygenation ↑ICP
Summary
 Comatose patient, secure and maintain airway by
ET intubation.
 Treat shock aggressively
 Hypoxia and hypovolaemia kill more patients than
brain injury.
 Secondary brain injury makes primary brain injury
worse
 If sedation or paralysis makes assessment difficult,
then treat patient until the brain can be assessed.
Spinal Trauma in ER
Spine Trauma
Suspicion of Spinal injury
 Mechanism of injury
 Unconscious Patient
 Neurologic deficits
 Spinal pain and tenderness
Spine Protection during evaluation and
transport
Protection is priority;
detection is secondary
Log Roll
4 persons
-1-maintain manual
in line immobilization
of head & neck
-2-shoulders & pelvis
-3-Pelvis & legs
-4- Directs & move
the spine board
Maintain neutral
alignment of spine
Spine injury Screening
Conscious Cooperative
Patient
Altered Consciousness or
Symptoms
• If no neck or spine pain or
tenderness
• If still no pain or tenderness
with voluntary movement
• No further evaluation or x-
ray necessary
Clear spine and remove
cervical collar.
• Radiographic
visualization of entire
spine
• Plain films
• CT scan of suspicious or
poorly visualized areas
Spine injury Screening
 Confirmation of Spine injury
 Clinical Signs
 Radiology- X Ray/ CT/ MR
 Presume Spinal Instability
 Early spine consult
Rule of thumb- N. deficit- Unstable
TRAUMA 2011TRAUMA 2011
Radiographic Evaluation
C. Spine: AP, Lat, open mouth (92%)
Thoracic & lumbar spine: AP, Lat
CT
10% of C. Spine pt. have a second # of V. column-10% of C. Spine pt. have a second # of V. column-
complete radiographic screeningcomplete radiographic screening
X-ray Evaluation
 C.spine- 4 lines
 Height of V. body, contour
 Pedicles, Sp.process, facets
 I/V disc space, Sp. Process inter space
 Pre vertebral soft tissue space <5mm at C3
TRAUMA 2011TRAUMA 2011
AP- Pedicles,
Facets, Sp. process,
V. body ht.
LAT- V. body height,
Angulation, disc space
Neurologic status
 Complete: No motor/ sensory function below the
level of injury
 Incomplete: Any motor/sensory below the level-
prognosis for recovery is better
 Perianal sensation may be the only sign of
incomplete SCI
Effects of Spinal Injury
Neurogenic Shock
 Cx and High Thoracic injuries- Cardiovascular
phenomenon
 Vasodilatation & pooling of blood- hypotension
 Bradycardia- No H.R. in response to hypotension
 I.V. fluids- fluid overload/ P. Edema
 Vasopressors / Atropine
Effects of Spinal Injury
Spinal Shock
 Neurologic phenomenon- Complete flaccidity &
loss of reflexes
 Gen. lasts 24- 48 hrs
 Anal & bulbo-cavernosus –first to return
TRAUMA 2011TRAUMA 2011
Management
Primary Survey
Airway: Protect C. Spine- Intubation if needed
Breathing: Oxygen, ventilatory support
Circulation:
-Hypovolemic shock- BP, HR, cool extremity-
fluids
-Neurogenic shock- BP, HR, warm extremity-
vasopressors
Disability: consciousness, GCS, pupils,
paresis/paraplegia
TRAUMA 2011TRAUMA 2011
Management
Secondary Survey
 History,mechanism of injury
 Reassess- consciousness, GCS
 Spine: palpation- entire spine- deformity,
Crepitus, tenderness, contusion
 Pain,paralysis/paresis-absent/present, location,
neurologic level
Management
Transfer Criteria-
 Unstable Fractures
 Neurologic deficits
AVOID UNNECESSARY DELAY
• Provide respiratory support as
needed
• Exclude other life- threatening
injury
• Properly immobilize entire
patient
• Avoid hypothermia
TRAUMA 2011TRAUMA 2011
Treatment - Summary in E.D.
 Protect from further injury:
 -Long spine board, C.Collar,log roll-neutral
alignment of spine
 -Minimize time on spine board- Decubitus ulcer
 Oxygen to all
 Fluid resuscitation & monitoring: CVP, Urinary
catheter, N.G. tube
 Steroids
TRAUMA 2011TRAUMA 2011
??
Jai Prakash Narain Apex Trauma CenterJai Prakash Narain Apex Trauma Center
All India Institute of Medical SciencesAll India Institute of Medical Sciences

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Er management of neurotrauma

  • 1. Dr Sumit Sinha Additional Professor Department of Neurosurgery, JPNATC, AIIMS, New Delhi
  • 3. ER Management of Neurotrauma Initial Management A (with C1) – B – C Initial Neuro-Assessment D - Glasgow Coma Scale Pupils Immediate Neurosurgical Management CT Scan - when? Neurosurgical Consult - when? Recognition and treatment of Herniation Other Considerations C-Spine, Bleeding, Extremity #, Rest of Spine Pitfalls
  • 4. Concept of ICP & CPP CPP = MAP – ICP CPP- Critical : < 50 mmHg ICP- When will ICP ?? -↑  Mass Lesions- Bleeding EDH, SDH, ICH  Cerebral Edema Cytotoxic, Vasogenic 10 mm Hg = Normal >20 mm Hg = Abnormal >40 mm Hg = Severe
  • 6. Classification of Head Injuries A. Blunt or Penetrating B. Based on GCS- Mild 14-15 Moderate 9-13 Severe 3-8 C. Morphology (Fracture and Intracranial)
  • 7. MANAGEMENT A- Airway with C-Spine PATENT ↓ OKAY Don’t Intubate until patient needs sedation for some other procedure THREATENED ↓ Remains Unresponsive GCS 8≦ (CANNOT PROTECT AIRWAY) Oral bleeding Base of Skull bleeding OBSTRUCTED ↓ Massive Maxillofacial Trauma Associated Neck Injury * If C-Spine Injury is suspected, intubation should be performed by the most experienced person available.
  • 8. MANAGEMENT B- Breathing HYPOXIA HYPERCARBIA Maintain PaO2 > 60 mmHg Maintain Normocarbia Maintain SpO2 > 95% PaCO2 - 35-40 mmHg
  • 9. MANAGEMENT C- Circulation TREAT HYPOTENSION Maintain MAP > 90 mm Hg USE Crystalloids – RL/ NS Ionotrope Infusion if needed
  • 10. Primary Aim of ER management Prevent Secondary Brain damage at all costs TREAT AGGRESSIVELY Hypotension (MAP > 90mmHg) Hypoxia (PaO2 < 60mmHg) Hypoglycemia Fever Raised Intra-cranial Pressure Seizures Early episodes of hypotension or hypoxia greatly increase morbidity and mortality from severe head injury
  • 11. MANAGEMENT D- Initial Neuro-Assessment  Key History  Mechanism of Injury  Response at scene → Neuro-exam at scene → Change in status  Note Glasgow Coma Scale (GCS 3-15)  Note Pupils : Size / Shape / Reaction  Note the symmetry of motor response- LATERALIZING SIGNS
  • 12. Head Injury Management High risk Moderate Risk ● GCS score still < 15 two hours after injury ● Neurologic deficit ● Open skull fracture ● Sign of basal skull fracture ● Extremes of age ● “Dangerous mechanism” ● Retrograde amnesia > 30 minutes in duration ● Severe headache ● Vomiting > 2 episodes Indications for CT
  • 13. Head Injury Management Indications for admission All Moderate and Severe HI Mild HI-  Unlikely to be carefully/ periodically checked at home  Other significant injuries  Child  PTA > 1hr.
  • 14. Head Injury Management Indications for Repeat CT  Patients failing to show expected recovery  Discordant clinical and initial CT findings  Deterioration  New deficits
  • 16. Medical Management  Controlled ventilation  Intubate if GCS<8 (Orotracheal)  Mannitol- 0.25-1.0 gm/Kg IV over 15 min (Not in Hypotensives)  Monitor Urine Output  Spine Cleared – Elevate the Head to 30°  Sedation/ Paralysis- Neurological examination before prolonged sedation / paralysis
  • 17. Other Considerations Seizure Focus/Post Traumatic Seizures (2-5%)  Load Phenytion ≈ 11-15mg/Kg IV slow with cardiac monitoring C-Spine  5%-20% of patients with severe HI will have C-spine injury  5%-10% with one spine # will have another one too  Therefore  C-Spine motion restriction and log-rolling till full spine cleared Control of Bleeding Immobilize other extremity fractures
  • 18. Surgical Management ● Scalp Wounds ● Possible site of major blood loss ● Direct pressure to control bleeding ● Occasional temporary closure
  • 19. Surgical Management Intracranial Mass Lesion ● Can be life-threatening if expanding rapidly ● Immediate neurosurgical consult ● Hyperventilation / mannitol ● Damage control craniotomy: transfer to neurosurgeon (rural / austere areas)
  • 20. Pitfalls  Alteration in consciousness is hallmark of head injury. Never attribute neurological abnormality to alcohol / drugs.  Assume spinal Injury till ruled out  No naso-gastric / naso-tracheal tube if base skull # suspected  Treat other life threatening bleeding first  Systolic pressure < 90 mmHg will lead to secondary brain Injury  Poor Ventilation and Oxygenation ↑ICP
  • 21. Summary  Comatose patient, secure and maintain airway by ET intubation.  Treat shock aggressively  Hypoxia and hypovolaemia kill more patients than brain injury.  Secondary brain injury makes primary brain injury worse  If sedation or paralysis makes assessment difficult, then treat patient until the brain can be assessed.
  • 23. Spine Trauma Suspicion of Spinal injury  Mechanism of injury  Unconscious Patient  Neurologic deficits  Spinal pain and tenderness
  • 24. Spine Protection during evaluation and transport Protection is priority; detection is secondary
  • 25. Log Roll 4 persons -1-maintain manual in line immobilization of head & neck -2-shoulders & pelvis -3-Pelvis & legs -4- Directs & move the spine board Maintain neutral alignment of spine
  • 26. Spine injury Screening Conscious Cooperative Patient Altered Consciousness or Symptoms • If no neck or spine pain or tenderness • If still no pain or tenderness with voluntary movement • No further evaluation or x- ray necessary Clear spine and remove cervical collar. • Radiographic visualization of entire spine • Plain films • CT scan of suspicious or poorly visualized areas
  • 27. Spine injury Screening  Confirmation of Spine injury  Clinical Signs  Radiology- X Ray/ CT/ MR  Presume Spinal Instability  Early spine consult Rule of thumb- N. deficit- Unstable
  • 28. TRAUMA 2011TRAUMA 2011 Radiographic Evaluation C. Spine: AP, Lat, open mouth (92%) Thoracic & lumbar spine: AP, Lat CT 10% of C. Spine pt. have a second # of V. column-10% of C. Spine pt. have a second # of V. column- complete radiographic screeningcomplete radiographic screening
  • 29. X-ray Evaluation  C.spine- 4 lines  Height of V. body, contour  Pedicles, Sp.process, facets  I/V disc space, Sp. Process inter space  Pre vertebral soft tissue space <5mm at C3
  • 30. TRAUMA 2011TRAUMA 2011 AP- Pedicles, Facets, Sp. process, V. body ht. LAT- V. body height, Angulation, disc space
  • 31. Neurologic status  Complete: No motor/ sensory function below the level of injury  Incomplete: Any motor/sensory below the level- prognosis for recovery is better  Perianal sensation may be the only sign of incomplete SCI
  • 32. Effects of Spinal Injury Neurogenic Shock  Cx and High Thoracic injuries- Cardiovascular phenomenon  Vasodilatation & pooling of blood- hypotension  Bradycardia- No H.R. in response to hypotension  I.V. fluids- fluid overload/ P. Edema  Vasopressors / Atropine
  • 33. Effects of Spinal Injury Spinal Shock  Neurologic phenomenon- Complete flaccidity & loss of reflexes  Gen. lasts 24- 48 hrs  Anal & bulbo-cavernosus –first to return
  • 34. TRAUMA 2011TRAUMA 2011 Management Primary Survey Airway: Protect C. Spine- Intubation if needed Breathing: Oxygen, ventilatory support Circulation: -Hypovolemic shock- BP, HR, cool extremity- fluids -Neurogenic shock- BP, HR, warm extremity- vasopressors Disability: consciousness, GCS, pupils, paresis/paraplegia
  • 35. TRAUMA 2011TRAUMA 2011 Management Secondary Survey  History,mechanism of injury  Reassess- consciousness, GCS  Spine: palpation- entire spine- deformity, Crepitus, tenderness, contusion  Pain,paralysis/paresis-absent/present, location, neurologic level
  • 36. Management Transfer Criteria-  Unstable Fractures  Neurologic deficits AVOID UNNECESSARY DELAY • Provide respiratory support as needed • Exclude other life- threatening injury • Properly immobilize entire patient • Avoid hypothermia
  • 37. TRAUMA 2011TRAUMA 2011 Treatment - Summary in E.D.  Protect from further injury:  -Long spine board, C.Collar,log roll-neutral alignment of spine  -Minimize time on spine board- Decubitus ulcer  Oxygen to all  Fluid resuscitation & monitoring: CVP, Urinary catheter, N.G. tube  Steroids
  • 38. TRAUMA 2011TRAUMA 2011 ?? Jai Prakash Narain Apex Trauma CenterJai Prakash Narain Apex Trauma Center All India Institute of Medical SciencesAll India Institute of Medical Sciences